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Correlation between serum HBV RNA and HBV DNA level, ALT activity in patients with chronic hepatitis B and HBV-related cirrhosis

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To investigate correlations between serum HBV RNA and HBV DNA level, ALT activity in naive patients with chronic hepatitis B and HBV-related cirrhosis. Subjects and methods: This cross-sectional study involved 39 chronic hepatitis B and 19 cirrhosis patients, aged 19 - 78 years. Serum samples at admission were used to quantify HBV RNA, HBV DNA level and ALT activity. Spearman’s correlation analysis was used for correlations of HBV RNA and HBV DNA level, ALT activity. For HBV RNA quantification, a specific real-time PCR technique was used with a lower limit of detection of 100 copies/mL. Results: The HBV RNA and HBV DNA detection rate was 61.54% and 100% in chronic hepatitis B patients and 42.11% and 89.47% in cirrhosis cases, respectively. HBV RNA level and ALT activity in chronic hepatitis B patients were significantly higher than in cirrhosis cases (6.29 ± 1.42 log10 copies/mL vs 5.1 ± 1.58 log10 copies/mL, p < 0.05 and 571.04 ± 574.94 U/l vs. 88.92 ± 155.06 U/l, p < 0.001, respectively). No significant HBV DNA level difference was found between those.

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CORRELATION BETWEEN SERUM HBV RNA AND HBV DNA LEVEL,

ALT ACTIVITY IN PATIENTS WITH CHRONIC HEPATITIS B AND

HBV-RELATED CIRRHOSIS

Nguyen Van Dien 1 ; Ho Huu Tho 2 ; Hoang Vu Hung 1

SUMMARY

Objectives: To investigate correlations between serum HBV RNA and HBV DNA level, ALT activity in naive patients with chronic hepatitis B and HBV-related cirrhosis Subjects and

methods: This cross-sectional study involved 39 chronic hepatitis B and 19 cirrhosis patients,

aged 19 - 78 years Serum samples at admission were used to quantify HBV RNA, HBV DNA

level and ALT activity Spearman’s correlation analysis was used for correlations of HBV RNA

and HBV DNA level, ALT activity For HBV RNA quantification, a specific real-time PCR

technique was used with a lower limit of detection of 100 copies/mL Results: The HBV RNA

and HBV DNA detection rate was 61.54% and 100% in chronic hepatitis B patients and 42.11%

and 89.47% in cirrhosis cases, respectively HBV RNA level and ALT activity in chronic hepatitis

B patients were significantly higher than in cirrhosis cases (6.29 ± 1.42 log 10 copies/mL vs 5.1 ± 1.58 log 10 copies/mL, p < 0.05 and 571.04 ± 574.94 U/l vs 88.92 ± 155.06 U/l, p < 0.001,

respectively) No significant HBV DNA level difference was found between those There was a

significant positive correlation between HBV RNA and HBV DNA level in chronic hepatitis B (r = 0.78; p < 0.001) and HBeAg (+), chronic hepatitis B (r = 0.81; p < 0.01) groups but not in

cirrhosis patients HBV RNA level significantly correlated with ALT activity in cirrhosis (r = 0.91;

p < 0.005) and HBeAg (+) CHB (r = -0.76; p < 0.001) patients Conclusion: HBV RNA level

correlated with HBV DNA level in chronic hepatitis B and HBeAg (+), chronic hepatitis B

patients, with ALT activity in cirrhosis and HBeAg (+), chronic hepatitis B patients

* Keywords: Chronic hepatitis B; Cirrhosis; Serum HBV RNA; HBV DNA; ALT

INTRODUCTION

Hepatitis B virus (HBV) infection remains

a global public health problem Worldwide,

an estimated 2 billion persons have been

infected with HBV, more than 240 million

persons have chronic infections, and

500,000 - 700,000 persons died as a

result of HBV infection [10] In Vietnam,

the prevalence of HBsAg (hepatitis B

surface antigen) in healthy subjects

ranges from 5.7 - 24.7% depending on

with HBV infection has remarkably improved with the advent of oral nucleos(t)ide analogues (NA)

It has been proposed that covalently closed circular (ccc) DNA level in the liver

is much more valuable in monitoring therapy compared to serum HBV DNA - one of most widely used marker in clinical practice

1 103 Military Hospital

2 Vietnam Military Medical University

Corresponding author: Nguyen Van Dien (nguyenvandien8290@gmail.com)

Date received: 20/12/2018

Date accepted: 18/01/2019

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However, intra-hepatic cccDNA

measurement seems ill-suited for clinical

use because it requires a liver biopsy

Recently, it is confirmed that HBV pgRNA,

an intermediate of HBV replication cycle,

is present in hepatitis B patients‟ sera It

gradually decreases during treatment but

is still detected over an extended period,

even undetectable serum HBV DNA level

goal is achieved, especially with NA drugs

[9] Recent studies have shown clinical

significance of serum HBV RNA that

includes: Estimate levels of cccDNA in the

liver, reflect the antiviral potency of NA

drugs; independently predict response to

treatment; guide NA discontinuation; and

monitor the emergence of viral mutation

during antiviral treatment Although the

clinical significance and nature of serum

HBV RNA have been intensively

investigated, little is known about the

factors influencing its level Additionally,

in Vietnam, quantification of serum HBV

RNA still has not been widely performed

In this study, we measured serum HBV

RNA and analyzed its characteristics in

CHB and HBV-related cirrhosis patients

Furthermore, serum HBV RNA level was

compared to serum HBV DNA and ALT

levels to clarify whether there exist any

clinical significance correlations between

them in these patients

SUBJECTS AND METHODS

1 Patients and samples

In this study, 58 adult treatment - naive

patients (39 chronic hepatitis B [CHB] and

19 HBV-related cirrhosis), who admitted

to Department of Infectious Diseases and

Department of Gastrointestinal Diseases,

103 Military Hospital, from 9 - 2016 to

11 - 2017, were recruited The diagnosis criteria were according to the guideline of American Association for the Study of

CHB patients and Bacon BR.„s criteria (Harrison's, 2008) [3] for cirrhosis patients

This study was approved by the Ethics Committee of Vietnam Military Medical University Written informed consent was obtained from each patient

Serum samples were collected at the admission, preserved in heparin-coated tubes and EDTA coated tubes depending on types of assays Samples for HBV RNA quantification were stored at -800C for further examination

2 Methods

* Quantification of HBV RNA:

This assay was performed at Department of Genomics, Institute of Biomedicine & Pharmacy, Vietnam Military Medical University using a protocol optimized from that of Jing Wang

et al (2016) [9] Extracted RNA (using

Bio-tek, Inc., USA) was used to synthesize cDNA, then quantified by real-time PCR (Rotor Gene™ Q,QIAGEN, Germany) in

20 µL reactions, including 3 µL of cDNA solution The experiments were performed using the protocol of 1 cycle at 95°C for

5 minutes, 45 cycles at 95°C for

30 seconds, 60°C for 15 seconds, and 72°C for 30 seconds The lower detection limit for the HBV RNA assay was determined as 100 copies/mL

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* Other laboratory tests:

HBsAg, HBV-e antigen, HBV-e antibody,

anti-HBc antibody, and hepatitis C virus

antibody were measured by Elecsys

HBsAg, Elecsys HBeAg, Elecsys

anti-HBe, Elecsys anti-HBs, Elecsys

anti-HBc - Roche kit, respectively that were

based on electrochemiluminescence

immunoassay principle

Serum HBV DNA level was extracted

from samples using E.Z.N.A.® Blood DNA

quantified by using real-time polymerase

chain reaction (PCR) technique with a 20 μL

volume on the Rotor Gene™ Q (QIAGEN, Germany) The limit of detection was

100 copies/mL

* Statistical analysis:

HBV nucleic acids were logarithmically transformed (log10) for analysis, and data are expressed as mean or median and range and percentage SPSS 22.0 software

for Window with student‟s t-test, Chi-square

tests or Fisher‟s exact tests, and Spearman‟s correlation test were used for statistical analysis as appropriate A p-value < 0.05 was considered to be statistically significant

RESULTS

1 Overall characteristics of study population

Table 1:

(A) CHB patients

(B) HBeAg (+), CHB patients

(C) HBeAg (-), CHB patients

(D) Cirrhosis patients

p value (A) vs (D)

p value (B) vs (C)

Age (year),

mean (range)

37.53 ± 12.47 (19 - 71)

31.94 ± 8.94 (20 - 58)

42.14 ± 13.51 (19 - 71)

56.90 ± 11.63 (33 - 78) < 0.001 < 0.01 Gender,

ALT (U/l) 571.04 ± 574.94

(55.4 - 2547)

627.11 ± 695.97 (104.8 - 2574)

522.98 ± 459.45 (55.4 - 1851.15)

88.92 ± 155.06 (19.24 - 723.2) < 0.001 > 0.05 HBV DNA

HBV DNA (log 10

copies/mL)

6.66 ± 1.76 (2.15 - 9.18)

7.44 ± 1.29 (5.09 - 9.18)

5.99 ± 1.86 (2.15 -8.08)

6.37 ± 2.35 (2.10 - 9.45) > 0.05 < 0.01

Cirrhosis patients were generally older than CHB patients Among CHB group , the mean age of HBeAg (-) patients were lower than HBeAg (+) ones There was no statistically significant diff erence in sex and HBV DNA detectability between the two study group and HBV DNA level in CHB patients was non-significantly higher than cirrhosis ones Further analysis in CHB patients showed a higher HBV DNA level in HBeAg (+) patients in comparison to HBeAg (-) subjects ALT activity was shown to be significantly higher in CHB patients compared to cirrhosis ones but not different among CHB patients with different HBeAg status

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2 Serum HBV RNA level in participants

A B

6.29

5.10

0

2

4

6

8

Serum HBV RNA level

Cirrhosis CHB

(n = 8) (n = 24)

5.62

HBV RNA

log10copies/ml

HBeAg (+) HBeAg (-)

Figure 1: Comparison of HBV RNA level among groups of patients

Comparison of HBV RNA level between CHB and cirrhosis patients (A); comparison

of HBV RNA level between HBeAg (+), CHB patients and HBeAg (-), CHB patients (B) HBV RNA was detected in 24/39 CHB (61.54%) and 08/19 cirrhosis patients (42.11%)

As shown in fig 1A, level of HBV RNA in CHB patients was significantly higher than in cirrhosis ones (p < 0.05) Among CHB patients, in relation to HBeAg status, HBV RNA was detected in the sera of CHB patients at the rate of 12/21 and 12/18 in HBeAg (-) and HBeAg (+) subjects, respectively (p > 0.05) The average level of HBV RNA of CHB patients with HBeAg (+) was observed to be significantly higher than of CHB

patients with HBeAg (-) (p < 0.05) (fig 1B)

3 Correlations between HBV RNA and HBV DNA level

3.50

5.50

7.50

9.50

HBV RNA

5.00 6.00 7.00 8.00 9.00 10.00

HBV RNA

5.00

6.00

7.00

8.00

9.00

10.00

4.00 5.00 6.00 7.00 8.00

HBV RNA

5.00 6.00 7.00 8.00 9.00

3.00 5.00 7.00 9.00

HBV RNA

Figure 2: Correlations between HBV RNA and HBV DNA level

(A: CHB patients; B: Cirrhosis patients; C: HBeAg (+), CHB patients; D: HBeAg (-), CHB patients HBV DNA (log 10 copies/mL); HBV RNA (log 10 copies/mL)

p < 0.05 Log 10 copies/mL p < 0.05

HBeAg (+) HbeAg (-)

n = 22 n = 12 Log 10 copies/mL

6.95 5.62

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In correlations analysis, we excluded patients with HBV RNA level under the limit of

detection In analyzing the entire study cohort, while the level of HBV RNA was

highlighted to be strongly correlated with that of HBV DNA in CHB patients (r = 0.78;

p < 0.001), there was no statistically significant relationship between them in cirrhosis

patients (r = 0.64; p > 0.05) (fig 2A & 2B) The HBV RNA - HBV DNA correlation in

CHB patients was then further analyzed and interestingly found that the positive

correlation between them was confirmed in the group of HbeAg (+) patients (r = 0.81;

p < 0.01) whereas no significant correlations was found in the others (r = 0.06; p > 0.05)

(fig 2C & 2D)

4 Correlations between HBV RNA level and ALT activity

A: r = -0.17; p > 0.05; n = 24 B: r = 0.91; p < 0.05; n = 8

0 500

1000

1500

2000

2500

3000

2.50 4.50 6.50 8.50

HBV RNA

20 40 60 80 100

2.00 4.00HBV RNA6.00 8.00

0 500

1000

1500

2000

2500

3000

4.00 5.00 6.00 7.00 8.00

HBV RNA

0 200 400 600 800 1000 1200 1400

2.50 4.50HBV RNA6.50 8.50

Figure 3: Correlations between HBV-RNA level and ALT activity

(A: CHB patients; B: Cirrhosis patients; C: HBeAg (+), CHB patients; D: HBeAg (-),

CHB patients HBV RNA (log 10 copies/mL); ALT (U/l)

In order to determine whether HBV

RNA level is linked to liver, we also tested

for associations between serum HBV RNA

level and liver injury in study population

In CHB patients, there was no statistically

significant relationship between ALT

activity and HBV RNA level (r = -0.17;

p > 0.05) (fig 3A) However, when stratifying

the patients according to HBeAg status,

a strongly negative correlation in the HBeAg (+), CHB subjects between them (r = -0.75; p < 0.01) (Spearman‟s correlation analysis) were observed

(fig 3C) We did not find any significant

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correlations between HBV RNA levels

and ALT activity (r = 0.152; p > 0.05)

in HBeAg (-), CHB patients (fig 3D)

Our analysis also highlighted a strong

and positive correlation between HBV

RNA level and ALT activity in the group of

patients with cirrhosis (r = 0.91; p < 0.05)

(8 patients) (fig 3D)

DISCUSSION

In this study, 61.54% (24/39) and

42.11% (08/19) detection rate for HBV

RNA were found in CHB and cirrhosis

patients, respectively HBV RNA detectability

in our study was higher than that in the

report by Huang et al (2015) (21/52 =

40.38% of the untreated CHB patients

with 46.5% of patients with HBeAg (+))

[4], but lower than in Yutang Li‟s study

(412/483 = 85.3% patients [5] This

difference may be attributed to the

differences between study populations

The patients in the current study had a

significantly lower HBeAg positive ratio

(18/39 = 46.15%) compared with their

patients (426/483 = 88.2%) Our findings

clearly showed that HBV RNA level was

significantly higher in CHB compared to

cirrhosis patients as well as in CHB

subjects with HBeAg (+) compared to

HBeAg (-) CHB ones This is in good

agreement with findings demonstrated in

other studies in CHB patients [8] There

was no significant difference between

CHB and cirrhosis patients in both HBV

DNA detectability and level However,

among CHB patients, we found a

significantly higher HBV DNA level in

subjects with HBeAg (+) compared to

others In addition, CHB patients had a

significantly higher ALT activity than cirrhosis ones Similar observations were also highlighted in previous study [1]

Interestingly, HBV RNA level strongly, positively correlated with HBV DNA level

in CHB patients and HbeAg (+), CHB patients Among CHB patients, while no significant correlations was revealed between HBV RNA level and ALT activity

in CHB patients and HBeAg (-), CHB patients, HBV RNA level was identified to strongly, negatively correlated with ALT activity in HBeAg (+) patients A non-significant correlation between HBV RNA and HBV DNA level and a strongly significant, positive correlation between HBV RNA and ALT activity were highlighted

in patients with cirrhosis

These were in line with previous studies conducted in CHB patients A strongly, positive correlation between HBV RNA and HBV DNA level were revealed by Akinori Rokuhara (2006) (r = 0.801;

p < 0.001 [7] and van Bommel F (2015) (r = 0.59; p < 0.001) [8] Among HBeAg (+) CHB patients, the relationship between HBV RNA and HBV DNA was also indicated by van Bommel F (2015) [8] The above results suggested that as an intermediate in HBV replication cycle, HBV pgRNA-containing virion is also produced and released into sera along with HBV DNA-containing virion (Dane particles) at a certain rate compared to Dane particles

In term of HBV RNA level - ALT activity correlation, in the same line, van Bommel

F (2015) revealed that HBV RNA level did not correlate with serum ALT activity

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(r = 0.01) [8] This apparent lack of correlation

between HBV RNA level and ALT activity

in CHB patients could be accounted for

the pathophysiology of chronic HBV

infection In immune clearance phase

(the predominance of our participants is

patients with exacerbation of CHB), while

HBV replication is partially suppressed,

infected hepatocytes is thought to be

damaged by the immune-mediated lysis

mechanism resulting into ALT elevation

However, that phenomenon differs among

patients depending on various factors

including HBeAg status In HBeAg positive

CHB patients, infected hepatocytes lysis

caused by sufficient immune clearance

results into ALT elevation and a decrease

in the level of viral markers, including

HBV DNA, HBV RNA, even leads to

seroconversion of some of them

Therefore, HBV RNA has a tendency to

negatively correlate with ALT activity

Further analysis in patients with HBeAg

negative CHB, it is thought that there is

an insufficient immune against HBV that

is characterized by ALT elevation without

decreasing in serum viral markers

Regarding cirrhosis ones, little is known

about serum HBV RNA to date

Our findings provide clues for further

investigation into the roles of serum HBV

RNA in natural history of HBV infection

and HBV infection management The

difference in serum HBV RNA level

between cirrhosis and CHB patients

suggests that it could help to have a more

precise view in the nature of viral

replication and pathogenesis of disease

among different phases of chronic HBV

infection Additionally, significant correlations found between serum HBV RNA and HBV DNA level, ALT activity indicate that serum HBV RNA could be a promising biomarker in HBV infection evaluation and management

This is the first publication on serum HBV RNA in Vietnam, however, the findings must

be interpreted in the context of a number of potential limitations Firstly, this is a cross-sectional study assessed data at one-time point of admission only Thus, it is difficult to establish a causal relationship between serum HBV RNA and liver histological changes, HBV DNA dynamics because it requires longitudinal observations Secondly, this study investigated a quite small sized sample which may have reduced the statistical power

CONCLUSION

This study demonstrates significant differences in baseline serum HBV RNA level between CHB and cirrhosis patients Serum HBV RNA level correlated with HBV DNA level in CHB and HBeAg (+) CHB patients, and ALT activity in patients with cirrhosis and HBeAg (+) CHB Serum HBV-RNA may help clinicians in managing persistent HBV infection

Acknowledgments

We thank all study participants for their donation of blood samples This work was supported by Department of Infectious diseases, 103 Military Hospital and Department of Genomics, Institute of Biomedicine and Pharmacy of Vietnam Military Medical University

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hepatitis B virus RNA is encapsidated pregenome RNA that may be associated with persistence of viral infection and rebound

J Hepatol 2016, 65 (4), pp.700-710

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Prevention and control of viral hepatitis infection: Framework for global action WHO, Geneva 2012

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