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20007 Received 19 August 1982/Accepted 16September1982 DNA synthesis, as measured by incorporation of[3H]TTP, was inhibited in Swiss mouse 3T3 cells treated with interferon and subsequen

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Vol 45, No 2 JOURNAL OF VIROLOGY, Feb 1983, p 895-897

0022-538X/83/020895-03$02.00/0

Copyright C 1983, American Society for Microbiology

Persistence of Interferon Action in Mouse Cells Permeabilized

with Lysolecithin

ELSBETH J LEEt AND THAZEPADATH SREEVALSAN*

DepartmentofMicrobiology, Georgetown University, Schools ofMedicine andDentistry, Washington,D.C.

20007 Received 19 August 1982/Accepted 16September1982

DNA synthesis, as measured by incorporation of[3H]TTP, was inhibited in

Swiss mouse 3T3 cells treated with interferon and subsequently permeabilized

with lysolecithin The degree of inhibition observed was similar in intact or

permeabilized cells The interferon-induced antiviral state was retained in

per-meabilized cells

Interferon (IFN) induces pleiotropic effectson

mammalian cells, including the inhibition of

DNAsynthesis and cell growth (7) The

mecha-nisms mediating these effects, however, remain

poorly definedatpresent Incorporationof

exo-geneouslyadded [3H]thymidineinto

acid-insolu-ble material is the usualtechnique employedfor

thedetermination of DNA synthesis in

mamma-lian cells Using thistechnique, several authors

haveshown that there is significantinhibition of

theincorporation of labeledthymidine into

cel-lularDNA in IFN-treated cells (1, 5, 6, 8) In

experiments with L1210cells,however,

Brouty-Boye and Tovey (2) have reported that IFN

inhibits the uptake of thymidine into cells, a

finding which has been confirmed with Swiss

mouse 3T3 cells (T Sreevalsan, unpublished

data) Therefore, a reduction in uptake could

accountfor the observed inhibition in the

incor-poration of [3H]thymidine into acid-insoluble

material in Swiss 3T3 cells treated with IFN(1,

6) Inthisreport, weexamined the effect of IFN

on DNAreplicationin permeabilized cells, into

which[3H]TTPcanbeintroduced directly If the

mechanism of inhibition of DNA synthesis is

dependentontheeffect ofIFN ontheability of

cellstotakeupthymidine,onewouldexpectthe

incorporation of[3H]TTP into cellular DNA in

permeabilized cells to be unaffected by IFN

Our results indicated that the inhibitory effects

ofIFN on DNA synthesis persisted even after

the cells werepermeabilized

The procedure used to permeabilize Swiss

3T3 cells was a modification of a method

de-scribedrecently (3, 4).Cells tobepermeabilized

were seeded in 33:mm dishes at 105 cells per

dish When cells wereconfluentand quiescent,

t Present address: Laboratory of Tumor Virus Genetics,

National Cancer Institute, National Institutes of Health,

Be-thesda, MD 20205

cultureswere stimulatedfor18 hwith Dulbecco modified Eagle medium containing 10% calf

serum asdescribedpreviously(6) Unstimulated culturesreceived medium containingno serum Thecells were permeabilized by using lysoleci-thin, and DNAsynthesis was measured by the

incorporation of[3H]TTPintotheacid-insoluble fraction as described by Castellot et al (3) Preliminary experiments indicated that,

com-pared with unstimulated cells, there is an

in-crease(50-to100-fold) in the ability of

stimulat-ed cells to synthesize DNA Additionally, [3H]TTPincorporation was linearup to 20 min

after permeabilization ofthe cells Thereafter, the ability of permeabilized cells to synthesize

DNA was gradually lost These findings were

similarto those reportedpreviously (3, 4)

Theresults in Table1 show thatincorporation

of[3H]thymidine by intact cells or [3H]TTP by permeabilized cellswasimpaired inmouse IFN-treated cultures The degree of inhibition of

DNA synthesis was similar in both intact and

permeabilized cells There was some (ca 20%) reduction of DNA synthesis when cells were treated with human lymphoblastoid IFN rather than mouse IFN (results not shown) This was notsurprising since it isnowknownthat human

IFN can act in cells of heterologous species,

includingmice (9) The results presented in Fig

1 show that the observed inhibitory effect of mouse IFN on DNA synthesis was dose depen-dent Again, comparable levels on inhibition

were observed inintact or permeabilized cells at

agiven doseofIFN

Experiments were performed to determine whether the antiviral state induced by IFN in cells could persist after permeabilization The results (Table 2) showed that the IFN-induced

antiviral state, as judged by resistance to

en-cephalomyocarditis virus replication, was

re-895

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896 NOTES

TABLE 1 Inhibitory effect of IFN on DNA

synthesis in intact or permeabilized 3T3 cellsa

Radioactivity in:

Treatment ~Intactcells Permeabilized

Treatment (CpMXcells cells(cpm x

106 cells) 104/MgOf

protein) Unstimulated 0.04 ± 0.01 0.05 ± 0.007

Serum + mouse 0.35 ± 0.02 0.58 ± 0.16

IFN

a Quiescent Swiss mouse 3T3 cells in 33-mm Nunc

dishes were prepared and then stimulated with fresh

medium containing serum as described previously (6).

Mouse IFN (specific activity, 2 x 105 U/mg of

pro-tein), prepared by described methods (6), was used at

1,000 U/ml The cultures were incubated for 18 h, at

which time DNA synthesis was ascertained Cultures

to be permeabilized were chilled by placing the dishes

on a metal tray in ice and then washed two times with

cold phosphate-buffered saline (pH 7.4) containing 2

mM CaC12 Dishes were then washed with cold buffer

containing 35 mMMgCI2and 1 mM CaC12

Monolay-ers were permeabilized in the same buffer (1 ml) with

60 to 80 ,ug oflysolecithin (Sigma Chemical Co., St.

Louis, Mo.) per ml for 2 min at 0°C (The

concentra-tion oflysolecithinrequired for optimal

permeabiliza-tion [>95%] varied depending on the batch of cells

used Therefore, for each set of experiments the

optimum concentration of lysolecithin required was

determined.) The degree of permeabilization was

as-sessed bydeterminingthe uptake of trypan bluebythe

cells After removal of theoverlyingsolution,

permea-bilized monolayers were incubated with the buffer

described above (1ml),withoutCaC12, containing0.25

mM dCTP, 0.25 mM dGTP, 0.25 mM dATP, 0.12 mM

GTP,0.12 mM UTP, 0.12 mMCTP,1.25 mMATP,10

mM phosphoenolpyruvate, and 5 ,uCi/ml of 60 ,uM

[methyl-3H]TTP tetrasodium salt (specific activity,

77.4 Ci/mmol; New England NuclearCorp., Boston,

Mass.) Intact cultures were incubated for 30 min with

0.25 ,uCi of 1 t±M[3H]thymidine (specificactivity, 52

Ci/mmol, New England Nuclear Corp.)

Permeabi-lized cells were incubated for 20 min at 37°C in

humidified 10% CO2 Monolayers were washed with

cold phosphate-buffered saline containing 2 mM

CaC12, followedbya 15-min incubation with cold 5%

trichloroacetic acid containing 0.2 M sodium PPi

Cultures were washed five times with cold 5%

tri-chloroacetic acid and then washed three times with

absolute ethanol The air-dried monolayers were

dis-solved in 0.1 N NaOH at 37°C for 30 min, and the

amount of radioactivity was determined by liquid

scintillation counting in acidified Ready Solv

(Beck-man Instruments, Inc., Palo Alto, Calif.) Values

represent the mean ± the standard error of the mean of

three experiments.

tained even after the cells were permeabilized

with lysolecithin Although the synthesis of

en-cephalomyocarditis virus-specific RNA in

con-trolpermeabilized cellswas lowcomparedwith

thatin intactcells,therewas asignificant

reduc-

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UNITS OF IFN

FIG 1 Inhibitory effect of IFN on DNA synthesis

in permeabilized cells Quiescent cultures (33 mm)

were prepared and stimulated as described in Table 1 Some of the cultures received mouse IFN (specific activity, 2 x 105 U/mg of protein) at the indicated concentration After 18 h of incubation, sets of

cul-tures were permeabilized, and DNA synthesis in cells

was measured by methods identical to those described

in Table 1 Values are expressed as a percentage of the yield of the control stimulated cells and represent the mean the standard error of the mean of three experiments Symbols: O, intact cells; 0, permeabi-lized cells.

TABLE 2 Persistence of IFN-induced antiviral activity in permeabilized 3T3 cells'

Radioactivity (cpm x103

of [3H]uridine per culture Treatment per 45min)in:

Intact Permeabilized cells cells

Serum + actinomycinD 10.70 1.90

+ virus Serum + actinomycin D 0.24 0.07 + virus + IFN

a Quiescent cultures (33 mm) were prepared and stimulated as described in Table 1 Wherever

indicat-ed, cultures received mouse IFN (1,000 U/ml)at the time of stimulation Afterapproximately15 h of

incu-bation,cultures werechallengedwith

encephalomyo-carditis virus and subsequently incubated for 6 h in media containing 2% dialyzed calf serum Cultures receiving 1p.gof actinomycin D per ml were incubated with the drug at this time After incubation, cultures were permeabilized as described in Table 1 This was followed by a 45-min pulse with [3H]uridine (1 ,uCi;

specific activity, 77 Ci/mmol). Cell-specific(no actino-mycin D) orvirus-specific(plusactinomycinD) acid-insoluble counts were expressed as counts per minute incorporated per culture.

J VIROL.

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NOTES 897

tion in the amount of viral RNA synthesis in

IFN-treated cultures Theseresultssuggestthat

theantiviralactivity induced by IFN in cellscan

persist evenafterthe cellsarepermeabilized

Themajormetabolic pathway for thedenovo

synthesis ofthymidylic acid in cells involves the

transfer of the methylene group from N5,N10

methylene-tetrahydrofolic acid to position 5 of

deoxyuridylic acid and is catalyzed by the

en-zymethymidylate synthetase Thesalvage

path-wayinvolves the transfer of the -y-phosphate of

ATPtothe5'position of thymidylic acid and is

catalyzed by theenzymethymidine kinase The

presentresultsclearly show thatintroduction of

TTP directly into cells did not abrogate the

inhibitory effects of IFN on DNA synthesis

This suggests that the anti-mitogenic effect of

IFN should reside, at least partly, on

mecha-nisms other than the reduced uptake of

thymi-dine Our results also show that the antiviral

activity induced by IFN in cells persisted even

afterthecellswerepermeabilized Thissuggests

that the factors mediating the inhibitory effects

of IFN are molecules which may be tightly

bound intracellularly

This work was supported by grant CD#56 from the

Ameri-can Cancer Society.

LITERATURE CITED

1 Balkwill, F R., and J Taylor-Papadimitriou 1978 Inter-feron affects both G0 and S+G2 in cells stimulated from quiescence to growth Nature (London) 274:798-800.

2 Brouty-Boye, D., and M G Tovey 1978 Inhibition by interferon of thymidine uptake in chemostat cultures of L1210 cells Intervirology 9:243-252.

3 Castellot, J J., Jr., M R Miller, D M Lehtomaki, and

A B Pardee 1979 Comparison of DNA replication and repair enzymology using permeabilized baby hamster kid-ney cells J Biol Chem 234:6904-6908.

4 Miller, M R., J J Castellot, Jr., and A B Pardee 1978 A permeable animal cell preparation for studying macromo-lecular synthesis DNA synthesis and the role of deoxyri-bonucleotides in S phase initiation Biochemistry 17:1073-1080.

5 Sokawa, Y., Y Watanabe, and Y Kawada 1977 Suppres-sive effects of interferon on the transition from a quiescent

to a growing state in 3T3 cells Nature (London) 268:236-238.

6 Sreevalsan, T., E Rozengurt, J Taylor-Papadimitriou, and

J Burchell 1980 Differential effect of interferon on DNA synthesis, 2-deoxyglucose uptake and ornithine decarbox-ylase activity in 3T3 cells stimulated by polypeptide growth factors and tumor promoters J Cell Physiol 104:1-9.

7 Taylor-Papadimitriou, J 1980 Effect of interferon on cell growth and function, p 13-46 In I Gresser (ed.),

Interfer-on 1980 Academic Press, Inc., New York.

8 Tovey, M G., D Brouty-Boye, and I Gresser 1975 Early effect of interferon on mouse leukemia cells cultivated in a chemostat Proc Natl Acad Sci U.S.A 72:2265-2269.

9 Weck, P K., E Rinderknecht, D A Estell, and N Steb-bing 1982 Antiviral activity of bacteria-derived human alpha interferon against encephalomyocarditis virus infec-tion in mice Infect Immun 35:660-665.

VOL.45, 1983

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