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THE ROLE OF SURGERY IN HEART FAILURE - PART 9 docx

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Abbreviations: DT, destination therapy; ePTFE, expanded polytetrafluoroethylene; LVAS, left ventricular assist system; TAH, total artificial heart... Lifetime Circulatory Support Must NotB

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Study MCSD Infection Bleeding Neurological Dysfunction Outcomes

Assist device References Type Indication N % Incid N % Incid N % Incid N % Incid BTT % Wean % Mort % Incid 1 year TxSv % AbioCor [61,62] Pulsatile DT 0 0 0 4 57.1 1.94 3 42.86 1.45 0 0 0.00 0 0 0 0 5 71.43 2.42 d NA d

Arrow LionHeart [32] Pulsatile DT d NA d d NA d 57 247.8 2.61 d NA d ddddddd NA ddddddd

Berlin Heart InCor [75] Flow BTT 0 0 0 4 26.7 0.64 4 26.67 0.64 3 20 0.48 5 33.33 1 6.667 6 40 0.967 d NA d

HeartMate I [94] Pulsatile BTT 8 25 1.36 4 12.5 0.68 2 6.25 0.34 3 9.38 0.51 20 62.5 1 3.125 6 18.75 1.023 d NA d

Heartmate I [95] Pulsatile BTT 57 58.8 3.06 20 20.6 1.08 2 2.062 0.11 12 12.4 0.65 74 76.29 2 2.062 24 24.74 1.29 69 93.2 HeartMate I [105] Pulsatile BTT 10 62.5 1.38 6 37.5 0.83 2 12.5 0.28 20 125 2.76 12 75 1 6.25 1 6.25 0.138 7 58.3 HeartMate I [106] Pulsatile BTT 41 36 3.11 28 24.6 2.12 16 14.04 1.21 4 3.51 0.30 57 50 3 2.632 34 29.82 2.576 d NA d

HeartMate I [107] Pulsatile BTT 125 44.6 1.45 31 11.1 0.36 75 26.79 0.87 3 1.07 0.03 188 67.14 10 3.571 82 29.29 0.953 158 84

HeartMate I [108] Pulsatile BTT d NA d d NA d d NA d d NA d 88 73.95 0 0 20 16.81 0.864 78 88.6 HeartMate I [43] Pulsatile BTT 15 29.4 0.78 d NA d 1 1.961 0.05 5 9.8 0.26 36 70.59 0 0 15 29.41 0.778 31 86.1 HeartMate II [109] Flow BTT/DT d NA d d NA d 1 6.667 0.14 1 6.67 0.14 1 6.667 0 0 2 13.33 0.274 d NA d

Jarvik FlowMaker [16] Flow DT 1 5.88 0.06 1 5.88 0.06 4 23.53 0.24 0 0 0.00 1 5.882 0 0 8 47.06 0.487 d NA d

Jarvik FlowMaker [110] Flow BTT/DT d NA d d NA d d NA d 10 9.8 0.17 ddddddd NA ddddddd

(CardioWest) [111] Pulsatile BTT 4 3.15 0.40 33 26 3.34 2 1.575 0.20 1 0.79 0.10 ddddddd NA ddddddd

Micromed DeBakey [31] Flow BTT 5 3.33 0.16 48 32 1.58 16 10.67 0.53 4 2.67 0.13 62 41.33 1 0.667 68 45.33 2.237 d NA d

Micromed DeBakey [15] Flow BTT 2 6.67 0.58 8 26.7 2.32 3 10 0.87 0 0 0.00 20 66.67 0 0 0 0 0 d NA d

Micromed DeBakey [30] Flow BTT 0 0 0.00 4 23.5 0.97 2 11.76 0.49 0 0 0.00 14 82.35 0 0 2 11.76 0.486 d NA d

Novacor LVAS [43] Pulsatile BTT 5 38.5 1.23 d NA d 3 23.08 0.74 0 0 0.00 9 69.23 0 0 2 15.38 0.493 7 77.8 Novacor LVAS [112] Pulsatile BTT d NA d 59 12.7 0.33 d NA d 0 0 0.00 155 33.41 21 4.526 147 31.68 0.821 d NA d

(ePTFE) [65] Pulsatile NA d NA d d NA d 9 10.23 0.31 d NA d ddddddd NA ddddddd

Novacor LVAS (Pol) [113] Pulsatile BTT 104 36.9 1.36 82 29.1 1.07 54 19.15 0.71 d NA d d NA d 82 29.08 1.071 d NA d

(Vasc) [113] Pulsatile BTT 58 28.7 1.15 59 29.2 1.17 23 11.39 0.46 d NA d d NA d 65 32.18 1.291 d NA d

CardioWest TAH [114] Pulsatile BTT 29 35.8 1.65 23 28.4 1.31 5 6.173 0.28 1 1.23 0.06 64 79.01 0 0 17 20.99 0.968 55 85.9 CardioWest TAH [115] Pulsatile BTT 5 11.9 0.51 8 19 0.81 4 9.524 0.40 2 4.76 0.20 11 26.19 0 0 24 57.14 2.425 d NA d

Totals 25 26.1 23.7 1.03 24.8 24.9 1.18 13.7 24.98 0.61 3.6 10.9 0.29 45.39 51.09 2.22 1.639 32.3 27.97 57.9 82 Because of differences in adverse event reporting criteria, data might be incomplete or interpreted differently and not accurately comparable for the experiences with different assist devices For an in-deep analysis, the authors suggest readers to refer to the original publication.

Abbreviations: DT, destination therapy; ePTFE, expanded polytetrafluoroethylene; LVAS, left ventricular assist system; TAH, total artificial heart.

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Lifetime Circulatory Support Must Not

Be Restricted to Transplant Centers

Oxford Heart Centre, John Radcliffe Hospital, Oxford, UK

The problem

Chronic heart failure affects around 5 million

North Americans and 7 million Europeans each

year, accounting for 2% of the total health care

budget in Western countries[1] The major

com-ponent of health care costs is repeated hospital

ad-missions to palliate intolerable symptoms and

escalate medical treatment It is estimated that

be-tween 250,000 and 500,000 patients in the United

States and approximately 2.2 million worldwide

are in the terminal phase of heart failure (Stage D,

New York Heart Association [NYHA] IV) and

refractory to maximum medical therapy [2]

With around 10% of the population older

than 65 years of age suffering systolic left

ven-tricular dysfunction, the number of patients

who have heart failure will double within the

next 25 years In this global context cardiac

transplantation is irrelevant Essentially

re-stricted to patients younger than 65 years of

age who do not have significant comorbidity,

fewer than 2,200 donor hearts per year are

made available in the United States and around

150 in the United Kingdom[3] In a population

constantly bombarded with media coverage of

medical advances, there will be escalating

de-mand for relief from severely symptomatic Stage

D disease Provided with an effective treatment,

most civilized health care systems are prepared

to intervene irrespective of cost The treatment

of advanced renal disease sets the precedent

Hemodialysis, which provides an overall 60%

2-year survival in the United States, is offered irrespective of age or transplant eligibility at a cost of around $60,000 per year [4]

The strategy of lifetime left ventricular assist device (LVAD) deployment is based on the success of mechanical bridge to transplantation

[5] First-generation LVADs were designed to re-place the failing left ventricle by providing stroke volume and pulsatile blood flow (Fig 1a, b)[6] Blood is actively withdrawn from the dilated chamber and pumped in a pulsatile manner to the ascending aorta at a rate of between 4 and

10 L/min In patients dying of cardiogenic shock these devices sustain life until a donor organ is available, provide symptomatic relief, reverse multiorgan dysfunction, and attenuate the cyto-kine and humeral responses to heart failure [7] Transplant outcomes are improved because termi-nally ill patients are in better condition to survive major surgery[8] In turn comes the observation that mechanical unloading of the failing heart and increased coronary blood flow have impor-tant beneficial effects on the diseased myocar-dium Reduced wall tension and stroke work result in decreased myocyte hypertrophy, apopto-sis, myocytolyapopto-sis, and fibrosis Myocyte genetic expression and metabolic processes revert toward normal[9] As a result LVADs can occasionally

be removed following functional improvement of the native heart (Fig 2)[10] Bridge to recovery occurs more often in inflammatory conditions, such as myocarditis, intoxication, or idiopathic dilated cardiomyopathy

With the exception of the United States, Germany, and France, bridge to transplantation

is an expensive and infrequent intervention In the study by Sharples and colleagues[11]evaluating the ventricular assist device program in the United

* Oxford Heart Centre, John Radcliffe Hospital,

Headley Way, Headington, Oxford OX3 9DU, United

Kingdom.

E-mail address: swestaby@AHF.org.uk

1551-7136/07/$ - see front matter Ó 2007 Elsevier Inc All rights reserved.

Heart Failure Clin 3 (2007) 369–375

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