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9HWHULQDU\ 6FLHQFH Expression of pituitary adenylate cyclase activating polypeptide and its type I receptor mRNAs in human placenta Phil-Ok Koh 1 , Chung-Kil Won 1 , Hae-Sook Noh 2 , Gye

Trang 1

9HWHULQDU\ 6FLHQFH

Expression of pituitary adenylate cyclase activating polypeptide and

its type I receptor mRNAs in human placenta

Phil-Ok Koh 1

, Chung-Kil Won 1

, Hae-Sook Noh 2

, Gyeong-Jae Cho 2

, Wan-Sung Choi 2,

*

1Department of Anatomy, College of Veterinary Medicine and Institute of Animal Medicine, Gyeongsang National University, Jinju 660-701, Korea

2

Department of Anatomy and Neurobiology, College of Medicine, Institute of Health Sciences, Gyeongsang National University, Jinju 660-751, Korea

Pituitary adenylate cyclase activating polypeptide

(PACAP) was first isolated from ovine hypothalamus and

was known to stimulate the release of growth factor in

various cells Recently, we reported the cellular

localization of PACAP and its type I (PAC 1 ) receptor in rat

placenta during pregnancy Placenta is a critical organ

that synthesizes several growth factors and angiogenic

factors for the fetal development and its own growth.

However, there is little information regarding the cellular

localization of PACAP and its receptor in human placenta

at various gestations The aim of the present study was to

define the expression and distribution of PACAP and

were expressed in stroma cells of stem villi and terminal

villi At the early stage, on 7 and 14 weeks, PACAP and

cells surrounding the blood vessels within stem villi These

genes were strongly expressed in stroma cells of stem villi

and terminal villi on 24 and 38 weeks The expression of

these genes was increased as gestation advanced, and

localized in the same areas Localization of PACAP and

play an important role, as an autoregulator or

findings strongly suggest that PACAP may have a critical

role in physiological function of the placenta for

gestational maintenance and fetal growth.

Key words: PACAP, receptor, placenta, human

Introduction

The placenta is an essential organ for the fetal development and the maintenance of pregnancy It is known that placenta synthesis the growth hormone [21] and several growth factors, such as basic fibroblast growth factor and insulin like growth factor [6,29] Also, placenta produces placenta growth factor (PlGF) and vascular endothelial growth factor (VEGF) [6,29], which are critical factors for the placental growth and fetal development As an important regulator of angiogenesis, VEGF contributes to the development and growth of the endothelium during the tissue growth [5,32] Also, another member of the VEGF family, PlGF, promotes endothelial cell proliferation in vitro [16] The previous study showed pituitary adenylate cyclase activating polypeptide (PACAP) stimulates the release of VEGF and acts as a trophic factor in various cells [7,8,17,31, 33] PACAP has considerable homology with vasoactive interstinal peptide (VIP) and growth hormone releasing hormone [17,31,33] Recently, it was reported that PACAP and PACAP receptor are present in both the human and rat placenta at term [23] Also, even more recently, we reported the cellular localization of PACAP and PACAP type I (PAC1) receptor in the rat placenta during pregnancy [14] Therefore, the existence of PACAP in placenta suggests that PACAP affects placental function

PACAP was originally isolated from ovine hypothalamus and was known to stimulate the production of cAMP in anterior pituitary cells [18] PACAP exists in two biologically active forms, PACAP 38 and PACAP 27, sharing the same N-terminal 27 amino acids [19] PACAP binds to three type Ireceptors Among these receptors, PAC1 receptor has high affinity with PACAP 38 and PACAP 27, very low affinity with VIP [12,28] But, VIP1 and VIP2 receptors have approximately equal high affinity for PACAP 38, PACAP

27, and VIP [11,12,28] PACAP and its receptor have been found in the central nervous system and its peripheral tissues, including the hypothalamus, pituitary gland, adrenal

*Corresponding author

Tel: 82-55-751-8716; Fax: 82-55-759-0779

E-mail: choiws@nongae.gsnu.ac.kr

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PACAP in the hypothalamus, pituitary, and gonads suggests

its roles in the reproductive system However, the existence,

localization of PACAP and PAC1 receptor genes in human

placenta at various gestations has been unknown Thus, the

present study was performed to determine the distribution of

PACAP and PAC1 receptor mRNAs in human placenta

Materials and Methods

Tissue preparation

Human placental tissue from legal abortions aged between

6-7 weeks post menstruation (pm) were collected from

normal pregnancies by curettages Second trimester placenta

from 14-24 weeks of gestation were obtained from induced

abortion of healthy pregnancies and term placenta (38-41

weeks pm) by caesarian section or normal delivery For in

situ hybridization studies, tissues were fixed with 4%

paraformaldehyde in 0.1M phosphate buffered saline (PBS)

and cryoprotected with 20% sucrose phosphate buffer for 24

hr Placental sections were cut in a cryomicrotome at a

thickness of 15µm, mounted on the Probe-on slides (Fisher

Scientific, USA), and stored at −70o

C Slides from each placenta were stained with hematoxylin and eosin for

general morphological observation

In situ Hybridization

All solutions were made with sterile water and glassware was

autoclaved to prevent contamination by RNase In situ

hybridization histochemistry was carried out, as described by

Angerer et al [1] Briefly, the slides were dried, washed with

0.1 M PBS, and treated proteinase K, TE buffer, and an

acetylation solution Sections were covered with prehybridization

buffer containing 50% deionized formamide and incubated at

37o

C for 1 hr After removal of the prehybridization buffer, the

slides were covered with the mixture containing the

prehybridization buffer, 50µg/ml yeast tRNA, 10 mM

dithiothreitol, and 35

S-labeled PACAP cRNA probe or PAC1

receptor cRNA probe [13] The slides were then covered with

cover glasses and incubated at 60o

C for 24 hr 35

S-UTP-labeled

probes were prepared using in vitro transcription kit (Promega,

USA) Antisense and sense cRNA probes were purified with a

Sephadex G-50 nick column (Pharmacia Biotech, Sweden) and

eluted with SET buffer (0.1% SDS, 1 mM EDTA, 10 mM Tris,

and 10 mM DTT) Tissue slides were posthybridized in a

posthybridization buffer Following a wash in 4× SSC for 30

min, the sections were then treated with ribonuclease A (50µg/

ml) at 37o

C for 10 min, washed twice in 2× SSC and 1 × SSC,

transferred to a wash buffer containing 0.1× SSC at 65o

C for

30 mins, and dehydrated in alcohol solutions with ascending

concentrations Slides were exposed to β-max hyperfilm

(Amersham, Sweden) for 4 days in light-tight cassettes at −

70o

C, and were dipped into NTB2 emulsion (1 : 1 dilution,

Eastman Kodak, USA), exposed at 4 for 2 weeks, developed in

Kodak D19 developer (1 : 1 dilution, Eastman Kodak, USA) at

15o

C, and counterstained with hematoxylin The slides were observed under a dark and a bright field microscope, and photographed

Results

The present study showed the expression and distribution

of PACAP and PAC1 receptor mRNAs in the human placenta

at various gestations In situ hybridization revealed the

expression of PACAP and PAC1 receptor mRNAs in stem villi and terminal villi Positive cells of PACAP mRNA were detected in stroma cells surrounding the blood vessels within stem villi on 7 weeks (Figs 1A & 2A) PACAP mRNA was expressed in stroma cells of stem villi on 14 week (Figs 1B & 2B) Furthermore, positive signals of PACAP mRNA were strongly observed in the stroma cells

of stem villi and terminal villi on 21 and 38 weeks (Figs 1C, 1D, 2C & 2D) Signals for PACAP mRNA in these cells were gradually increased as gestation advanced However, PACAP mRNA was very weakly expressed in cytotrophoblast cells and syncytiotrophoblast cells There were no detectable signals in negative control with a sense probe (Fig 1E) The hybridization of adjacent sections with PACAP and PAC1 receptor cRNA probes showed that PACAP and PAC1

receptor mRNAs were expressed in the same areas Positive signals of PAC1 receptor mRNA were detected in stroma cells of stem villi on 7 and 14 weeks (Figs 3A, 3B, 4A & 4B) PAC1 receptor mRNA was strongly expressed in stroma cells of stem villi and terminal villi on 21 and 38 weeks

Fig 1 Dark-field photomicrographs of PACAP mRNA expression

in the human placenta from 7 (A), 14 (B), 24 (C), and 38 (D)

weeks gestation by in situ hybridization A and B: Positive signals

were moderately observed in stroma cells of stem villi C and D: Positive cells were strongly detected in stroma cells of stem villi and terminal villi E: No positive signals were detected in negative control with a sense probe Bar = 200µm

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(Figs 3C, 3D, 4C & 4D) As similar like to the expression

pattern of PACAP, positive signals for PAC1 receptor in these

cells became strong as gestation advanced But, PAC1

receptor mRNA was very weakly expressed in cytotrophoblast

cells and syncytiotrophoblast cells No positive signals of

PAC1 receptor mRNA was detected in negative control with

a sense probe (Fig 3E)

Discussion

In the present study, we showed the cellular localization of PACAP and PAC1 receptor mRNAs in human placenta on 7,

14, 24, and 38 weeks Recently, we reported evidence that PACAP and PAC1 receptor mRNAs were expressed in decidual cells, chorionic vessels, and stroma cells of

chorionic villi in the rat placenta [14] Also, Scaldaferri et al.

[23] demonstrated the presence of PACAP and PAC1

receptor in both the human and the rat placenta at term, using RT-PCR and immunohistochemistry techniques They showed the expression of PACAP at term placenta, but they did not offer any information as to the distribution of PACAP and PAC1 receptor in human placenta at various

gestations, during pregnancy In this study, we utilized in situ

hybridization to determine the existence of PACAP and PAC1 receptor mRNAs in human placenta Our data showed the expression of PACAP and PAC1 receptor mRNAs in stroma cells of stem villi and terminal villi As gestation advanced, the expression of PACAP and PAC1 receptor mRNAs was increased in these cells

It is known that placenta produces several growth factors such as insulin like growth factor and basic fibroblast growth factor Especially, VEGF and placenta growth factor (PlGF), which are essential factor for the placental growth and fetal development [5,32] VEGF was expressed in stroma cells within villi in human placenta [26] Localization of VEGF

in stroma cells demonstrates that VEGF play an important role in the physiological growth and function of the vascular system in the villous stroma Furthermore, PACAP that acts

as a growth factor in various cells, stimulates VEGF release

Fig 2 Bright-field photomicrographs of PACAP mRNA

expression in the human placenta from 7 (A), 14 (B), 24 (C), and

38 (D) weeks gestation by in situ hybridization A and B: PACAP

mRNA was strongly expressed in stroma cells of stem villi C

and D: Positive signals were strongly detected in stroma cells of

stem villi Arrows indicate the positive cells Bar = 20µm

placenta from 7 (A), 14 (B), 24 (C), and 38 (D) weeks gestation

by in situ hybridization A and B: Positive signals were

moderately observed in stroma cells of stem villi C and D:

Positive cells were strongly detected in stroma cells of stem villi

and terminal villi E: No positive signals were detected in

negative control with a sense probe Bar = 200µm

Fig 4 Localization of PAC1 receptor mRNAs expression in the human placenta from 7 (A), 14 (B), 24 (C), and 38 (D) weeks

gestation by in situ hybridization A and B: PAC1 receptor mRNA was strongly expressed in stroma cells of stem villi C and D: Positive signals were strongly detected in stroma cells of stem villi Arrows indicate the positive cells Bar = 20µm

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endothelial cells by promoting cell proliferation and

permeability In this study, PACAP was strongly expressed

in stroma cells of stem villi and terminal villi PACAP

mRNA was localized in the stroma cell surrounding the

blood vessel on 14 and 24 weeks Also, PACAP mRNA was

expressed in the whole stroma cells on 38 weeks

Furthermore, PACAP 38 immunostaining was detected in

stroma cells of stem and terminal placental villi [23] Both

PACAP and VEGF were expressed in stroma cells of

terminal villi Thus, our data suggest that PACAP stimulates

the release of VEGF and promotes the growth of placenta

In the previous studies, VIP/PACAP neuropeptide family

regulates the blood flow and hormone secretion in human

placenta [4,9,30] Steenstrup et al [30] reported that PACAP

were expressed in the uteroplacental unit, where it causes a

concentration dependent relaxation on stem villous and

umbilical cord arteries These results suggest that PACAP

mediates the placental growth and fetal development during

the pregnancy period

In the previous studies, many researchers demonstrated

that PACAP acts an autocrine and/or paracrine regulator in

various tissues, including ovarian granulose cells, testicular

Leydig cells, and placental tissue [15,20,22,24] Also, in our

results, PACAP and PAC1 receptor mRNAs were expressed

in the same areas Although our data did not elucidate the

physiological role and action mechanism of PACAP in

human placenta, the localization of PACAP and its PAC1

receptor in the same areas strongly suggest that PACAP may

act as an autoregulator or pararegulator via its PAC1 receptor

in stem villi and terminal villi during pregnancy In

conclusion, our findings suggest that PACAP may have an

important role in physiological function of the placenta for

gestational maintenance and fetal growth

Acknowledgments

This work was supported by the Brain Korea 21 Project,

the Ministry of Education of Republic of Korea, and was

partly supported by the grant from the Korea Science and

Engineering Foundation (KOSEF R04-2003-000-10062-0)

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