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Cervical SpineAbstract The cervical spine often becomes involved early in the course of rheumatoid arthritis, leading to three different patterns of instability: atlantoaxial subluxation

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Cervical Spine

Abstract

The cervical spine often becomes involved early in the course of rheumatoid arthritis, leading to three different patterns of instability: atlantoaxial subluxation, atlantoaxial impaction, and subaxial subluxation Although radiographic changes are common, the prevalence of neurologic injury is relatively low The primary goal of treatment is to prevent permanent neurologic injury while avoiding potentially dangerous and unnecessary surgery Strategies include patient education, lifestyle modification, regular

radiographic follow-up, and early surgical intervention, when indicated Magnetic resonance imaging is indicated when neurologic deficit (myelopathy) occurs or when plain radiographs show atlantoaxial subluxation with a posterior atlantodental interval≤14 mm, any degree of atlantoaxial impaction, or subaxial stenosis with a canal diameter≤14 mm Surgery should be

considered promptly for any of the following: progressive neurologic deficit, chronic neck pain in the setting of radiographic instability that does not respond to nonnarcotic pain medication, any degree of atlantoaxial impaction or cord stenosis, a posterior atlantodental interval≤14 mm, atlantoaxial impaction represented

by odontoid migration≥5 mm rostral to McGregor’s line, sagittal canal diameter <14 mm, or a cervicomedullary angle <135°

Rheumatoid arthritis (RA) is a chronic inflammatory autoim-mune disorder characterized by poly-arthritic disturbance of peripheral joints and early involvement of the cervical spine RA is relatively com-mon, affecting 0.5% to 1.5% of the

US population and twice as many women as men.1The etiology of the disease is unknown but is likely to

be multifactorial, with a relatively strong genetic component RA re-duces life expectancy, and half of all afflicted patients become disabled within 10 years of diagnosis.2 The course of the disease in any given pa-tient, however, is unpredictable and may be relentlessly progressive or

characterized by intermittent flares and remissions Current medical treatment involves early and aggres-sive use of disease-modifying anti-rheumatic drugs, such as methotrex-ate, antimalarial drugs, sulfasalazine, and gold Early clinical studies sug-gest that pharmacologic blockade of tumor necrosis factor-αwith etaner-cept or infliximab can preserve joint function and favorably affect the nat-ural history of the disease.3

Chronic synovial inflammation leads to progressive destruction of the joints, ligaments, and bone, par-ticularly in the atlantoaxial region Eventually, this process leads to clinical manifestations of pain,

de-David H Kim, MD, and

Alan S Hilibrand, MD

Dr Kim is Assistant Clinical Professor of

Orthopaedic Surgery, Department of

Orthopaedic Surgery, Tufts University

School of Medicine, Boston, MA, and

The Boston Spine Group, New England

Baptist Hospital, Boston Dr Hilibrand is

Associate Professor, Departments of

Orthopaedic Surgery and Neurosurgery,

and Director of Medical Education for

the Department of Orthopaedic Surgery,

Jefferson Medical College, Thomas

Jefferson University, Philadelphia, PA,

and The Rothman Institute, Philadelphia.

None of the following authors or the

departments with which they are

affiliated has received anything of value

from or owns stock in a commercial

company or institution related directly or

indirectly to the subject of this article:

Dr Kim and Dr Hilibrand.

Reprint requests: Dr Hilibrand, The

Rothman Institute, 925 Chestnut Street,

Philadelphia, PA 19107-4216.

J Am Acad Orthop Surg

2005;13:463-474

Copyright 2005 by the American

Academy of Orthopaedic Surgeons.

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formity, instability, and neurologic

deficits Three characteristic

pat-terns of instability resulting from

rheumatoid involvement of the

cer-vical spine have been described; each

may occur in isolation or in

combi-nation: atlantoaxial subluxation,

at-lantoaxial impaction, and subaxial

subluxation Each form of

instabili-ty can lead to compression of or

in-jury to the spinal cord or brainstem

and can progress to paralysis or

death Treatment strategies for the

rheumatoid cervical spine include

patient education and lifestyle

mod-ification, periodic assessment for

ra-diographic signs of increased risk of

neurologic injury, and early surgical

intervention to prevent permanent

neurologic injury

Epidemiology

After the hands and feet, the cervical

spine is the most common site of

disease involvement in RA.4

De-pending on the population and

diag-nostic criteria, between 17% and

86% of patients with RA have

evi-dence of cervical spine disease.5

Sev-eral studies of patients with RA

sug-gest that the cervical spine becomes

involved early in the course of the

disease, often within the first 2 years

following diagnosis.6A prospective

study of 103 Finnish patients with

rheumatoid factor–positive RA

re-vealed a 20-year incidence of

atlan-toaxial subluxation of 23%,

atlanto-axial impaction of 26%, and subatlanto-axial

subluxation of 19%.7

Pathophysiology

The cervical spine is composed of 22

separate synovial joints, all potential

targets of rheumatoid disease.5The

synovial joints between the

trans-verse atlantal ligament and the

odon-toid process, as well as those between

the anterior atlas arch and the

odon-toid, are frequently involved The

oc-cipitoatlantal and atlantoaxial

artic-ulations are the only segments in the

spine without intervertebral disks,

which may account for the high prev-alence of disease in the upper cervi-cal spine in patients with RA

Atlantoaxial Subluxation

Rheumatoid involvement of the synovial joints in the cervical spine

is characterized by formation of pan-nus, an inflammatory tissue with variable fibrous content Synovitis and pannus formation can weaken the transverse, alar, and apical liga-ments The weight of the head, par-ticularly with neck flexion, contrib-utes to the repetitive strain of these ligaments, leading to stretching or rupture and the onset of

atlantoaxi-al subluxation Transverse ligament weakening and rupture occur most commonly at the site of a synovial bursa separating the ligament from the posterior surface of the odontoid

Inflammation also leads to decalcifi-cation and occasional rupture of the ligamentous insertion sites on the atlas

Erosion of the odontoid process, a hallmark of RA, may occur

anterior-ly at its synovial joint with the arch

of C1, posteriorly at its synovial joint with the transverse ligament, and at the tip of the odontoid in re-lation to the apical ligament inser-tion Asymmetric patterns of ero-sion can lead to various radiographic instability patterns, including an-teroposterior, rotatory, or lateral sub-luxations (Table 1) Both rotatory and lateral subluxation patterns can result in torticollis

Atlantoaxial subluxation may oc-cur in up to 49% of RA patients and usually manifests as anterior sub-luxation of the atlas (C1) on the

ax-is (C2).8This increased anterior at-lantodental interval and decreased posterior atlantodental interval is apparent on lateral cervical spine ra-diographs, particularly with the neck in flexion (Figure 1) As sublux-ation increases with time, the space available for the spinal cord

decreas-es, which may compress or injure the spinal cord However, in patients with a large spinal canal, an anterior

atlantodental interval >10 mm may

be seen without any apparent neuro-logic sequelae

The reducibility of atlantoaxial subluxation, either with extension positioning or external traction, is important in planning treatment Initially, subluxation may be purely dynamic, appearing only on flexion views Eventually, pannus may be-come interposed between the

anteri-or atlas arch and the odontoid, con-verting a dynamic subluxation into a fixed one

Atlantoaxial Impaction

Involvement of the atlanto-occipital and atlantoaxial joints can lead to weakening and collapse of the lateral masses, with rostral mi-gration of the odontoid process and atlantoaxial impaction Atlantoaxial impaction also has been referred to

as superior migration of the odon-toid, cranial settling, and basilar in-vagination The prevalence of atlan-toaxial impaction is reportedly between 5% and 32% and generally

is thought to follow the appearance

of atlantoaxial subluxation.5 Com-pared with other instability patterns, atlantoaxial impaction appears to carry the worst prognosis and a much higher risk of myelopathy The symptomatology of atlanto-axial impaction is highly variable Compression of the C1 and C2 nerve roots leads to occipitocervical pain Ventral pressure on the medulla ob-longata can injure local cranial nerve nuclei or cause sudden death by

Table 1 Patterns of Rheumatoid Arthritis in the Cervical Spine

Atlantoaxial subluxation Anterior

Posterior Lateral Rotatory Atlantoaxial impaction Subaxial subluxation

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compressing the respiratory center.

The anterior spinal artery and

verte-bral arteries also can be

compro-mised, leading to neurologic deficits,

vertebrobasilar insufficiency, or

tran-sient ischemic attacks.9,10With

pro-gression of atlantoaxial impaction

and greater penetration of the

odon-toid process into the foramen

mag-num, the degree of atlantoaxial

sub-luxation may decrease in a process

referred to as “pseudostabilization.”5

However, any reduction in

atlan-toaxial subluxation with the

pro-gression of atlantoaxial impaction

actually carries a worse prognosis

because of the increased risk of

brainstem injury and sudden death.11

Subaxial Subluxation

Destabilization of the facet joints

as a result of weakening of the facet

capsules and interspinous ligament

results in anterior subaxial

sublux-ation Although marked

degenera-tive disk changes are consistently

present, synovitis has not been

ob-served in disk or annular tissue

Therefore, anterior spinal disease

does not appear to contribute

direct-ly to the development of subaxial

subluxation This instability pattern

is a relatively late manifestation of

cervical spine disease and is

ob-served in 20% to 25% of patients with RA.5 It is also the most fre-quently observed new instability pattern following upper cervical fu-sion in RA patients Subaxial sub-luxation occurs most frequently at the C2-3 and C3-4 levels and

typical-ly affects multiple adjacent levels, yielding a characteristic “staircase”

deformity and associated kypho-sis.6,12 Spinal cord compression oc-curs anteriorly from the proximal edge of the vertebral body distal to the slip or posteriorly from the neu-ral arch of the slipped vertebrae Sub-axial subluxation may not be appar-ent on radiographs with the patiappar-ent

in neutral position but should be-come apparent, when present, on flexion or extension views

Clinical Presentation

Rheumatoid involvement of the cer-vical spine is often asymptomatic

Neck pain, the most frequent com-plaint, may be present in 40% to 80%

of patients.5 Characteristically, pa-tients describe the pain as a deep ache radiating into the occipital, retro-orbital, or temporal areas The tem-poral pattern is typically mechanical and readily distinguishable from the typical pain of rheumatoid synovitis

Pain referred to the face, ear, or sub-occiput originates from irritation of the C2 nerve root supply to, respec-tively, the nucleus of the spinal trigeminal tract, greater auricular nerve, or greater occipital nerve.13 Some patients with atlantoaxial subluxation report the sensation of the head falling forward during neck flexion.14A clunking sensation also can occur during neck extension with spontaneous reduction of atlan-toaxial subluxation; this has been la-beled a positive Sharp-Purser test.15 Stiffness, crepitus, and painful range

of motion also are common com-plaints Sleep apnea may be caused

by brainstem compression

associat-ed with atlantoaxial impaction.16 Objective neurologic signs are present less frequently than pain but may be found in 7% to 34% of pa-tients.17,18In contrast to

radiograph-ic changes, whradiograph-ich appear early in the course of RA, neurologic deficits usually appear later, most

common-ly in late middle-age patients They may be difficult to elicit in patients with more advanced disease Ex-tremity weakness is thought to re-sult from advancing joint involve-ment with neurologic symptoms Signs of cervical myelopathy, the clinical manifestation of spinal cord

Figure 1

A 65-year-old woman with rheumatoid arthritis and progressive cervical myelopathy A, Lateral radiograph of the cervical spine

reveals atlantoaxial subluxation The anterior atlantodental interval measures 4 mm and the posterior atlantodental interval

measures 14 mm Sagittal (B) and axial (C) T2-weighted MRI scans reveal significant additional reduction in space available for

the cord to 7 mm because of a large soft-tissue pannus posterior to the odontoid process

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compression, should be sought,

in-cluding a wide-based spastic gait,

clumsy hands, a visible change in

handwriting, or difficulty

manipu-lating buttons or handling coins

Other classic physical findings

asso-ciated with myelopathy include

hy-perreflexia, a positive Babinski test,

and a positive Hoffmann sign Mild

motor and reflex deficits may be

im-possible to identify in patients with

significant pain and deformity of

ex-tremity joints Lhermitte’s sign, the

sensation of electric shocks traveling

down the torso or upper extremities

on neck flexion, suggests

myelopa-thy Urinary retention, followed by

overflow incontinence, is a

symp-tom of severe myelopathy

Occa-sionally, injury to the pyramidal

tract decussation can manifest as a

“cruciate paralysis,” with varying

degrees of upper extremity weakness

that may be symmetric or

asymmet-ric.10This injury pattern is often

dif-ficult to distinguish from central

cord syndrome

Radiographic

Evaluation

Plain Radiography

Appropriate preoperative

screen-ing for cervical spine disease in

pa-tients with RA is a controversial

subject No radiographic screening

protocol is universally accepted

Nevertheless, certain groups of

pa-tients should be considered strong

candidates for preoperative

radio-graphic screening (Table 2) All

pa-tients with RA should undergo an initial series of cervical spine radio-graphs, including an anteroposterior view and lateral views with the pa-tient in the neutral position as well

as in flexion and extension These radiographs are recommended before any surgical procedure requiring in-tubation One study of 113 RA pa-tients undergoing elective hip or knee arthroplasty found that 61%

demonstrated evidence of

instabili-ty, defined by at least 3 mm of dy-namic atlantoaxial subluxation, by atlantoaxial impaction (according to Ranawat’s method), or by subaxial subluxation, although only 50% of patients were symptomatic.19

Atlantoaxial Subluxation

Traditionally, an anterior atlanto-dental interval >5 mm was consid-ered diagnostic of atlantoaxial sub-luxation However, several studies have demonstrated that the anterior atlantodental interval does not cor-relate with the risk of neurologic injury.19-21Boden et al20showed that measurement of the posterior atlan-todental interval appears to be a more reliable predictor of

neurolog-ic defneurolog-icit in patients with atlantoax-ial subluxation; a value≤14 mm has been suggested as an indication for surgical stabilization (Figure 2) The posterior atlantodental interval also may be used to predict neurologic re-covery after surgery

Atlantoaxial Impaction

Several radiographic

measure-ment techniques have been

suggest-ed as means to gauge the severity of atlantoaxial impaction The original definition of atlantoaxial impaction was protrusion of the odontoid above the margins of the foramen magnum, also known as McRae’s line Because identifying the mar-gins of the foramen magnum on plain radiographs is difficult, alter-native landmarks have been estab-lished (Figure 3) McGregor’s line is defined on a lateral radiograph by the hard palate and the base of the oc-ciput (opisthion) Protrusion of the tip of the odontoid above

McGre-Table 2

Indications for Anteroposterior and Lateral Radiographs of the Cervical

Spine* in Patients With Rheumatoid Arthritis

Prolonged cervical symptoms >6 months

Neurologic signs or symptoms

Scheduled procedures requiring endotracheal intubation in patients who

have not had cervical radiographs in the last 2 to 3 years

Rapidly progressive carpal or tarsal bone destruction

Rapid overall functional deterioration

* Neutral, flexion, and extension

Figure 2

Diagrammatic representation of atlantoaxial subluxation typically seen in patients with rheumatoid arthritis The posterior atlantodental interval (PADI)

is measured from the posterior margin

of the odontoid process to the anterior margin of the posterior arch of C1 (Reproduced with permission from Boden SD, Dodge LD, Bohlman HH, Rechtine GR: Rheumatoid arthritis

of the cervical spine: A long-term analysis with predictors of paralysis

and recovery J Bone Joint Surg Am

1993;75:1282-1297.)

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gor’s line >4.5 mm is considered diagnostic of atlantoaxial impac-tion.20

The presence of odontoid erosion can make all of these radiographic measurements inaccurate For this reason, other techniques have been developed to diagnose atlantoaxial impaction on plain radiographs, even with significant odontoid ero-sion.22These include the Ranawat method, designed to assess the ex-tent of collapse at the atlantoax-ial articulation, and the Redlund-Johnell method, which uses the dis-tance between the anterior axis base and McGregor’s line22(Figure 4)

Subaxial Subluxation

The radiographic appearance of subaxial subluxation is characteris-tic and includes not only sagittal plane listhesis of sequential verte-bral bodies but also posterior ele-ment changes, including facet joint erosions and widening, whittling, or spindling of the spinous processes (Figure 5) There are multiple def-initions of subaxial subluxation

Figure 3

Radiographic landmarks for assessing atlantoaxial impaction in patients with

rheumatoid arthritis On a lateral radiograph, atlantoaxial impaction is diagnosed by

protrusion of the odontoid tip proximal to McRae’s line or 4.5 mm above

McGregor’s line (Adapted with permission from Riew KD, Hilibrand AS, Palumbo

MA, Sethi N, Bohlman HH: Diagnosing basilar invagination in the rheumatoid

patient: The reliability of radiographic criteria J Bone Joint Surg Am

2001;83:194-200.)

Figure 4

Methods to assess atlantoaxial impaction on plain radiographs A, Ranawat method A line (a) is drawn across the transverse

axis of the atlas, and a connecting line (b) is drawn through the vertical axis of the odontoid from the center of the C2 pedicle

radiographic shadow Values (x)<15 mm in men and <13 mm in women are diagnostic for atlantoaxial impaction B,

Redlund-Johnell method A line (a) is drawn between McGregor’s line (b) and the midpoint of the inferior end plate of C2 (c) A value (x)<34 mm in men and <29 mm in women is diagnostic for atlantoaxial impaction (Adapted with permission from Riew KD, Hilibrand AS, Palumbo MA, Sethi N, Bohlman HH: Diagnosing basilar invagination in the rheumatoid patient: The reliability of

radiographic criteria J Bone Joint Surg Am 2001;83:194-200.)

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Yonezawa et al12described subaxial

subluxation as >4 mm or 20%

listhe-sis of vertebral body diameter

Magnetic Resonance

Imaging

Although plain tomography,

cine-radiography, computed tomography

(CT), and CT myelography have

been used to follow the rheumatoid

cervical spine, these studies have

been supplanted by the use of

mag-netic resonance imaging (MRI) MRI

provides the most detailed

defini-tion of the craniocervical juncdefini-tion,

including the size of rheumatoid

pannus The finding of odontoid

ero-sion on MRI is considered highly

specific for RA and may clarify the

diagnosis in otherwise nonspecific

cases.23

The space-occupying effect of the

periodontoid pannus can be

visual-ized clearly on MRI Both T1- and

T2-weighted images provide

excel-lent visualization of brainstem or

spinal cord contour Increased signal

intensity within the spinal cord on

T2-weighted images may represent

edema, gliosis, or myelomalacia and

has been associated with poor

neuro-logic recovery following surgery.24

The cervicomedullary angle can

be measured on MRI by drawing a

line along the anterior aspect of the

cervical spinal cord and the medulla

This angle normally is between 135°

and 175° (Figure 6) With progressive

craniocervical disease, the

brain-stem angulates ventrally over the

displaced odontoid process, leading

to increased obliquity of the

cervi-comedullary angle One study

re-ported a 100% correlation between a

cervicomedullary angle <135° and

neurologic signs of cervicomedullary

compression, myelopathy, or C2

ra-diculopathy.25

The utility of flexion-extension

or so-called dynamic MRI evaluation

has been debated.26A theoretic

ad-vantage is the ability to identify,

when the patient is in the neutral

position, potentially significant

dy-namic cord compression before

sta-tic compression is apparent One study suggested that cord diameter

in cervical flexion <6 mm is a risk factor for neurologic deficit.27 How-ever, a theoretic risk of sudden death exists as a result of prolonged cervi-cal flexion in a patient with severe instability; therefore, the recommen-dation for routine dynamic MRI in these patients may need to change

Natural History

Understanding of the natural

histo-ry of the rheumatoid cervical spine

is limited Most studies have been handicapped by small sample size or retrospective design Oda et al28 re-viewed the records of 49 patients fol-lowed radiographically for a mini-mum of 5 years and identified a progressive pattern of cervical sub-luxations The earliest instability pattern is most often a reducible at-lantoaxial subluxation.6 Transition from reducible subluxation to an ir-reducible subluxation often ac-companies atlantoaxial impaction, which appears, on average, 6 years after atlantoaxial subluxation.11 Sub-axial subluxation occurs less

fre-quently than do the other two pat-terns and generally develops after atlantoaxial impaction.29

Forty percent to 80% of patients with rheumatoid subluxations dem-onstrate radiographic progression.17 Neurologic deficits occur much less frequently and do not correlate well with radiographic progression.17 Rana30retrospectively reviewed the records of 41 patients with atlanto-axial subluxation who were followed for 10 years; 61% of patients had no change, 27% had progressive sublux-ation, and 12% had improvement at final follow-up Boden et al20 retro-spectively reviewed the records of 73 patients followed for an average of 7 years In 31 patients, all treated non-surgically, no significant neurologic deficit developed during the observa-tion period Of the 42 patients (58%)

in whom paralysis developed, 7 were managed nonsurgically Six of the

Figure 5

Lateral radiograph demonstrating subaxial subluxation across the C3 through C5 segments, with associated facet joint erosions and spinous process changes

Figure 6

Midsagittal T-2-weighted MRI scan of the craniocervical junction in a patient with atlantoaxial impaction The cervicomedullary angle (CMA) is defined by the angle subtended between a line parallel to the long axis

of the brainstem (a) and a line parallel

to the cervical spinal cord (b)

Trang 7

seven patients treated nonsurgically

experienced progressive neurologic

deterioration, and all seven died

within 4 years of the onset of

paral-ysis, five from cord compression Of

the 35 patients who underwent

sur-gery, 25 (71%) experienced marked

neurologic improvement

Despite the general impression

that rheumatoid involvement of the

cervical spine does not contribute to

increased mortality in this patient

population, the high rate of sudden

death observed in patients with

un-treated myelopathy argues against

this conclusion.31When myelopathy

appears, the mortality rate increases

dramatically; without surgery, most

patients die within 1 year.32An

au-topsy study of 104 patients with RA

identified 11 with atlantoaxial

sub-luxation and notable spinal cord

compression; most had experienced

sudden death.33 This finding

sug-gests that the mortality rate from

cervical spine instability in patients

with RA may be as high as 10%

Grading systems have been

devel-oped to assess disease severity as well as treatment outcomes The Ranawat grading system is widely used in clinical studies but has been criticized for lacking the ability to discriminate mild degrees of myel-opathy34(Table 3) The Zeidman and Ducker modification of the Nurick myelopathy scale provides more dif-ferentiation, with an assessment of gait and hand function, and may be more useful10(Table 4) However, its failure to account for severity of ra-diculopathic complaints

significant-ly limits this system for guiding sur-gical decision making

Risk Factors and Predictors of Recovery

Several risk factors for progression of atlantoaxial subluxation have been suggested, including male sex, rheu-matoid factor seropositivity, higher initial C-reactive protein level, presence of subcutaneous nodules, and advanced peripheral joint dis-ease, specifically rapid loss of carpal

height.6,18,28,36,37HLA-DR4 and B-27 seropositive antibodies do not ap-pear to be significant risk factors.6 Whether corticosteroid treatment represents an independent risk fac-tor remains controversial.38Patients with any degree of atlantoaxial im-paction or cord compression on MRI are at significant risk of neurologic injury and should be considered strong candidates for prophylactic decompression and/or stabiliza-tion.37,39

Plain radiography is an efficient and inexpensive means of monitor-ing disease progression Boden et

al20found that the posterior atlanto-dental interval and subaxial sagittal canal diameter correlated with the presence and severity of paralysis, whereas the anterior atlantodental interval did not Using a standard tube distance of 6 feet (1.8 m), 96%

of patients with atlantoaxial sublux-ation and paralysis demonstrated a posterior atlantodental interval≤13

mm In contrast, 60% of the same patients had an anterior atlantoden-tal interval <9 mm All patients with subaxial subluxation and paralysis demonstrated a subaxial sagittal ca-nal diameter≤13 mm

Boden et al20found the posterior atlantodental interval to be a predic-tor of surgical outcome Postopera-tively, no patient experienced signif-icant neurologic recovery when the preoperative posterior atlantodental interval measured <10 mm In pa-tients with isolated atlantoaxial sub-luxation, a posterior atlantodental interval of at least 10 mm predicted improvement of at least one Rana-wat class In the setting of atlanto-axial subluxation and atlantoatlanto-axial impaction, recovery required a pre-operative posterior atlantodental in-terval≥13 mm All patients with a preoperative posterior atlantodental interval and subaxial sagittal canal diameter measuring at least 14 mm experienced complete motor recov-ery

Multiple studies have suggested that the degree of preoperative

neu-Table 4

Zeidman and Ducker Modification of Nurick Grading Scale for

Myelopathy10

Grade Radiculopathy Myelopathy Gait Hand Function

0 Present Absent Normal Normal

I Present Present Normal Slight

II Present Present Mildly abnormal Functional

III Present Present Severely abnormal Unable to button

IV Present Present With assistance

only

Severely limited

V Present Present Nonambulatory Useless

Table 3

Ranawat Grading Scale for Myelopathy35

II Weakness, hyperreflexia, altered sensation

IIIA Paresis and long-tract signs, ambulatory

IIIB Quadriparesis, nonambulatory

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rologic deficit is also a strong

predic-tor of postoperative neurologic

re-covery.40,41Factors with no apparent

predictive value in terms of recovery

include age, sex, duration of

paraly-sis, and preoperative anterior

atlan-todental interval.42

Nonsurgical Treatment

Patients with rheumatoid

involve-ment of the cervical spine benefit

from early, aggressive medical

inter-vention as well as regular follow-up

Because of the prevalence and early

appearance of cervical spine

involve-ment, screening cervical spine

radio-graphs should be considered in all

patients with RA Regular follow-up

radiography should then be

per-formed in patients with any

evi-dence of cervical spine involvement,

especially when they have advanced

peripheral joint disease At least one

study has demonstrated that early,

aggressive combination

disease-modifying antirheumatic drug

ther-apy can prevent or delay

develop-ment of atlantoaxial subluxation.43

Soft cervical collars are

appropri-ate treatment of symptomatic

pa-tients with relatively minor

occipi-tocervical pain and may occasionally

represent the treatment of choice in

elderly or debilitated patients who

are poor surgical candidates.44

Unfor-tunately, orthoses may be

problem-atic in patients with

temporoman-dibular joint involvement Although

they offer excellent relief of

symp-tomatic neck pain, soft cervical

col-lars do not provide much limitation

of motion and probably do not alter

the natural history of cervical spine

disease.5Progressive neurologic

de-terioration has been observed in

pa-tients with spinal cord myelopathy

treated with a soft collar.20

A comprehensive program of

pa-tient education, physical therapy,

collars, practical aids, symptomatic

treatment, and disease-modifying

antirheumatic drugs achieves

signif-icant (P < 0.001) lasting pain relief in

most patients.45Patients should be

taught to avoid cervical flexion

Physical therapy should focus on isometric strengthening of neck muscles and overall postural train-ing Narcotic medication may be ap-propriate for short-term relief of acute pain, but when chronic

narcot-ic use is required for pain relief, then surgical treatment should be consid-ered

Surgical Management

The goals of surgical treatment of rheumatoid disease of the cervical spine are to achieve spinal stability through a solid fusion and to decom-press involved neural structures

The most commonly accepted indi-cations for surgical treatment of RA

in the cervical spine include neuro-logic deterioration and intractable pain with spinal instability (Table 5)

In addition, Boden et al20 proposed surgery, even without neurologic findings, when patients demonstrate one of three radiographic risk fac-tors: (1) atlantoaxial subluxation with a posterior atlantodental inter-val≤14 mm; (2) atlantoaxial impact represented by odontoid migration

≥5 mm rostral to McGregors’s line;

or (3) subaxial subluxation with sag-ittal canal diameter≤14 mm

Patients with RA are a challenging surgical population Those requiring

surgery suffer from a systemic illness and may be significantly malnour-ished and debilitated In addition to cervical spine disease, micrognathia and temporal mandibular disease make standard intubation difficult and anesthesia hazardous Excessive trauma caused by standard intuba-tion has been associated with a 14% incidence of upper-airway obstruc-tion following extubaobstruc-tion.46This rate can be reduced to 1% with fiberoptic assistance Skin lesions and corticos-teroid use notably increase the rate of wound complications and infections Poor structural bone quality may ren-der standard surgical fixation tenu-ous and unreliable

Atlantoaxial Subluxation

When atlantoaxial subluxation is reducible, a variety of posterior fu-sion techniques are possible, includ-ing Gallie or Brooks wirinclud-ing, Magerl transarticular screws, or Harms C1-2 lateral mass fixation.4 Fixa-tion strategies incorporating Magerl screws appear to provide

particular-ly stable fixation; a fusion rate of 95% has been reported with use of transarticular screws.47The need for postoperative halo-vest stabilization depends on the quality of surgical fixation

When atlantoaxial subluxation is nonreducible, transarticular screw

Table 5 Indications for Surgery in Patients With Rheumatoid Arthritis With Cervical Spine Involvement

Progressive neurologic deficit (eg, weakness, gait disturbance, loss of fine motor coordination)

Mechanical neck pain unresponsive to nonnarcotic pain medication (in the setting of radiographic evidence of AAS, AAI, or SAS)

Radiographic risk factors of impending neurologic injury PADI≤14 mm in the setting of AAS

AAI represented by odontoid migration≥5 mm rostral to McGregor’s line Sagittal canal diameter≤14 mm in patients with SAS

Any degree of AAI or cord stenosis

A cervicomedullary angle <135°

AAI = atlantoaxial impaction, AAS = atlantoaxial subluxation, PADI = posterior atlantodental interval, SAS = subaxial subluxation

Trang 9

fixation in combination with C1

laminectomy should be considered,

especially when the patient has

pos-terior cord compression An

occipi-tocervical fusion is an alternative

MRI studies revealing significant

pannus resorption after spinal

stabi-lization indicate that, if bone is not

impinging anteriorly on the cord,

then odontoid resection may be

un-necessary.48 Sublaminar wire

fixa-tion is contraindicated when

atlan-toaxial subluxation cannot be

reduced

Atlantoaxial Impaction

Because the risk of neurologic

in-jury is high with atlantoaxial

impac-tion, early surgery following

identi-fication of this condition has been

recommended.41 Neurologic deficit

or evidence by MRI of cord

compres-sion is a strong indication for

sur-gery.13The use of preoperative halo

traction has been recommended to

reduce the degree of atlantoaxial

im-paction and obviate foramen

mag-num decompression or odontoid

re-section An occipitocervical fusion

can be performed using wires or

screws attached to the occiput below

the superior nuchal line and

con-nected to fixation in the subaxial

spine (Figure 7) When traction is

un-successful, symptomatic

decompres-sion with a transoral odontoid

resec-tion or C1 laminectomy, along with

posterior stabilization, is required.4

An alternative technique for

multi-level instability including

atlantoax-ial impaction is a long rod loop fixed

with occipital and cervical laminar

wires without attempted fusion

Subaxial Subluxation

Evidence from clinical studies

suggests that patients with subaxial

subluxation and neurologic deficits,

or asymptomatic patients with a

subaxial canal diameter ≤14 mm,

should be considered for surgical

sta-bilization.20 Preoperative halo-vest

immobilization can provide

tempo-rary stabilization and reduction of

displacement, often with immediate

improvement in neurologic defi-cits.49Reducible subluxations can be fused anteriorly or posteriorly, but optimal treatment of irreducible subluxations is anterior decompres-sion and fudecompres-sion.23,34Posterior fusion should be strongly considered fol-lowing any laminectomy50 (Figure 8) The extent of fusion required is often not apparent but is a critical decision because RA is progressive

Instability patterns can occur in combination at multiple levels of the cervical spine When atlantoax-ial subluxation and subaxatlantoax-ial sublux-ation occur concurrently, fusion should be extended to the lowest in-volved level to minimize the risk of junctional degeneration.51Some in-vestigators recommend more exten-sive fusions, at times including the entire cervical spine and extending caudally to T1 or T2.52,53

The extent of postoperative im-mobilization depends on the type of surgical fixation used Wiring tech-niques typically require rigid

immo-bilization in a halo-vest or four-poster orthosis for 8 to 12 weeks.5 Following plate and screw fixation, a cervical orthosis may be sufficient New plate, rod, and screw instru-mentation techniques with less

rig-id fixation appear to have improved fusion rates and maintenance of alignment but may increase the risk

of neurovascular injury

Outcomes of Surgery

Over the past 10 years, outcomes of surgery on the rheumatoid cervical spine have improved markedly, largely because of earlier diagnosis of myelopathy and surgical referral.54 More aggressive medical manage-ment of RA and a decrease in corti-costeroid use may be additional con-tributing factors

Most studies favor surgery over nonsurgical management for pa-tients with neurologic deficits.55,56 One of the strongest predictors of postoperative neurologic recovery

Figure 7

A 56-year-old woman with rheumatoid arthritis and atlantoaxial impaction

A,Preoperative midsagittal T2-weighted MRI scan demonstrating penetration of the odontoid through the foramen magnum with impingement on the brainstem

B,Postoperative radiograph demonstrating reduction of the occipitoatlantoaxial relationship and occipitocervical instrumented fusion using a plate-and-screw system

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appears to be preoperative

neurolog-ic status Nonambulatory patients

demonstrate higher complication

rates and lower overall survival.57

Boden et al20reported a retrospective

review of the records of 35 patients

and found that all Ranawat class II

patients had notable neurologic

im-provement after surgery compared

with 62% of class III patients

Casey et al58reported results from

their prospective study of 116

pa-tients with atlantoaxial impaction

Transoral odontoid resection was

performed for anterior bony

com-pression; otherwise, laminectomy

and instrumented fusion were done

A higher revision surgery rate was

noted in patients without fusion to

the occiput Occipitocervical fusions

failed because subaxial instability

below the level of fusion progressed

Satisfactory results were more

like-ly in patients with better

preopera-tive neurologic function Overall,

marked neurologic improvement

oc-curred in 45% and significant pain

relief in 97% of patients A high rate

of perioperative complications was attributed to the general debility of the patient population; the perioper-ative mortality rate was 10%

Surgical results for subaxial sub-luxation may be less favorable

Olerud et al52reported a small retro-spective study of 16 patients with subaxial subluxation and varying de-grees of myelopathy who underwent anterior and/or posterior decompres-sion and fudecompres-sion Although neck pain was typically relieved, arm pain im-proved less reliably Patients with myelopathy had the worst prognosis, and four of five with severe myelop-athy died within 3 months of surgery

The authors recommend early sur-gery for subaxial subluxation, before significant myelopathy develops

The overall complication rate is markedly increased in patients with

RA Wound complications, includ-ing infection, may occur in up to 25% of patients Generalized os-teopenia correlates with systemic disease activity and compromises fixation strength, leading to

in-creased rates of instrumentation fail-ure.52Successful outcomes are pred-icated on achieving solid fusions, and pseudarthrosis has been associ-ated with a less favorable clinical re-sult because of persistent

instabili-ty.20 Recurrent instability in the form of subaxial subluxation at more caudal levels may occur either

as a result of the natural progression

of the disease or as a result of in-creased stress from an adjacent fu-sion Current perioperative

mortali-ty rates have been reported to be between 5% and 10%, with in-creased rates in patients having coin-cident cardiovascular disease or at-lantoaxial impaction.34,41,58,59

Summary

In most patients with RA, neck pain occurs without significant

neurolog-ic defneurolog-icit Multimodality therapy, in-cluding patient education, physical therapy, and active medical treat-ment, can be very effective in con-trolling symptoms and limiting

dis-Figure 8

A,Preoperative lateral cervical radiograph of a 46-year-old woman with RA myelopathy and subaxial subluxation of C4 on C5

B, Preoperative sagittal T1-weighted MRI scan revealing cervical cord compression at level of subluxation C, Postoperative

radiograph following laminectomy and instrumented fusion using C1-2 transarticular screws and lateral mass screws from C3

to C5

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