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The multi-replication protein A RPA system – a newperspective Kengo Sakaguchi, Toyotaka Ishibashi*, Yukinobu Uchiyama and Kazuki Iwabata Department of Applied Biological Science, Tokyo U

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The multi-replication protein A (RPA) system – a new

perspective

Kengo Sakaguchi, Toyotaka Ishibashi*, Yukinobu Uchiyama and Kazuki Iwabata

Department of Applied Biological Science, Tokyo University of Science, Chiba, Japan

Replication protein A (RPA) is a single-stranded DNA

(ssDNA)-binding protein complex comprising a

hetero-trimeric combination of a large (70 kDa), middle

(32 kDa) and small (14 kDa) subunit [1,2]

Function-ally, RPA corresponds to an alternative form of a

bacterial ssDNA-binding protein (SSB) Until 2005,

only one copy of the RPA complex was thought to be

present in eukaryotes [1–9] Indeed, preliminary

analysis of the genomes of mammals and yeastindicated that they encoded a single copy of eachsubunit of the RPA complex [1,2] However, werecently found that higher plants have at least threedifferent species of complex (types A, B and C), eachdisplaying a different biological function [10–12] Orig-inally, we intended to investigate the plant repairsystem [13–43], but during the course of this study we

Keywords

convergent evolution; DNA polymerases;

eukaryotic DNA metabolism; meiotic pairing

and recombination; multi-RPA system;

O sativa and A thaliana; paralog ⁄ ortholog/

analog/heterolog; Rad51 ⁄ DMC1 ⁄ Lim15;

replication protein A; RPA subunits (70, 32

and 14 kDa)

Correspondence

K Sakaguchi, Department of Applied

Biological Science, Faculty of Science and

Technology, Tokyo University of Science,

2641 Yamazaki, Noda, Chiba 278 8510,

Department of Biochemistry and

Microbiology, University of Victoria, Victoria,

Canada

(Received 11 September 2008, revised 26

November 2008, accepted 5 December

2008)

doi:10.1111/j.1742-4658.2008.06841.x

Replication protein A (RPA) complex has been shown, using both in vivoand in vitro approaches, to be required for most aspects of eukaryoticDNA metabolism: replication, repair, telomere maintenance and homolo-gous recombination Here, we review recent data concerning the functionand biological importance of the multi-RPA complex There are distinctcomplexes of RPA found in the biological kingdoms, although for a longtime only one type of RPA complex was believed to be present in eukary-otes Each complex probably serves a different role In higher plants, threedistinct large and medium subunits are present, but only one species of thesmallest subunit Each of these protein subunits forms stable complexeswith their respective partners They are paralogs as complex Humans pos-sess two paralogs and one analog of RPA The multi-RPA system can beregarded as universal in eukaryotes Among eukaryotic kingdoms, para-logs, orthologs, analogs and heterologs of many DNA synthesis-relatedfactors, including RPA, are ubiquitous Convergent evolution seems to beubiquitous in these processes Using recent findings, we review the compo-sition and biological functions of RPA complexes

Abbreviations

ATR, ataxia telangiectasia mutated and Rad3-related; dsDNA, double-stranded DNA; MMS, methyl methanesulfonate; NER, nucleotide excision repair; PCNA, proliferating cell nuclear antigen; pol a, DNA polymerase a; RPA, replication protein A; SC, synaptinemal complex; SSB, single-stranded DNA-binding protein; ssDNA, single-stranded DNA.

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serendipitously discovered the involvement of RPA

[10–12] Interestingly, RPAs are not necessarily

com-pletely independent complexes Only one copy of the

small subunit was found, whereas there were three sets

of the large and middle subunits [10–12] The mode of

action of these RPA complexes seems to be universal,

at least in Plantae Each RPA complex must be

inde-pendently related to various DNA synthetic events

within the plant Because DNA replication and repair

are generally very similar between animals and plants

[13,44–66], the role of the RPA complex should be

reconsidered in the light of this new finding Therefore,

we retrospectively searched reports about screening for

RPA homologs in animals and fungi Humans carry

two homologs of the middle subunit (HsRPA2 and

HsRPA4) [67–69] Moreover, Richard et al recently

reported that the two human SSB homologs (hSSB1

and hSSB2) possess a domain organization that is

closer to archaeal SSB than to RPA [70] Although the

genetic and biochemical characteristics of hSSB1 are

totally different from those of human RPA, both are

critical for genomic stability [70] Thus, like Plantae,

the human DNA repair enzymes also function as a

multiple system Furthermore, the multi-RPA or SSB–

RPA mixed system is presumably universal in

eukary-otes Here, in the light of these recent discoveries, we

review the function and structure of the RPA

com-plexes

There are many reports in the literature concerning

the role of RPAs RPA is ubiquitous and essential for

a wide variety of DNA metabolic processes, including

DNA replication, repair and recombination [1] In

par-ticular, RPA is required for cross-over during meiosis

[71–74] According to a recent report [75], the large

and middle subunits of human RPA may act as an

independent prognostic indicator of colon cancer, as

well as therapeutic targets for regulation by tumor

sup-pressors involved in the control of cell proliferation

Thus, despite the previous studies on RPA, there are

many new areas of research involving this complex

that still need to be addressed

History of RPA studies

We begin this review by summarizing studies that first

identified RPA as a factor necessary for SV40

replica-tion in vitro [76–79] RPA is required for activareplica-tion of

the pre-replication complex to form the initiation

com-plex, and for the ordered loading of essential initiator

functions, such as DNA polymerase a–primase (pol a)

complex, to the origins of replication [76–79] The

gen-eral role of RPA has been studied in great detail in

mammals and yeasts [1,2] It was originally thought

that the RPA complex was evolutionarily conservedthroughout eukaryotes and that the function is funda-mental irrespective of DNA synthesis Many data wereobtained on the assumption that there is just one RPAcopy RPA accumulates along stretches of ssDNA gen-erated during DNA replication and repair (Fig 1A)[1,5–8,79–87] RPA also plays an essential role inDNA repair and is required for nucleotide excision

A

B

Fig 1 (A) RPA in the DNA replication (B) The role of RPA in NER.

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repair (NER) [88–90] During strand elongation in

DNA replication⁄ repair, RPA stimulates the action of

DNA polymerases such as pol a, pol d, pol e, pol k

and pol j [5–8,80,81,85–87] Conversely, pol f is not

under the influence of RPA, suggesting that

RPA-dependent ssDNA stretching is not always essential for

DNA polymerization [88] RPA interacts with XPA at

sites of DNA damage, stimulating XPA–DNA contact

and recruiting the incision proteins ERCC1⁄ XPF and

XPG to the damaged site (Fig 1B) [89–91] These

pro-cesses include damage detection and signaling,

tran-scriptional responses, DNA damage checkpoints and

apoptosis [4,7] RPA is known to interact specifically

with numerous transcription, replication and repair

proteins including T antigen, the tumor suppressor

p53, the transcription factors Gal4 and VP16, DDB,

uracil DNA glycosylase, recombinases and the DNA

helicases, Bloom’s and Werner’s proteins

RPA is also a checkpoint protein that has been

iden-tified by the generation of a mutant in the large

sub-unit in yeast [92] In addition, RPA was found to be

necessary for the removal of oxidized base lesions from

genomic DNA in long-patch base excision repair

[93,94] RPA also interacts with Rad51 and Rad52,

thereby playing a role in initiating homologous

recom-bination events [95–111] In the repair of double-strand

breaks by homologous recombination in

Saccharomy-ces cerevisiae, RPA stimulates DNA strand exchange

by Rad51 protein, provided that RPA is added to a

pre-existing complex of Rad51 protein and ssDNA

RPA is also implicated in forming the meiotic

recom-bination nodules [112–118] Furthermore, RPA has a

specific interaction with the tumor suppressor p53

[119–121] and promotes DNA binding and chromatin

association of ataxia telangiectasia mutated and

Rad3-related (ATR) in vitro via ATR interacting protein

[122] RPA is also required to recruit and activate

Rad17 complexes for checkpoint signaling in vivo

[123] Thus, the functions of RPA are surprisingly

ambiguous Namely, RPA functions in a wide range of

systems from DNA replication to DNA damage and

stress responses (biochemical and cell biological) as

well as cross-over in meiosis [1,2]

It is thought that the major interaction between

RPA and DNA occurs through the RPA70kDa

sub-unit, and the role of the RPA32kDa and RPA14kDa

subunits is supplementary [124] Indeed, RPA70kDa is

the major subunit of the complex having four

ssDNA-binding domains in the middle of the subunit By

contrast, RPA32kDa and RPA14kDa each possess a

single DNA-binding domain, displaying only weak

binding affinity [2,125] The contact surfaces in RPA

have been elucidated for several of its binding

part-ners The results of these studies suggest that proteinsfrom distinct processing pathways may use a smallnumber of common sites to bind RPA and remodelthe mode of DNA binding [124]

The RPA32kDa subunit is phosphorylated duringprogression of the cell cycle and in response to a widevariety of DNA-damaging agents, such as ionizingradiation, UV and camptothecin [120,126–128] RPAphosphorylation stimulated by DNA damage promotesDNA binding and chromatin association of ATR

in vitro via ATR interacting protein [83,122,129] RPA

is also required for recruitment and activation of theRad17 complexes during checkpoint signaling in vivo.RPA may function in the sensing of DNA damage[111] In budding yeast, the middle subunit (32 kDa)becomes phosphorylated in reactions that require theMec1 protein kinase, a central checkpoint regulatorand homolog of human ATR [71–74] However, themeiosis-specific protein kinase Ime2 is required fornormal meiotic progression [130] A natural target ofIme2 activity is also the middle subunit of RPA [130].Ime2-dependent RPA phosphorylation first occursearly in meiosis The middle subunit is not supplemen-tary, but is a signal acceptor for sensing various struc-turally specific DNA sites Furthermore, RPA32kDa isreportedly related to viral DNA replication [124,131].There is almost no information concerning themolecular role of the RPA14kDa subunit It is knownthat RPA14kDa contains one weak DNA-bindingdomain, which may slightly modify the mode of DNAbinding of RPA

Consequently, it was generally believed that themajor roles of RPA had been elucidated However, atthis stage, it was not known that RPA representedmore than one molecular species Thus, most research-ers did not consider the possibility of orthologs, para-logs, analogs and heterologs of the RPA complex

Multi-RPA systems

In contrast to the intensive studies of RPA in mals and yeasts, until 2001 little was known about thisprotein in plants Plants are affected by various envi-ronmental stress factors For example, DNA in plants

mam-is continuously damaged by UV irradiation from light UV is known to induce DNA damage [13],although plants generally have a higher tolerance for

sun-UV than animals Field-grown crops such as wheat arealso known to suffer continuous UV-induced DNAdamage Furthermore, the formation of reactiveoxygen species in cells due to UV irradiation, bioticstresses and secondary metabolism, causes cellularcomponents, including DNA, to be oxidized and there-

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fore susceptible to oxidative modification In addition,

the fidelity and integrity of DNA are constantly

chal-lenged by chemical substances in the environment,

ion-izing radiation and errors that occur during DNA

replication or proofreading This accumulated damage

blocks a number of critical processes, such as

tran-scription and replication, and can eventually cause cell

death Thus, UV damage can reduce the growth and

yield of plant crops Indeed, there is no difference

between the abilities of animals and plants to remove

damaged DNA [13] Plants have evolved several

DNA-repair pathways [13] Whereas previous studies on

DNA repair have focused mostly on animals and yeast

cells, recent analyses of UV tolerance and DNA repair

have addressed the responses of plants to

environmen-tal factors and the mechanisms of stress resistance in

plants [13] An additional basis for molecular analyses

has been provided by the completion of

genome-sequencing projects in model plants such as rice and

Arabidopsis Completed genome sequences allow the

identification of entire gene groups related to DNA

repair in higher plants In order to better understand

the mechanisms of DNA protection and plant DNA

repair systems, we attempted to isolate the gene

encod-ing plant RPA Surprisencod-ingly, analysis of rice revealed a

new type of RPA complex gene [10–12]

In 1997, an ortholog of the RPA70kDa subunit

(Os-RPA1) was isolated from deepwater rice (Oryza sativa

L cv Pin Gaew 56), and its expression was induced

by gibberellin [132] To use the OsRPA1 protein for

plant DNA replication studies, we aimed to clone the

cDNA and obtain the recombinant protein from rice

(O sativa L cv Nipponbare) Although we failed to

clone the OsRPA1 cDNA, we unexpectedly obtained

cDNA of the RPA70kDa subunit alternative The new

alternative gene differed greatly from OsRPA1, having

closer homology with its counterpart in

Arabdop-sis thaliana reported in the database [10] We found

that A thaliana also has a homolog of OsRPA1,

sug-gesting that two different RPA types are universally

present in seed plants [10] Rice has two different types

of RPA70kDa subunit, renamed OsRPA70a (newly

found) and OsRPA70b (OsRPA1), respectively [10]

We discovered their homologs in A thaliana, and

described the substantial properties of the T-DNA

insertion lines [11] Transcripts of OsRPA70a are

expressed in proliferating tissues, such as root tips and

young leaves that contain meristem, but also more

weakly in the mature leaves, whereas OsRPA70b is

expressed mostly in proliferating tissues [10]

The existence of these genes gives rise to an

intrigu-ing evolutionary question Why do mammals and yeast

have only one copy of the gene for the RPA70kDa

subunit in their genome? Furthermore, is only the est subunit of the RPA complex duplicated in plant,and what are the roles of the two RPA types? Interest-ingly, when the RPA70a subunit lacked the T-DNAinsertion or RNA interference (RNAi), the line could

larg-be viable [10–12] The surviving mutant was logically normal except for hypersensitivity towardssome mutagens, such as UV and methyl methanesulfo-nate (MMS) [10–12] Plants are naturally exposed to

morpho-UV for much longer than animals or yeast [133–135]and depend on sunlight for their development Becauseseed plants synthesize DNA under relatively high levels

of UV irradiation, the RPA system might be morecomplicated in plants than in animals

Therefore, we attempted to screen for rice RPA genes

in the genome (O sativa L cv Nipponbare) We foundthree different genes encoding the largest (RPA70kDa)and middle subunits (RPA32kDa), but only onegene encoding the smallest (RPA14kDa) [12] EachOsRPA70s and OsRPA32s gene was not a pseudogene

or redundant gene We designated the subunits from rice

as OsRPA70a, OsRPA70b, OsRPA70c, OsRPA32-1,OsRPA32-2, OsRPA32-3 and OsRPA14 [12] TheRPA70bsubunit is the ubiquitous RPA70 subunit found

in all eukaryotes [10] The various subunits do not domly associate with other subunits, but form a distinctcomplex Three different RPA complexes (A, B or Ctype) were composed of these subunits in vivo Types A,

ran-B and C were OsRPA70a–OsRPA32-2–OsRPA14,OsRPA70b–OsRPA32-1–OsRPA14 and OsRPA70c–OsRPA32-3–OsRPA14, respectively [11,12] Only thesmallest subunit is common to all the complexes.Because the system was also present in A thaliana[11,12], these properties may be universal in higherplants In conclusion, higher plants have a multi-RPAsystem [11,12]

The RPA complexes are spatially segregated inplants Type A is localized to the chloroplast, whereastypes B and C are found in the nuclear region [11] Inhuman and yeast cells, the middle subunit exists in thenucleus and cytoplasm, whereas the large subunit ispresent only in the nucleus [11] The RPA32kDa sub-units probably exist as each protein alone (OsRPA32-

1, OsRPA32-2, OsRPA32-3 or OsRPA14) or as freeheterodimer complexes such as OsRPA32-1–OsRPA14,OsRPA32-2–OsRPA14 and OsRPA32-3–OsRPA14[11,12]

In rice, co-regulation of OsRPA70b and OsRPA32-1during the cell cycle, and regulation of OsRPA32-1 inresponse to UV has been reported [43] RPA70kDahas been reported to be unstable when not in a com-plex Because expression of OsRPA70a was observed

at both the mRNA and protein levels, we suggest that

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the rice genome contains another protein, distinct from

OsRPA32-2 that might form a stable complex with

OsRPA70a As described earlier, the RPA32kDa

sub-unit is phosphorylated in response to cell-cycle phase

transitions and a wide variety of DNA-damaging

agents, suggesting that RPA activities are regulated by

the extent of phosphorylation [120,126–128] Rice had

three different RPA32kDa subunits This infers the

existence of independent phosphorylation systems that

control each type of RPA complex Does the

phos-phorylation occur on the same RPA complex?

Are such phenomena limited in the RPA system?

Drosophila has two paralogs of proliferating cell

nuclear antigen (PCNA) and a ‘heterolog’ (Rad9–

Rad1–Hus1) [65,136,137] Moreover, the fungus

Coprinus cinereus generates two different PCNAs by

alternative splicing, although there is only a single

copy of the gene in the genome [138] Even the

plural-izing recipe of PCNA is also phylogenetically

diversi-fied The roles of PCNA are probably diversified, and

a division of labor occurs [65] Like RadA and hSSB,

we also found another FEN-1-like analog, SEND-1

and GEN [25,63,66] All are transcribed and translated

and therefore do not represent pseudogenes

Knock-down of one of their genes in the same category seems

to lead to lethality, although there is little published

data on this subject The diversification must be closely

related to the point at which biochemical control

sys-tems divide [65] Similar considerations probably apply

to the multi-RPA system

Phylogenetic aspects of multi-RPA

systems

Sophisticated studies are required to verify whether

a specific subunit (OsRPA32-1, OsRPA32-2 or

OsRPA32-3) is responsible for phosphorylational

control Furthermore, which RPA complex corresponds

to the RPA found in mammals and yeast? Are no other

RPA types present in animals and yeasts? Whether

mammals and yeasts evolved a multi-RPA system,

which was subsequently lost over evolutionary time is so

far unclear We have investigated the plant multi-RPA

system in terms of phylogenetics

Two large RPA subunits, RPA70 and RPA32, and a

small subunit, RPA14, are relatively well conserved

among eukaryotes (Fig 2A) The deduced amino

acid sequence among OsRPA70a, OsRPA70b and

OsRPA70c showed low identity levels ( 50%) between

them [12] Similarly, the deduced amino acid sequence

among OsRPA32-1, OsRPA32-2 and OsRPA32-3 was

compared; each type also displayed low identity levels

[12] In the system, the sequence homologies among the

OsRPA70kDa subunits and among the OsRPA32kDasubunits were low [12] The B type complex wasthought to be ubiquitous in eukaryotes [12]

RPA70kDa has two RPA ssDNA-binding domains,DBD-A and DBD-B for binding ssDNA, and a third,DBD-C, which displays only weak ssDNA-bindingactivity (Fig 2B) RPA70kDa also contains the DBD-

F domain, which has been shown to interact withmultiple proteins and to interact weakly with DNA(Fig 2B) The primary amino acid sequences ofDBD-A, DBD-B, DBD-C and DBD-F domains arevery similar [12] RPA32kDa has only a single ssDNA-binding domain (DBD-D) [12] Furthermore, all thedomains have high levels of sequence homology withtheir counterparts in human and yeast RPAs [12] TheDBD-E domain is in the RPA14kDa subunit, and isalso highly conserved [12]

In yeast, RPA1 (largest subunit) can only bind tothe RPA2⁄ 3 dimer (middle and smallest subunitdimer) The DBD-C and DBD-D regions of rice arequite similar to the DBD-C and DBD-D regions of

S cerevisiae [139], but OsRPA14 has only low larity to RPA3 This sequence divergence mayaccount for the differences in binding observedbetween the yeast and rice proteins Rice DBD-A andDBD-B domains are more conserved than DBD-Cand DBD-F, implying that the primary function ofOsRPA70a and OsRPA70b is to bind DNA, and thatthis function has been conserved during evolution,even though the secondary functions of these proteinsmay have diverged Based on this analysis the B typecomplex corresponds to the mammalian and yeastRPA

simi-In plant, human and yeast, the domains of DBD-Aand DBD-B are more homologous than those ofDBD-C and DBD-F, and the biochemical characteris-tics are common among OsRPA70a, OsRPA70b andOsRPA70c It is well established that the RPA70kDasubunit accumulates along stretches of ssDNA gener-ated by stalled replication forks and⁄ or DNA damage[1,82–84] In the RPA70kDa subunit, DBD-A andDBD-B possess the strongest ssDNA-binding activity.Indeed, DBD-A and DBD-B were the first to be iden-tified as DNA-binding domains [12] DBD-C andDBD-D have a weak ssDNA–binding activity [12],whereas DBD-F interacts physically with the tumorsuppressor p53 and nucleosome remodeling complexFACT The interaction with DBD-F can also contrib-ute to an additional binding of structurally distortedDNA (i.e damaged DNA) By analogy, the primaryfunction of all the OsRPA70kDa subunits must be tofind special regions of DNA with which to bind Isthere a divergence in biochemical function among the

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various domains? What is the specialization of hSSBs

(analogs of RPA), which appeared by convergent

evolution [70]?

Furthermore, why are the middle subunits diversified

phylogenetically? As discussed earlier, the major role

of the middle subunits is not to bind to DNA,

although they may be involved in the controlling signal

via phosphorylation Indeed, in humans, HsRPA2

interacts with uracil–DNA glycosylase and XPA, but

HsRPA4 does not [67–69] Moreover, the small

sub-unit is presumably responsible for linking the other

subunits (large and middle) The driving force behind

the diversification of the small subunit is an interesting

question that needs to be addressed

The phylogenetic data suggest that the multi-RPA

(or the SSB–RPA mixed) systems are universal in

eukaryotes However, it is important to establishwhether plants have paralogs or orthologs of hSSB Inparticular, we need to investigate the in vivo functions

of each of the A, B and C types of plant multi-RPAsystems

In vivo roles of the multi-RPA system

If the multi-RPA system is unique in plants, some ofthe in vivo roles may also be specific for plants.OsRPA70a (type A complex) is localized in the chloro-plast, but OsRPA70b (type B) and OsRPA70c (type C)are found in the nuclear compartment [12] The type Asystem is thought to be plant specific, whereas types Band C could be universal Fortunately, the homologs

of OsRPA70a, OsRPA70b and OsRPA70c were found

A

B

Fig 2 (A) Pairwise comparison of each OsRPA subunit with human (HsRPA), Schizosaccharomyces pombe (SpRF-A) and Drosophila melanogaster (DmRPA) (B) Domain structures of OsRPAs Each color box indicates each DBD domain shown as the lower half of the figure DBD domain are classified into A, B, C, D, E and F.

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to be present in A thaliana (AtRPA70a, AtRPA70b

and AtRPA70c) [11,12]

Interestingly, the AtRPA70a deletion mutant

(SALK017580) was lethal, but the AtRPA70b deletion

mutant (SALK088429) was viable and hypersensitive

to UV and MMS [12] Therefore, type A may be

essential for DNA replication and transcription (and

also DNA repair) in the chloroplast Type B may have

at least some role in nuclear DNA repair [12]

Intrigu-ingly, the AtRPA70c deletion mutant does not appear

to be viable Type C shows nuclear localization, and

the AtRPA70c deletion mutant may be lethal,

suggest-ing that type C is essential for DNA replication and

transcription (and possibly DNA repair) in the nucleus

[12]

To investigate the function of the various proteins,

RNAi of AtRPA70a and AtRPA70b were performed

[140–143] The RNAi-mediated knockdown of

AtRPA70aalso displayed lethality However, RNAi of

AtRPA70bwas viable and did not differ in phenotype

from wild-type RT-PCR analysis was also carried out

using total RNA extract from seedlings of atrpa70b

mutant and the AtRPA70b RNAi line No atRPA70b

transcript could be detected Furthermore, western blot

analysis of total proteins from seedlings of wild-type

and atrpa70b mutant indicated very little AtRPA70b

[12]

These results indicated that AtRPA70a (probably,

the AtRPA70a–AtRPA32-2–AtRPA14 complex) has

an essential role, probably in DNA replication in the

chloroplast, whereas AtRPA70b (the

AtRPA70b–At-RPA32-1–AtRPA14 complex) is not essential under

normal growth conditions However, it is known that

yeast rpa70 mutants are very sensitive to mutagens

such as UV and MMS [11,12] To determine whether

AtRPA70b is similarly involved in mutagen tolerance,

the mutagen sensitivity of atrpa70b mutant and the

AtRPA70b RNAi line was tested When 1-week-old

seedlings were exposed to various UV-B doses and

then grown for an additional week in the absence of

UV-B, there were no remarkable morphological

differ-ences between wild-type, atrpa70b mutant and

AtRPA70b RNAi line seedlings, although leaf

yellow-ing was somewhat increased in the mutant and RNAi

seedlings [11,12] Compared with wild-type, the

amounts of chlorophyll (a + b) were decreased in

atrpa70b and the AtRPA70b RNAi lines [11,12]

One-week-old seedlings were also grown on MS medium

containing various concentrations of MMS or H2O2

After 1 week, growth of the wild-type plants was

inhibited by UV-B, MMS or H2O2 Compared with

wild-type plants, the growth of atrpa70b mutant and

AtRPA70bRNAi line seedlings was more inhibited by

UV-B, and was completely stopped by MMS [11,12].Mutants showed little increase in sensitivity to H2O2.Like the yeast rpa70 mutants, the atrpa70b mutantand AtRPA70b RNAi line are more sensitive thanwild-type to UV and MMS, suggesting that At-RPA70b is involved in the repair system for DNAdamaged by these mutagens [11,12]

The lethality of both the T-DNA insertion mutantand the RNAi line of AtRPA70a indicate that theAtRPA70a–AtRPA32-2–AtRPA14 complex plays anessential role, such as DNA replication, in the chlorop-lasts of living cells (Fig 3) By contrast, the mutantand RNAi line of AtRPA70b were viable but showedhigh sensitivity to UV and MMS, suggesting involve-ment of the AtRPA70b–AtRPA32-1–AtRPA14 com-plex in the repair of damaged DNA (Fig 3) However,AtRPA70c deletion was thought to be lethal, suggest-ing that the AtRPA70c–AtRPA32-3–AtRPA14 com-plex may function mainly in nuclear DNA replicationand transcription (Fig 3) Subcellular localizationanalysis suggested that the type A RPA complex isrequired for chloroplast DNA metabolism, whereastypes B and C function in nuclear DNA metabolism[12]

Recently, RPA70 and RPA32 subunits from plantshave been reported to play a role in viral and transpo-son DNA syntheses [131,144] It will be intriguing toinvestigate how the RPA complex functions in thesemechanisms Higher plants may have evolved thetype A for the chloroplast to offer protection againsthigh levels of UV irradiation Indeed, as mentionedearlier, plants are exposed to UV radiation for muchlonger than animals or yeast Higher plants depend onexposure to sunlight, including UV, for their develop-ment because their energy is derived from photosyn-thesis Thus, the repair system in subcellular organelles

is presumably much more efficient in plants than inanimals and yeast

The human homologs of RPA32, HsRPA2 andHsRPA4 [67] may correspond to OsRPA32-1 (type B)and OsRPA32-3 (type C) of plants, respectively,although only the middle subunit is diversified Inter-estingly, hSSB1 did not localize to replication foci inS-phase cells and hSSB1 deficiency did not influenceS-phase progression [70] Depletion of hSSB1 abro-gated the cellular response to DSBs, including activa-tion of ATM and phosphorylation of ATM targets,after ionizing radiation [70] Ionizing radiation andanti-cancer drugs can induce DNA DSBs, which arehighly cytotoxic lesions Cells deficient in hSSB1 exhib-ited increased radiosensitivity, defective checkpointactivation and enhanced genomic instability coupledwith a diminished capacity for DNA repair Thus,

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hSSB1 must influence diverse endpoints in the cellular

DNA damage response In this way, hSSB1 resembles

the type B system

Why are they not always found? The multi-RPA

types may resemble each other biochemically because

most of the subunits (large and⁄ or middle) display a

significant degree of similarity In many eukaryotes, the

multi-RPA system may diversify by exchanging some

subunits For example, some of the non-homolog(s) of

hSSB1 are derived from convergent evolution

Further-more, ubiquitous RPA (type B) is dispensable and can

easily be analyzed using the knockdown mutant,

whereas the type C or HsRPA complex (or hSSB2) is

lethal However, very few researchers have studied these

mutants Interestingly, the same phenomena was found

in Drosophila PCNAs, where the major PCNA is a

homolog of the ubiquitous PCNA in eukaryotes but is

dispensable [65] Subsequently we analyzed the

proper-ties of these proteins in more detail The role of the

miner subunit is not well understood because the

knockdown mutant is, as yet, unavailable [65]

A new perspective for RPA complexes

If multi-system RPAs are found to be universal each

of the corresponding functions should be reconsidered

Nuclear RPAs may be divided into two categories: (a)

replication⁄ transcription (plant C type), and (b)

repair⁄ recombination (plant B type) The large subunit

may function as an agent for ssDNA stretching [1,2],

whereas the middle subunit may act as a signal

trans-duction acceptor The small subunit may be a

connect-ing factor for formconnect-ing the heterotrimeric complex.Indeed, the small subunit mostly exists as a hetero-dimer with the middle subunit, whereas the largest sub-unit can be stabilized by binding to the dimer [10–12].Genetic knockdown of the type 1 RPA increases thelethality (i.e the type C), but type 2 RPA can surviveunless the DNA is damaged (i.e type B) Therefore,subunit variety and function of the various subunits ofRPA must be reconsidered in view of these new find-ings For example, human RPA interacted with XPA

at sites of DNA damage, stimulated XPA–DNA action, and recruited the incision proteinsERCC1⁄ XPF and XPG to the damaged site [89] TheRPA must be a complex with HsRPA2, which corre-sponds to type B In NER and long-patch base exci-sion repair, type B may be responsible for thesefunctions in eukaryote kingdoms

inter-The reported biological functions of mammalian andyeast RPA are mostly involved in meiosis The middlesubunit has an important role in regulating synaptine-mal complex (SC) formation and meiotic recombina-tion at meiotic prophase, mainly at zygotene andpachytene [71–74,114,115,130] The protein factors,such as DNA polymerases and recombinases, aremajor proteins involved in meiotic prophase events.Nevertheless, RPA is known biochemically to interact

in vitro with DNA polymerases and recombinases[6–8,13,31,40–42,44,72,85–88,138,145–169]

In fulfilling its biosynthetic roles in nuclear tion and in several types of repair, DNA polymerase isassisted by RPA In eukaryotes, recent investigationshave revealed at least 14 types of DNA polymerase

replica-Fig 3 Hypothetic model of the cellular function of A-, B- and C-type RPA com- plexes.

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(pol a, b, c, d, e, f, g, h, i, j, k, l, m and p) [45,170].

In a sense, all are analogs of each other RPA is

reported to interact with at least pol a, d, e, k and j

[3,5–8,76,80,81,85–88] RPA contributes to the high

fidelity of the polymerases during DNA synthesis Of

the polymerase species, pol a, d and e replicate DNA

during S phase, but pol a is replication specific [80]

All the other polymerases are involved in DNA repair

and recombination [81] We reported that in meiosis

two categories of DNA polymerases (a) pol a complex

and (b) pol k and l were expressed [165,168] The

former is for replication at zygotene (or SC formation)

and the latter is for repair and recombination at late

zygotene to pachytene (Fig 4) [155,165,168,171–173]

Using a D-loop recombination intermediate substrate,

we observed that either pol k or pol l can promote

the primer extension of an invading strand present in a

D-loop structure [168] Both could fully extend the

primer in the D-loop substrate, suggesting that the

D-loop extension is an activity that is intrinsic to

the polymerases [168]

Two orthologs of the recombinases, Rad51 and

Lim15⁄ Dmc1, are present in meiosis [44,114,115,152–

154,161,162,167] These recombinases occur at late

leptotene to early zygotene (Fig 4) The interaction of

RPA and Rad51 is well established Another meiotic

role of RPA was also found At meiotic prophase (lateleptotene to early zygotene), with RPA, the homology-search recombinase complex is involved in homologouschromosome synapsis, preventing the formation ofsuperfluous reciprocal recombinant events (Fig 4)[114,115] Both Rad51 and Lim15⁄ Dmc1 were identi-fied as being involved in this process, although thespecific function of each protein is not yet known [44].Are the DNA polymerase and recombinasefunctions mediated by one species of RPA complex?Interestingly, dephosphorylation of transformed nod-ule-associated histone H2AX chromatin occurs at thistime This suggests annealing of single strands orrepair of DSBs By a similar mechanism, if the middlesubunit of RPA is also dephosphorylated, RPA wouldlose the function of maintaining the noncross-overcondition We must also consider the role of the multi-RPA system during the meiotic prophase events

It is known that a small amount of DNA replicates

at zygotene (pairing DNA synthesis) and that therepair synthesis of DNA occurs at pachytene (cross-over DNA synthesis) [172,173] The two sequentialDNA synthesis reactions play a role in the progression

of meiosis It is possible that a complex of RPA andpol a differs from the recombination-dependent RPA.Because DNA polymerase searches for the RPA–Fig 4 Hypothetic model of meiotic cell cycle and its relation to RPA.

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ssDNA complex structure on the DNA, RPA

complexed with pol a are probably functionally

inde-pendent from RPA complexed with other repair

polymerases Pol k and l were thought to be involved

in the ‘crossover DNA synthesis’ for DNA

recombina-tion Because the pol k(or the pol l)-deficient mutant

is viable, RPA may be like the type B or HsRPA2

type However, ‘pairing DNA replication’ appears to

be specific for SC formation At that stage, the DNA

polymerase a-catalytic subunit and primase are

pre-sumably also present [165] This replication could be

the basis for SC extension and formation of the

transi-tion nodules [44] Indeed, this process probably

requires RPA, such as the type C form (Fig 4)

During prophase, DNA polymerases as well as

paralogs and orthologs of PCNA, recombinases

and perhaps RPA are required (Fig 4) [42,44,45,

151,152,155,157,159,160,165,168,171] Electron

micros-copy data [115,117] suggest that meiotic functions

in vivo are shared by each of the paralogs and

ortho-logs, and maybe also the analogs and heterologs

Indeed, control of the biological process could be more

finely tuned by sharing function amongst paralogs,

orthologs, analogs and heterologs

Background for the screening of

multiple protein systems involved in

DNA metabolism

We have studied many protein factors in DNA

replica-tion⁄ repair and their relation to the meiotic system in

higher plants (O sativa and A thaliana) [13–

43,45,156,171], a fungus (C cinereus) [44,138,145–155,

157–169] and an arthropod (Drosophila melanogaster)

[44–66] Each of the materials represents the biological

kingdom of plant, fungus and animal, respectively

Our research aimed to comprehensively understand

these DNA synthesis-related events in phylogenetically

diverse species In addition to RPA, we elucidated

many of the related factors, such as Rad51,

Lim15⁄ Dmc1, RadA, PCNA, DDB, XRCC1, Rad2

family nucleases and special nucleases, DNA

polyme-rases, ORC1, RFC, RecQ, DNA ligases, CAF-1,

mtTFA, Rrp1, Mer3, Snm1, Rad6, SUMOylation

fac-tors (Aos1, Uba2, Ubc9, SUMO), leucine

aminopepti-dase and 26S proteasome-related factors (Jab1, Sgt1,

DnaJ) (Table 1) During the course of our

experi-ments, we frequently observed that protein factors

involved in the same DNA metabolic processes are not

always homologs in eukaryotic cells Although the

paralogs and orthologs are ubiquitous, evolutionally

different factors were often found to be involved in

the same biosystems, which are referred to as ‘analogs’

and ‘heterologs’ Indeed, convergent evolution might

be ubiquitous in eukaryotic DNA metabolic processes.According to definition, ‘homolog’ is a gene related to

a second gene by descent from a common ancestral DNAsequence ‘Ortholog’ is a gene in different species thatevolved from a common ancestral gene ‘Paralog’ is agene related by duplication within a genome Orthologsretain the same function in the course of evolution,whereas paralogs evolve new functions ‘Analog’ is a genethat has common activity but not a common origin

‘Heterolog’ is a gene that differs in both origin and ity Heterolog does not classify homolog, ortholog, par-alog or analog It may be also said that heterolog is used

activ-as a synonym of ‘just different protein (gene)’, bactiv-asically.For example, PCNA is not one copy [65,138,159];two PCNA paralogs and one PCNA-like heterotrimer(Rad9–Rad1–Hus1) (‘analog’ or ‘heterolog’) werefound in Drosophila [65,136,137] Rad9–Rad1–Hus1 isfound universally in eukaryotes Plant SYCP1 andyeast Zip1 mediate the same role in meiosis, despitedisplaying no significant homology (‘analog’ or ‘het-erologs’) [174,175] Similarly, human mus81–Eme1 isfunctionally the same as Escherichia coli RuvC (‘ana-log’ or ‘heterologs’) [176–178] In plants, two recA-likeprotein paralogs (Rad51 and Lim15⁄ Dmc1) as well as

a prokaryotic recA homolog (RadA) were found logs’) [42] Furthermore, this is not the plastid compo-nent [42] As described earlier, in addition to the twosubtypes of RPA (HsRPA2 and HsRPA4) two humanSSB homologs are also present (‘analogs’) [70] More-over, in human, five Rad51 paralogs (Rad51B,Rad51C, Rad51D, Xrcc2 and Xrcc3) have been found[179–181] Two FEN-1 paralogs (FEN-1a and FEN-1b) and one analog (SEND-1) were found in plants[25,26], and another FEN-1 analog occurs in Drosoph-ila (GEN) [63,66] DNA polymerases, especially forDNA repair, are greatly diversified in eukaryotes[76,182,183] DNA polymerase b (pol b) for shortpatch base excision repair are found only in verte-brates [45]; plant short patch base excision repair usespol f instead [33,39,45] However, as yet, a recBCDhomolog has not been found in the eukaryotic recom-bination process Prokaryotic homologs such as RadAand hSSB are often found in eukaryotes (‘analog’ or

(‘ana-‘heterolog’), although there are the eukaryotic tional alternatives [42,70] All the protostomic animalslack any X family DNA polymerases essential fordevelopment of the nervous and immune system [45]

func-In Drosophila, AP endonuclease 1 homolog (Rrp1)binds to pol f [64] Plant XRCC1 lacks the polymer-ase-binding domain [33,39] Therefore, factor variation(orthologs, paralogs, ‘analogs’ and ‘heterologs’) seems

to be ubiquitous in eukaryotic DNA metabolism

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