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Acid-base disturbances in patients with chronic kidney disease stage 4, 5

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To investigate rate, characteristics of acid-base disturbances in patients with chronic kidney disease stage 4, 5 who were diagnosed for the first time. Subjects and methods: 182 patients with chronic kidney disease stage 4, 5 due to some causes without alkalization or replacement therapy within a month. Assessment of acid-base disturbances based on results of arterial blood gas test according to the criteria of Berend K, which include pH, HCO3 - and PaCO2. Additionally, metabolic acidosis was identified when HCO3 - level < 22 mmol/L and renal tubular acidosis was diagnosed according to the criteria of Yaxlay J (2016) based on urinary pH and HCO3 - . Results: 140 patients (76.9%) had acid-base disturbances with all types according to Berend K, including: Metabolic acidosis (79.3%); respiratory acidosis (3.6%); metabolic alkalosis (1.41%); respiratory alkalosis (13.6%) and mixed acid-base disturbances (2.1%). There were 152 patients (83.5%) with HCO3 - level < 22 mmol/L, in which HCO3 - level decreased mildly (71.1%); moderately (26.3%) and severely (2.6%).

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ACID-BASE DISTURBANCES IN PATIENTS WITH CHRONIC KIDNEY DISEASE STAGE 4, 5

Nguyen Duc Phat 1 ; Hoang Trung Vinh 2 ; Pham Van Tran 2

SUMMARY

Objectives: To investigate rate, characteristics of acid-base disturbances in patients with chronic kidney disease stage 4, 5 who were diagnosed for the first time Subjects and methods:

182 patients with chronic kidney disease stage 4, 5 due to some causes without alkalization or replacement therapy within a month Assessment of acid-base disturbances based on results of arterial blood gas test according to the criteria of Berend K, which include pH, HCO 3 - and PaCO 2 Additionally, metabolic acidosis was identified when HCO 3

-

level < 22 mmol/L and renal tubular acidosis was diagnosed according to the criteria of Yaxlay J (2016) based on urinary pH and HCO 3

- Results: 140 patients (76-.9%) had acid-base disturbances with all types according

to Berend K, including: Metabolic acidosis (79.3%); respiratory acidosis (3.6%); metabolic alkalosis (1.41%); respiratory alkalosis (13.6%) and mixed acid-base disturbances (2.1%) There were 152 patients (83.5%) with HCO 3

-

level < 22 mmol/L, in which HCO 3

-

level decreased mildly (71.1%); moderately (26.3%) and severely (2.6%) Type 2 of renal tubular acidosis accounted for the highest rate (48.7%), while type 1 accounted for the lowest rate (2.6%) Conclusions: Patients with chronic kidney disease stage 4, 5 had occurence of all types of acid-base disturbances, in which metabolic acidosis accounted for the highest rate Renal tubular

acidosis was also seen in all types, in which type 2 accounted for the highest rate

* Keywords: Chronic kidney disease; Acid-base disturbances; Metabolic acidosis; Renal tubular acidosis

INTRODUCTION

Chronic kidney disease (CKD) is

increasing in all countries, due to many

different causes and causes severe

consequences for patients and society

CKD damages many organs, causing many

disorders of metabolism - endocrine

Disturbances of acid-base balance in

general and metabolic acidosis in particular

are common manifestations, especially

in the late stage of the disease, affect

progression of disease and life of patients

Metabolic acidosis usually occurs when the glomerular filtration rate is less than

25 - 30 mL/min, equivalent to stage 4, 5

of CKD However, it also depends on many factors and is individualized Disturbances of acid-base balance are defined by arterial blood gas Although the concept of disturbances of acid-base balance was proposed by Arrhenius in

1880, so far knowlege of manifestations, causes and progress have remained unclear, incomprehensible and controversial

1 Kiengiang Department of Health

2 103 Military Hospital

Corresponding author: Hoang Trung Vinh (hoangvinh.hvqy@gmail.com)

Date accepted: 14/11/2018

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Identifying the manifestations of disturbances

of acid-base balance corresponding to

clinical types of disorder as well as

evaluating degree of the disturbances

provide basis of appropriating diagnosis,

treatment and prognosis Therefore, we

conducted this study for purpose: To

investigate rate, characteristics of

acid-base disturbances in patients with CKD

stage 4, 5 who were diagnosed for the

first time

SUBJECTS AND METHODS

1 Subjects

182 patients with CKD stage 4, 5 were

enrolled in our study

* Selected criteria:

+ Causes of CKD: Hypertension,

diabetes mellitus, chronic glomerulonephritis,

chronic pyelonephritis, polycystic kidney

disease

+ Diagnosed with CKD for the first

time

+ Agree with enrolling in the study

* Excluded criteria:

+ Having any combined diseases or acute complications

+ Having combined chronic diseases

or severe complications such as heart failure grade 3, 4; cirrhosis, bronchial asthma, chronic obstructive pulmonary disease, respiratory failure

+ Acute renal failure

2 Methods

* Study design: Prospective, descriptive,

cross-sectional

* Study contents:

+ Ask for history of the disease and clinical manifestations

+ Examine the organs or parts of the body

+ Perform biochemistry test and arteria blood gas test

* Criteria for diagnosis and classifications: Table1: Categories of CKD according to KDIGO (2012)

diseases and risk factors, slow the progression of kidney disease

2

Kidney damage with

Control risk factors and combined diseases, slow the progression of kidney disease

Diagnose and treat complications caused by kidney disease

treatments

(or hemodialysis)

Obligatory substitution therapy (if there is hyperuricemia syndrome)

(Source: KDIGO (2012): Definition and classification of CKD)

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Table 2: Reference values of arterial blood gas indice tested on the GEM

Prernier 3,000

Table 3: Classification of acid-base disturbances by Berend K

Mixed acid-base disturbances when:

Table 4: Classification of renal tubular acidosis

decreased

Normal or mildly decreased

Normal or mildly to

Primary

defective

position

Impaired

the distal tubule

Reabsorbtion of

proximal tubule

Both proximal and distal tubule injuried

Defects in the ability to produce and secrete amoni ion

* Statistical analysis:

+ SPSS software version 17.0 was used to analyse data

+ Research contents do not violate medical ethics

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RESULTS

* Distribution of patients by age group

(n = 182):

≤ 40 years old: 19 patients (10.4%);

41 - 50 years old: 21 patients (11.5%);

51 - 60 years old: 37 patients (20.3%);

61 - 70 years old: 61 patients (33.5%);

> 70 years old: 44 patients (24.2%)

* Distribution of patients by CKD causes

(n = 182):

68 patients (37.4%) had hypertension,

16 patients (8.8%) had diabetes mellitus,

59 patients (32.4%) had hypertension

combined to diabetes mellitus, and

39 patients (21.4%) had primary structural

kidney diseases (primary structural kidney

diseases comprise chronic glomerulonephritis,

chronic pyelonephritis, polycystic kidney

disease) Majority of the patients was

hypertensive and/or diabetic patients

Table 5: Rates of acid-base disorders

and types of disorders

(n)

Percentage (%)

Non acid-base

disorders

Rate of acid-base disorders was higher

than rate of non disorders Simple

acid-base disorders accounted for higher rate

compared to mixed disorders

* Distribution of patients with acid-base

disorders by type of disorders (n = 140):

111 patients (79.3%) had metabolic acidosis; 5 patients (3.6%) had respiratory acidosis; 2 patients (1.4%) had metabolic alkalosis; 19 patients (13.6%) had respiratory alkalosis; 3 patients (2.1%) had mixed acid-base disorders Metabolic acidosis accounted for the highest rate

Metabolic alkalosis accounted for the lowest rate

Table 6: Rate and severity of decreased

HCO3- level

Mild decrease (15 - < 22)

Moderate decrease (10 - 14.9)

Severe decrease (< 10)

+ Rate of decreased HCO3- level was higher than that of normal HCO3- level

+ Rate of mildly decreased HCO3- level was the highest, rate of severely decreased HCO3- level was the lowest

* Distribution of patients by types of renal tubular acidosis (n = 152):

Type 1: 4 patients (2.6%); type 2:

74 patients (48.7%); type 3: 34 patients (22.4%); type 4: 40 patients (26.3%)

Type 2 of renal tubular acidosis accounted for the highest rate, type 1 accounted for the lowest rate

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DISCUSSION

1 General characteristics of patients

Two factors are ages of patients and

CKD causes that can impact on the

appearance and severity of acid-base

disturbances Ages of patients ranged

from 28 to 79, in which age group of 61 -

70 accounted for the highest rate and age

group of ≤ 40 accounted for the lowest

rate Age may be a factor related to

occurrence and severity of acid-base

disorders In older patients, acid-base

disturbances seem to occur more frequent

and be at more severe degree Age is

related to the causes of CKD, in which

hypertension and diabetes mellitus are the

two diseases occuring with the highest rate,

they can appear alone or in combination

In our study, hypertension was found in

127 cases, accounting for 69.8%, while

diabetes was found in 75 cases, accounting

for 41.2% Primary renal structural diseases,

including chronic glomerulonephritis, chronic

pyelonephritis, polycystic kidney disease

were seen with the lower rate (21.4%)

2 Acid-base disorders in patients with CKD stage 4, 5

According to the classification of Berend K, our study indicated that 76.9%

of patients had acid-base disorders, in which majority was simple acid-base disorders (97.7%) Among patients with base imbalance, all types of acid-base disturbances were seen, however metabolic acidosis was reported with the highest rate (79.3%), metabolic alkalosis accounted for the lowest rate (1.4%) Respiratory alkalosis and respiratory acidosis were found in 13.6% and 3.6% of patients, respectively There were

3 patients (2.1%) who had mixed acid-base disturbances Our results showed that all types of acid-base disturbances can be seen in patients with CKD stage 4,

5 due to some causes, in which metabolic acidosis was the most popular rate, some forms of all-cause alkalosis, although metabolic acidosis is the most common Our results were similar to those of many authors reported

Table 7: Rate of metabolic acidosis by some authors

acidosis

Our study (2018)

CKD stage 4, 5 without alkalization or renal replacement therapy

accounted for 71.1%

Vu Thi Thu Huong (2014)

[1]

CKD with all stages without renal replacement therapy

42.1% matched criteria for metabolic acidosis, 60.3% of patients had

Stage 1: 1.1%; stage 2: 2.7%; stage 3a:

27.9%; stage 3b: 19.2%; stage 4: 9.4%;

stage 5: 1.4%

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Costa de Oliveira (2015) [5]

End stage renal disease (ESRD) treated by maintenance hemodialysis

94.7%, in which 10.3% of patients had

-: 20.18 ± 4.93 mmol/L

hemodialysis

< 22 mmol/L

If diagnosis of metabolic acidosis only

based on HCO3- level < 22 mmol/L, our

study showed 152 cases (83.5%) having

metabolic acidosis, which was higher than

that based on the criteria of Berend K,

which simultaneously based on both pH

and HCO3- In fact, the rate of metabolic

acidosis is even higher Majority of

authors also identified metabolic acidosis

only based on HCO3- level < 22 mmol/L

[9, 10] Among patients with HCO3- level

< 22 mmol/L, 71.1% of patients with mild

decrease, 26.3% with moderate decrease

and only 2.6% with severe decrease This

classification was also used by Costa de

Oliveira C.M for evaluation [5] Patients

with metabolic acidosis were classified

into injured types related to tubular renal,

by which type 2 of renal tubular acidosis

caused by defect in proximal tubule

accounted for the highest rate (48.7%),

while type 1 caused by defect of distal

tubule accounted for the lowest proportion

(2.6%)

CONCLUSIONS

By studying 182 patients with chronic

kidney disease stage 4, 5 who were

diagnosed for the first time, we had the

following comments:

+ 76.9% of patients had acid-base disturbances, in which 97.9% were simple acid-base disorders

+ Among patients with acid-base disturbances, all types of acid-base disturbances were seen with the different rates, including: Metabolic acidosis with the highest rate (79.3%); respiratory alkalosis 13.6%; respiratory acidosis 3.6%; metabolic alkalosis 1.7% and mixed acid-base disorders 2.1%

+ Among patients with decreased HCO3- level corresponding to criteria for metabolic acidosis, patients with mild decrease accounted for the highest rate (71.1%); moderate decrease accounted for 26.3%; severe decrease accounted for the lowest rate

+ Type 2 of renal tubular acidosis was seen with the highest rate (48.7%), followed

by type 4 (26.3%); type 3 (22.4%) Type 1 was recorded with the lowest rate (2.6%)

REFERENCE

1 Vu Thi Thu Huong Survey of some

arterial blood gas parameters in patients with chronic renal failure Master Thesis in Medicine Vietnam Military Medical University 2014

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2 Kraut J.A, Madias N.E Metabolic

acidosis of CKD: An update AJKD 2016,

67 (2), pp.307-317

3 Harambat J, Kunzmann K, Azukaitis K

et al Metabolic acidosis is common and

associates with disease progression in

children with chronic kidney disease Kidney

Int 2017, 92 (6), pp.1507-1514

4 Chen W, Abramowitz M.K Treatment of

metabolic acidosis in patients with CKD Am J

Kidney Dis 2014, 63 (2), pp.311-317

5 Costa de Olivevira C.M, Lustosa C,

Cristino E.F et al Metabolic acidosis and its

association with nutritional status in hemodialysis

J Bras Nefrol 2015, 37 (4), pp.1/10-7/10

6 Sajgure A.D, Dighe T.A, Korpe J.S et al

The relationship between metabolic acidosis

and nutritional parameters in patients on

hemodialysis Indian J Nephrol 2017, 27 (3), pp.190-194

7 Liborio A.B, Leite T.T Disturbances in

acid-base balance in patients on hemodialysis Hemodialysis INTECH 2013, Chapter 10, pp.211-224

8 Sternlicht H, Melamed M.L Alkalinization

to retard progression of chronic kidney failure Nutritional Management of Renal Disease

2013, Chapter 18, pp.257-262

9 Raphael K.L Metabolic acidosis and

subclincal metabolic acidosis in CKD JASN

2018, 29 (2), pp.376-382

10 Goraya N, Simoni J, Jo C.H et al A

comparison of treating metabolic in CKD stage 4 hypertensive kidney disease with fruits and vegetables or sodium bicarbonate Clin J Am Soc Nephrol 2013, 8 (3), pp.371-381

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