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The posterior cuff muscles provide dynamic posterior gleno-humeral stability, with the supraspina-tus functioning as a powerful abduc-tor of the arm in the scapular plane and the infrasp

Trang 1

Skeletally Immature Athlete

Frank S Chen, MD, Veronica A Diaz, MD, Mark Loebenberg, MD, and Jeffrey E Rosen, MD

Abstract

Shoulder and elbow injuries in the

skel-etally immature are becoming more

frequent as more children and

ado-lescents participate in recreational and

competitive athletics requiring

repet-itive overhead motion Although most

of these injuries result from chronic

overuse, traumatic injuries to the

shoul-der and elbow also occur The injury

patterns in these patients are distinct

because their developing physes are

relatively weak Whereas injuries in

adults tend to involve ligamentous and

soft-tissue structures, injuries in the

skeletally immature commonly involve

the physes as well

Shoulder and Elbow

Anatomy

To manage these injuries effectively,

clinicians should understand

func-tional shoulder and elbow anatomy

in children and adolescents, as well

as the normal developmental

se-quence of the primary and secondary ossification centers, which represent potential sites of injury

Shoulder

The proximal humeral physis, which has formidable growth and re-modeling potential,1,2contributes ap-proximately 80% of the longitudinal growth of the upper extremity It is composed of three primary ossifica-tion centers—the humeral head, the greater tuberosity, and the lesser tu-berosity—that coalesce between the ages of 5 and 7 years to form a sin-gle proximal humeral epiphysis Sub-sequently, the proximal humeral phy-sis fuses approximately between the ages of 14 and 17 years in females and

16 and 18 years in males.1,3

The capsuloligamentous and mus-cular structures of the shoulder pro-vide static and dynamic stability of the glenohumeral joint.4-6The static stabilizers function primarily at the extremes of the range of motion

(ROM) as they reciprocally tighten and loosen to limit humeral head translation The dynamic stabilizers provide stability during the midrange

of motion, when the static stabilizers are lax The dynamic stabilizers con-tract in a coordinated pattern to pro-vide concavity-compression of the humeral head within the glenoid cav-ity, limiting abnormal translation.4-6

Different portions of the joint cap-sule, glenohumeral ligaments, and gle-noid labrum provide static gleno-humeral stability, depending on the position of the arm.4,6The anterosu-perior capsule, coupled with the struc-tures of the rotator interval, limit in-ferior and posterior translation of the

Dr Chen is Attending Physician, Sports Medi-cine Department, Palo Alto Medical Foundation, Palo Alto, CA Dr Diaz is Resident, University

of Miami/Jackson Memorial Hospital, Miami, FL.

Dr Loebenberg is Consultant Surgeon, Assaf Harofeh Medical Center, Tel Aviv University School of Medicine, Tzrifin, Israel Dr Rosen is Assistant Professor, Orthopaedic Surgery, and Di-rector, Child & Adolescent Sports Medicine, Sports Medicine/Arthroscopic Surgery, NYU–Hospital for Joint Diseases, New York, NY.

None of the following authors or the departments with which they are affiliated has received anything

of value from or owns stock in a commercial com-pany or institution related directly or indirectly

to the subject of this article: Dr Chen, Dr Diaz,

Dr Loebenberg, and Dr Rosen.

Reprint requests: Dr Rosen, NYU–Hospital for Joint Diseases, Suite 2, 305 Second Avenue, New York, NY 10003.

Copyright 2005 by the American Academy of Orthopaedic Surgeons.

The intensity of training and competition among young athletes can place them at

increased risk of acute and chronic injuries, which occur in patterns unique to the

skeletally immature athlete Prompt recognition and treatment of these injuries are

critical to prevent long-term functional disability and deformity Children and

ad-olescents participating in recreational and organized sports are particularly

suscep-tible to a broad spectrum of shoulder and elbow injuries involving both osseous and

soft-tissue structures Understanding the relevant functional anatomy,

biomechan-ics of throwing, and pathophysiology of injury can help the clinician manage

com-mon acute traumatic injuries, some of which may result in chronic problems

Over-use injuries occur more frequently than do acute, traumatic injuries, and early

recognition, coupled with appropriate treatment or prevention, can help restore and

maintain normal shoulder and elbow function.

J Am Acad Orthop Surg 2005;13:172-185

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humeral head in the adducted arm.4,7

The middle glenohumeral ligament

functions to limit anteroposterior (AP)

translation in the abducted arm at the

midrange of external rotation The

in-ferior glenohumeral ligament, a

com-plex structure possessing anterior and

posterior bands with an interposing

axillary pouch, serves as the

prima-ry restraint to AP as well as to

infe-rior translation in the abducted and

maximally externally rotated arm.4,7

The posterior capsule, which does not

have any direct posterior ligamentous

reinforcements, is important in

lim-iting posterior humeral translation in

the adducted, internally rotated, and

forward-flexed arm.4,5Overall static

stability is further enhanced by the

la-brum, which deepens the concavity

of the glenoid socket in both the AP

and superior-inferior dimensions The

labrum also acts as an anchor point

for the capsuloligamentous structures

and the long head of the biceps

ten-don Labral injury or detachment is

commonly associated with injury to

the associated capsuloligamentous

structures.4-6

The dynamic stabilizers are the

ro-tator cuff, the long head of the biceps,

the deltoid, and the scapulothoracic

muscles The posterior cuff muscles

provide dynamic posterior

gleno-humeral stability, with the

supraspina-tus functioning as a powerful

abduc-tor of the arm in the scapular plane

and the infraspinatus and teres

mi-nor combining to externally rotate and

flex the humerus.5,6,8 The

subscapu-laris, in addition to functioning as an

internal rotator of the humerus, is an

important dynamic anterior

stabiliz-er, given its confluence with the

an-terior capsule.5,6,8The long head of the

biceps is important in preventing

an-terior and superior humeral head

translation.9Finally, the deltoid and

scapulothoracic muscles—including

the trapezius, levator scapulae,

ser-ratus anterior, and both rhomboid

muscles—function to position the

scapula to provide maximum

stabil-ity at the glenohumeral articulation.4-6

Elbow

Skeletal maturation of the elbow occurs at primary and secondary os-sification centers within the distal hu-merus, radius, and ulna Six second-ary ossification centers represent potential sites of injury (1) The capi-tellum has a variable ossification pat-tern but usually appears by age 2 years in males (2) Ossification of the radial head occurs next, between age

4 and 5 years, followed by (3) the me-dial epicondyle between age 6 and 7 years and (4) the trochlea between age

9 and 10 years (5) The olecranon ap-pears next, usually by age 11, fol-lowed by (6) the lateral epicondyle during adolescence.10,11Ossification rates are highly variable and also dif-fer by sex; rates in females usually precede those of males by 6 to 12 months Therefore, clinicians should obtain radiographs of the contralat-eral side to evaluate elbow injuries in the skeletally immature patient

The osseous anatomy of the el-bow allows flexion-extension and pronation-supination through the ulnohumeral and proximal radioul-nar articulations, respectively In full extension, the elbow possesses a normal valgus-carrying angle of 11°

to 16° This bony configuration pro-vides approximately 50% of the overall stability of the elbow, primar-ily against varus stress in the ex-tended elbow The anterior joint cap-sule, the ulnar collateral ligament (UCL) complex, and the lateral col-lateral ligament complex provide the remainder of elbow stability.12,13

The UCL complex consists of three main portions: the anterior bundle, posterior bundle, and oblique bundle (transverse ligament) The anterior bundle is functionally the most im-portant in providing stability against valgus stress and is further

subdivid-ed into distinct anterior and

posteri-or bands, possessing reciprocal functions.12-14The anterior band is the primary restraint to valgus stress at lesser degrees of flexion and is more susceptible to injury in the extended

elbow.13,14The posterior band, because

of its primary stabilizing role at

high-er degrees of elbow flexion, is func-tionally more important than the an-terior band in the overhead throwing athlete

Originating from the medial epi-condyle, the flexor-pronator muscu-lature provides dynamic valgus sta-bility of the elbow.15From proximal

to distal, this muscle mass includes the pronator teres, flexor carpi radi-alis, palmaris longus, flexor digi-torum superficialis, and flexor carpi ulnaris Electromyographic (EMG) and biomechanical studies have shown the pronator teres, flexor dig-itorum superficialis, and flexor carpi ulnaris muscles, which form muscu-lotendinous units overlying the UCL complex, to be primarily responsible for maintaining dynamic valgus sta-bility.13,15

The lateral collateral ligament com-plex is less well understood than the medial ligamentous structures It is composed of three distinct portions: the radial collateral ligament, the lat-eral UCL, and the accessory latlat-eral col-lateral ligament.12,14The lateral UCL has been shown to be the primary re-straint against rotatory subluxation of the ulnohumeral joint; injury to this structure allows posterolateral rota-tory instability to develop.14The ra-dial collateral ligament is reportedly

an important secondary restraint of the lateral elbow, along with the ex-tensor muscles, including the exten-sor digitorum communis, brachiora-dialis, and extensor carpi radialis longus and brevis.12,14These muscles impart dynamic stability to the

later-al elbow EMG studies have shown that they exhibit complex, interdepen-dent firing patterns throughout the throwing motion; thus, they may be vulnerable to overuse injuries.8,15

Biomechanics of Throwing

The overall motion and kinematics of throwing in adolescents are similar to

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those in adults, with stresses that are

similar but of lesser absolute

magni-tude.16 Proper throwing mechanics

can and should be taught at a young

age, along with strengthening of the

upper extremity, to reduce injury

rates

Although specific techniques of

overhead throwing vary with

differ-ent sports, the basic motion is

simi-lar The baseball pitch has been the

most studied and can be divided into

five main stages8,17(Fig 1) In stage

1, or windup, the elbow is flexed and

the shoulder is in slight internal

ro-tation; muscular activity is minimal

Stage 2, or early cocking, begins

when the ball leaves the

nondomi-nant gloved hand and ends when the

forward foot contacts the ground The

shoulder begins to abduct and rotate

externally This stage entails early

activation of the deltoid followed by

activation of the supraspinatus,

in-fraspinatus, and teres minor

mus-cles.5,6,8,17

Stage 3, or late cocking, is

charac-terized by further shoulder abduction

and maximal external rotation as well

as increasing elbow flexion and

fore-arm pronation Activity levels of the

supraspinatus, infraspinatus, and

te-res minor reach their peak during the

midportion of this phase, and

sub-scapularis and periscapular

muscu-lar activity increase.6,8,17Tremendous

shear forces are generated across the

anterior shoulder, predominantly by

the rotator cuff muscles.16,17The long

head of the biceps and the

subscap-ularis also contribute to dynamic

an-terior shoulder stability during late

cocking.8,9

In stage 4, or acceleration, the

shoulder musculature generates a

large forward force on the extremity,

resulting in internal rotation and

ad-duction of the humerus coupled with

rapid elbow extension.5,6,17,18Working

in concert with the periscapular

mus-cles, the subscapularis exhibits high

activity during this stage.5,6,17,18Stage

4 ends with ball release as

tremen-dous valgus stresses are generated

about the medial elbow struc-tures.13,18The anterior bundle of the UCL bears most of these forces Sec-ondary supporting structures, such as the flexor-pronator musculature, fa-cilitate transmission of these signif-icant stresses Most elbow injuries oc-cur during this stage because these forces are concentrated on the

medi-al elbow structures Bmedi-all release medi-also generates tremendous compression and rotatory stresses laterally in the radiocapitellar articulation, and pow-erful triceps contraction imparts ten-sile forces in the posterior compart-ment.13,19

In stage 5, or follow-through, all excess kinetic energy is dissipated as the upper extremity decelerates rap-idly The stage ends when all motion

is complete Forceful deceleration of the upper extremity occurs as the el-bow reaches full extension and the shoulder is maximally internally ro-tated.8,13The biceps and brachialis ex-hibit high activity levels during this phase, as does the posterior cuff mus-culature, which contracts

eccentrical-ly to stabilize the glenohumeral joint.8,9The deltoid, latissimus dorsi, and subscapularis muscles contribute

to shoulder stability and prevent

hu-meral head subluxation Tremendous torque is generated across the gleno-humeral joint as the arm rapidly de-celerates.5,18

Acute Injuries of the Shoulder and Elbow

Traumatic osseous and soft-tissue in-juries of the shoulder and elbow in the skeletally immature athlete span

a wide range of injury patterns, some

of which may lead to chronic insta-bility The most commonly observed acute injury patterns in this popula-tion are glenohumeral dislocapopula-tions and acute medial epicondylar fractures

Traumatic Glenohumeral Dislocations

Although relatively uncommon, traumatic shoulder dislocations do oc-cur, primarily during collision sports The incidence is as high as 7% in young athletes participating in ice hockey.20In addition, up to 40% of all primary shoulder dislocations occur

in patients younger than 22 years.20

AP, axillary, and lateral views of the shoulder always should be obtained because associated fractures of the

gle-Figure 1 Phases of the throwing motion in baseball (Adapted with permission from DiGiovine NM, Jobe FW, Pink M, Perry J: An electromyographic analysis of the upper

ex-tremity in pitching J Shoulder Elbow Surg 1992;1:15-25.)

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noid rim or Hill-Sachs lesions may

oc-cur Magnetic resonance imaging

(MRI), MR arthrography, or

comput-ed tomography arthrography may

demonstrate a Bankart lesion or

la-bral detachment (Fig 2) Intra-articular

contrast medium can be used to

out-line and properly visualize the labrum;

without contrast medium, these

struc-tures cannot be fully evaluated

La-bral injury or detachment usually

de-notes concomitant injury of the

associated capsuloligamentous

struc-tures, which can result in distinct

in-stability patterns, depending on the

region of capsulolabral injury.4-6,17

Recurrent instability after a

trau-matic injury in the skeletally

imma-ture patient is common; rates range

from 25% to 90% in adolescents and

up to 100% in patients with open

phy-ses.21,22Surgical intervention may be

indicated when symptoms of

insta-bility persist despite 4 to 6 months of

nonsurgical management—a brief

pe-riod of immobilization followed by

dy-namic shoulder stabilization with

del-toid, rotator cuff, and scapular muscle

strengthening Because recurrence

rates are high in this population and

because arthroscopic stabilization

tech-niques have advanced, early

stabili-zation increasingly is being

recom-mended for athletes with traumatic

instability with labral detachments or

bony Bankart lesions

Arthroscopic techniques now

ap-pear to produce functional results

comparable to those of open Bankart

or anterior capsulolabral

reconstruc-tion procedures.23-29Arthroscopy has

the potential advantages of better

vi-sualization of the capsulolabral

com-plex and other intra-articular

struc-tures, less surgical dissection (which

decreases scarring), less damage to

surrounding tissues (which

decreas-es morbidity), and earlier and more

rapid rehabilitation with improved

ROM, especially in external

rota-tion.25,26,29Arthroscopic suture anchors

can be used for labral repair, and

cap-sular pathology can be addressed

con-comitantly with suture

capsulorrha-phy to maximize functional outcome and minimize the risk of recurrence (Fig 3)

Postoperative shoulder immobili-zation is generally maintained for the first 10 to 14 days, followed by a gressive ROM and strengthening pro-gram In patients with anterior insta-bility, shoulder abduction and external rotation in the 90°–90° position over-head should be avoided in the early postoperative period Therapy is di-rected at strengthening the rotator cuff, deltoid, and scapulothoracic muscles

to provide dynamic stabilization of the shoulder These techniques and pro-tocols can achieve results compara-ble to those of traditional open sta-bilization techniques, so that the patient may be allowed to return to athletic activity at 3 to 6 months.23-29

Medial Epicondylar Fractures

Avulsion fractures of the medial epicondyle result from extreme val-gus loads or violent muscle contrac-tions during the throwing motion and commonly occur in adolescents as the medial epicondyle begins to fuse.11,21

Patients may report feeling a “pop”

or “giving way” of the elbow, followed

by acute pain; they also may describe locking or catching of the elbow Ex-amination reveals tenderness and swelling over the medial epicondyle with decreased ROM and valgus in-stability.11Plain radiography shows avulsion of the medial epicondylar apophysis with varying degrees of dis-placement, depending on the force of the trauma.10,11Type 1 fractures (a large fragment that may involve the entire epicondyle) occur in younger children, and type 2 fractures (small fragments)

in adolescents older than 15 years of age with fused physes.10,11

Treatment is guided by the extent

of fracture displacement Minimally displaced fractures are treated with immobilization for 2 to 3 weeks, fol-lowed by a rehabilitation protocol, in-cluding protected active and active-assisted ROM exercises.10,11Nonunions have been reported as a result of

in-adequate immobilization and activ-ity modification because repetitive traction from resumed throwing leads

to residual motion and stress at the fracture site, inhibiting physeal fusion Late surgical excision may be

indicat-ed for pain For patients with fractures displaced >5 mm, valgus stability should be tested clinically and, if nec-essary, should include valgus stress radiography A medial joint line open-ing >2 to 3 mm is considered abnor-mal In the presence of instability or marked rotation or displacement of the medial epicondyle fragment, sur-gical reattachment by open reduction and internal fixation (with smooth Kir-schner wires) is indicated to restore valgus stability Anatomic reduction may prevent late sequelae, such as radiocapitellar degenerative

chang-es.10,11,21

Chronic Overuse Injuries

Although skeletally immature ath-letes sustain a variety of acute shoul-der and elbow injuries, most of these are chronic overuse injuries

second-Figure 2 Anterior labral detachment Axial T2-weighted MRI scan demonstrating avul-sion of the anterior labrum (curved arrow) and the fluid between the glenoid (G) and the displaced labrum S = subscapularis tendon (Reproduced with permission from Kingston S: Diagnostic imaging of the upper

extrem-ity, in Jobe FW [ed]: Operative Techniques in

Up-per Extremity Sports Injuries St Louis, MO:

Mosby, 1996, p 46.)

Trang 5

ary to cumulative stresses from

repet-itive overhead throwing motion

Chronic injuries occur

predominant-ly in baseball players, but participants

in other sports involving similar

over-head activity, such as football, tennis,

swimming, or volleyball, also are

sus-ceptible These injuries occur in

spe-cific patterns depending on the nature

of the repetitive stresses and the

de-velopmental anatomy of the athlete

Injuries of the Shoulder

Shoulder and elbow injuries increase

in frequency during the mid to late

teenage years As the athlete matures

and gains strength, the shoulder is

subjected to greater stresses during

the throwing motion The most

com-mon overuse injuries include Little

League shoulder, rotator cuff

tendini-tis, and glenohumeral instability—

anterior, posterior, and

multidirec-tional

Little League Shoulder

Little League shoulder is

epiphysi-olysis of the proximal humerus

sec-ondary to repetitive microtrauma from

overhead activity Patients present

with diffuse shoulder pain that is

worse with throwing Arecent increase

in the throwing regimen often pre-cedes the onset of symptoms.21,30,31

Findings include tenderness and swelling over the anterolateral shoul-der, with weakness on resisted abduc-tion and internal rotaabduc-tion External ro-tation contractures with decreased internal rotation also may develop Ra-diographs usually reveal proximal physeal widening, best appreciated on

an AP view taken with the shoulder

in external rotation Depending on the severity of the condition, radiographs also may demonstrate metaphyseal demineralization and fragmentation coupled with physeal irregularity and periosteal reaction.21,30,31

Treatment involves an initial period

of 2 to 3 months of rest and activity modification, followed by a progres-sive throwing program The protocol calls for a light tossing schedule and gradually progresses with increasing distance and velocity This protocol has shown excellent results, with up

to 91% of patients remaining asymp-tomatic.21Because of the great remod-eling potential of the proximal hu-merus, long-term consequences are rare However, problems can occur and may include premature physeal clo-sure with resultant humeral length

dis-crepancy or angular deformity, as well

as subsequent Salter-Harris fractures

of the proximal humeral epiphysis Factors that contribute to the de-velopment of Little League shoulder include excessive throwing, poor technique, and muscle-tendon imbal-ance Coaches, trainers, and parents should be aware of the American Academy of Orthopaedic Surgeons (AAOS) guidelines for pitching (Ta-ble 1) Developing proper throwing mechanics and limiting the number

of pitches and innings thrown are cru-cial for preventing Little League shoulder Control, not speed, should

be emphasized in training regimens

In addition, educating coaches and players about appropriate stretching, strengthening, and conditioning and proper throwing mechanics is vital

Rotator Cuff Tendinitis and Impingement

Adolescent overhead athletes— especially those involved in baseball, swimming, and tennis—often sustain tendinitis or strains of the rotator cuff

as a result of outlet impingement, cumulative tensile overload, and instability associated with internal impingement32-35(Fig 4) Patients with rotator cuff damage usually present

Figure 3 Arthroscopic repair of a Bankart lesion with suture anchors The patient is in the lateral decubitus position A, Elevator used to free the labrum, which has healed medially on the glenoid neck B, Mobilization of the dissected labrum onto the glenoid rim C, Fixation

of the labrum to the glenoid with a suture anchor after arthroscopic knot tying.

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with anterolateral shoulder pain that

worsens with continued activity In

ad-dition, they may report mild stiffness

and weakness in the involved

extrem-ity Physical examination should

in-clude provocative impingement

ma-neuvers and testing of ROM because

active internal rotation may be present

secondary to a tight posterior

cap-sule.32,33The Neer impingement sign

is pain elicited by forcing the arm into

a position of maximal forward

eleva-tion The Hawkins impingement sign

is pain elicited by forcible internal

ro-tation with the arm forward

elevat-ed to 90°, which produces pain when

the supraspinatus tendon impinges on

the coracoacromial ligament or ante-rior acromion Pain also may be present with resisted supraspinatus testing, although significant weakness

is not typically noted unless an un-derlying tear is present

Examination for concomitant gle-nohumeral instability is important be-cause treatment must be geared to-ward all etiologic factors Standard radiographic studies, including AP, outlet, and axillary views, typically

do not show any marked osseous ab-normalities MRI is the imaging study

of choice for evaluation of rotator cuff damage Increased signal in the ten-don and inflammation in the

sub-acromial space may be noted within the insertion of the supraspinatus ten-don, in cases of tendinitis MRI also may show evidence of partial or full-thickness tears (Fig 5), although these are not commonly observed in ado-lescents.2

Initial treatment of rotator cuff in-jury is nonsurgical, consisting of rest, ice, nonsteroidal anti-inflammatory drugs (NSAIDs), and physical ther-apy The physical therapy program focuses on ROM and strengthening

of the shoulder muscles to correct un-derlying muscular imbalance and to provide dynamic glenohumeral sta-bility Proper rehabilitation is crucial not only to relieve pain and expedite return to play but also to prevent pro-gression to partial or full-thickness tears that might require surgical in-tervention Stretching is important to establish and maintain full ROM, es-pecially in patients with tight poste-rior capsules with limited internal ro-tation A strengthening program is instituted to increase strength in the rotator cuff as well as in the scapular stabilizers During the acute phase of tendinitis, exercises should be per-formed below shoulder level to avoid rotator cuff outlet impingement, with gradual progression as symptoms

de-Table 1

Pitching Recommendations for the Young Baseball Player

Age Maximum Pitches per Game Maximum Games per Week

Reproduced with permission from Pasque CB, McGinnis DW, Griffin LY: Shoulder, in

Sullivan JA, Anderson ST (eds): Care of the Young Athlete Rosemont, IL: American

Academy of Orthopaedic Surgeons, and Elk Grove Village, IL: American Academy of

Pediatrics, 2000, p 347.

Figure 4 Swimmers subject their shoulders to excessive forces during both (A) the front crawl-stroke (cuff impingement, arrows) and (B)

the backstroke (anterior capsular tension) In panel B, the arrows indicate the pull of the rotator cuff on the proximal humerus (Adapted

with permission from Wilkens KE: Shoulder injuries: Epidemiology, in Stanitski CL, DeLee JC, Drez D Jr [eds]: Pediatric and Adolescent Sports

Medicine Philadelphia, PA: WB Saunders, 1994, p 181.)

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crease Usually, nonsurgical treatment

allows gradual return to competition

For patients who do not respond to

an initial 6- to 12-week period of

mod-ified activity and physical therapy, an

MRI should be considered to

evalu-ate for partial or full-thickness tears

and other intra-articular damage

Arthroscopic treatment of rotator

cuff injury is reserved for injuries that

do not respond to nonsurgical

man-agement; results have been mixed in

young athletes with regard to pain

re-lief and return to sports Surgical

suc-cess depends on the nature of the

un-derlying rotator cuff damage as well

as any associated problems, such as

instability or labral tears.2,35It is

portant to distinguish between

im-pingement and concomitant

under-lying instability because failure to

address these subtle instability

pat-terns may compromise functional

re-sults True outlet impingement is

ex-tremely rare in adolescents, who

typically present with secondary or

internal impingement as a result of

subtle instability patterns These

pat-terns may be related to rotator cuff fa-tigue, to superior labral anterior-posterior (SLAP) lesions involving the superior biceps–labral anchor complex,

or to true instability and are asciated with articular-sided partial-thickness rotator cuff tears Arthroscopic acro-mioplasty is rarely performed alone

in this population; rather, subacromial bursectomy and débridement are usu-ally accompanied by procedures that address the associated damage (ie, débridement of partial-thickness ro-tator cuff tears and repair of SLAP/

labral lesions).36Progressive ROM and strengthening exercises may be initi-ated early after surgery As a general rule, however, arthroscopy should be

a last resort in the treatment of rota-tor cuff injuries in the adolescent ath-lete and undertaken only when spe-cific, clearly defined damage can be addressed

Anterior Glenohumeral Instability

Anterior instability usually results from chronic overload injuries in the athlete engaged in overhead sports

Excessive, repetitive external rotation during the overhead motion places tremendous stress on the anterior cap-sular and ligamentous structures, causing microtrauma that leads to lig-amentous laxity Initially, the rotator cuff and periscapular muscles com-pensate However, these dynamic stabilizers fatigue with repeated ac-tivity, and anterior glenohumeral translation ensues, with subsequent development of instability Secondary impingement of the rotator cuff an-terosuperiorly against the coracoac-romial arch during forward flexion may occur, causing tendinitis or even undersurface tears.31Furthermore, as the humeral head translates anteriorly with shoulder abduction and exter-nal rotation, interexter-nal impingement of the rotator cuff also may occur33(Fig

6) Normally, with the shoulder in the apprehension position, the distance between the rotator cuff and the pos-terosuperior glenoid rim is small As

the static stabilizers become lax and the dynamic stabilizers fatigue, in-creased anterior glenohumeral trans-lation with the arm in the apprehen-sion position pinches the cuff against the posterosuperior glenoid rim, pro-ducing internal impingement Con-comitant posterior capsular contrac-tures caused by repetitive stress may further exacerbate the impinge-ment.5,24

Athletes typically present with de-creased throwing effectiveness and pain, especially during late cocking and early acceleration They also may report a “dead arm.” On examination, load-and-shift (Fig 7) and fulcrum tests may not demonstrate anterior laxity Mild anterior apprehension with a positive relocation test may be present, indicating internal impinge-ment Loss of internal rotation also may be present, secondary to a tight posterior capsule.5,6,17Athletes with associated rotator cuff damage may have appropriate findings Usually, in the absence of a traumatic injury, plain radiography shows no signif-icant abnormalities MRI may show increased signal within the posterior cuff, consistent with fraying in cases

of internal impingement, and if MRI

is performed with contrast medium,

it may show redundancy in the an-terior capsule Usually, labral damage

is not present unless there has been

a traumatic episode Routine use of MRI for instability secondary to over-use is not needed unless the clinician suspects associated damage Treatment of anterior instability begins with an initial period of rest followed by physical therapy and a home exercise program that empha-sizes strengthening and conditioning

of the rotator cuff, deltoid, and scap-ular muscles Both concentric and ec-centric exercises are included as well

as stretching of the posterior capsule

if tightness is present Improper throwing mechanics also must be cor-rected Athletes are allowed to return gradually to throwing once stability, strength, and endurance have

im-Figure 5 Oblique coronal MRI scan of the

shoulder after intra-articular injection of

gad-olinium, demonstrating partial-thickness

un-dersurface rotator cuff tear (arrow)

(Repro-duced with permission from Kingston S:

Diagnostic imaging of the upper extremity,

in Jobe FW [ed]: Operative Techniques in

Up-per Extremity Sports Injuries St Louis, MO:

Mosby, 1996, p 35.)

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proved (usually within 3 months).

With well-supervised physical

ther-apy, most will be able to return to their

prior level of activity in 6 months.2,37

If symptoms persist despite 4 to 6

months of well-supervised

nonsurgi-cal management, surgery may be

in-dicated Arthroscopy may reveal

stretching of the inferior

glenohumer-al ligament and anterior capsule,

la-bral fraying, or undersurface cuff

tears.38,39Débridement alone of the

ro-tator cuff and posterosuperior glenoid

is inadequate to address the

under-lying pathology; either open or ar-throscopic anterior capsuloligamen-tous reconstruction is recommended for the best functional outcomes An arthroscopic anteroinferior suture capsulorrhaphy is often sufficient to address the underlying damage, and current arthroscopic techniques have results comparable to those of open stabilization.29,36Arthroscopy allows excellent visualization of the capsu-lolabral complex with minimal inva-siveness, which can decrease morbid-ity and, more important, minimize

external rotation loss

postoperative-ly, which is critical in the overhead athlete The lax inferior glenohumeral ligamentous complex and anteroin-ferior capsule are imbricated arthro-scopically and tightened using mul-tiple nonabsorbable sutures Suture anchors may be placed along the gle-noid rim to repair a labral detachment

as well as to perform capsular plication24-27(Fig 3, C)

Postoperative isometric strength-ening exercises are started early Sling immobilization may be discontinued after 10 to 14 days, followed by pro-gressive ROM and strengthening ex-ercises The deltoid, rotator cuff, and scapular muscles are targeted to pro-vide dynamic stability and restore nor-mal glenohumeral and scapulothoracic rhythm Return to full, unrestricted

Figure 6 Progression of injury in internal impingement A, The normal position of the

hu-meral head in the glenoid during abduction to 90° in the scapular plane and maximal

ex-ternal rotation B, Anterior translation (curved arrow) leads to subluxation of the humeral

head and hyperangulation C, This in turn leads to skeletal, labral, and tendinous lesions.

Inset: The posterosuperior region of the glenoid (broken line) is where impingement occurs.

(Reproduced with permission from Jobe CM, Pink MM, Jobe FW, Shaffer B: Anterior

shoul-der instability, impingement, and rotator cuff tear: Theories and concepts, in Jobe FW [ed]:

Operative Techniques in Upper Extremity Sports Injuries St Louis, MO: Mosby, 1996, p 175.)

Figure 7 Load-and-shift test for anterior in-stability of the shoulder With the patient

seat-ed, the examiner stabilizes the scapula with one hand and then applies a compressive force to the glenohumeral joint (arrows) and measures anteroposterior excursion (dotted lines) (Adapted with permission from Mc-Farland EG, Shaffer B, Glousman RE, Conway

JE, Jobe FW: Anterior shoulder instability, im-pingement, and rotator cuff tear: Clinical and

diagnostic evaluation, in Jobe FW [ed]:

Op-erative Techniques in Upper Extremity Sports In-juries St Louis, MO: Mosby, 1996, p 185.)

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activity may take up to 6 to 12 months

in the throwing athlete

Posterior Glenohumeral

Instability

Although not as common as

ante-rior pathology, posteante-rior instability is

increasing in incidence as a result of

chronic microtrauma to the posterior

structures from repetitive overhead

activity Less commonly, a single

trau-matic episode may result in

posteri-or capsular injury and subluxation,

which may be missed if lateral and

axillary radiographs are not

ob-tained.20Repetitive eccentric

contrac-tion during the deceleracontrac-tion and

follow-through stages of throwing

stretches the posterior capsule and

produces microtears within the

pos-terior cuff Together, these factors can

contribute to development of

poste-rior instability.37,40Typically, athletes

present with pain during the

decel-eration phase of throwing, and pain

may be elicited on examination with

the arm in flexion, adduction, and

in-ternal rotation as the shoulder is

pos-teriorly subluxated Usually, in the

ab-sence of a posterior labral tear, neither

plain radiography nor MRI shows

any damage

Initial treatment is nonsurgical and

includes physical therapy to

strength-en the posterior rotator cuff and

scapu-lar muscles, especially the

infraspina-tus, teres minor, and posterior deltoid

Proper throwing mechanics are

em-phasized along with leg and trunk

strengthening to transfer some of the

throwing stresses to the lower

extrem-ities Usually, athletes are able to

re-turn to throwing after 4 to 6 months

of rehabilitation Recurrent or

recal-citrant symptoms may require

surgi-cal intervention, with either open or

arthroscopic posterior capsulorrhaphy

to imbricate the redundant posterior

capsule.41,42

Multidirectional Shoulder

Instability

Multidirectional instability (MDI)

is characterized by symptoms of

sub-luxation in more than one direction (anterior, posterior, or inferior) in the absence of a major traumatic event

Commonly, MDI affects athletes par-ticipating in sports that involve repet-itive shoulder abduction and exter-nal rotation Competitive swimmers, especially those swimming the but-terfly stroke, and gymnasts often exhibit symptoms of MDI.2,31,37

Affect-ed athletes typically possess under-lying physiologic glenohumeral lax-ity that is exacerbated by repetitive microtrauma or by a traumatic insult, resulting in inability to maintain dy-namic stability Athletes may report

a dead arm as well as a sensation of the shoulder dislocating and sponta-neously reducing Symptoms may be vague but usually correlate with the direction of instability Athletes with anterior instability describe pain with the arm in the overhead, abducted, and externally rotated position Those with posterior instability typically re-port pain with the arm in the forward-elevated and internally rotated posi-tion, such as when pushing open heavy doors Patients with inferior in-stability may report discomfort when they carry heavy objects with the arm

at the side Occasionally, secondary rotator cuff symptoms also may be re-ported in conjunction with instabil-ity.31

On physical examination, general-ized ligamentous laxity may be present, with findings such as elbow and metacarpophalangeal joint hy-perextension The affected shoulder demonstrates increased

glenohumer-al translation in multiple directions

Comparing the affected shoulder with the contralateral shoulder is mandatory, and there may be multi-ple positive findings on load-and-shift, relocation, and fulcrum tests and apprehension maneuvers Typ-ically, a sulcus sign significant for in-ferior laxity is also present It is im-portant to determine the direction or directions of increased

glenohumer-al translation that actuglenohumer-ally replicate the patient’s symptoms because

lax-ity does not necessarily indicate in-stability Imaging studies are often un-remarkable; plain radiographs usually show no osseous abnormalities unless the patient has had an actual dislo-cation, in which case a humeral head

or glenoid defect may be observed MRI arthrography with intra-articular contrast medium may show a redun-dant or patulous capsule with in-creased capsular volume, usually with

no evidence of labral damage Initial treatment consists of rest and wet heat before, and ice after, ac-tivity Most importantly, the patient should begin rehabilitation that em-phasizes strengthening of the rotator cuff, deltoid, and scapulothoracic musculature to provide dynamic sta-bility Surgery is indicated when there are residual symptoms after a min-imum of 6 months of therapy Usu-ally, the loose redundant capsule is reconstructed and imbricated in the direction or directions of predomi-nant instability (anterior, inferior, or posterior, or combinations of these) Because current arthroscopic capsu-lorrhaphy techniques can achieve re-sults similar to those of open inferior capsular shifts, these are typically pre-ferred in overhead athletes.43,44 Ther-mal energy to “shrink” the redundant capsule is not indicated, given the failure rates for MDI;45-47rather, su-ture capsulorrhaphy techniques to eliminate redundancy by imbricating the capsule and reducing its overall volume are preferable Occasionally, these techniques are augmented with suture anchors along the glenoid rim for additional fixation.43,44Suture cap-sulorrhaphy may be accomplished for the anterior and posterior capsule as well as the rotator interval, depend-ing on the nature of the injury Patients should be counseled re-garding treatment goals, including ini-tial shoulder “tightening,” during which the shoulder is immobilized for

a period of 2 to 4 weeks while gentle isometric exercises are performed This

is followed by gradual increase in ROM and strengthening over an

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ex-tended period, with return to

unre-stricted activity by 6 months

Injuries of the Elbow

Elbow injuries occur more

frequent-ly than shoulder injuries, with 50%

to 75% of adolescent baseball players

reporting elbow pain.2Most of these

injuries result from chronic repetitive

stresses; they can be limited by

de-creasing the frequency and duration

of throwing and by improving

pitch-ing mechanics Although these

inju-ries are most common in pitchers,

they also occur frequently in other

overhead athletes.2,21

Little League Elbow

Initially described as an avulsion

fracture of the medial epicondyle,

Lit-tle League elbow is a general term

re-lating to several abnormalities in the

elbow of the young overhead athlete,

including medial epicondylar

avul-sion, medial epicondylar apophysitis,

and accelerated apophyseal growth

with delayed closure of the

epicondy-lar growth plate.2,10,11,21Little League

elbow results from repetitive valgus

stresses and tension overload of the

medial structures Repetitive

contrac-tion of the flexor-pronator

muscula-ture stresses the chondro-osseous

or-igin, leading to inflammation and

subsequent apophysitis Affected

ath-letes are usually younger than age 10

years and typically report a triad of

medial elbow pain, decreased

throw-ing effectiveness, and decreased

throwing distance.2,10,11,21 Patients

may exhibit medial swelling, focal

tenderness over the medial

epi-condyle, and occasional flexion

con-tractures Although results of plain

ra-diography are sometimes normal,

radiographic changes include

irreg-ular ossification of the medial

epi-condylar apophysis early in the

dis-ease process, followed by accelerated

growth, marked by apophyseal

en-largement, separation, and

eventual-ly fragmentation.2,10,11,21

Generally, treatment consists of 2

to 4 weeks of rest and NSAIDs, fol-lowed by stretching and strengthen-ing exercises of the elbow, with grad-ual return to throwing at 6 weeks if the athlete is symptom free.10 Occa-sionally, symptoms may persist for extended periods, typically because

of inadequate rest or activity modi-fication In these instances, brief splint

or cast immobilization may be nec-essary, and the patient should not re-sume throwing until the following season.10Other factors contributing to exacerbation of symptoms include a high number of pitches thrown and innings pitched as well as improper throwing mechanics, all of which should be addressed and monitored closely in young overhead athletes

Ulnar Collateral Ligament Injuries and Valgus Instability

UCL injuries are uncommon in skeletally immature athletes Pa-tients with this injury report medial elbow pain that is exacerbated dur-ing the late cockdur-ing and acceleration stages of throwing Examination for valgus stability is performed with the elbow flexed 25° to 30° to unlock

the olecranon from its fossa as a val-gus stress is applied; this maneuver tests the anterior band of the ante-rior bundle of the UCL The poste-rior band is tested by the milking maneuver (Fig 8), performed by pulling the patient’s thumb with the forearm supinated, shoulder ex-tended, and elbow flexed more than 90°.47Usually, results of plain radi-ography are normal unless late changes associated with chronic lax-ity and valgus extension overload have developed Valgus stress views also may be obtained to assess sta-bility; a medial joint opening >2 mm wide indicates instability (Fig 9) However, MRI is more useful and provides good visualization of the UCL as well as of the surrounding structures19(Fig 10) Recently, com-puted tomography arthrography also has been used adjunctively to evaluate undersurface tears of the UCL as well as other intra-articular structures

Initial treatment of UCL injuries in-cludes a short period of immobiliza-tion coupled with ice and NSAIDs to control pain Once the acute inflam-mation subsides, a supervised

ther-Figure 8 Elbow examination for medial instability A, The examination for valgus stability

is done with the elbow flexed 25° to 30° (to unlock the olecranon) testing the anterior band

of the anterior bundle of the ulnar collateral ligament The examiner firmly grasps the

pa-tient’s elbow and forearm applying varus-valgus stress while palpating the UCL B, The

milk-ing maneuver tests the posterior band of the anterior bundle of the ulnar collateral ligament The maneuver is performed by applying downward and valgus stress with the forearm su-pinated, and elbow flexed more than 90° (Adapted with permission from Kvitne RS, Jobe

FW: Ligamentous and posterior compartment injuries of the elbow, in Jobe FW [ed]:

Oper-ative Techniques in Upper Extremity Sports Injuries St Louis, MO: Mosby, 1996, p 415.)

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