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Tissue homogenates from 60 specimens submitted to the Veterinary Diagnostic Center were evaluated by polymerase chain reaction (PCR) for detection of bovine viral diarrhea virus (BVDV)..[r]

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Investigation Journal of Veterinary Diagnostic

http://vdi.sagepub.com/content/6/1/44 The online version of this article can be found at:

DOI: 10.1177/104063879400600109

1994 6: 44

J VET Diagn Invest

Beverly J Schmitt, Osvaldo J Lopez, Julia F Ridpath, Judith Galeota-Wheeler and Fernando A Osorio Evaluation of PCR for Diagnosis of Bovine Viral Diarrhea Virus in Tissue Homogenates

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Evaluation of PCR for diagnosis of bovine viral

diarrhea virus in tissue homogenates

Beverly J Schmitt, Osvaldo J Lopez, Julia F Ridpath, Judith Galeota-Wheeler, Fernando A Osorio

Abstract Tissue homogenates from 60 specimens submitted to the Veterinary Diagnostic Center were evaluated by polymerase chain reaction (PCR) for detection of bovine viral diarrhea virus (BVDV) Conventional virus isolation procedures showed the specimens contained BVDV The BVDV RNA was extracted from the homogenates and subjected to a reverse transcription reaction followed by PCR amplification The PCR product was blotted onto a nylon membrane and hybridized with a 30-base pair oligonucleotide probe labeled with 32P One set of PCR primers detected BVDV in 46/60 (77%) of the tissue homogenates An additional set of primers was used to detect 10/11 samples that had escaped detection with the first set of primers The results indicate that BVDV can be detected by PCR directly out of tissue homogenates generated in a diagnostic setting

Bovine viral diarrhea virus (BVDV) is an important using the polymerase chain reaction (PCR) tech-pathogen of cattle causing a variety of diseases that nique.1,2,5,6,10,11,15,23,24,26

range in severity from mild inapparent infections to Application of PCR in a diagnostic setting requires fatal mucosal disease.20,21 BVDV is classified within adaptation for use on clinical samples Previous reports the family Flaviviridae and is a member of the genus on the use of PCR for detection of BVDV have not Pestivirus The viral genome consists of a single-strand- addressed the problems associated with clinical

spec-ed nonpolyadenylatspec-ed RNA about 12.5 kb in length.8 imens The purpose of this study was to evaluate PCR Isolates of BVDV are divided into 2 biotypes, cyto- detection of BVDV in field specimens using primers15,22

that amplify sequences near opposite ends of the BVDV genome.

pathic and noncytopathic, based on effects in cell

cul-ture.25 Infections of cattle with noncytopathic BVDV

during early gestation may cause abortion or birth of

calves immunologically tolerant to the infecting

vi-rus 12,17 These calves are persistent carriers of BVDV

and can be a source of infection to the herd.4

Super-infection of persistently infected cattle with a

cyto-pathic BVDV may induce mucosal disease.4,7

Detec-tion of persistently infected cattle is important for

control of BVD In addition, contamination of fetal

bovine serum and other bovine products used in cell

culture work with BVDV presents a problem in

pre-paring veterinary biologics.3,19 Traditionally, virus

iso-lation with confirmation by immunofluorescence

has been the method used to detect BVDV in clinical

samples and in contaminated cell cultures and

biolog-ical products These techniques are time

consum-ing and require the use of cell culture Several reports

have described rapid diagnostic tests for BVDV

From the Department of Veterinary Science, University of

Ne-braska, Lincoln, NE 68583 (Schmitt, Lopez, Galeota-Wheeler, Oso-

rio), and the Virology Cattle Research Unit, National Animal

Dis-ease Center, Agricultural Research Service, USDA, Ames, IA 50010

(Ridpath) Current address (Schmitt): Diagnostic Virology

Labora-tory, National Veterinary Services Laboratories, Veterinary Services,

Animal and Plant Health Inspection Service, USDA, Ames, IA

Received for publication March 26, 1993

Materials and Methods

Preparation of clinical samples Tissue samples from

cat-tle with clinical signs of BVD were submitted to the Uni-versity of Nebraska Veterinary Diagnostic Center virology laboratory All samples came from the state of Nebraska between 1988 and 1990 The samples were prepared as fol-lows Minimum essential medium (MEM) with 2% horse serum was added to tissue at a ratio of 3:l and then ho-mogenized in a stomacher for 2-4 min The homogenate was centrifuged at 2,000 rpm for 30 min at 4 C, after which the supernatant was poured into 15-ml snap cap tubes After initial detection of BVDV by virus isolation, homogenates were preserved by freezing at -70 C

Cell cultures Tissue homogenates were inoculated onto

primary bovine lung cells and bovine turbinate cellsa that were free of BVDV Cultures were maintained in MEM sup-plemented with 10% horse serum in Leighton tubes Cell monolayers were stained for BVDV antigen using fluores-cein-labeled antibodya and examined by fluorescent micros-copy Tissue homogenates that were positive for BVDV were used for further analysis by PCR

Titration of homogenates Before the PCR was used, the

titer of infectious BVDV was determined using microtitra-tion plates and bovine lung cells For noncytopathic BVDV samples, serial dilutions of virus were inoculated onto bovine lung cells and then infected with a cytopathic BVDV (Singer 44

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Use of PCR in diagnosis of BVDV infections 45

strain) End point was determined by observing inhibition

of cytopathic effect?

Primers for PCR The primers used in this study are listed

in Table 1 Primer set 115 amplifies a 205-base segment that

contains a BstIII site The primers were synthesizedb and

purified by either polyacrylamide gel electrophoresis (PAGE)

or high-performance liquid chromatography (HPLC) A

sec-ond set of primers was used on samples that were not

am-plified by primer set 1 with the exception of 3 samples that

lacked sufficient quantity for testing These primers are

lo-cated at the 5' end immediately upstream of the open reading

frame 22

Extraction of BVDV RNA from tissue homogenates. A

100-µl volume of tissue homogenate was added to 450 µ1 of

STE buffer (100 mM NaCl, 50 mM Tris, 5 mM

ethylene-diaminetetraacetic acid [EDTA]; pH 8) in a microcentrifuge

tube Proteinase K was added at a concentration of 20 µg/

ml, the solution was incubated at room temperature for 10

min, and 6 µl of 10% sodium dodecyl sulfate (SDS, 0.2% wt/

vol) was added After incubation at 56 C for 20 min, phenol/

chloroform extractions were done, and the nucleic acid was

precipitated with 60 µl of 3 M sodium acetate (pH 5.2) plus

1 ml cold (-20 C) 100% ethyl alcohol The precipitated RNA

was pelleted by centrifugation for 15 min at 12,000 x g at

4 C, washed with cold 70% ethanol, and dried in a vacuum

desiccator

Reverse transcription (R T) After resuspension of the RNA

pellet in 9 µl of sterile distilled water, 1 µl (40 µmol) of each

primer was added and the sample was incubated for 5 min

at 70 C The sample was chilled on ice for 1 min and 3 µl

of deoxynucleoside triphosphatesc (7.5 µM), 4 µl Schimke

solution (10 mM dithiothreitol, 10 mM MgCl,, 70 mM KCl,

80 mM Tris; pH 8), 1 µl of reverse transcriptased (19.6 u/ml),

and 1 µl of RNase inhibitore (28 u/µl) were added The

re-sulting RT reaction mixture was incubated at 42 C for 2 hr

Reaction mixtures were then chilled on ice for immediate

testing or stored at -20 C

PCR Viral strains NADL and Singer were used as

pos-itive controls for PCR Negative controls consisted of MEM

with 1) 10% horse serum and gentamicin and 2) tissue

ho-mogenate from a gnotobiotic calf A mixture of PCR reagents

without template was used as control for detection of

con-tamination with extraneous DNA

A master mix was made of the following reagents: distilled

water, 58.5 µl; 10x buffer, 9.5 µl (15 mM MgCl2, 400 mM

Tris-HCl, 500 mM KCl, 1% gelatin); dNTPS, 16 µl of

equi-molar mixture of dNTPs (5 mM); 3 µl (40 µmol) of each

primer A 5-µl aliquot of RT reaction mixture was added;

the resulting mixture was heated to 95 C for 5 min and chilled

on ice Then 2.5 units of Taq polymerasee in 5 µl of 1 x buffer

(10 x Promega buffer diluted 1: 10) were added for PCR

am-plification A layer of 60 µl of Nujol mineral oilf was added

The PCR program consisted of 1 min at 95 C (denaturation),

1 min at 55 C (primer annealing) and 2 min at 72 C (primer extension) for 40 cycles Primer set 2 was used at a concen-tration of 15 µmol per reaction during both RT and PCR All other parameters remained as previously described

Nucleic acid hybridization. PCR products using primer set 1 were analyzed by electrophoresis in 2% agaroseg in TBE buffer (0.045 M Tris borate, 0.001 M EDTA) Gels were blotted onto a nylon membrane, denatured, and cross-linked

by UV light The hybridization reaction used a 30-base probe with the following sequence: ACCTAAACCGAAGCAGGT-TACCAAGGAAGC The probe was end labeled with 32P using T4 polynucleotide kinase.h Unincorporated label was removed by chromatography using a Sephadex G-50 spin column Prehybridization was done in 5 x SSPE-1 x BP (2% bovine serum albumin, 2% polyvinylpyrrolidone)-1%

sodi-um dodecyl sulfate (SDS) for 30 min at 55 C The hybrid-ization was performed using the same buffer with addition

of at least 4 x 106 cpm probe/ml for 0.5 hr at 55 C The nylon filter was washed in 1% SSPE-1% SDS 3 times for 5 min at room temperature and 1 time for 1 min at 55 C The filter was dried, exposed to radiographic film, and developed Analysis of the amplified product from primer set 2 was performed using a probe made from a 365-base pair segment

of the BVDV-Singer genome’ and cloned into the pBluescript

II KS +/- vectorj The probe aligned with bases 182-545

of the BVDV NADL genome Phage T3 polymerase with

32

P-UTP was used to radiolabel the probe

Prehybridization was for at least 4 hr at 55 C in hybrid-ization buffer consisting of 6 x standard saline citrate (SSC),

2 x Denhardt’s reagent, 0.1% SDS, and 100 u/ml denatured salmon sperm Hybridization with the same buffer was done

at 55 C overnight The blots were washed twice for 5 min at room temperature in 1 x SSC and 0.1% SDS followed by 2 washes for 30 min in 0.2 x SSC and 0.1% SDS at 55 C Autoradiography was performed by exposing the blot to ra-diographic film

Results

Titration of BVDV infectivity in clinical samples.

Fifty-five of 60 tissue homogenates had sufficient quan-tity to be tested for virus concentration In 23 ho-mogenates, the concentration of BVDV (either cyto-pathic or noncytocyto-pathic) was 101.7-103.3CCID,,/ml The concentration of BVDV in 32 homogenates was

< 101.7 CCID50/ml

PCR results using primer sets 1 and 2 Using PCR

primer set 1, 46/60 homogenates (77%) were positive for BVDV (Table 2) Forty-six isolates were positive

by PCR using the UNL primers, for a detection level

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of 77% (46/60) Primer set 2 allowed detection of BVDV

in 10/11 homogenates that were negative using primer

set 1 These complementary primers boosted the

de-tection level to 93% (56/60).

Discussion

Practical application of PCR for diagnosis of BVDV

requires that it be useful for field samples such as tissue

homogenates Consideration should be given to the

sensitivity of the test and ability of the primers to

identify most, if not all, BVDV strains In this study,

PCR was evaluated for direct detection of BVDV in

tissue homogenates from field cases of BVD Using 2

sets of primers, 55/60 tissue homogenates positive for

BVDV by virus isolation were positive by PCR The

PCR procedure used here required a single

amplifi-cation cycle and used primers derived from either the

5' noncoding region or the 3' coding region of the viral

genome.

In an earlier study, a PCR technique was evaluated

on tissue samples without cell culture passage.2 These

authors tested their set of nested primers targeting the

genomic region of gp48 on 8 organ suspensions

posi-tive for BVDV by virus isolation They were able to

detect BVDV in the suspensions only after second stage

amplification by PCR.

In the present study, primer set 2 detected 3 samples

that had detectable virus titer but were negative with

primer set 1 Primer set 2 may amplify a more

con-served area of the BVDV genome The genomic

di-versity of BVDV is well documented and presents a

challenge to researchers looking for a nucleic acid-based

test that will detect all isolates 13,14,22

Further work in the development of a diagnostic

PCR procedure for detection of BVDV should focus

on the selection of a single universal set or mixture of

primers that can detect all strains of BVDV Also,

re-sitivity of PCR as a diagnostic test Routine use of PCR in a diagnostic setting will require the use of non-radioactively labeled oligonucleotide probes in con-firmatory hybridizations of amplified products.

Acknowledgements

We thank Dr E Berry (North Dakota State University, Fargo, ND) for sequence information and M Palmer and D Reichenbach for their excellent technical assistance The use

of a particular manufacturer’s product does not constitute

an endorsement on behalf of the USDA

1

2

3

4

5

6

7

8

9

verse transcriptases that act as DNA polymerases dur-

ing both reverse transcription and PCR can streamline

the procedure by combining the 2 reactions in 1 tube.18 10.

Preventing the RNA destroying activity of RNases by

addition of RNAase inhibitor in sample preparation 11.

could also be an important step in increasing the

sen-Sources and manufacturers

National Veterinary Services Laboratories, Ames, IA

Genosys, The Woodlands, TX

Pharmacia, Alameda, CA

Life Sciences, St Petersburg, FL

Promega, Madison, WI

Perkin-Elmer, Norwalk, CT

FMC, Rockland, ME

Biolabs, Beverly, MA

Dr E Berry, North Dakota State University, Fargo, ND Stratagene, La Jolla, CA

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