(BQ) Part 2 book Textbook of endodontology has contents: Root canal instrumentation, root canal illing materials, root canal illing techniques, the root canal treated tooth in prosthodontic reconstruction, nonsurgical retreatment, nonsurgical retreatment,... and other contents.
Trang 1Part 3
Endodontic Treatment Procedures
169
Trang 3Chapter 10
Endodontic emergencies
Peter Jonasson, Maria Pigg, and Lars Bjørndal
Introduction
The most frequent causes of tooth-related pain are pulpal
and periapical inflammation due to bacterial infection
Notably, the underlying major etiological mechanisms
behind a painful inflamed pulp or apical periodontitis
are not different from “cases” without pain The focus
is still on the bacterial infection, but an acute clinical
expression has appeared and needs immediate attention
Very often, the dentist has to consider management of
emergencies under time pressure, either as unscheduled
consultations squeezed in between regular patients, or
due to a sudden complication This requires clinical skill,
not least regarding proper diagnostics to ascertain that
the source of pain has been correctly identified
Emer-gency treatment is often a compromise and a temporary
solution, and needs to include information to the patient
about the need for later completion of the treatment and
expected outcome
The aim of emergency treatment is to achieve pain
relief and/or infection control while at the same time
maintaining good prognosis for the subsequent
treat-ment, and to prevent adverse sequelae This usually
includes removing the cause of pain but may also
include drainage in case of purulent infection and
swelling (see Core concept 10.1)
After a general introduction on diagnostics and
emer-gency principles, this chapter will cover the
manage-ment of patients with acute pain or complications
origi-nating from the tooth or the surrounding tissues
General diagnostic considerations and
emergency principles
A general medical and local dental anamnesis and a
careful clinical examination is the basis for diagnosis
and treatment (see Chapter 4), and should in the gency situation focus on the chief complaint (Fig 10.1).The pain history often provides important informa-tion regarding endodontic conditions, and the patient’sdescription is a vital part of diagnosis The type andduration of symptoms may give some indication of theconditions (see Core concept 10.2)
emer-Pain varies in intensity and may be mild, ate, or severe Pain quality also varies along a spec-trum from sharp, intermittent attacks over pulsating orthrobbing sensations to a continuous dull ache How-ever, the intensity and quality of pain per se wasreported to have poor correlation to the diagnosis [1],and the evidence that symptoms are useful as markers
moder-of disease severity is insufficient overall [2] Caution isthus needed when the information is used to make adiagnosis
Correct diagnosis is fundamental for adequate ment, but is not always easy to achieve in the acute situ-ation Several teeth may display pathosis, but to achievesymptom relief the offending tooth must be identified(see Core concept 10.3)
treat-The etiology and pathogenesis behind emergency scenarios
A high proportion of patients seeking emergency dentalappointments because of pain have symptoms of pul-pal or periapical disease [3] In one study it was foundthat the most frequent reason for performing root canaltreatment was vital symptomatic carious teeth (Fig 10.2)[4, 5], but fractures or cracks in the tooth substance(Fig 10.3) from traumatic injuries or leakage in gapsalong the margins of restorations or from chemical orthermal insult subsequent to dental treatment are othercauses (see Core concept 10.4)
Textbook of Endodontology, Third Edition Edited by Lars Bjørndal, Lise-Lotte Kirkevang, and John Whitworth.
© 2018 John Wiley & Sons Ltd Published 2018 by John Wiley & Sons Ltd.
Companion Website: www.wiley.com/go/bjorndal/endodontology
171
Trang 4172 Endodontic Treatment Procedures
Core concept 10.1 Overall strategy for emergency
visits
An overall strategy for handling emergency visits includes the
following:
r Identify the pain cause (diagnosis, location)
r Eliminate the pain cause (infection, inflammation)
r Consider need of drainage
r Consider risk of infection spreading (need for antibiotics)
r Consider need for further pain relief (analgesics) and later
treatment
Once the carious bacterial front reaches the innermost
dentin and the pulp [6], the dentin barrier function is
lost and the pulp is no longer able to resist bacterial
invasion Consequently, microorganisms will enter the
pulp space and over time advance in apical direction, as
the pulp tissue gradually breaks down and the necrotic
zone progresses Bacterial products induce an
inflam-matory reaction in the periradicular tissues (apical
peri-odontitis) adjacent to canal orifices, mainly in the apical
part of the tooth The degree of inflammation and the
presence and character of symptoms will depend on the
Core concept 10.2 Taking a pain history
A thorough pain history should include the following information:
r Exacerbating or relieving factors
quantitative (number) and qualitative (virulence) nature
of the microorganisms as well as the host inflammatoryresponse (see Chapter 7)
Most acute endodontic conditions develop neously, but in conjunction with certain treatments theremay be a higher risk of postoperative pain (flare-up) Ifthe host defense is insufficient to contain the infectionwithin the root, bacteria may enter the periapical tis-sues and cause a massive acute inflammatory response,which usually includes pain and swelling The process
sponta-of swelling can be categorized as either an abscess or
Diagnosis and treatment
Vital pulp and apical periodontitisNecrotic, infected pulp
Caries excavation (- pulp exposure)
Systemic infection spread
(- pus - swelling) Access cavity preparation and cleaning of pulp chamber
(+/- pus +/- swelling) Complete chemomechanical cleaning of root canal system +/- incision for drainage
(- moderate swelling,
- signs of spread to deeper structures) Removal of source of infection + systemic antibiotics (+ severe swelling, breathing difficulties, + signs of spread to deeper structures) Removal of source of infection + systemic antibiotics Hospitalization +/- surgical drainage
Paraclinical examination:
Radiology
Fig 10.1 Flowchart of the emergency diagnostic process and endodontic treatment modalities.
Trang 5Endodontic emergencies 173
Core concept 10.3 Clinical assessment of tooth
pain and the potential parameters involved Extraoral
r Pulp vitality (cold, electricity)
r Pain provocation (percussion, apical palpation, temperature)
cellulitis (see later) An abscess is a localized
accumula-tion of pus and swelling within the soft tissue and the
pathway follows the route of least resistance (Fig 10.4)
It is usually very painful If the amount of pus increases,
the pressure in the tissue rises and the abscess may
eventually perforate the bone and periosteum (Fig 10.5)
and burst, or establish a fistula through the oral mucosa
or skin, allowing drainage of the infection and
dimin-ishing the risk of infectious spread A fistula may also
advance along the periodontal space mimicking a deep
periodontal pocket The choice of endodontic emergency
treatment should be based on location of the infection
and consideration of the risk for systemic spreading
Although this is rare, it is important to diagnose and treat
deep infections of dental origin early, since they may
lead to serious conditions and even, in very rare cases,
death The influence of local anatomy is well
under-stood, but there is limited literature describing other
risk factors contributing to the spread of odontogenic
infections
Acute pain from pulpitis
Two types of primary nociceptors are responsible for
pulpal pain: A- and C-fibers The A-fibers have free
nerve endings in the peripheral pulp and inner dentin,
and normally respond to thermal or mechanical
stim-uli (when pulp or dentin is exposed) with sharp and
intense pain The C-fibers are located deeper in the
pulp, and are mainly sensitive to extreme temperaturesand chemical stimulation During pulpal inflammation,particularly in late stages, release of proinflammatorymediators such as bradykinin and histamine activate theC-fibers, resulting in dull, aching pain [7] (see Chap-ter 3) Presence of such pain can thus be considered a sign
of pulpitis During inflammation, the excitation olds of pain neurons drop due to central and periph-eral changes [8–10] and inflamed teeth are overall moresensitive The inflammation is further potentiated by therelease of neuropeptides from the nociceptive neuronsthemselves, often referred to as neurogenic inflamma-tion [11, 12] Since the inflammatory reaction in pulpitislargely occurs inside the tooth, it is mainly factors able
thresh-to directly stimulate the pulpal nerves that will inducepain, such as changes in temperature (cold/hot), osmoticpressure (sweetness), or mechanical manipulation of anopen carious lesion (chewing)
Acute pain from apical periodontitisSensory trigeminal nerves innervate the periradiculartissues Periodontal mechanoreceptors are responsiblefor tactile sensation and the ability to sense tooth load(e.g., objects between the teeth) and do not normally sig-nal pain However, in an inflammatory state, normallypainless activities, such as chewing or tooth brushing,become painful Perception of pain on stimulation that
is normally nonpainful in nature (such as light sion, pressure, or touch) is known as allodynia Hyper-algesia, a decrease in pain threshold to, for example,heat, is another example of how the nociceptive responseincreases in the presence of bacteria-induced inflamma-tion Functional plasticity on peripheral and central lev-els of the nervous system explains these phenomena[13, 14]
percus-Since the sensitized nociceptors in apical periodontitisare located outside the tooth, pain is usually provoked
by activities stimulating the periodontal tissues, such asbiting or chewing Palpation of the alveolar process close
to the tooth apex will also elicit pain, especially whenthere is local spread of infection with extreme inflamma-tory activity (e.g., an abscess)
Symptomatic pulpitis – vital pulp
Anamnesis and pain history
In an emergency situation patients will be in differentdegrees of pain and thus needing different urgency fortreatment A situation suggestive of a progressing pulpinflammation is when the tooth first becomes increas-ingly more sensitive to cold air or cold drinks andfood products, which subsequently turns into shorter
Trang 6174 Endodontic Treatment Procedures
Fig 10.2 Mandibular molar with (a) extremely deep caries, (b) penetrating radiographically into the pulp, and with apical radiolucency, (c) pronounced bleeding
of the pulp and the focal presence of pus is noted (d) prior to preparation of an endodontic working restoration and an aseptic working field Source: Reproduced from [4] with permission from Springer.
or longer periods of lingering pain elicited by the same
stimuli The intermittent character of the pain
experi-ence is a characteristic feature and contributes in the
dif-ferential diagnosis from other painful conditions In the
most severe case, excruciating pain may linger for hours
Pain may occur spontaneously or be provoked by hot or
cold drinks and foods In the end stage, prior to
com-plete breakdown of the pulp, patients may find that cold
water alleviates the symptoms However, symptoms and
circumstances vary widely and careful examination is
necessary to confirm the diagnosis and match findings
to the pain description (Table 10.1)
Relevant examination – clinical and radiographic
If the pain history indicates a vital inflamed pulp,the examination should focus on confirming this andlocalizing the tooth Deep caries, fracture of tooth orrestoration, and cracks may be observations that sup-port the diagnosis, as well as pain provocation of air
Trang 7Core concept 10.4 Reasons for emergency
r Pulp exposure because of caries, iatrogenic injury, or trauma in an
otherwise nonpainful tooth
r Symptomatic apical periodontitis
r Midtreatment or posttreatment pain associated with pulpectomy,
root canal disinfection or retreatment
r Flare-up subsequent to root canal treatment
blasting or careful probing of the defect, to which a vitalpulp should respond Pain can sometimes be provoked
by tooth percussion [1, 15] Prolonged spontaneous painattacks and lingering pain sensations after pulp provo-cation are regarded as signs of severe inflammation,but the scientific support for this is weak [2] If a toothcrack is suspected (sometimes referred to as “crackedtooth syndrome”; see Chapter 3), transillumination andselective loading of cusps by biting on a wooden spat-ula, cotton roll, or instrument specially designed for thepurpose (FracFinderTM, Tooth Slooth®) can be helpful(Fig 10.6) Typically, sharp pain is elicited when the pres-sure is released after biting with moderate force Injury
to the pulp may also be caused by restorative treatment,
Trang 8176 Endodontic Treatment Procedures
Mylohyoid muscle
Sublingual space,
in sublingual tissue above mylohyoid muscle (1)
Palatal abscess (6) Maxillary sinus
Vestibule
Buccal space between buccinator muscle and overlying skin (3)
Submandibular space below mylohyoid muscle (2)
Buccinator muscle
(4) (5)
Fig 10.4 Common pathways of an
api-cal abscess The route depends on the
location of the infection process in
rela-tion to the surrounding anatomical
struc-tures: (1) sublingual space in the
sublin-gual tissue above the mylohyoid muscle; (2)
submandibular space below the mylohyoid
muscle; (3) buccal space between
buccina-tor muscle and overlying skin; (4) vestibule;
(5) maxillary sinus; (6) palatal abscess.
typically carried out within a fairly short period of time
(weeks) prior to the onset of symptoms
Identifying the offending tooth is an important
pri-mary task but may be a demanding diagnostic challenge
(see Chapter 4) The primary reason is that findings other
than the patient’s report of pain are rarely present If
there is no overt deep carious lesion (the most common
cause of painful pulpitis) the clinician may be faced with
the difficult task of assessing which one of several teeth
is affected (see Case study 10.1)
Radiographic examination should focus on
identify-ing possible compromise of the pulp If the pain is in
(c)
Fig 10.5 Acute and chronic apical abscess (a) Drainage through the
alve-olar bone, subperiostal/submucosal abscess (b) Drainage by fistula formation.
(c) Drainage along the periodontal ligament.
the posterior region, the bite-wing projection has severaladvantages It is superior for detection of caries and deeprestorations, and also allows simultaneous assessment ofupper and lower jaw, which is important since referredpain is fairly frequent The inability of the patient tocorrectly locate the painful tooth is explained by thefunctional convergence of the trigeminal sensory path-ways In pulpitis cases, the patient’s report of whichtooth is painful should always be supported by clearfindings of pathology to eliminate the risk of treating thewrong tooth Since the pain originates from intradentaltissue, abnormal periapical radiographic findings are
Table 10.1 Prevalent clinical findings associated with reversible and irreversible pulpitis
Pulp condition
Diagnostic factors
Reversible pulpitis
Irreversible pulpitis
Pulp response to cold and electricity Yes Yes
Tenderness to biting/percussion Possible Yes Radiographic signs of apical bone
destruction
Possible Possible
Trang 9Endodontic emergencies 177
Fig 10.6 Diagnosis of a cracked second molar (a) Pain is elicited on selective loading of cusps by biting on a specially designed instrument (FracFinder ® ) (b)
No radiographic evidence of a crack; but (c) clinically, a crack is clearly identified following removal of a superficial restoration.
not expected and thus a periapical radiograph should
not be the first choice However, it should be noted
that periapical radiolucency can be found in association
with teeth with pulpitis of various degree, and should
not be regarded as a certain sign of pulp necrosis
(see Fig 10.2b) Also, tenderness to percussion of theoffending tooth and even of the neighboring teeth may
or may not be observed in the final stages of pulpalinflammation
Case Study 10.1
Pulpitis may be accompanied by severe painful
symp-toms Although the cause is often a deep carious lesion,
painful pulpitis may also follow pulp capping or
restora-tion close to the pulp Especially in a dentirestora-tion that is
fully and properly restored the offending tooth may be
difficult to identify This case demonstrates the dilemma
the clinician may be faced with in cases like this
The emergency patient, a 55-year-old woman, had
suf-fered from excruciating pain over several days The pain
varied from none to intense, which is typical in cases of
painful pulpitis Also suggestive of pulpitis was that the
pain was poorly localized and was felt to variably
origi-nate from the lower as well as from the upper jaw on the
right hand side Occasionally, pain radiated peripherally
to involve the temporal region There was no clear
asso-ciation with intake of hot and cold drinks or food, and
pain was not aggravated by biting or chewing
Paraceta-mol gave pain relief, albeit only for a few hours
The patient, who was a regular attendant to the clinic,
was well restored and had no obvious carious lesion
(a,b,c) Tooth 47 had received a ceramic restoration
about 2 years previously Teeth 45, 47, and 48 could
be tested for pulp sensitivity but the other teeth wererestored with full cover crowns, and tests were incon-clusive The three teeth showed no response to electricityand unclear response to cold testing Periodontal condi-tions were fair with no periodontal pockets There was
no pain on percussion or apical palpation According tothe radiographs there were no signs of caries
Because of the inconclusive anamnestic, clinical, andradiographic findings, emergency treatment was post-poned because of the risk of entering the wrong tooth.Three days later the patient was seen again afterhaving been on analgesics She reported spontaneous,intermittent, and pulsating pain localized to the lowerjaw Drinking coffee induced sustained pain Tooth
47 showed some percussion sensitivity After isolatingtooth 47 with rubber dam, provocation with warm waterinduced severe persistent pain of high intensity A sub-sequent nerve block in the lower jaw gave pain relief.Upon accessing the pulp of tooth 47, the causative toothwas confirmed by the finding of abundant bleeding (d)
Trang 10178 Endodontic Treatment Procedures
Emergency management of reversible
(saveable pulp)/irreversible pulpitis
(nonsaveable pulp)
Patients with pulpal pain may require a pulpectomy
pro-cedure whereby the entire pulp tissue is removed (see
Chapter 6) and replaced with a root filling, but this
deci-sion should be taken only after careful consideration of
the causes and the extent to which the pain condition
can be alleviated by a more conservative approach
aim-ing to preserve the pulp and re-establish nonpainful and
healthy conditions in the long term
Reversible pulpitis – saveable pulp
Cases where the pulp is not exposed and the pain
presents as hypersensitivity or only short-lasting pain
to external stimuli are especially amenable to a
con-servative or “wait and see” approach One example is
postoperative hypersensitivity after a restorative
proce-dure; the symptoms to temperature changes and
chew-ing are often of a temporary nature and will disappear
without active treatment, or following adjustment of
hyperocclusion If symptoms are pronounced or have
persisted for some time, removal of the composite
restoration and replacement with a temporary
restora-tion may solve the problem, by blocking exposed
denti-nal tubules and removing residual stress from composite
polymerization shrinkage
Vital teeth with exposed dentin may become
hyper-sensitive to external stimuli, most frequently
temper-ature changes and tooth brushing The patient may
experience considerable discomfort and request an
emergency appointment Exposed dentinal tubules
are susceptible to thermally induced volume changes
of fluid in the dentinal tubules, leading to nociceptor
activation Discomfort often hinders proper cleaning,
and retention of biofilm and bacterial products may
together with neurogenic inflammation induce chronic
inflammatory changes in the pulp
The treatment strategy should initially be
conserva-tive Professional cleaning and re-establishment of good
hygiene routines may in itself lead to significant and
per-manent pain relief [16] On failure to achieve pain relief,
toothpastes with strontium or potassium salts, which
block fluid movement by precipitation of salt crystals,
may be tried [1, 17] If ineffective, the next step is to seal
the dentinal tubules with fluoride varnish, dentin primer
and resin, or a composite restoration Dentin
hypersensi-tivity can be very difficult to control, indicating that the
materials either have no permanent effect and/or that
the inflammatory changes are so profound that a
natu-ral healing process is prevented Endodontic treatment
should be a last resort
Clinical procedure 10.1 Emergency pulpotomy
1 Ensure appropriate local anesthesia.
2 Prepare access cavity to the pulp and remove the coronal pulp with
a bur.
3 Irrigate with copious amounts of water or NaOCl (0.5–2.5%).
4 Control hemorrhage by pressure with cotton pellets In case of fuse bleeding, soak pellets in 3% hydrogen peroxide or an aqueous mixture of Ca(OH)2.
pro-5 Restore access cavity with a temporary filling.
6 Perform pulpectomy as soon as possible.
Irreversible pulpitis – nonsaveable pulp
If the pulp condition is deemed to be of an irreversiblenature, the first step in the emergency treatment is toexpose the pulp If there is a carious lesion, all cari-ous dentin should be excavated first From then on sev-eral options are available, although time pressure oftendecides the choice of treatment Pulpectomy with com-plete debridement of the root canals will offer a highprobability of pain relief [18–20] However, with timeconstraints pulpotomy and removal of the coronal pulptissue without penetrating into the radicular pulp tissue
is an efficient treatment with a comparable probability ofpain relief [18–22] (see Clinical procedure 10.1) Pulpo-tomy is reported to give total or partial pain relief inmore than 90% of cases [18] This is a temporary measureuntil time is available for pulpectomy; if pain from thetooth was previously not relieved by pulpotomy, pulpec-tomy should be performed
In general, a cotton pellet should not be placed inthe access cavity during temporization, as it reduces thethickness of the temporary seal, which increases the risk
of bacterial leakage and reinfection of the pulp chamber[23, 24] The patient should be made aware that postop-erative tenderness or a slight dull pain in the affectedregion can be expected for a couple of days after theemergency procedure Analgesics are usually effective,but if severe pain continues, the patient is advised to seek
a new appointment For a pharmacological approach(see Advanced concept 10.1) [25]
Advanced concept 10.1
Recently, a randomized clinical trial found that a pharmacological approach using intraosseous methylprednisolone injection relieved pain caused by acute pulpitis more effectively than did pulpotomy during a 7-day period before proper endodontic treatment was per- formed [25] The concept is interesting and could have relevance espe- cially in regions where access to dental resources is low However, adequate diagnosis is important This requires good knowledge about etiology and training in differential diagnosis of dental pain.
Trang 11Endodontic emergencies 179
Symptomatic apical periodontitis with or
without acute abscess
Anamnesis and pain history
A chief complaint is localized pain, at first only present
on chewing, but eventually developing into persistent,
severe pain disturbing night sleep The patient has
usually no doubt which tooth is affected The tooth
may feel elevated or even loose, and there may be
soft-tissue swelling However, considerable variation in
symptoms should be expected, since the transition from
a necrotic pulp to acute periapical disease occurs along a
continuum
Relevant examination – clinical
and radiographical
The tooth and periradicular tissue should be tested
with palpation, percussion, periodontal probing,
mobil-ity, analysis of occlusion and articulation, and
transillu-mination It can be difficult clinically to estimate the
tran-sition between symptomatic apical periodontitis and the
initial stages of an abscess formation The tooth may be
tender to percussion and the mucosa and bone
overly-ing the apical region may be sensitive to palpation
Peri-odontal probing will reveal drainage of an abscess or
fis-tula formation through the periodontal ligament space
(Fig 10.5) Differential diagnosis in relation to localized
marginal periodontitis is important and is done by pulp
vitality assessment (see Chapter 4) In addition, a narrow
isolated periodontal pocket may be caused by a vertical
root fracture
A periapical radiographic examination of the tooth is
mandatory The apical radiolucency may be restricted
to a minor widening of the periodontal space Notably,
while inflammatory changes in soft tissue develop fast,resorption of periapical bone is a slower process, whichmay not yet be detectable in radiographs at the acutestage Radiographs in two horizontal angulations may
be helpful if one image does not show clear pathology
In case of a fistula along the periodontal pocket, graphic changes can be completely absent For morecomplicated cases when the intraoral examination isinconclusive, cone beam computed tomography may beconsidered [26], but should not be the first choice inemergency cases
radio-Emergency management of symptomatic apical periodontitis
The strategy for the biomechanical instrumentation ried out in pain cases is the same as in symptom-free cases In case of extrusion of the tooth from thesocket due to inflammatory exudate in the periodontalligament, the tooth may be very tender and even loose.Therefore, in addition to local anesthesia, it may be nec-essary to stabilize the tooth during access Pus may ormay not appear spontaneously (Fig 10.7) After accesspreparation, an aseptic working field is prepared withrubber dam and disinfection, and the standard root canaldisinfection is carried out (see Chapters 13–15) Calciumhydroxide is applied in the cleaned canal, and propertemporary sealing is mandatory A moist cotton pelletcan be used to wipe the walls of the access cavity cleanfrom calcium hydroxide, allowing a proper tight seal
car-If time is insufficient for a full root canal disinfection,
an access cavity should be performed, removing theinfected crown pulp and exposing the canal orificesfollowed by irrigation of the pulp chamber with water
or sodium hypochlorite and placement of a temporary
Fig 10.7 Symptomatic apical periodontitis in a mandibular molar (a) Temporary restoration of questionable quality is seen radiographically (b) Pus emerges spontaneously from the cavity (c) Due to a cotton pellet placed in the cavity, the temporary restoration (arrow) was insufficient and leakage of bacteria from the oral environment occurred.
Trang 12180 Endodontic Treatment Procedures
filling [20, 22] Cleaning of the pulp chamber has
shown pain relief for a large proportion, but not to the
same magnitude as complete root canal disinfection
[20, 22, 27–29] The patient should be scheduled for
completion of root canal treatment as soon as possible
to preclude recurrent pain This concept is not
recom-mended in patients with infection associated with either
pus or swelling
Drainage of pus through the root canal
If the periapical process is purulent, drainage through
the root canal (Fig 10.7) is often obtained within
min-utes, but it can be difficult to predict If pus does not
emerge from the root canal during instrumentation as
expected, once the canal has been properly cleaned it
is advisable to penetrate the apical constriction with a
handfile (size 10–15) in order to optimize drainage Care
should be taken not to overinstrument the apical
fora-men region
The tooth should never be left open between
appoint-ments [30]; it should be sealed with a tight temporary
restoration, after placement of calcium hydroxide In
very rare cases the drainage may continue, but even in
such cases the tooth can be sealed By the time of the next
appointment the initial removal of infection has usually
arrested pus formation
Acute apical abscess – incision and drainage
The classical approach to achieving drainage is by
inci-sion (Fig 10.8) The right timing of abscess inciinci-sion is
often described as “when it appears soft and fluctuant,”
that is, when the pus is located in the submucosa, as
opposed to a more diffuse increase of volume or even a
hard swelling, indicating that the periosteum has started
to elevate The decision whether to incise or not is based
on clinical assessment and experience It is advisable to
Fig 10.8 Drainage of a fluctuant submucosal abscess is provided by the
use of a scalpel Pus is released on careful incision (approx 1 cm cut).
monitor symptom development carefully, incise whenpossible, and refrain from antibiotics unless systemicspread is identified
Cellulitis
Cellulitis is an infection spreading in the connective sue with diffuse erythematous swelling Cellulitis is usu-ally painful and may be accompanied by malaise andfever Pathogens and related inflammatory exudate mayspread along the spaces of the head and neck within thefascial planes (see Fig 10.4)
tis-Such advanced systemic spread of endodontic tions is rare, but if not properly assessed and adequatelyhandled it may lead to serious complications and be life-threatening (see also Chapter 25) [31, 32]
infec-Posttreatment emergency
A painful condition may remain after emergency ment or arise postoperatively by an initially nonpainfultooth The latter condition is termed endodontic flare-up.Teeth with preoperative pain and teeth with apical peri-odontitis are more prone to developing postoperativepain and discomfort [33–35] The causes include micro-bial, mechanical, and/or chemical injury to the pulp orperiradicular tissues Contamination due to not apply-ing a rubber dam, an unsatisfactory temporary restora-tion (Fig 10.7a), or displacement of carious dentin andbacterial plaque into the pulpal or periapical tissue arekey factors [35–37] In combination with inappropriateintracanal medication, incomplete instrumentation, non-instrumented canals, and apical overinstrumentation, it
treat-is easy to comprehend that conditions for bacterial tiplication in the root canal system are created It should
mul-be emphasized that complications of this nature should
be rare in properly managed clinical practice [36, 38](see Core concept 10.5) Cracked tooth (see Fig 10.3c–e)substance and hyperocclusion are other factors to con-sider when examining a patient for causes of a post-endodontic emergency
After assessment of potential causes, the first step is toassess the need to carry out a re-entry procedure Post-operative pain often resolves spontaneously and may
be controlled simply by pain medication and a tion of the occlusal forces Re-entry is only relevant iffurther removal of inflamed or infected pulp tissue can
reduc-Core concept 10.5
Adherence to basic endodontic principles – including aseptic ment, complete removal of accessible pulpal tissue, and filling of canal to proper length – favors pain relief and precludes endodontic flare-ups.
Trang 13be performed, and when it is deemed necessary, the
endodontic procedure should follow the same strictly
aseptic routine as described above, which includes
rub-ber dam application and disinfection Moreover, in cases
with abscess formation a re-entry will allow for drainage
of pus
An endodontic flare-up may also be associated with an
overfilled root canal Normally, a small extrusion of root
filling material does not cause more than slight
tender-ness, which usually subsides over a couple of days If a
severe pain condition has developed, with apical
tender-ness and some swelling, there is often a bacterial cause
where microorganisms have been pushed into the
peri-odontal tissues along with the root filling material
How-ever, a flare-up may also occur when the root filling is
short of apex (Fig 10.9)
Non-endodontic tooth pain – conditions
of differential diagnostic interest
Non-odontogenic pain presenting as toothache
chal-lenges the diagnostic ability of the clinician Although
most toothache is odontogenic and endodontic, the
den-tist needs to be aware of and able to identify other
con-ditions with similar symptoms, in order to avoid
mis-diagnosis (see Core concept 10.6) A brief description of
the most relevant differential diagnoses is given below,
but several other conditions are possible (see Chapters 4
and 22)
Marginal (periodontal) abscessMarginal periodontitis is generally not painful, butsometimes a marginal abscess occurs Symptoms andclinical findings of marginal abscess are throbbing pain,marginal swelling, tenderness to percussion and palpa-tion, and a local deep pocket The pulp is normally vital(responds to pulp testing) and bone destruction associ-ated with the tooth apex is absent (atypical, lateral bonedestruction may be seen) A history of periodontal dis-ease should be a warning sign Emergency treatmentincludes careful scaling and irrigation of the affectedpocket [39]
Referred pain from other teeth ornondental structures
Referred pain originates in a different body structurethan where it is perceived by the patient The phe-nomenon is explained by the complex innervation pat-terns of sensory nerves In the central nervous system(CNS), the afferents converge and signals from some-times quite remote tissues use the same pathways; thisleads to an inability to discriminate between pains of dif-ferent origin, such as another ipsilateral tooth, mastica-tory or neck muscles and jaw joints [40, 41], sinus dis-ease [42], neurovascular headache [43], or even cardiacmuscle ischemia [44, 45] Failure to find clinical or radio-graphic evidence of pathology in the pain region shouldresult in inquiry about muscle tenderness, ear, nose, and
Trang 14182 Endodontic Treatment Procedures
Core concept 10.6 Overview of findings suggestive
of odontogenic pain (of dental origin) and non-odontogenic pain
Odontogenic pain Non-odontogenic pain
Evidence of dental or
periodontal pathosis is
present (e.g., caries, leakage,
fracture, crack, pocket,
lingering to dull, constantly
aching and throbbing
Poorly localized pain, appears at different locations at different times
Unilateral and localized pain Bilateral pain or multiple pain sites
Pain level affected by
temperature change
Pain co-occurring with headache
Pain evoked on tooth
percussion, pressure and/or
Pain level affected by changes in posture
throat infection, headaches, and heart disease, and
clin-ical examination should include assessment of
oppos-ing teeth and provocation of muscles If muscle
palpa-tion increases the tooth pain, this may indicate that the
tooth pain has a muscular origin Tender or painful teeth
are frequently reported in association with bruxism and
with sinusitis (upper premolars and first molar)
Idiopathic or neuropathic tooth pain
Patients with long-lasting pain complaints sometimes
schedule an emergency appointment, and such chronic
problems should be distinguished from acute tooth pain
Symptoms and clinical signs overlap with inflammatory
pain, and in the emergency situation it is wise to be
vig-ilant Pain that does not resolve as expected, for
exam-ple lingering pain after seemingly adequate endodontic
treatment, should signal caution The pain may be
neu-ropathic, caused by trauma or an earlier dental
proce-dure, or idiopathic (of unknown origin) In the absence
of explanatory clinical or radiographic findings, the
den-tist should avoid invasive treatment A later
appoint-ment for more comprehensive assessappoint-ment should be
scheduled, or the patient referred to an endodontist or
specialist in orofacial pain Differential diagnostic effortsmay include diagnostic anesthesia [46], a somatosensoryscreening examination [47], and extended radiographicexamination or imaging [48, 49]
Management of patients with acute dental pain
From a psychological perspectiveAcute pain is often accompanied by anxiety and emo-tional distress The experience of pain is complex innature, and the sensory input from tissue injury orinflammation is only one dimension Affective factors(such as stress, anxiety, fear, and mood) and cogni-tive factors (such as attention, control, pain beliefs, andexpectations) are other dimensions that play an impor-tant role for the total impact of pain and the patient’sability to cope Stress and fear of treatment amplify pain,and in the emergency situation the dentist should there-fore strive to inspire confidence and make the patient feelsafe If the patient is stressed and anxious, the dentistmay overestimate the severity of the condition (in terms
of tissue damage), and thus prompt a more invasive cedure than is needed A very important aspect of theemergency management is to listen carefully to the his-tory reported by the patient before any intraoral exam-ination and treatment By spending sufficient time onlistening and asking relevant questions many mistreat-ments may be avoided
pro-AntibioticsIntake of antibiotics does not provide pain relief inirreversible pulpitis [50] Use of antibiotics should berestricted to situations when the bacterial infection hasspread outside the tooth Fever, malaise, and tendernessand swelling of regional lymph nodes are signs of sys-temic spread, and thus indications for antibiotic treat-ment Abscess formation adjacent to the offending tooth
is a sign of local spread, and removal of the infectionand drainage is the recommended primary treatmentsince evidence is scarce that antibiotics have an addi-tional effect [51] Antibiotics are generally not required,but careful assessment of surrounding tissues should bedone to exclude deeper spread (see Chapter 25).Prescription of antibiotics as a solitary emergencytreatment may sometimes be necessary if it is not pos-sible to remove the infection (anesthetic failure, psycho-logical concerns, or physical obstacles) but should beavoided as far as possible to minimize the risk of devel-opment of resistant bacterial strains (both within theindividual and in the community) and must in all cases
be followed by causal treatment soon after
Trang 15Endodontic emergencies 183
Analgesics
Pain relief is usually the patient’s major concern
Appro-priate management of the infection is often enough but
in some cases pharmacological supplements are needed
Nonsteroidal anti-inflammatory drugs (NSAIDs) are the
medications of choice, since they are considered effective
and safe, and are available without prescription
Ibupro-fen in combination with acetaminophen (paracetamol)
is reported to be more effective than ibuprofen alone
[52] Patients should be encouraged to seek pain relief
whenever needed Longer duration of preoperative pain
predicts persisting pain after root canal treatment [53]
Preoperative analgesia may potentiate the effect of local
anesthesia, decrease postoperative pain, and lower the
risk of sensitization of the pain system and chronic pain
development [54–56]
References
1 Seltzer S, Bender IB, Ziontz M The dynamics of pulp
inflammation: Correlation between diagnostic data and
actual histologic findings in the pulp Oral Surg Oral Med.
Oral Pathol 1963; 16: 969–77.
2 Mej`are IA, Axelsson S, Davidson T, Frisk F, Hakeberg
M, Kvist T, et al Diagnosis of the condition of the
den-tal pulp: a systematic review Int Endod J 2012; 45: 597–
613.
3 Hasselgren G, Calev D Endodontics emergency treatment
sound and simplified NY State Dent J 1994; 60: 31–3.
4 Bjørndal L Vital Pulp Therapy for Permanent Molars Berlin:
Springer, 2017.
5 Bjørndal L, Laustsen MH, Reit C Root canal treatment in
Denmark is most often carried out in carious vital molar
teeth and retreatments are rare Int Endod J 2006; 39:
785–90.
6 Reeves R, Stanley HR The relationship of bacterial
pene-tration and pulpal pathosis in carious teeth Oral Surg Oral
Med Oral Pathol 1966; 22: 59–65.
7 N¨arhi MV The characteristics of intradental sensory units
and their responses to stimulation J Dent Res 1985; 64
(Spec No): 564–71.
8 Byers MR, Taylor PE, Khayat BG, Kimberly CL Effects of
injury and inflammation on pulpal and periapical nerves.
J Endod 1990; 16: 78–84.
9 Khayat BG, Byers MR, Taylor PE, Mecifi K, Kimberly CL.
Responses of nerve fibers to pulpal inflammation and
peri-apical lesions in rat molars demonstrated by calcitonin
gene-related peptide immunocytochemistry J Endod 1988;
14: 577–87.
10 N¨arhi M, Yamamoto H, Ngassapa D Function of
intraden-tal nociceptors in normal and inflamed teeth In:
Den-tine/Pulp Complex (Shimono M, Maeda T, Suda H,
Taka-hashi K, eds.) Tokyo: Quintessence, 1996: 136–40.
11 Brain SD Sensory neuropeptides: their role in
inflamma-tion and wound healing Immunopharmacology 1997; 37:
133–52.
12 Chiu IM, von Hehn CA, Woolf CJ Neurogenic tion and the peripheral nervous system in host defense and
inflamma-immunopathology Nat Neurosci 2012; 15: 1063–7.
13 Hu JW Tooth pulp In: Clinical Oral Physiology (Miles
TS, Nauntofte B, Svensson P, eds.) Copenhagen: Quintessence, 2004: 141–64.
14 Trulsson M, Essick G Mechanosensation In: Clinical Oral
Physiology (Miles TS, Nauntofte B, Svensson P, eds.).
Copenhagen: Quintessence, 2004: 165–98.
15 Dummer PM, Hicks R, Huws D Clinical signs and
symp-toms in pulp disease Int Endod J 1980; 13: 27–35.
16 Hovgaard O Dentin hypersensibilitet: fysiologi og behandling.
PhD thesis, Institut for Oral Anatomi og slære, ˚ Arhus Tandlægehøjskole, 1988.
Tandsygdom-17 Poulsen S, Errboe M, Lescay Mevil Y, Glenny AM sium containing toothpastes for dentine hypersensitivity.
Potas-Cochrane Database Syst Rev 2006; 3: CD001476.
18 Asgary S, Eghbal MJ A clinical trial of pulpotomy vs.
root canal therapy of mature molars J Dent Res 2010; 89:
1080–5.
19 Oguntebi BR, DeSchepper EJ, Taylor TS, White CL, Pink FE Postoperative pain incidence related to the type of emer-
gency treatment of symptomatic pulpitis Oral Surg Oral
Med Oral Pathol 1992; 73: 479–83.
20 Bjerk´en E, Wennberg A, Tronstad L Endodontisk
akutbe-handling Tandl¨akartidningen 1980; 72: 314–19.
21 Hasselgren G, Reit C Emergency pulpotomy: pain
reliev-ing effect with and without the use of sedative dressreliev-ings J.
Endod 1989; 15: 254–6.
22 Molander A, Nilsson A, Reit C Effekter av endodontisk
akutbehandling Tandl¨akartidningen 2004; 96: 48–54.
23 Beach CW, Calhoun JC, Bramwell JD, Hutter JW, Miller
GA Clinical evaluation of bacterial leakage of endodontic
temporary filling materials J Endod 1996; 22: 459–62.
24 Laustsen MH, Laren T, Reit C, Bjørndal L den af temporære endodontiske fyldningsmaterialer En
Bakterietæthe-klinisk og mikrobiologisk undersøgelse Tandlægebladet
2004; 108: 888–94.
25 Bane K, Charpentier E, Bronnec F, Descroix V,
Gaye-N’diaye F, Kane AW, et al Randomized clinical trial of
intraosseous methylprednisolone injection for acute
pulpi-tis pain J Endod 2016; 42: 2–7.
26 Patel S, Durack C, Abella F, Roig M, Shemesh H,
Lam-brechts P, et al European Society of Endodontology tion statement: the use of CBCT in endodontics Int Endod.
posi-J 2014; 47: 502–4.
27 Fouad AF, Rivera EM, Walton RE Penicillin as a
supple-ment in resolving the localized acute apical abscess Oral
Surg Oral Med Oral Pathol Oral Radiol Endod 1996; 81:
590–5.
28 Henry M, Reader A, Beck M Effect of penicillin on operative endodontic pain and swelling in symptomatic
post-necrotic teeth J Endod 2001; 27: 117–23.
29 Nusstein JM, Reader A, Beck M Effect of drainage upon access on postoperative endodontic pain and swelling in
symptomatic necrotic teeth J Endod 2002; 28: 584–8.
30 Tj¨aderhane LS, Pajari UH, Ahola RH, B¨ackman TK, Hietala
EL, Larmas MA Leaving the pulp chamber open for drainage has no effect on the complications of root canal
therapy Int Endod J 1995; 28: 82–5.
Trang 16184 Endodontic Treatment Procedures
31 Carter L, Starr D Alarming increase in dental sepsis Br.
Dent J 2006; 200: 243.
32 Sepp¨anen L, Rautemaa R, Lindqvist C, Lauhio A Changing
clinical features of odontogenic maxillofacial infections.
Clin Oral Invest 2010; 14: 459–65.
33 Ali A, Olivieri JG, Duran-Sindreu F, Abella F, Roig M,
Garcia-Font M Influence of preoperative pain intensity on
postoperative pain after root canal treatment: a prospective
clinical study J Dent 2015: 8.
34 Sadaf D, Ahmad MZ Factors associated with
postopera-tive pain in endodontic therapy Int J Biomed Sci: IJBS.
2014; 10: 243–7.
35 Walton R, Fouad A Endodontic interappointment
flare-ups: a prospective study of incidence and related factors.
J Endod 1992; 18: 172–7.
36 Imura N, Zuolo ML Factors associated with endodontic
flare-ups: a prospective study Int Endod J 1995; 28: 261–5.
37 Rosenberg PA, Babick PJ, Schertzer L, Leung A The effect
of occlusal reduction on pain after endodontic
instrumen-tation J Endod 1998; 24: 492–6.
38 Trope M Flare-up rate of single-visit endodontics Int.
Endod J 1991; 24: 24–6.
39 Herrera D, Alonso B, de Arriba L, Santa Cruz I, Serrano C,
Sanz M Acute periodontal lesions Periodontol 2000 2014;
65: 149–77.
40 Simons DG, Travell JG, Simons LS Travell & Simons’
Myofas-cial Pain and Dysfunction: The Trigger Point Manual, vol 1,
2nd edn Philadelphia, PA: Lippincott Williams & Wilkins,
1999.
41 Wright EF Referred craniofacial pain patterns in patients
with temporomandibular disorder J Am Dent Assoc 2000;
131: 1307–15.
42 Hansen JG, Højbjerg T, Rosborg J Symptoms and signs
in culture-proven acute maxillary sinusitis in a general
practice population Acta Pathol Microbiol Immunol Scand.
2009; 117: 724–9.
43 Alonso AA, Nixdorf DR Case series of four different
headache types presenting as tooth pain J Endod 2006; 32:
1110–13.
44 Kreiner M, Alvarez R, Waldenstr ¨om A, Michelis V, Mu ˜niz
R, Isberg A Craniofacial pain of cardiac origin is associated
with inferior wall ischemia J Oral Fac Pain Headache 2014;
46 List T, Leijon G, Helkimo M, ¨ Oster A, Svensson P Effect
of local anesthesia on atypical odontalgia – a randomized
controlled trial Pain 2006; 122: 306–14.
47 Baad-Hansen L, Pigg M, Ivanovic SE, Faris H, List
T, Drangsholt M, et al Chairside intraoral qualitative
somatosensory testing: reliability and comparison between
patients with atypical odontalgia and healthy controls J.
Orofac Pain 2013; 27: 165–70.
48 Pigg M, List T, Abul-Kasim K, Maly P, Petersson A A comparative analysis of magnetic resonance imaging and radiographic examinations of patients with atypical odon-
talgia J Oral Fac Pain Headache 2014; 28: 233–42.
49 Pigg M, List T, Petersson K, Lindh C, Petersson A tic yield of conventional radiographic and cone-beam com- puted tomographic images in patients with atypical odon-
Diagnos-talgia Int Endod J 2011; 44: 1092–101.
50 Nagle D, Reader A, Beck M, Weaver J Effect of systemic
penicillin on pain in untreated irreversible pulpitis Oral
Surg Oral Med Oral Pathol Oral Radiol Endod 2000; 90:
pain Cochrane Database Syst Rev 2013; 6: CD010210.
53 Nixdorf DR, Law AS, Lindquist K, Reams GJ, Cole E,
Kan-ter K, et al Frequency, impact, and predictors of
persis-tent pain after root canal treatment: a national dental PBRN
study Pain 2016; 157: 159–65.
54 Campiglia L, Consales G, De Gaudio AR Pre-emptive
anal-gesia for postoperative pain control: a review Clin Drug
Invest 2010; 30(Suppl 2): 15–26.
55 Dahl JB, Kehlet H Preventive analgesia Curr Opin
Anaes-thesiol 2011; 24: 331–8.
56 Reader A, Nusstei J, Drum M Successful Local
Anesthe-sia for Restorative Dentistry and Endodontics Chicago, IL:
Quintessence, 2011.
Trang 17Chapter 11
Controlling the environment – the
aseptic working field
Merete Markvart and Pia Titterud Sunde
Background
The microbial etiology of pulp and periapical disease is
well established, and it is no surprise that infection
con-trol is central to effective clinical management Studies
have shown convincingly that the presence of
microor-ganisms in the root canal at the time of filling is
associ-ated with impaired periapical healing [1] and it is
cru-cial that the root canal system is protected from the oral
microflora during treatment Probably the single most
effective way of preventing contamination is to isolate
all teeth that are undergoing endodontic treatment with
a well-sealing rubber dam
History and evidence
The benefits of tooth isolation with a rubber sheet were
first described by Sanford Christie Barnum in 1864 [2],
with the rubber dam punch introduced in 1882 and
con-tinuous technological development since that time
The routine isolation of teeth with rubber dam is
recommended in professional treatment quality
guide-lines for both adults and children [3–5] and should be
considered the standard of care for all endodontic
proce-dures In common with many elements of contemporary
endodontic practice, including the choice of irrigant
solution or root canal filling material, there is little hard
evidence to prove a causal link between the use or
nonuse of rubber dam and the outcome of endodontic
treatment The established professional support for this
simple and inexpensive measure to promote asepsis and
protect patients from endodontic materials and
instru-ments, however, makes such studies both unnecessary
and unethical to perform [6] Although the data are not
strong, inferences on treatment outcome may be drawn
from a large population-based study which revealed a
small but statistically significant improvement in tooth
survival when rubber dam was employed for primary
endodontic treatment compared with non-isolated trols [7] Similar inferences have also been made for theuse of rubber dam isolation during nonsurgical retreat-ment [8] In patient-centered terms, the nonuse of rubberdam was identified as one of the risk factors for contin-ued symptoms in a small cohort of patients referred forspecialist care after commencing endodontic treatment[9], and a small retrospective review found a signifi-cant, negative impact on periapical health when teethreceiving posts were not isolated with rubber dam [10].The benefits of rubber dam isolation go beyond thecreation of an aseptic working environment Unpro-tected patients do, unfortunately, swallow and aspirateendodontic instruments from time to time [11–13] andisolation of the working environment with rubber dam
con-is the only sure protection (see Chapter 25 for more mation on the management of swallowed or inhaledendodontic instruments) The rubber dam also functions
infor-to promote the use of antimicrobial irrigants such assodium hypochlorite [14], which is foul-tasting and maydamage unprotected soft tissues [15]
The microbial content of air turbine aerosols producedduring operative procedures, including access cavitypreparation, is significantly reduced by rubber dam iso-lation, thereby reducing the risk of cross-infection in thedental practice [16–19]
Despite all of these advantages, and the routine cation of rubber dam in dental school, dentists in manycountries abandon the use of rubber dam when theyenter general practice [6, 14, 20, 21] (see Chapter 26 formore information on transition shock and the pressuresthat come to bear on professionals as they enter clini-cal practice) In a recent study with 10-year follow-upamong Danish general dental practitioners, there was
appli-an encouraging increase in the application of rubberdam for endodontic procedures from 4% to 29%, but theproportion of general dental practitioners preparing anaseptic working field was still low [22]
Textbook of Endodontology, Third Edition Edited by Lars Bjørndal, Lise-Lotte Kirkevang, and John Whitworth.
© 2018 John Wiley & Sons Ltd Published 2018 by John Wiley & Sons Ltd.
Companion Website: www.wiley.com/go/bjorndal/endodontology
185
Trang 18186 Endodontic Treatment Procedures
Core concept 11.1 Advantages of establishing the
aseptic working field
r The application of rubber dam facilitates disinfection of the
work-ing field.
r The aseptic working field lowers the risk of contamination of the
root canal system.
r Contrast and vision in the working field are enhanced.
r The risk of the patient swallowing instruments is reduced.
r The patient is protected against caustic and foul-tasting irrigants
such as sodium hypochlorite.
Preparing teeth for rubber dam isolation
and the development of an aseptic
working field
The benefits of developing an aseptic working field are
summarized in Core concept 11.1
All plaque and calculus should be removed before
establishing the aseptic working field Removal of
plaque and calculus can be done with periodontal
curettes and/or ultrasound, followed by a rubber cup
with pumice All carious dentine and defective/leaking
restorations should be removed before accessing the
pulp chamber, and a new, clean bur should be used to
enter the pulp space in an effort to minimize
contamina-tion with infected material
Teeth scheduled for endodontic treatment are often
compromised by caries, large restorations, or cracks and
will require occlusal reduction and cuspal coverage aspart of their prosthodontic reconstruction (see Chapter16) Fragile, undermined cusps may be at risk of frac-ture while treatment proceeds and should be reducedfrom the outset Teeth with extensive substance loss, sub-gingival cavity margins or ingrowth of gingival tissuescan be difficult to isolate, and in such cases an endodon-tic “working restoration” is placed This may involveperforming a local gingivectomy, before rebuilding withcomposite resin or glass ionomer cement (Fig 11.1a,b) Ifthe pulp chamber has been opened during initial excava-tions, it is wise to place sterile cotton wool, foam sponge,
or a soft cement such as Cavit (3M) in the chamber beforebuilding the working restoration On occasions, it may
be difficult to place a conventional matrix and a per ring or orthodontic band may need to be cementedand filled with a hard setting material such as IRM®
cop-(Dentsply) or zinc phosphate cement, before adjustingthe occlusion On occasions, teeth with little undercutcan be modified by the addition of composite resin totheir buccal and lingual surfaces All approaches shouldallow the stable application of a rubber dam clamp andprovide the tooth with walls that will allow the accesscavity to be flooded with an appropriate volume of irri-gant during treatment
Access preparationThe timing of rubber dam isolation is contentious Manyadvocate opening the access cavity without rubber damisolation in an effort to avoid any loss of orientationand the risk of overcutting or perforation (Fig 11.2)
Fig 11.1 (a) A maxillary first molar that requires a working restoration before isolation with rubber dam and endodontic treatment (b) The same tooth after rebuilding with Ketac Molar ® and isolation with rubber dam.
Trang 19Controlling the environment 187
Fig 11.2 Perforation of a maxillary molar during access cavity preparation
with the rubber dam in situ The mesiobuccal canals were also not found due
to disorientation.
Others advocate access cavity preparation to the brink
of pulp exposure before applying the dam, yet others
routinely place the rubber dam from the outset unless
the anatomy seems particularly challenging (see
Chap-ter 12) Decision-making is based on the balance of risks,
with iatrogenic damage and microbial contamination
being prominent considerations From a purely aseptic
point of view, having a disinfected rubber dam in place
before entry to the pulp chamber is probably wise
Prerequisites before rubber dam isolation are
summa-rized in Clinical procedure 11.1
Rubber dam isolation
Rubber dam isolation represents the first step in
estab-lishing an aseptic working field Rubber dam sheets
are available in thicknesses from 0.15 to 0.35 mm (often
referred to as light, medium, heavy, extra heavy), and
in individual sheets or rolls For the endodontic
treat-ment of a single tooth, sheets of 5 in × 5 in in a medium
Clinical procedure 11.1 Prerequisites before
application of rubber dam
r Local anesthesia.
r Plaque, calculus, and carious dentine should be removed.
r Insufficient restorations should be replaced.
r If there is substantial loss of tooth structure, the tooth must be
rebuilt with glass ionomer cement or composite.
r Undermined cusps must be adjusted to avoid tooth fracture
between appointments.
r Preparation of the access cavity and identification of the root
canal entrances (note that this action is not a prerequisite in all
cases).
Bow
Jaws Wing
Forceps hole
Contact points
Fig 11.3 The key features of a winged rubber dam clamp.
thickness are commonly used Concerns about latexallergy [23, 24] have encouraged many to switch to latex-free polyethylene or polyvinylchloride products.Rubber dam clamp
Clamps are generally designed with two jaws and fourpoints (Figs 11.3 and 11.4a–e) that are necessary to makestable and secure contact with the tooth to be isolated.Clamps with multiple points are available to make sta-ble contact with unusually shaped or damaged teeth(Fig 11.4f) The jaws may be flat (bland), for use onteeth with good undercuts, or deeply festooned (active),for use on teeth that are partially erupted or with lim-ited undercut, often engaging and gaining stability justbelow the gingival margin (Fig 11.4e) Clamps are avail-able for application to anterior teeth, premolars, andmolars, with the differences relating largely to size anddistance between the jaws Clamps specific to maxillaryand mandibular teeth are also available, as are wingedand wingless versions to accommodate different place-ment techniques (see later) Clamps with a matt finishmay avoid reflection and glare from the operating light.Even new rubber dam clamps may fracture during use[25], and many advocate tying dental floss to all rubberdam clamps before use so that fragments can be safelyretrieved Clamps usually fracture across the bow, andthe simple act of passing floss down the forceps hole onone side, and back up the hole on the other side beforetying in a loop is sufficient
Punches and forceps
A rubber dam punch is used to make holes in the rubberdam sheet (Fig 11.5a) Some punches allow the prepa-ration of a single size of hole, while others have anadjustable platform that allows holes of different diam-eters to be punched To obtain a tight seal around thetooth, the hole must be punched cleanly; a defect in thecut may cause the dam to split and tear as it is stretched
Trang 20188 Endodontic Treatment Procedures
Fig 11.4 (a) An Ivory 9 clamp designed for anterior teeth (b) Winged premolar clamp (c) Winged lower molar clamp with four-point jaws (d) Wingless lower molar clamp (e) Molar clamp with deeply festooned (active) jaws (f) Molar clamp with multiple contact points.
over the tooth The hole should be made in a position
that will ensure positioning of the rubber dam sheet
cen-trally over the mouth to protect the oral opening from
instruments and materials Stamps are available to
indi-cate hole-positioning for individual teeth, or the sheet
can be offered to the mouth and marked to indicate
opti-mal hole-position
Forceps are used to expand the clamp and position
it on the tooth (Fig 11.5b) Sometimes the retaining
grooves on the beaks of the forceps are too deep, and it
(a)
(b)
Fig 11.5 (a) A multi table rubber dam punch, capable of creating holes of
different sizes for different types of teeth (b) Rubber dam forceps.
may be difficult to disengage the forceps from the clampafter placement on the tooth The tips of the forceps caneasily be thinned with a bur or stone to ensure that theywill slip smoothly off the clamp when positioned
Rubber dam frameThe rubber dam frame holds the loose ends of the rubbersheet away from the tooth during treatment, protectingthe oropharyngeal opening and retracting soft tissues.They are available in closed loop and open U-designs(Fig 11.6) and fabricated in metal or plastic The appli-cation of a frame that optimally retracts the dam and
Fig 11.6 Rubber dam frames; a closed frame in plastic and open frames in both metal and plastic.
Trang 21Controlling the environment 189
Fig 11.7 Application of the DryDam ® to a lower right first premolar The
rubber bands round the ears retract the dam instead of a frame.
maximizes working space and visualization is
particu-larly important at the back of the mouth
Frameless rubber dams, such as Dry-Dam© (Directa)
are fixed with rubber bands around the patient’s ears
(Fig 11.7), and include a backing paper to limit contact of
the rubber with facial skin and to absorb small amounts
of moisture that may otherwise be uncomfortable The
hole must be carefully placed according to tooth position
if the Dry-Dam is to be worn comfortably and protect the
oropharynx
Application of the rubber dam
The clamp is usually placed with its jaws on the
buc-cal and lingual sides of the tooth and the bow
posi-tioned distally Although manufacturers suggest
pat-terns of clamp for specified types of teeth, it is important
to choose a clamp that will fit stably and securely on the
tooth, and to confirm this by trying it on the tooth before
use Sometimes creativity is called for in clamp selection
(Fig 11.8)
Winged technique
In this technique, the hole in the rubber dam is stretched
over the wings of the clamp (Fig 11.9a), before carefully
applying it the tooth with forceps, sliding the points of
the clamp below the coronal undercut and positioning
just above the gingival margin (Fig 11.9b) The rubber
is then disengaged from the wings with a blunt
instru-ment (Fig 11.9c), before flossing it through the proximal
contacts to form a tight seal around the neck of the tooth
(Fig 11.9d)
Wingless technique
Here, the bow of the clamp is engaged through the hole
in the dam (Fig 11.10), before applying it to the tooth
Fig 11.8 The aseptic working field of a first upper molar prepared to receive
a ceramic crown but afterwards needing endodontic treatment The absence
of undercuts required creative clamp selection; in this case an Ivory 9 “incisor” clamp The field is disinfected with iodine.
with forceps as before The rubber dam sheet is thenpassed over the clamp and flossed through proximalcontacts as in the winged technique
With both winged and unwinged designs, it is sible to place the clamp first, before stretching a largehole over the entire clamp, or to place the rubber damfirst before applying the clamp over the top When teethare well-approximated, and especially at the front ofthe mouth and around porcelain restorations that may
pos-be damaged by rubpos-ber dam clamps, it may pos-be ble to avoid clamps and retain the rubber dam withinterproximal stabilizing cords such as Wedjets (Coltene)(Fig 11.11) Experienced practitioners develop their ownmethods and tricks; the important thing is to find asimple method that works for you and that provides agood seal
possi-The frame is then applied, taking care not to placeunnecessary tension on the clamp If there is a risk ofleakage at the margins of the dam, small gaps can besealed with a light-curing material such as Opaldam(Ultradent) or a nonhardening putty such as Oraseal®
(Ultradent) Agents of this sort may not seal as perfectly
as an ideally placed rubber dam sheet, but may help
in challenging situations [26] Leakage can also be vented by tying tight dental floss ligatures around therubber dam at the neck of the tooth (Fig 11.12)
pre-Disinfection of the working field
Having isolated the tooth with a well-sealing rubberdam, the working field should be disinfected, includingthe tooth, clamp, and rubber dam to a radius of 2–3 cmfrom the tooth
Trang 22190 Endodontic Treatment Procedures
Fig 11.9 (a) Rubber dam engaged on the clamp wings (b) Positioning the clamp and rubber dam to engage undercuts (c) Disengaging the dam with a blunt instrument (d) Flossing through the contacts secures a tight seal.
A popular method includes scrubbing with 30%
hydrogen peroxide on a cotton bud until no more
effer-vescence is seen, before scrubbing again with 10% iodine
tincture [27] Methods involving 30% hydrogen
perox-ide followed by 2.5% sodium hypochlorite [26] have
shown similar results, as have chlorhexidine/alcohol
preparations
Key steps in the development of an aseptic working
field are summarized in Clinical procedure 11.2
Fig 11.10 Engaging the rubber dam during isolation with a wingless
clamp The bow is pushed from the back of the rubber dam sheet through
the hole, allowing visualization of the jaws as they are applied to the tooth.
Aseptic working procedures
Asepsis should be maintained throughout the entireoperative procedure, and members of the dental teamshould pay attention to hand hygiene and the use ofpersonal protective equipment, including gloves
Fig 11.11 Isolation of three anterior teeth with a single floss ligature and Wedjets placed interproximally.
Trang 23Controlling the environment 191
Fig 11.12 Placement of the rubber dam on the partially erupted central
incisor of a 7-year-old patient The dam was fixed with a floss ligature and two
clamps were placed over the rubber dam sheet on the first primary molars.
Although there is limited evidence on microbial
trans-mission from dental personnel to the root canal, skin
commensals including Staphylococcus aureus and
Pro-pionibacterium acnes have been identified in recent
investigations on the ecology of endodontic infections
[28, 29]
It goes without saying that endodontic instruments
should be sterilized before use, and efforts should be
made to avoid touching the parts of instruments that
will enter the root canal Equally, paper points for
dry-ing root canals should be sterile and gutta points should
be sterilized by immersion in sodium hypochlorite
or chlorhexidine/alcohol for 2 minutes before use
[30–32]
Radiographic images and rubber dam
Contamination of the working field with saliva is a
par-ticular risk during radiographic exposure, and efforts
should be made whereever possible leave the frame in
position (Fig 11.13)
The principles of aseptic working are summarized in
Clinical procedure 11.3
Clinical procedure 11.2 Application and
disinfection of rubber dam
1 Tie floss to the selected clamp before trying it on the tooth.
2 Try on the clamp on the tooth for stability Select a different clamp
if necessary.
3 Choose a frame that enhances the view of the working field.
4 Place the hole central in the rubber dam sheet.
5 Apply the rubber dam by the preferred method.
6 Disinfect the working field by scrubbing the tooth, the clamp and
2–3 cm of rubber dam surrounding the teeth with disinfectant.
Fig 11.13 Exposing a radiograph without losing control of the aseptic ronment The image sensor is held firmly in a hemostat and the dam frame only partially disengaged.
envi-Coronal sealingDue to time constraints, or an active decision to apply anintracanal medicament, root canal treatment may be con-ducted over more than one visit, and require the place-ment of a provisional coronal restoration This shouldprovide a good seal and be durable in function, butshould be readily distinguished from tooth tissue andeasily removed Some materials that are marketed forprovisional restoration expand on setting and may risktooth fracture and leakage [33, 34]
Teeth with substantial tissue loss are best managed bythe placement of a “working restoration” as describedpreviously, sometimes with the support of a copperring or orthodontic band, rather than relying on theprecarious bond of unsupported cement material Con-ventional access cavities within teeth or through work-ing restorations can then be sealed by blocking canalentrances with 2 mm thickness of a soft cement like Cavit
Clinical procedure 11.3 Working aseptically
r Both the operator and the dental nurse should pay attention to
hand hygiene and should wear gloves and a face mask.
r New, sterile burs should be used to enter the pulp chamber.
r Only sterilized instruments should be used in root canals The
working parts of instruments that will enter root canals should be handled with sterile tweezers or mosquito forceps to avoid con- tamination This includes rubber stop adjustment and exchanging rotary instruments in the handpiece.
r The rubber dam frame should not be removed for radiographic imaging.
r Paper points and tubs for irrigants should be sterile.
r Gutta-percha cones should be disinfected for 2 minutes in sodium
hypochlorite before use.
Trang 24192 Endodontic Treatment Procedures
Fig 11.14 Canal plugs of Cavit to protect the root fillings from coronal
leakage if the provisional restoration is lost.
(Fig 11.14), before closing with the maximum
possi-ble thickness of well-adapted hard cement, such as
the polymer-reinforced zinc oxide eugenol cement, IRM
(Dentsply) The canal entrance plugs of Cavit will
pro-vide an additional layer of protection if the provisional
restoration is lost Similarly, canal plugs of Cavit or IRM
can also be placed in canal openings to protect
perma-nent root fillings against coronal leakage
Restorative considerations are discussed in
Chap-ter 16
References
1 Sj ¨ogren U, Figdor D, Persson S, Sundqvist G Influence
of infection at the time of root filling on the outcome of
endodontic treatment of teeth with apical periodontitis Int.
Endod J 1997; 30: 297–306.
2 Barnum SC Following history of the discovery of the dam.
Can J Dent Sci 1877; 4: 88–9.
3 American Association of Endodontists AAE Position
State-ment Dental Dams 2017 Available from: https://www
.aae.org/uploadedfiles/publications_and_research/
guidelines_and_position_statements/
dentaldamstatement.pdf (accessed October 13, 2017).
4 European Society of Endodontology Quality guidelines for
endodontic treatment: consensus report of the European
Society of Endodontology Int Endod J 2006; 39: 921–30.
5 American Academy of Pediatric Dentistry Guideline on
pulp therapy for primary and young permanent teeth
Pedi-atr Dent 2008; 30(7 Suppl.): 170–4.
6 Ahmed HM, Cohen S, L´evy G, Steier L, Bukiet F Rubber
dam application in endodontic practice: an update on
crit-ical educational and ethcrit-ical dilemmas Aust Dent J., 2014;
59: 457–63.
7 Lin PY, Huang SH, Chang HJ, Chi LY The effect of rubber
dam usage on the survival rate of teeth receiving initial root
canal treatment: a nationwide population-based study J.
Endod 2014; 40: 1733–7.
8 Van Nieuwenhuysen JP, Aouar M, D’Hoore W
Retreat-ment or radiographic monitoring in endodontics Int.
Endod J 1994; 27: 75–81.
9 Abbott PV Factors associated with continuing pain in
endodontics Aust Dent J 1994; 39: 157–61.
10 Goldfein J, Speirs C, Finkelman M, Amato R Rubber dam use during post placement influences the success of root
canal-treated teeth J Endod 2013; 39: 1481–4.
11 Israel HA, Leban SG Aspiration of an endodontic
instru-ment J Endod 1984; 10: 452–4.
12 Kuo SC, Chen YL Accidental swallowing of an endodontic
file Int Endod J 2008; 41: 617–22.
13 Susini G, Pommel L, Camps J Accidental ingestion and aspiration of root canal instruments and other dental for-
eign bodies in a French population Int Endod J 2007; 40:
585–9.
14 Whitworth JM, Seccombe GV, Shoker K, Steele JG Use of rubber dam and irrigant selection in UK general dental
practice Int Endod J 2000; 33: 435–41.
15 H ¨ulsmann M, Hahn W Complications during root canal
irrigation – literature review and case reports Int Endod J.
2000; 33: 186–93.
16 Wong RC The rubber dam as a means of infection control
in an era of AIDS and hepatitis J Indiana Dent Assoc 1988;
67: 41–3.
17 Forrest WR, Perez RS The rubber dam as a surgical drape:
protection against AIDS and hepatitis Gen Dent 1989; 37:
236–7.
18 Samaranayake LP, Reid J, Evans D The efficacy of rubber dam isolation in reducing atmospheric bacterial contami-
nation ASDC J Dent Child 1989; 56: 442–4.
19 Harrel SK, Molinari J Aerosols and splatter in dentistry: a brief review of the literature and infection control implica-
tions J Am Dent Assoc 2004; 135: 429–37.
20 Mala S, Lynch CD, Burke FM, Dummer PM Attitudes of
final year dental students to the use of rubber dam Int.
Endod J 2009; 42: 632–8.
21 Anabtawi MF, Gilbert GH, Bauer MR, Reams G, Makhija
SK, Benjamin PL, et al Rubber dam use during root
canal treatment: findings from The Dental Practice-Based
Research Network J Am Dent Assoc 2013; 144: 179–86.
22 Markvart M, Bjorndal L Ten year follow-up study on the adoption of endodontic technology amongst Danish
general dental practitioners Int Endod J 2016; 49: 90,
R163.
23 Burke FJ, Wilson MA, McCord JF Allergy to latex gloves in
clinical practice: case reports Quintessence Int 1995; 26(12):
859–63.
24 Hamann CP, Turjanmaa K, Rietschel R, Siew C, Owensby
D, Gruninger SE, et al Natural rubber latex
hypersensitiv-ity: incidence and prevalence of type I allergy in the dental
professional J Am Dent Assoc 1998; 129: 43–54.
25 Zinelis S, Margelos J In vivo fracture of a new rubber-dam
clamp Int Endod J 2002; 35: 720–3.
26 Ng YL, Spratt D, Sriskantharajah S, Gulabivala K ation of protocols for field decontamination before bacte- rial sampling of root canals for contemporary microbiology
Evalu-techniques J Endod 2003; 29: 317–20.
Trang 25Controlling the environment 193
27 Moller AJ Microbiological examination of root canals and
periapical tissues of human teeth Methodological studies.
Odontol Tidskr 1966; 74(Suppl.): 1–380.
28 Sunde PT, Olsen I, Debelian GJ, Tronstad L Microbiota
of periapical lesions refractory to endodontic therapy.
J Endod 2002; 28: 304–10.
29 Niazi SA, Clarke D, Do T, Gilbert SC, Mannocci F,
Beighton D Propionibacterium acnes and Staphylococcus
epidermidis isolated from refractory endodontic lesions
are opportunistic pathogens J Clin Microbiol 2010; 48:
3859–69.
30 Gomes BP, Vianna ME, Matsumoto CU, Rossi Vde P,
Zaia AA, Ferraz CC, et al Disinfection of gutta-percha
cones with chlorhexidine and sodium hypochlorite Oral
Surg Oral Med Oral Pathol Oral Radiol Endod 2005; 100:
512–17.
31 Kayaoglu G, G ¨urel M, Om ¨url ¨u H, Bek ZG, Sadik B ination of gutta-percha cones for microbial contamination
Exam-during chemical use J Appl Oral Sci 2009; 17(3): 244–7.
32 Fern´andez R, Cadavid D, Zapata SM, Alvarez LG, Restrepo
FA Impact of three radiographic methods in the outcome
of nonsurgical endodontic treatment: a five-year follow-up.
J Endod 2013; 39: 1097–103.
33 Tennert C, Eismann M, Goetz F, Woelber JP, Hellwig E, Polydorou O A temporary filling material used for coro- nal sealing during endodontic treatment may cause tooth
fractures in large Class II cavities in vitro Int Endod J 2015;
Trang 27Chapter 12
Access and canal negotiation: the first
key procedural steps for successful
endodontic treatment
Ove A Peters and Ana Arias
Introduction
An ideal access cavity should promote high-quality
root canal treatment, facilitating the efficient removal
of vital and necrotic pulp tissue, thorough disinfection
and complete obturation of the canal system At the
same time as much structural dentin as possible should
be preserved to maintain the mechanical strength of
the tooth Anatomical knowledge is necessary to avoid
iatrogenic errors, and practitioners should cultivate a
deep understanding of tooth development and
post-eruption reaction patterns that may influence pulp
anatomy, and carefully analyze each case individually
before and during treatment
The access cavity is a critical element of
tooth-preserving endodontic and restorative treatment, and
demands the following essential stages:
r Analysis of the tooth with preoperative radiographs,
and occasionally three-dimensional (3D) imaging
r Access cavity preparation, including conservative
entry to the pulp chamber and identification of all
canal entrances
r Initial canal negotiation, coronal flaring, and working
length determination
Principles of tooth development
and tooth anatomy
Teeth are formed by epithelial and mesenchymal tissues,
which interact sequentially during early development of
the crown (late bell stage) and later during root
devel-opment as cells from the cervical loop proliferate in an
apical direction In the crown, epithelial/mesenchymal
interactions continue throughout dental development,
and result in the external tooth surface and internal pulp
spaces having similar shapes Similarly in the root, theodontoblasts continue to deposit dentin symmetricallyaround the periphery of the pulp, and as a consequence,the inner and outer contours of the root are similar incross-section External landmarks therefore reflect inter-nal anatomy, and this can provide valuable hints foraccess cavity design [1]
The following rules provide further anatomical ance for canal location once the pulp chamber has beenunroofed [2–4]:
located in the center of the tooth at the level of thecemento-enamel junction (CEJ)
cham-ber are concentric to the external surface of thetooth at the level of the CEJ, (i.e., the external rootsurface anatomy reflects the internal pulp chamberanatomy)
of the clinical crown to the wall of the pulp chamber
is the same throughout the circumference of the tooth
at the level of the CEJ – the CEJ is the most consistent,repeatable landmark for locating the position of thepulp chamber
equidistant from a line drawn in a mesial–distaldirection, through the pulp chamber floor (exception:maxillary molars)
line perpendicular to a line drawn in a mesial–distaldirection across the center of the floor of the pulpchamber (exception: maxillary molars)
cham-ber floor is always darker than the walls Secondaryand tertiary dentin particular often has a whitishappearance
Textbook of Endodontology, Third Edition Edited by Lars Bjørndal, Lise-Lotte Kirkevang, and John Whitworth.
© 2018 John Wiley & Sons Ltd Published 2018 by John Wiley & Sons Ltd.
Companion Website: www.wiley.com/go/bjorndal/endodontology
195
Trang 28196 Endodontic Treatment Procedures
are always located at the junction of the walls and the
floor
are located at the angles in the floor–wall junction
are located at the terminus of the root developmental
fusion lines
Over time, secondary and tertiary dentin deposition
may reduce the overall volume of the pulp chamber and
complicate access cavity preparation, and these
chal-lenges may be compounded by the development of pulp
stones within the diminished pulp space
In order to prepare the access cavity conservatively
and in the right place, knowledge is needed of the
number and location of canal entrances, and the
inci-dence and location of common anatomical variations
(see Chapter 13 for more information on 3D pulp
anatomy)
Individual analysis of the tooth,
preoperative radiographs, and additional
CBCT scans in complex cases
Well-angulated radiographic images are mandatory to
facilitate safe and efficient access, negotiation of the
root canal system, and to minimize the risk of mishaps
that may result from unexpected anatomical complexity
Periapical films and bite-wings (for posterior teeth)
pro-vide a first orientation of pulp chamber and root canal
location Although two radiographs with different
angu-lations are often sufficient to develop a 3D picture of the
tooth to be treated, cone beam computed tomography
(CBCT) images may occasionally be justified to
evalu-ate the potential for extra canals, complex morphologies,
and/or dental anomalies [5, 6]
Images should be studied carefully, ideally with nification, to reveal coronal interferences to canal entryand instrumentation such as:
mag-r small pulp chambers;
r canals that have narrowed or become obliterated
coronally;
r bulges of dentin covering canal entrances;
r presence of pulp stones;
r canals that narrow within the root; and
r possible bifurcations or lateral topographical exits of
the apical foramen
Anatomical complexity and potential challenges mayencourage referral to a colleague with specialist skills inendodontics
Rubber dam isolation
For optimal aseptic working, the rubber dam should be
in place before commencing access cavity preparation(see Chapter 11) Yet, there are certain clinical situations,particularly where treatment is undertaken by inexperi-enced clinicians, when accessing before rubber dam iso-lation (Fig 12.1) may have benefits:
r The height of the clinical crown and the long axis of
the tooth may be better visualized, reducing the risks
of furcal or cervical perforation
r Accessing the wrong tooth is less likely As shown
in Fig 12.1, a further recommendation to avoid thisunlikely mishap is to mark the correct tooth with
a sterile pen before starting the access preparation.This also provides information regarding the tooth’slong axis
Irrespective of the sequence, it should be kept in mindthat a well-adapted rubber dam is a key element of infec-tion control in endodontics
Fig 12.1 Markings with sterile red pen to demonstrate the tooth’s long axis before and after rubber dam placement.
Trang 29Access and canal negotiation 197
Core concept 12.1 Evaluation criteria for
traditional access cavity preparations
r Root canal orifices are exposed and clearly visible
r All root canals can be scouted with an endodontic explorer (or
small hand files), in a straight line and without indicating
instru-ment deflection
r The access cavity fully unroofs the pulp chamber, including all
anatomical ramifications
Access cavity preparation
Although glossy advertisements may suggest that
suc-cessful endodontics is all about the latest instrument for
canal preparation, it should be noted that all treatment
steps may be compromised if the access cavity is not
ade-quate, and its importance cannot be overemphasized
In practical terms, access cavity preparation can be
divided in two steps:
1 External outline of the access cavity performed with
high-speed burs until the pulp chamber is
pene-trated
2 Internal refinement where canal orifices should be
identified and straight-line access to the root canal
should be achieved with the goal of maximum
dentin preservation
Standardized access cavity outlines (see Core concept
12.1) for each tooth are frequently utilized in
teach-ing and are helpful to novice students as they prepare
their first preclinical access cavities (Fig 12.2) Defining
cavity shapes in this way helps students to know where
Fig 12.2 Canal orifice locations for the preparation of access cavities.
Source: Adapted from [7].
to cut and reduces some of the risks of inappropriateand excessive tissue loss As stated before, a more tooth-specific approach may help to ultimately achieve thegoal of not sacrificing more tissue than is absolutely nec-essary In this case, 3D software tools enable the visual-ization of transparent models or images, disclosing theposition and dimensions of the pulp chamber and canalentrances in relation to crown and root morphology(Fig 12.3) Such reconstructions define a convenienceform, in which the smallest possible dimensions of theaccess cavity are dictated by the precise location of canalentrances on the pulpal floor The widest dimensionsare situated in the most coronal portion, promoting a
“straight-line” access to the orifice(s) of the root canal,including the removal of dentin to extend straight-lineaccess directly to the apical foramen, or to the primarycurvature of the root canal These concepts facilitatesubsequent treatment procedures and minimize proce-dural errors (Fig 12.3)
(a)
(b)
Fig 12.3 Microcomputed tomography renderings of access cavities in a maxillary central incisor, seen in the clinical and mesiodistal view Note the difference in angulation of the tooth long axis and the entry of the access into the crown seen in the mesio-distal view (a) Traditional access (b) Minimal invasive access.
Trang 30198 Endodontic Treatment Procedures
It should be recognized that overly large access
cavi-ties eliminate crucial structural dentin and may
compro-mise the biomechanical integrity of the tooth Minimal
preparation is more readily achieved with
magnifica-tion, enhanced lighting, and highly flexible instruments,
making it possible to move from the stereotypical and
often excessive designs (round/triangular in anterior
teeth, oval in premolars and triangular/quadrilateral in
molars) illustrated in most textbooks [8, 9] Many teeth
have suffered considerable tissue loss prior to
endodon-tic intervention, and it may be even more important in
such cases to adopt a thoughtful, deliberate, and
con-servative approach to access in order to avoid further
unnecessary tissue loss and structural weakening
Figure 12.4a shows procedural steps for traditional
and conservative access to a mandibular molar (see
Clin-ical procedure 12.1) Millions of successfully preserved
teeth are testament to the fact that endodontic treatment
involving traditional access can be successful, but
con-temporary concepts are encouraging the use of
magni-fication and minimal intervention Where magnimagni-fication
is not available, as in most undergraduate programmes
and many private practices, students and practitioners
are encouraged to work as conservatively as they
rea-sonably can
The steps outlined in Fig 12.4b include the initial
preparation of a smaller version of the final access
cav-ity shape deep into dentin, and penetration into the
pulp chamber towards the largest pulp horn This may
be done with a cylindrical or slightly tapered diamond
bur The outline is then refined, unroofing the
cham-ber with a non end-cutting bur, which is unlikely to
damage the chamber floor or walls If magnification is
Clinical procedure 12.1 Access cavity preparation
Irrespective of the specific tooth and root canal anatomy, the main
steps in access cavity preparation involve the following:
1 Visualizing the pulp chamber space in three dimension and in
relation to the clinical crown.
2 Penetration of the enamel or restoration with an appropriate bur.
3 Mapping out a small version of the definitive access cavity shape
deep in dentin with opening of the pulp chamber towards the
largest pulp horn.
4 Refining the access:
– with a bur with a noncutting tip.
– with ultrasonic tips and under magnification for higher dentin
preservation.
5 Balancing the access cavity size between the demands for an
adequate convenience form and optimized structural strength.
The final access cavity must reveal all root canal orifices and
should function as a reservoir for irrigation solutions during
subse-quent shaping Importantly, it should provide adequate space for root
canal instruments to the canals.
available, conventional high- and low-speed burs may
be less desirable [3], and practitioners may prefer toselectively unroof the chamber with ultrasonically ener-gized tips that improve visual access, while providinghigh cutting efficiency, combined with safety and con-trol [10] Alternatives include special burs with long, nar-row shanks to enhance visualization, and small roundtips for controlled cutting (e.g., Munce discovery burs,
or Meisinger Goose-Neck burs)
In conventional access procedures, ultrasonic tips areuseful for access refinement, location of MB2 canals inupper molars and accessory canals in other teeth, loca-tion of calcified canals in any tooth, and the removal ofattached pulp stones [10–12] Micro-instruments (micro-openers, micro-debriders) were designed to facilitatesuch procedures under magnification [13] The combina-tion of the operating microscope (magnification and illu-mination together) and specifically designed ultrasonictips has allowed these steps to be optimized (Fig 12.5)[11, 14]
The concept of minimally invasiveaccess cavities
As Gluskin et al [8] have stated, “the concept of
mini-mally invasive endodontics calls for the treatment andprevention of pulpal pathoses and apical periodontitis,while causing the least amount of change to the dentalhard tissues This preserves the strength and function ofthe endodontically treated tooth with the intent that itwill last the patient’s lifetime.” Access cavity prepara-tion is the first invasive step of a root canal treatmentand therefore it plays a crucial role in this treatmentapproach
Endodontically treated teeth are more frequentlyextracted because of fracture than because of persis-tent apical pathosis [15] and, as we have seen, efforts
to maintain tooth structure are beneficial This conceptchanges access cavity design from a completely unroof-ing and coronally divergent preparation to the selectivepreservation of part of the chamber roof and pericer-vical dentin [16], prioritizing the removal of caries andrestorative material ahead of tooth structure, while con-serving dentin mainly at the cervical level [9] The finalgoal is the long-term functional survival of the tooth, rec-ognizing that no current restorative material can replacethe mechanical characteristics of dentin lost from areasthat suffer intensive stress–strain forces during mastica-tion or parafunctional activity [17]
Preserving pericervical dentin (4 mm above and belowthe crestal bone) seems to be crucial for the distri-bution of functional stresses and hence tooth strengthand long-term viability of the entire complex [17] Theconservation of the dentin in the cingulum of incisors
Trang 31Access and canal negotiation 199
(a)
(b)
Fig 12.4 Two different versions of access cavity preparations in the same simulated mandibular molar (a) Traditional access (b) Minimally invasive access Note the use of ultrasonic tips and small files to scout the root canal in both (phase 1).
(pericingulum dentin) also seems to improve functional
stress distribution [9], although the conservation of
dentin has not yet been correlated with an increase in
fracture resistance in this group of teeth [18] Finite
ele-ment analysis has, however, demonstrated the reduced
masticatory stresses achieved by preserving even smallamounts of radicular dentin [19]
Although it preserves tissue, the use of minimallyinvasive endodontics [8] does not mean that treatmentgoals must be compromised, and access preparation
Trang 32200 Endodontic Treatment Procedures
Fig 12.5 Microcomputed tomography of
mini-mally invasive access cavity preparation in a natural
mandibular molar Right: The same access in
transpar-ent image to show the conservation of the pulp
cham-ber walls when access to both distal canals is gained.
should not be so restrictive as to impede the location
and entry of instruments into all canal orifices or safe
and efficient cleaning and shaping procedures Balance
is needed, with judgments based on clinical
experi-ence Cavities cut within composite or amalgam can
often be slightly larger and the benefits of removing old
restorative materials in their entirety are discussed in
Chapter 11
Difficulties accessing teeth with mineralized
pulp chambers
Access may be difficult in traumatized teeth, in older
patients where pulp chambers and root canal entrances
have been reduced by dentin apposition, or where they
are obliterated by pulp stones [20] The apposition of
new hard tissue starts in the coronal portion of the root
canal and is followed by gradual narrowing of the pulp
space [21]
Pulp canal mineralization is not pathologic itself
and its identification on a radiograph does not require
endodontic intervention per se, yet pulp infection and
necrosis may subsequently develop, and require
treat-ment Occasionally, teeth may become discolored by the
apposition of dentin, and in the absence of clinical signs
or symptoms of disease, root canal treatment is again
not generally indicated If, on the other hand, signs and
symptoms of disease do develop, or if a decision is made
to attempt internal bleaching of the tooth to improve its
color, root canal treatment may in these circumstances
be justified Accessing such teeth requires great skill and
experience and there is a high risk of causing massive
tissue loss, perforating the crown or root, or promotingfracture risk Specialist referral should be considered insuch cases
The use of magnification, ideally an operative scope, often allows the visualization of a central gray,translucent area, fully embedded in darker tertiarydentin or bone-like matrix [4] These structures areindicative of the original canal orifices In other cases,there is no color change to indicate where the root canalsused to be and dyes may help to detect the fine residualfilaments of pulp tissue or tracts of organic material [22]that histology suggests are always present (Fig 12.6)
micro-Fig 12.6 Use of ophthalmic fluorescein in a calcified canal to dye organic rests in search of the root canal.
Trang 33Access and canal negotiation 201
The use of long ultrasonic tips optimizes visualization
when working deep under the microscope and reduces
unnecessary dentin removal [10] Progress has
tradi-tionally been monitored by periodically exposing
peri-apical radiographs from different angles (Fig 12.7) and
this helps to control against mesiodistal or buccolingual
deviation
Three-dimensional imaging technology is
recom-mended to avoid mishaps in complex cases, with the
potential to create 3D printed templates that will guide
the access of burs into calcified root canals, analogous
to the guide sleeves used routinely during implant
placement [23–25] Virtual planning and precise
cut-ting has the potential to preserve tooth structure and
avoid iatrogenic perforations in calcified teeth (see alsoChapter 23)
Access as a diagnostic step
It is desirable to confirm not only the etiology of pal or periapical pathosis in teeth requiring endodontictreatment, but also to assess their restorability Often thisrequires the complete removal of the existing restora-tion to exclude or confirm leakage, fractures, and otherpossible causes [26] The merits of removing crownsand other restorative materials before access to identifycaries, cracks, or fractures was discussed in Chapter 11,
Trang 34pul-202 Endodontic Treatment Procedures
Advanced concept 12.1
Initial coronal flaring may progress in a stepwise manner, moving
for-ward and backfor-ward between phases 1 and 2 This may be especially
helpful when shaping root canals with substantial curvatures or
S-shaped root canals, which often present their most challenge anatomy
in the apical third Here, mishaps are often avoided by flaring the
coro-nal two-thirds of the root cacoro-nal before negotiating the apical third.
This pre-enlargement procedure can be conceptualized by imagining
the root canal as two separate canals After scouting the coronal part
of the canal with an ISO 8 or 10 file (phase 1), the straight coronal
portion (first part of the curvature) is first flared as an uncomplicated
canal This will allow easier negotiation and subsequent enlargement
of a shorter but more difficult apical third after removing the
restric-tive dentin in the coronal and middle thirds.
and this process forms an important element of the
diag-nostic and treatment planning process
Canal negotiation
After selectively unroofing the canal entrances and
ensuring that instruments can approach them without
stress, it is time to begin canal negotiation The pulp
chamber may be flooded with sodium hypochlorite
to secure hemostasis, commence disinfection, and help
lubricate the path of instruments as they enter deeper
canal anatomy In uncomplicated cases, it may be
possi-ble to advance a small hand file (size ISO 10 or smaller)
with gentle watch-winding motions directly to the root
canal terminus and estimate the working length with
an electronic apex locator Many canals can then be
enlarged without special difficulty, following the
prin-ciples outlined in Chapter 13
In more complicated cases where it is not
possi-ble to advance directly to the canal terminus,
negoti-ation may be conducted in a number of phases, with
episodes of exploration and enlargement that will
facil-itate the stepwise progression of instruments to the
api-cal constriction and facilitate safe canal enlargement
(see also Advanced concept 12.1) Although this process
is distinct from the outlining and unroofing processes
described for access cavity preparation, it is
undoubt-edly an element of endodontic access, extending from
the pulp chamber to allow instrument access into the
complexities of the canal system The principles of this
often multiple-phased process are described in the
fol-lowing sections:
r Pre-flaring the superficial part of the canal (phase 1)
r Coronal flaring of the coronal half to two-thirds of the
canal (phase 2)
r Negotiation to the canal terminus and determination
of working length (phase 3)
Pre-flaring the superficial part
of the canal (phase 1)
It is tempting to open root canal entrances with driven nickel–titanium (Ni-Ti) files without first explor-ing or “scouting” the anatomy, but the temptationshould be resisted if instrument fractures and other iatro-genic errors such as ledges are to be avoided The coro-nal portion of root canals should always be exploredwith hand files, often sizes ISO 15 or 20, to determinehow deeply they can enter without force Often someapproximation can be made from the preoperative radio-graph, but in particularly narrow canals, the radiographmay not be helpful and even smaller instruments may
engine-be needed for initial exploration engine-before gently openingwith hand files to size ISO 15 or 20 The confirmationthat small instruments can enter should ensure a safepathway for the fragile tips of canal-enlarging instru-ments, and defines the apical limit of a so-called “safetyzone” which may be opened or “flared” with a range
of instruments including Ni-Ti orifice shapers, increasedtaper Ni-Ti files, or Gates Glidden drills Instrumentationshould be accompanied by copious irrigation to removecutting debris The act of pre-flaring improves straight-line access and removes interferences from the coronalpart of the canal that may impede deeper explorationand canal enlargement
Coronal flaring of the coronal half totwo-thirds of the canal (phase 2)Pre-flaring (phase 1) often allows small hand instru-ments to penetrate deeper into the canal, usually withgentle watch-winding and filing motions against thecanal walls to gain entry to the middle or apical third
of the canal Engine-driven or hand Ni-Ti instrumentsmay then be used to flare the coronal half to two-thirds
of the canal, accompanied once again by copious tion In very challenging curved or narrow canals, thisphase may itself progress in a series of steps and requiresboth patience and light touch
irriga-In common with pre-flaring, coronal flaring allowsdeeper exchange of irrigant and removes further inter-ferences that may impede entry to the apical third
Negotiation to the canal terminus anddetermination of working length (phase 3)
At this stage it is often possible to advance small, curved hand instruments to the root canal terminus withlight watch-winding and filing motions and determinethe working length with an electronic apex locator Thismay be confirmed with a working length radiograph
pre-It is then common practice to develop a “glide-path”
by progressively advancing stainless-steel hand files of
Trang 35Access and canal negotiation 203
size ISO 15 or 20 to working length, and lightly opening
the space to ensure that they will “glide” smoothly in
and out of the canal without resistance This once again
ensures the safe passage of larger hand- or engine-driven
Ni-Ti instruments, which will complete the canal
flar-ing from coronal orifice to workflar-ing length Alternatively,
rotary or reciprocating instruments specifically designed
for the purpose of glide path preparation may be used
Detailed shaping protocols, including final apical
canal instrumentation (phase 4) will again be considered
in Chapter 13
References
1 Hargreaves HM, Goodis HE, Tay RT Seltzer and Bender’s
Dental Pulp, 2nd edn Chicago, IL: Quintessence, 2002.
2 Ingle JI, Bakland LK, Baumgartnet JC Endodontics, 6th edn.
Hamilton, ON: BC Decker, 2008.
3 Peters OA Accessing root canal systems: knowledge base
and clinical techniques ENDO 2008; 2: 87.
4 Krasner P, Rankow HJ Anatomy of the pulp-chamber
floor J Endod 2004; 30: 5–16.
5 Arens DE, Gluskin AH, Peters CI, Peters OA Practical
Lessons in Endodontic Treatment Chicago, IL: Quintessence,
2009.
6 AAE and AAOMR Joint position statement: Use of cone
beam computed tomography in endodontics 2015 update.
J Endod 2015; 41: 1393–6.
7 Carlsen O Dental Morphology Copenhagen: Munksgaard,
1987.
8 Gluskin AH, Peters CI, Peters OA Minimally invasive
endodontics: challenging prevailing paradigms Br Dent.
J 2014; 216: 347–53.
9 B ´oveda C, Kishen A Contracted endodontic cavities: the
foundation for less invasive alternatives in the
manage-ment of apical periodontitis Endod Topics 2015; 33: 169–86.
10 Plotino G, Pameijer CH, Grande NM, Somma F Ultrasonics
in endodontics: a review of the literature J Endod 2007; 33:
81–95.
11 Rampado ME, Tj¨aderhane L, Friedman S, Hamstra SJ.
The benefit of the operating microscope for access cavity
preparation by undergraduate students J Endod 2004; 30:
863–7.
12 G ¨orduysus MO, G ¨orduysus M, Friedman S Operating
microscope improves negotiation of second mesiobuccal
canals in maxillary molars J Endod 2001; 27: 683–6.
13 B ¨urklein S, Sch¨afer E Minimally invasive endodontics.
Quintessence Int 2015; 46: 119–24.
14 Sheets CG, Paquette JM Ultrasonic tips for conservative
restorative dentistry Dent Today 2002; 21: 102–4.
15 Bor´en LD, Jonasson P, Kvist T Long-term survival of endodontically treated teeth at a public dental specialist
clinic J Endod 2015; 41: 176–81.
16 Clark D, Khademi J Modern molar endodontic access and
directed dentin conservation Dent Clin North Am 2010;
54: 249–73.
17 Clark D, Khademi JA Case studies in modern molar
endodontic access and directed dentin conservation Dent.
Clin North Am 2010; 54: 275–89.
18 Krishan R, Paqu´e F, Ossareh A, Kishen A, Dao T, Friedman
S Impacts of conservative endodontic cavity on root canal instrumentation efficacy and resistance to fracture assessed
in incisors, premolars, and molars J Endod 2014; 40:
1160–6.
19 Bonessio N, Arias A, Lomiento G, Peters OA Effect of root canal treatment procedures with a novel rotary nickel titanium instrument (TRUShape) on stress in mandibular
molars: a comparative finite element analysis Odontology
21 Smith JW Calcific metamorphosis: a treatment dilemma.
Oral Surg Oral Med Oral Pathol 1982; 54: 441–4.
22 Malhotra N, Mala K Calcific metamorphosis
Litera-ture review and clinical strategies Dent Update 2013; 40:
48–58.
23 Zehnder MS, Connert T, Weiger R, Krastl G, K ¨uhl S Guided endodontics: accuracy of a novel method for guided access cavity preparation and root canal location.
study Int Endod J 2016; 49: 790–5.
26 Abbott PV Assessing restored teeth with pulp and periapical diseases for the presence of cracks, caries
and marginal breakdown Aust Dent J 2004; 49:
33–9.
Trang 37Chapter 13
Root canal instrumentation
Lars Bergmans and Paul Lambrechts
Introduction
Accurately prepared root canals that allow effective
elimination of soft- and hard-tissue elements,
disin-fection, and obturation of the root canal system are
critical to successful endodontic treatment The
proce-dure, which is referred to as “cleaning and shaping”
[1], is often difficult and time-consuming and
there-fore requires a systematic approach to avoid errors such
as underpreparation and iatrogenic injury that may
adversely affect the clinical prognosis In this chapter
concepts for effective root canal instrumentation are
reviewed and clinical guidelines are presented, based on
root canal system anatomy and final shaping objectives
Principles of root canal instrumentation
Root canal instrumentation is accomplished by the use
of endodontic instruments and (antimicrobial)
irrigat-ing solutions under aseptic workirrigat-ing conditions A
pri-mary objective of this chemomechanical preparation in
teeth with vital or necrotic pulps is shaping the root
canal space and it is generally accepted that the most
appropriate final root canal shape is a tapered
(coni-cal) preparation with the smallest diameter near the root
tip, and the widest at the canal entrance Special
atten-tion should therefore be paid to the apical level and
the original path of the canal As a general rule, the
removal of root dentin should be centered within the
ini-tial root canal anatomy, and during canal enlargement
soft-tissue elements, which potentially provide substrate
for the growth of remaining microorganisms, will also be
removed
Root canal instrumentation may be carried out using
hand-held or engine-driven rotary or reciprocating
instruments, which come in many configurations The
quality, sizing, and physical properties of endodontic
instruments and the materials used for their facture are generally well defined according to ISO(International Organization for Standardization) andANSI (American National Standards Institute) stan-dards Instrument properties (e.g., stiffness) relate to
manu-type of alloy (stainless steel versus nickel–titanium
(Ni-Ti)), degree of taper (conicity), and cross-sectionaldesign
Stainless-steel files are inherently stiff, and thisincreases with increasing instrument size An instrumentthat is too stiff will cut more on the convex (outer) sidethan on the concave (inner) side, thereby straighteningthe curve (Fig 13.1) The resulting “hour-glass shape”and canal aberrations (e.g., ledge, zip, and perforation)leave important portions of the root canal wall uninstru-mented and create an irregular canal shape that is diffi-cult to clean, disinfect, and fill properly
Over time, researchers and clinicians have found avariety of methods to overcome the challenges cre-ated by stiff stainless-steel instruments, and as a result,various movements for instrument manipulation andapproaches to shaping the canal have been proposed.Although skillful operators can handle these demandingtechniques, shaping a curved root canal with stainless-steel hand files remains a time-consuming and challeng-ing exercise
Besides adaptations in file design and use, the problem
of instrument stiffness has been reduced by the tion of instruments manufactured from Ni-Ti alloys [2].Ni-Ti’s unique property of superelasticity allows Ni-Tifiles to be worked in curved canals with less lateral forceagainst the canal walls All such files are made from Niti-nol, an equi-atomic Ni-Ti alloy (using about 56 wt% Niand 44 wt% Ti) with a low modulus of elasticity and greatresistance to plastic deformation (Note: The symbols ofthe metals were combined with the place of invention(Naval Ordnance Laboratory, Silver Springs, MD, USA)
introduc-to create the acronym NiTiNOL.) Today, a wide array of
Textbook of Endodontology, Third Edition Edited by Lars Bjørndal, Lise-Lotte Kirkevang, and John Whitworth.
© 2018 John Wiley & Sons Ltd Published 2018 by John Wiley & Sons Ltd.
Companion Website: www.wiley.com/go/bjorndal/endodontology
205
Trang 38206 Endodontic Treatment Procedures
Straighten
Fig 13.1 The stiff instrument tends to straighten within the curved root
canal (a), causing ledge formation (b), zipping (c), or perforation (d).
Ni-Ti file systems are employed in general and specialist
practices, the majority being engine-driven in
continu-ous clockwise rotation or reciprocation
The driving force behind this development is the belief
that engine-driven Ni-Ti files, applied in a crown-down
sequence (see further later) could improve both the
qual-ity and efficacy of root canal preparation Before
enter-ing the excitenter-ing field of Ni-Ti instrumentation, some
basic preparation concepts will be reviewed, including
straight-line access and the matching of shaping
objec-tives with individual tooth anatomy
Root canal system anatomy
Root canal(s) versus root canal system
The internal complexity of teeth has been investigated
by a variety of methods As early as 1917, Hess [3]
stud-ied the internal anatomy of thousands of teeth using
an impression/replica technique and concluded that
the pulp anatomy often represented a complex system
composed of a central area (root canals with round,
oval or irregular cross-sectional shape) with lateral
complexities (fins, anastomoses, and accessory canals),
which in terms of volume were greater than the central
“root canals.” This observation challenges conventional
approaches to root canal cleaning and shaping, since
much of the anatomical complexity will be
inaccessi-ble to both instruments and irrigants/medicaments, and
may therefore continue to harbor necrotic pulp tissue as
well as infectious elements (see Fig 15.35) A
postop-erative endodontic image that demonstrates a smooth,
conical shape is generally too simplistic and
underesti-mates the challenges of root canal instrumentation and
disinfection
In dental practice, complete visualization of the pulp
system is rarely possible, since even an operating
microscope is limited to the straight part of the root
canal (above the curve) Traditionally, radiographic
techniques have been considered helpful adjuncts tovisualize root morphology, but radiographs also havelimitations related to resolution, geometric distortion,and two-dimensional (2D) projection Even though theparalleling technique with orthogonal and eccentricprojections improves our three-dimensional (3D) under-standing, much of the lateral anatomy, especially in thebuccolingual plane, will remain invisible (Fig 13.2) This
Trang 39Dig-Root canal instrumentation 207
Fig 13.3 (a) Digital photograph of an upper premolar with a single root that is severely curved toward its terminus (b,c) Micro-CT images showing the internal anatomy of the root (d) A detailed view on the anatomical complexity of the apical part.
limited perception of canal system anatomy may cause
procedural difficulties and may invite clinicians to
fol-low an unthinking 2D-based approach where
instru-mentation to the final working length early in the
shap-ing procedure often causes procedural mishaps
As 3D imaging (cone beam computed tomography
(CBCT)) has become established in specialist dental
practice, the unique anatomy of complex root canal
sys-tems can be observed from axial, sagittal, and coronal
perspectives [4] That is not to say that all canal
sys-tems should be 3D imaged and international guidelines
(SEDENTEXCT guidelines [5], American Association of
Endodontists (AAE)/American Academy of Oral and
Maxillofacial Radiology (AAOMR) [6], and European
Society of Endodontology (ESE) [7] recommend that this
should be reserved for teeth with a high frequency of
extra canals, and cases where complex morphology is
suspected
Even in these cases, caution should be exercised before
exposing patients, particularly young patients, to 3D
imaging; high-resolution, limited field-of-view images
are generally preferred Aside from radiation concerns,limitations for the use of CBCT as a standard procedureinclude potential artefact generation and high levels ofscatter and noise [8] Therefore, CBCT should be usedonly when the need for imaging cannot be met by lowerdose 2D radiography
Root canal curvatureMost root canals are curved and, in addition, curved rootcanals are often relatively narrow when compared totheir straight counterparts Curvatures typically becomemore pronounced and exhibit their greatest anatomi-cal complexity toward their apical terminus (Fig 13.3).Root canal curvature can be described by level (coro-nal, middle, or apical), angle and radius [9] Most cur-vatures are multiplanar and are thus expressed in bothmesiodistal and buccolingual (or buccopalatal) planes(Fig 13.4)
The fact that root canals are curved and narrow inmature teeth makes them difficult to clean and shape
Trang 40208 Endodontic Treatment Procedures
(a) (b)
(c) (d)
Fig 13.5 Micro-CT data of an upper premolar (a,b) Renderings of the outer
root surface with a mesial invagination (c,d) Visualizations of the inner root
anatomy Notice that the root canals that join or diverge deviate from their
initial path, while the resulting angle is different for the canals involved.
The risks of canal straightening and the creation of errors
are related to the level and severity of the curvature, with
abrupt apical curvatures and double curvatures (the
S-shape) being especially difficult to negotiate and shape
In addition, canals that join or diverge always deviatefrom their initial path (Fig 13.5)
The creation of straight-line access, the use of flexibleendodontic instruments and the precurving of files areessential measures to prepare curved canals (see furtherlater, and Chapter 12 on canal negotiation)
Cross-sectional shape and diameterRoot canals may be round, oval, or irregular (ribbon-shaped) on cross-sectional view Oval and irregularshapes are common in the coronal two-thirds of rootcanals, whereas the round variant is often restricted tothe apical part (Fig 13.6) When two or more canalsare present in the same root, anastomoses and fins(lateral extensions) are frequently observed (Fig 13.7),while some root canals may present with extreme cross-sectional shapes This applies especially to the C-shapedcanal (Fig 13.8), which is more prevalent in certainethnic groups [10] Oval and irregular cross-sectionalshapes in particular challenge root canal cleaning andshaping since large parts of the lateral anatomy are oftenout of reach because most endodontic instruments aredesigned to stay centered (see Fig 15.35)
Root canal diameter is related to the concept of ity or “taper.” Observing the root canal diameter at con-secutive levels along the root gives an idea of its overallconicity, though the exact diameter and taper will varyfor each point along the central axis Usually root canalsare wide in the coronal part and relatively narrow api-cally Immature teeth and roots that have experiencedinternal resorption may appear different, and the deposi-tion of secondary and tertiary dentin may alter root canaldiameter generally or locally
(c)
(d)
(e)
Fig 13.6 (a,b) Visualizations of a lower
premolar scanned with micro-CT (c–e)
Corre-sponding slices at different horizontal levels
(indicated by the yellow lines) reveal the
rib-bon (c), oval (d), and round (e) cross-sectional
shape of the canal.