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Consequently, injury to a por-tion of the talus may lead to degen-erative changes of the articular sur-faces and subsequent loss of range of motion ROM.1The talus has a lim-ited reparati

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Aaron K Schachter, MD, Andrew L Chen, MD, MS, Ponnavolu D Reddy, MD, and Nirmal C Tejwani, MD

Abstract

Early accurate diagnosis of

osteochon-dral lesions of the talus is important

because optimal ankle joint function

requires talar integrity.1 Boyd and

Knight2showed that the tibiotalar

ar-ticulation is subjected to more load per

unit area than any other joint in the

body Consequently, injury to a

por-tion of the talus may lead to

degen-erative changes of the articular

sur-faces and subsequent loss of range of

motion (ROM).1The talus has a

lim-ited reparative capacity because a large

portion of it is covered by articular

cartilage, which limits the vascular

supply.3-6

Osteochondral fractures refer to

le-sions affecting the articular cartilage

of the talar dome and the underlying

subchondral bone.7Originally

refer-red to as “osteochondritis dissecans,”

these lesions were thought to be

is-chemic.8 Subsequent studies have

shown that, in most cases, these

injuries are sequelae of previous

trauma.7,9-13Moreover, the results of

these studies showed that the

lo-cation of the lesion could be

pre-dicted, based on the mechanism of injury

Etiology and Epidemiology

Fractures of the talar body account for approximately 1% of all fractures in the human body.14A large proportion

of these are transchondral or com-pression fractures of the talar dome, which are often unrecognized be-cause frequently they are associated with other, more obvious injuries to the foot and ankle Alexander and Lichtman11observed that 28% of these injuries were associated with other fractures These coexisting fractures, approximately half of which involve the ankle malleoli, may overshadow

a significant osteochondral defect of the talus.1Similarly, Van Buecken et

al15 reported that these injuries (ie, transchondral talar fractures) were as-sociated with 6.5% of ankle sprains

Berndt and Harty7found that 57% of talar dome lesions were located me-dially and 43% laterally Others have

reported a fairly equivalent distribu-tion9(Fig 1)

In a series of 70 patients (71 frac-tures), all lateral talar dome injuries were associated with a traumatic event, whereas only 64% of medial ta-lar dome injuries were attributed to trauma.10Others have corroborated these results, reporting that all

later-al lesions were associated with

trau-ma (Fig 1, B and C) but only 82% of medial lesions were11(Fig 1, A) Al-though trauma usually is the cause

of injury, it may not be a single event but may consist of a series of

repeat-ed, less intense injuries.12In a com-prehensive review of published stud-ies of 500 patients, Flick and Gould13 reported that a history of trauma was present in 98% of lateral dome lesions

Dr Schachter is Resident, Department of Ortho-paedic Surgery, NYU–Hospital for Joint Dis-eases, New York, NY Dr Chen is in private practice, Littleton, NH Dr Reddy is Fellow, Department of Orthopaedic Surgery, NYU–Hos-pital for Joint Diseases Dr Tejwani is Associate Professor, Department of Orthopaedic Surgery, NYU–Hospital for Joint Diseases.

None of the following authors or the departments with which they are affiliated has received anything

of value from or owns stock in a commercial com-pany or institution related directly or indirectly

to the subject of this article: Dr Schachter, Dr Chen, Dr Reddy, and Dr Tejwani.

Reprint requests: Dr Tejwani, Bellevue Hospital–

NB, 21 W 37, 550 First Avenue, New York, NY 10016.

Copyright 2005 by the American Academy of Orthopaedic Surgeons.

Osteochondral lesions of the talus occur infrequently and usually represent late

se-quelae of ankle trauma Because of the functional significance of the talus and its

limited capacity for repair, correct early diagnosis is important Osteochondral

frac-tures should be suspected in patients with chronic ankle pain, especially those with

a prior ankle injury Historically, plain radiographs have been used to stage lesions;

more recently, magnetic resonance imaging and arthroscopy have been used

Non-surgical management remains the mainstay of treatment of acute, nondisplaced

os-teochondral lesions Surgical management is reserved for unstable fragments or

fail-ure of nonsurgical treatment Recent advances in osteochondral grafting have allowed

reconstruction of the talar dome, leading to more predictable relief of pain and

im-provement of function.

J Am Acad Orthop Surg 2005;13:152-158

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but in only 70% of medial dome

le-sions Persistent ankle pain in the

ab-sence of any recognized trauma should

heighten suspicion of a talar

osteo-chondral lesion

Mechanism of Injury

Berndt and Harty7originally described

two possible mechanisms for

osteo-chondral fractures of the talus

Com-pressive injury to an ankle positioned

in dorsiflexion and inversion (eg, a

di-rect tibiotalar impact) may crush the

subchondral bone of the lateral talar

dome, with or without overlying

car-tilage damage Alternatively,

subject-ing the plantarflexed ankle to impact

forces of inversion and external

rota-tion can produce osteochondral

inju-ries to the medial talar surface O’Farrell

and Costello16also suggested that the

medial talar osteochondral lesion is

caused by a combination of inversion

with plantar flexion Other mechanisms

thought to account for lesions in other

locations17include impaction of the

dome against the lateral malleolus,

re-sulting in lateral dome lesions, or

against the posterior tibial lip,

result-ing in medial defects.7,18

Classification

In 1959, Berndt and Harty7proposed

a staging system based on

radio-graphic findings: stage I, small area

of subchondral bone compression;

stage II, osteochondral fragment par-tially detached; stage III, osteochon-dral fragment completely detached but not displaced; stage IV, osteo-chondral fragment completely de-tached and displaced Other grading systems based on more recent radio-logic techniques also have been de-scribed For example, Anderson et

al19and Ferkel et al20used magnetic resonance imaging (MRI) to classify talar osteochondral injury

With interest in and use of ankle arthroscopy increasing, some have questioned the accuracy of classifica-tion based solely on plain tomogra-phy Pritsch et al21graded lesions ac-cording to articular injury visualized during ankle arthroscopy, ranging from grade I (intact appearance) to grade III (frayed appearance) The au-thors found that radiographic and ar-throscopic findings correlated

poor-ly Fifty percent of lesions classified

as stage IV according to the Berndt and Harty system were found to be intact viewed through the arthroscope

However, direct visualization of an in-tact articular surface does not permit the underlying bone to be examined;

thus, the extent of a bony lesion may

be underestimated Pritsch et al21 em-phasized that treatment protocols should be based on the integrity of the articular cartilage and that plain radiographs may not necessarily show

the critical elements of talar osteochon-dral injury This concept under-scores that MRI and ankle arthroscopy have the potential to play important roles in the evaluation of these injuries

Clinical Evaluation

The first step in assessing ankle pain should be a meticulous clinical eval-uation to differentiate among the many potential diagnoses, including liga-mentous injury, fractures of the fib-ula, and fractures of the tibial plafond

In patients with acute injury, the an-kle and foot may be swollen and pain-ful, limiting the specificity of the ex-amination Nonetheless, the ankle and foot should be palpated to identify any discrete locations of tenderness A careful neurovascular assessment is also essential ROM of the injured an-kle and hindfoot should be compared with ROM in the contralateral lower extremity Testing for instability should

be performed, including an anterior drawer test with the ankle in both plantar flexion and dorsiflexion The ankle also should be subjected to inversion and eversion stress testing Although ankle sprains are more common than osteochondral injuries

of the talus, talar osteochondral

inju-ry should be included in the differ-ential diagnosis of chronic ankle pain The index of suspicion for talar osteo-chondral injury should be high in the

Figure 1 Plain radiographs of posttraumatic osteochondral injuries (arrows) A, Mortise view of a medial injury B, Anteroposterior view

of a lateral lesion of the talus C, Oblique view of a lateral lesion of the talus.

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setting of ankle pain without any

rec-ognized trauma or with persistent

an-kle pain after an acute injury has

re-solved

Radiographic Evaluation

Plain Radiography

Standard radiographic views of the

ankle should be obtained, including

anteroposterior, lateral, and mortise,

with weight bearing if possible Canale

and Kelly10proposed that the talar

pro-file may be visualized better with an

ankle radiograph made with 15° of

foot pronation and with the tube

an-gled 75° cephalad Nonstandard

pro-jections that allow further elucidation

of the talar profile include the

mor-tise view of the ankle in plantar

flex-ion to visualize posteromedial lesflex-ions

and stress views to detect

ligamen-tous laxity DeLee22suggested that

ra-diography cannot identify

cartilagi-nous defects or grade I (nondisplaced)

lesions The inability of plain

radiog-raphy to visualize nonosseous

struc-tures limits its usefulness.10

Computed Tomography

Computed tomography (CT) allows

the integrity of the subchondral bone

to be assessed in multiple planes, and

CT is often invaluable in preoperative

planning10,23(Fig 2) However, CT is

limited in its ability to visualize

cer-tain osteochondral lesions, especially

cartilaginous or nondisplaced (grade

I) lesions In comparing the use of CT

and MRI to evaluate possible

osteo-chondral talar defects identified by

bone scintigraphy, Anderson et al19

re-ported that, although CT and MRI were

90% concordant in identifying lesions

visible by plain radiography, CT was

capable of identifying only 4 of 14

le-sions that were not evident on plain

radiographs (stage I), whereas MRI

demonstrated all 14

Bone Scintigraphy

Anderson et al19examined the role

of bone scintigraphy in the evaluation

of patients with posttraumatic ankle disability, reporting a 57% incidence

of osteochondral defects of the talar dome, despite normal appearance on radiography The authors found bone scintigraphy to be useful in evaluat-ing ankle injuries when radiographs appear to be normal but suggested that a positive bone scan be followed

up with MRI Urman et al24 found that when bone scans showed abnor-mal uptake in the talar dome on at least one view, the scan was 94% sen-sitive and 96% specific for osteochon-dral injury

Magnetic Resonance Imaging

MRI allows multiplanar evalua-tion and offers the advantage of

vi-sualizing the surface of articular car-tilage and subchondral bone as well

as edema and other features of the surrounding soft tissue (Fig 3) MRI also allows early subchondral dam-age (stdam-age I lesions) to be

detect-ed.25,26In one study,19MRI allowed grade I osteochondral lesions to be identified correctly in all 14 patients who had had abnormal results on bone scintigraphy MRI findings also have been found to correlate closely with visual findings during arthros-copy.20

Radiographic Protocol

Stone27proposed a radiographic protocol for evaluating osteochondral lesions of the talus Plain radiography

Figure 2 Computed tomography scans of a medial talar osteochondral lesion A, Coronal view demonstrating extent of the lesion B, Axial view showing the medial malleolar lesion

(arrow).

Figure 3 MRI scans of an ankle A, Sagittal T2-weighted scan demonstrating an anterior talar dome lesion B, Coronal T1-weighted scan demonstrating a medial talar dome defect.

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should be used for initial evaluation

of patients presenting with acute

an-kle injury with hemarthrosis or with

significant tenderness at the bony

landmarks of the ankle Any

osteo-chondral lesion identified should

then be evaluated with CT to

deter-mine the size, shape, location, and

de-gree of displacement Persistence of

pain despite normal appearance on

plain radiographs warrants further

investigation, such as MRI or bone

scintigraphy The MRI scans should

be scrutinized for associated injuries

(eg, ligamentous disruption) that may

be responsible for, or contribute to,

persistent ankle pain

Treatment

Nonsurgical

Generally, nonsurgical treatment

in-volves an initial period of no weight

bearing with cast immobilization,

fol-lowed by progressive weight bearing

and mobilization to full ambulation

by 12 to 16 weeks.1Berndt and Harty7

found that, after acute osteochondral

injury, nonsurgical treatment with

im-mobilization nearly always resulted

in healing Many authors have

sug-gested that the decision to operate

should depend on the grade of the

le-sion Berndt and Harty grade I and

II lesions should be managed

non-surgically for up to 1 year to allow

for resolution before resorting to

surgery.9,11,15,27-29Nevertheless, a

meta-analysis of 14 studies with a total of

201 patients showed only a 45%

suc-cess rate of nonsurgical treatment of

grade I, grade II, and medial grade

III talar osteochondral lesions (not all

injury types were specified).30

Non-surgical treatment of chronic lesions

had a success rate of 56%.30Shelton

and Pedowitz29reported just 25%

sat-isfactory results for nonsurgical

treat-ment of grade II and III lesions

Surgical

Failure of nonsurgical

manage-ment or the presence of advanced

grade III or IV lesions often necessi-tates surgical intervention

Excision, Drilling, and Curettage

Excision may be done with either open or arthroscopic techniques Ex-cision of the subchondral fragment and curettage of the lesion’s surface

is done to remove debris, fibrous tis-sue, or devitalized cartilage This pro-cedure has been recommended in the treatment of chronic lesions and le-sions with necrotic material.22 Vascu-lar access channels are then created

in the underlying subchondral bone using an awl, drill, or arthroscopic shaver This allows marrow elements

to migrate into the site of injury and produce fibrocartilage to cover the le-sion.1,27,30,31

Generally, arthroscopic excision and drilling can be done as outpatient pro-cedures They are associated with fa-cilitated rehabilitation, a quick return

to normal function, and minimal wound-healing complications.27 Trans-articular drilling may be indicated in the treatment of a symptomatic os-seous lesion underlying an intact car-tilaginous surface.27However, expo-sure can be limited by the topography

of the joint as well as by the diffi-culty in reaching posterior lesions through standard anterior portals

Transarticular drilling is also user de-pendent and requires specialized equipment and arthroscopic instru-ments

Results of excision and drilling have been encouraging compared with those of nonsurgical treatment In a review of 16 studies with a total of

165 patients, Tol et al30found excision, curettage, and drilling to have had good to excellent results in 88% of pa-tients with grade III and higher lesions

However, simple excision and curet-tage without drilling (9 studies, 111 patients) had a success rate of 78%, whereas excision alone (5 studies, 63 patients) had a success rate of 38%.30 The success rate of excision alone has been shown to be lower than that of nonsurgical treatment (45%).32

Internal Fixation

Internal fixation of osteochondral lesions may be done using a variety ofmethods,includingscrews,Kirsch-ner wires, and bioabsorbable

devic-es.33,34DeLee22proposed that internal fixation is indicated when the injury occurs acutely and the fracture is

larg-er than one third the size of the re-spective dome Stone27suggested that the lesion should be at least 7.5 mm

in diameter and that the patient be young for surgical fixation

The surgical approach is determined

by the location of the lesion Lesions

on the lateral aspect of the talar dome may be approached using either an anterolateral or posterolateral approach; anteromedial lesions may be accessed through an anteromedial approach Posteromedial lesions may require os-teotomy of the medial malleolus for adequate exposure Tochigi et al35 sug-gested using an anterolateral tibial os-teotomy to allow access to centrolat-eral talar lesions Excision of loose fragments with débridement and curet-tage of the lesions should be performed before internal fixation of the osteo-chondral fragment.22

Current Trends

Osteochondral Allograft

Fresh-frozen osteochondral al-lografts have been used to repair os-teochondral lesions of the talus Gross

et al36reported retrospectively on a series of nine patients in whom they had implanted talar osteochondral al-lografts to avoid the donor-site mor-bidity associated with autograft har-vest Only four of the patients had an identifiable previous trauma to the af-fected ankle One of the procedures was performed for a traumatic open fracture with talar osteochondral de-ficiency The authors’ indications for performing the procedure included a lesion with a diameter of at least 1 cm and a depth of at least 5 mm as well

as inability to perform primary repair

of the fragment

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Following open débridement of

the lesion to a bleeding host bed,

prefashioned fresh-frozen cadaveric

osteochondral allografts were

trans-planted using one or two

minifrag-ment cancellous screws for graft

fixation Postoperative management

included cast immobilization for 2

weeks, with subsequent ROM

exer-cises and a patellar tendon–bearing

brace for 1 year At a mean follow-up

of 11 years, six of the nine grafts

remained intact The three failures

resulted from fragmentation and

resorption of the allograft

Osteochondral Autograft

Osteochondral autografts use

car-tilage and subchondral bone

harvest-ed from host non–weight-bearing

ar-ticular surfaces Both the knee and the

talus itself have been used as donor

sites.37-42

Assenmacher et al43reported on

arthroscopically assisted autograft

transplantation in nine patients with

talar dome lesions The lesions were

graded using both MRI and

arthros-copy Unstable lesions were identified

by radiographic evidence of grade III

or IV injury or by the arthroscopic

vi-sualization of a loose fragment

Pa-tients considered to have unstable

le-sions underwent autograft of the talar

osteochondral defect with a plug

har-vested from the ipsilateral distal

fe-mur The lesions were débrided

be-fore the grafts were placed After

adequate débridement, grafting was

performed through an arthrotomy

The prefashioned graft was inserted

into a reamed recipient site and

packed into place Postoperatively,

patients remained non–weight

bear-ing for 7 weeks, with ROM exercises

begun 10 days postoperatively There

were no complications or revision

surgery, and results were good or

ex-cellent as assessed with the American

Orthopaedic Foot and Ankle

Soci-ety (AOFAS) ankle-hindfoot score

Follow-up MRI demonstrated that

the grafts were incorporated in all

subjects at 9 months postoperatively

Scranton and McDermott44reported

on osteochondral autograft replace-ment of stage IV talar lesions in 10 pa-tients The distal femur was used as

a donor site; average increase in the AOFAS ankle-hindfoot score was 27 points Mendicino et al38corroborated these results; they reported a high level

of patient satisfaction, excellent return

to function, and no donor site com-plaints with the use of distal femoral osteochondral autografts to treat stage III and IV lesions

The anterior talar dome also has been used as a donor site for osteo-chondral autograft of the talus Lee39 reported no postoperative complica-tions and cited advantages of the ta-lar dome donor site that include elim-ination of a second surgical site, decreased intraoperative time, and a close match in shape of the graft to the native recipient site

Mosaicplasty autograft also has been described, with reconstruction

of the osteochondral defect using multiple, small-diameter plug au-tografts as opposed to one large, pre-fashioned graft Hangody and col-leagues41,42reported on the outcomes

of talar mosaicplasty, with the

medi-al or latermedi-al femormedi-al condyle as the do-nor site In 36 patients, multiple grafts

of 4.5 × 3.5 mm were harvested to re-constitute the talar defects, which av-eraged 1 cm in diameter (Fig 4) Us-ing the Hanover scorUs-ing system, ankle function was judged to be good

or excellent in 34 of the 36 patients (94%) at follow-up of 2 to 7 years.42 All patients returned to full activity

Functional performance of osteo-chondral grafts has been evaluated

Histologic analysis by Giannini et

al40 showed that, unlike drilled le-sions, which heal with fibrocarti-lage,41,45 osteochondral autografts heal with type II collagen at the re-cipient site Type II collagen is char-acteristic of hyaline cartilage Gian-nini et al40reported an increase in the AOFAS ankle-hindfoot score from an average of 32 preoperatively to 91 postoperatively in eight patients

Summary

Because talar integrity is important for ankle function and because artic-ular repair is difficult, osteochondral lesions may result in functional im-pairment and, later, chronic ankle pain and functional debilitation Os-teochondral injury should be sus-pected in cases of recalcitrant ankle pain once more common causes, such as ligamentous injury, are ruled out Although occasionally the etiol-ogy of such lesions is unidentifiable, the majority arise as late sequelae of ankle trauma Lateral lesions are thought to be secondary to com-bined forceful dorsiflexion and ever-sion of the ankle, whereas medial in-juries may result from combined ankle inversion and plantar flexion

If an osteochondral talar defect is suspected but plain radiographs fail

to reveal the lesion, MRI may be use-ful for identifying early, nondis-placed lesions

Early (grade I or II) lesions may be amenable to nonsurgical treatment; more severe (grade III or IV) lesions

or those for which nonsurgical man-agement has failed may necessitate surgical intervention Subchondral drilling is done for stable lesions to encourage fibrocartilage growth, and osteochondral grafts may be used to restore the talar articular surface for unstable or larger defects To date, functional outcomes reported for

os-Figure 4 Mosaicplasty Intraoperative pho-tograph showing osteochondral autografts (arrows) at the site of a talar dome defect.

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teochondral grafting of the talus

gen-erally have been favorable Recent

re-search has suggested that healing of

such grafts may occur with a predom-inance of type II collagen, but further investigation is necessary to

deter-mine whether these weight-bearing surfaces are biomechanically similar

to hyaline cartilage

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