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Lecture Clinical procedures for medical assisting (4/e): Chapter 1 – Booth, Whicker, Wyman

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Chapter 1 - Principles of asepsis. Our bodies are amazing structures that defend us against infections under normal circumstances. As you read this chapter you will learn about disease-causing microorganisms, how the body defends itself against infections, and ways that infections might occur.

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Principles of

Asepsis

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infectious disease prevention.

1.2 Identify the types of microorganisms that

cause disease.

1.3 List some infectious diseases, and identify

their signs and symptoms.

1.4 Discuss the importance of preventing

antibiotic resistance in a health-care setting.

1.5 Describe ways you can help prevent

antibiotic resistance in health-care settings.

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1.7 Explain how the body’s defenses protect

against infection.

1.8 Describe the cycle of infection.

1.9 Identify and describe the various methods of

disease transmission.

1.10 Explain how you can help break the cycle of

infection.

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– Antibiotic-resistant organisms

– Importance of patient education on the proper use of antibiotics

You will learn about:

– Disease-causing

microorganisms – How the body fights

disease – Ways infections occur

structures that defend us 

against infections under

normal circumstances

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Edward Jenner (1749–1823) • Developed first effective

of puerperal fever to women in childbirth

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Louis Pasteur (1822–1895) • Helped develop the germ

theory of infectious disease, stating that disease is

caused by microorganisms

Joseph Lister (1827–1912) • Helped develop germ theory

• Introduced aseptic techniques through the use of antiseptics

on wounds, surgical sites, and surgical instruments

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Robert Koch (1843–1910) • Developed a set of proofs,

known as Koch’s postulates, claiming that microbes cause disease

Sir Alexander Fleming

(1881–1955)

• Discovered penicillin

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• Threat of infection still present

– New infectious diseases

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though great advances have been made in

controlling infections over the past century?

ANSWER: The threat of infection is still present because

of new diseases and diseases that have become resistant

to treatments.

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• Pathogens

– Microorganisms capable of causing disease

– Evade host defenses

• People avoid infections most of the time

– Many microorganisms are beneficial or harmless

– Normal defenses resist infection

– Conditions are not favorable for pathogens to grow

and be transmitted

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Prions* • Infectious particle

made of protein

• No nucleic acid

• Reproduction unknown

Pr P

Creutzfeldt-Jakob disease Mad cow disease

* Experts disagree as to whether prions are directly responsible 

for disease or merely aid an unknown agent in causing disease

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Viruses • DNA or RNA

surrounded by protein coat

• Reproduced in living cells

• Very small

zoster virus Chickenpox

Varicella-Bacteria • Single-celled

• Reproduce quickly

• Mostly asexual reproduction

Vibrio cholerae Cholera

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Protozoans • Single-celled

• Reproduction mostly asexual

Entamoeba histolytica Amebic dysentery

Fungi • Multicellular

• Reproduction is sexual and asexual

Candida albicans Candidiasis

Helminths • Multicellular

parasitic

• Contain specialized organs

• Sexual reproduction

Enterobius vermicularis Pinworms

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exposed to microorganisms What are the reasons for

this?

ANSWER: This is because:

many microorganisms are beneficial or harmless

we have normal defenses to resist infection

conditions are not favorable for the pathogen to grow

and be transmitted

Correct!

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symptoms of common

infectious diseases

can help protect

against exposure

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• Itchy rash  fluid-filled blisters

• Slight fever, headache, general malaise

• Spread by direct, indirect, droplet, or airborne transmission

• Isolate until all blisters have scabbed over

• 1996 – live vaccine approved

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• Characterized by a harsh, barking cough, difficulty breathing, hoarseness, and low-grade fever

• Most common in infants and young children

• Symptoms lessened by humidification of air, rest, and clear fluids

• Commonsense precautions to prevent spread

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– 35 – 50% of teens develop mononucleosis

• Symptoms – fever, sore throat, swollen lymph nodes

• Virus remains dormant for life

• Occasionally reactivates as tumors

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• Monitor closely – bacterial meningitis

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• Spread through blood or fecal-oral route

• Acquired immune deficiency syndrome

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(Flu) body aches, upper respiratory congestion

• Isolation and commonsense precautions

• Vaccines

– Live, attenuated virus – nasal spray – Inactivated virus – IM injection

• Annual vaccination

– People at risk for complications

• People older than 50 years old

• People in close contact with persons at risk for complications

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(Rubeola) • Spread by droplets or direct transmission

• Initial symptom of fever develops 8 to 13 days after exposure, followed by a

characteristic itchy rash 14 days after exposure

• Isolation for 7 days after rash appears

• Keep children under 3 years old away from anyone with the disease

• Reportable to state or county health dept

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coverings of brain and spinal cord and the fluids around them

• Viral – milder form

– Clears in 1 to 2 weeks without treatment – Aseptic meningitis

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requiring immediate treatment

– Vaccination available for people in high-risk groups

– Symptoms – red, blotchy rash, confusion, delirium, light sensitivity, headache, fever and chills, nausea and vomiting, sleepiness, stiff neck – May spread through exchange of respiratory and throat secretions

– Reportable to state or county health dept.

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– Primarily affects salivary glands

• Incubation – 2 to 3 weeks

• Pain related to inflammation of parotid gland and fever

• Isolate until glandular swelling stops

• Reportable to state or county health dept

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(Whooping

quick short coughs, characteristic “whoop”

during inhaled breath following coughing fit

• Isolate for 3 weeks following onset of spasmodic coughs

• Reportable to state or county health dept

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herpes virus

• Infants and young children

• Incubation 5 to 15 days

• Symptoms – sudden, high fever; sore throat;

swollen lymph nodes; rash

Rubella (German

Measles) • Highly contagious viral disease

• Direct or droplet transmission

• Incubation 16 to 18 days

• Symptoms – fever and itchy rash

• Vaccination available

• Reportable

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pharyngitis

(strep throat) – Sore throat, swelling of pharyngeal mucosa, fever, headache, nausea,

abdominal pain – Treat with antibiotics

• Scarlet fever

– Bacteria becomes systemic – Characteristic “strawberry rash”

– Incubation 7 to 10 days – Isolate 7 days

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pharyngitis

(cont.) – Occurs after apparent recovery from strep throat– Autoimmune disorder – antibodies to

streptococci cross-react with heart tissues – Symptoms – carditis, ECG changes, joint pain and inflammation, fever

• Acute post-streptococcal glomerulonephritis

– Inflammation of glomerulus of the kidney resulting

in inadequate filtering of the blood – Symptoms – swelling of hands and feet, decreased urine output, hypertension, protein in urine

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following a contaminated puncture wound

• Incubation – 3 to 21 days

• Late symptoms – lockjaw, paralysis

• No isolation needed, but reportable

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lungs

• Symptoms – night sweats, productive cough, fever, chills, fatigue, unexplained weight loss, diminished appetite, bloody sputum

• Incidence – higher in urban centers

• Transmission

– Mycobacterium tuberculosis

– Droplet

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(cont.)

– Early diagnosis, prompt treatment

– Compliance with treatment regimen

• Preventing TB

– Vaccination – BCG (not used in the U.S.)

– Causes false-positive with TB skin test

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• Patient measures

– Covering mouth

– Proper disposal of

tissues– Take medication as

directed– Avoid close contact

with others– Air out their room

techniques

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ANSWER: The spread of many infectious diseases can be

limited or prevented by using commonsense precautions:

Using tissues when coughing or sneezing

Washing hands frequently

Using disposable dishware

can be limited or prevented?

Excellent! 

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• VRE – vancomycin-resistant enterococci

• VISA – vancomycin-intermediate S aureus

• VRSA – vancomycin-resistant S aureus

• ESBLS – extended-spectrum beta-lactamases

• PRSP – penicillin-resistant Streptococcus

pneumoniae

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– Most common in non-hospital health-care facilities

• Community-associated MRSA

– Increasing in incidence

• PRSP

– Common in patients seeking care in physicians’

offices and clinics (pediatrics)

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by drug-resistant organisms

– Advanced age

– Invasive procedures

– Prior use of antibiotics

– Repeated contact with health-care system

– Severity of illness

– Underlying diseases or conditions

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antibiotic-resistant microorganisms

– Prevent infection

– Diagnose and treat infection

appropriately – Use antibiotics carefully

– Prevent transmission of

infections

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resistant microorganisms?

ANSWER: Strategies to reduce the incidence of

antibiotic-resistant microorganisms include:

Prevent infections

Diagnose and treat infections appropriately

Use antibiotics carefully

Prevent transmission

Good Job!

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disease-• Damage is caused by:

– Depleting nutrients – Reproducing themselves – Making body cells the target of body’s own defenses

– Producing toxins

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through 4 stages of illness:

– Incubation – begins at first exposure; ends when first

symptom appears

– Prodromal – begins at first onset of symptoms;

generally short

– Invasion – numbers of organisms are greatest;

symptoms are most pronounced– Convalescent – patient regains normal health status

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ANSWER: The four stages of illness are:

Incubation – begins at first exposure; ends when first

symptom appears

Prodromal – begins at first onset of symptoms; generally

short

Invasion – numbers of organisms are greatest;

symptoms are most pronounced

Convalescent patient regains normal health status

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of being resistant to

pathogens and the

disease they cause

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in the body

– Provide a barrier against pathogens

– Normally live in balance

– Become pathogenic when host’s defenses are

compromised

• Opportunistic infections

– Infections occurring when a host’s resistance is low

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• Begin tissue repair

• Destroy invading microorganisms

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1.Initial constriction, then dilation of blood vessels, causing redness and heat

2.Fluid leakage from local vessels  swelling3.Scar tissue formation

– Chronic inflammation

• Damage to tissues

• Loss of function

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– White blood cells ( phagocytes )

engulf and digest pathogens – Three types

• Neutrophils – found in pus

• Monocytes – formed in bone marrow and become

• Macrophages when they migrate to specific tissues

– Found in lymph nodes, liver, spleen, lungs, bone marrow, and connective tissue

– Deliver antigens (foreign substances) to lymphocytes

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– T cells activate B cells to produce antibodies

to neutralize an antigen – Memory B cells respond quickly to produce

antibodies in later invasions – Specific antibodies are produced in response

to specific antigens – Antibodies attract phagocytes, which destroy

antigens

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– Active – body produces own antibodies

• Natural active

• Artificial active

– Passive – antibodies that are produced

outside body enter the body

• Natural passive

• Artificial passive

• Complement

– Proteins activated by antibodies

– Helps white blood cells destroy pathogens

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immunity?

ANSWER: Active immunity is long-term immunity in which

the body produces its own antibodies.

Passive immunity results when antibodies produced

outside the body enter the body.

Both can be natural or artificial.

Impressive!

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human body capable of sustaining

pathogen growth

– Subclinical case – unnoticeable infection

microorganisms become pathogenic

into the body

Click for Cycle 

of Infection

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leaves the host

–Nose, mouth, eyes, or ears

–Feces or urine

–Semen, vaginal fluid, or other

reproductive discharge –Blood or blood products

Click for Cycle 

of Infection

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Click for Cycle 

of Infection

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– Enter through any

cavity lined with mucous membrane

• Mouth, nose, vagina, rectum

• Ears, eyes, intestinal tract, urinary tract, reproductive tract, breaks in the skin

Click for Cycle 

of Infection

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