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Spectrophotometric titration The binding of ThDP to apoTK, and the formation of a catalytically active holoenzyme, is accompanied by the appearance of a new absorption band in the 290–34

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Donor substrate regulation of transketolase

Olga A Esakova1, Ludmilla E Meshalkina1, Ralph Golbik2, Gerhard Hu¨bner2and German A Kochetov1

1 A N Belozersky Institute of Physico-Chemical Biology, Moscow State University, Moscow, Russia; 2 Martin-Luther-University Halle-Wittenberg, Halle/Saale, Germany

The influence of substrates on the interaction of

apotrans-ketolase with thiamin diphosphate was investigated in the

presence of magnesium ions It was shown that the donor

substrates, but not the acceptor substrates, enhance the

affinity of the coenzyme either to only one active center of

transketolase or to both active centers, but to different

degrees in each, resulting in a negative cooperativity for

coenzyme binding In the absence of donor substrate,

neg-ative cooperativity is not observed The donor substrate did

not affect the interaction of the apoenzyme with the inactive

coenzyme analogue, N3¢-pyridyl-thiamin diphosphate

The influence of the donor substrate on the coenzyme–

apotransketolase interaction was predicted as a result

of formation of the transketolase reaction intermediate 2-(a,b-dihydroxyethyl)-thiamin diphosphate, which exhib-ited a higher affinity to the enzyme than thiamin diphos-phate

1 The enhancement of thiamin diphosphate’s affinity

to apotransketolase in the presence of donor substrate is probably one of the mechanisms underlying the sub-strate-affected transketolase regulation at low coenzyme concentrations

Keywords: 2-(a,b-dihydroxyethyl)-thiamin diphosphate; regulation of enzyme activity; spectrophotometric titration; thiamin diphosphate; transketolase

Transketolase (TK, EC 2.2.1.1), containing divalent cations

and thiamin diphosphate (ThDP) as cofactors, catalyzes

one of the key reactions of the pentosephosphate pathway

in carbohydrate transformation, namely the cleavage of a

carbon–carbon bond adjacent to a carbonyl group in

ketoses (donor substrates) with subsequent transfer of a

two-carbon unit to aldoses (acceptor substrates) [1] The TK

enzyme is a homodimer with two active centers located at

the interface between the contacting surfaces of the

mono-mers The active centers are characterized by the same

enzymatic activity, regardless of the divalent cation used as a

cofactor A negative cooperativity in ThDP binding is

observed in the presence of calcium ions [2–5] However,

contrasting data have been published regarding the affinity

of the coenzyme to the apoenzyme of TK (apoTK) in the

presence of magnesium ions

negative cooperativity, albeit slightly pronounced [5], while

others call into question the nonequivalency of the enzyme’s

active centers on ThDP binding [6,7]

ThDP–apoTK binding requires at least a two-step mechanism [8]

TKþ ThDP ƒƒƒƒ! TKThDP ƒƒƒƒƒƒƒƒ ƒƒƒƒ!kþ1

k 1

TK-ThDP ðScheme 1Þ The first step, fast and easily reversible, yields an inter-mediate: a catalytically inactive, primary TKÆÆÆThDP com-plex The second step is slow and accompanied by conformational changes necessary for the formation of the catalytically active holoenzyme, TK*-ThDP The initially identical TK active centers become nonequivalent in the course of ThDP binding It has been inferred [9] that the nonequivalency of the TK active centers in coenzyme binding is determined by the increase of the backward conformational transfer rate constant (k)1in Scheme 1) for the one active center with respect to the other The X-ray data have shown that the structures of apoTK and holoenzyme of transketolase (holoTK) differ in the position

of two loops in the two subunits (residues 187–198 and 383–394) – they are relatively flexible in the apoenzyme and structured in the holoenzyme These two loops are charac-terized by high mobility, and in holoTK they directly contact the coenzyme [10–12] It cannot be ruled out that the interdependent counter-phase movement of these loops determines the alternative destabilization of the secondary complexes (TK*-ThDP in Scheme 1) of the TK active centers with the coenzyme [9]

As already known, 2-(a,b-dihydroxyethyl)-thiamin diphosphate (DHEThDP) is an intermediate of the TK reaction for donor substrate transformation According to the data obtained by X-ray crystallography [13], there are additional bonds of DHEThDP to amino acid residues in

Correspondence to G A Kochetov, A N Belozersky Institute of

Physico-Chemical Biology, Moscow State University, 119992,

Moscow, GSP-2, Russia Fax: +7 95 939 31 81,

Tel.: +7 95 939 14 56, E-mail: kochetov@genebee.msu.su

Abbreviations: apoTK, transketolase apoenzyme; DHEThDP,

2-(a,b-dihydroxyethyl)-thiamin diphosphate; holoTK, transketolase

holoenzyme; HPA, hydroxypyruvic acid; ThDP, thiamin diphosphate;

TK, transketolase from Saccharomyces cerevisiae; X5P, xylulose

5-phosphate.

Enzyme: transketolase (EC 2.2.1.1).

(Received 29 June 2004, revised 10 August 2004,

accepted 3 September 2004)

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the active center of the enzyme compared to ThDP The

enzymatically generated intermediate displays a higher

affinity to apoTK than ThDP [14] Based on these data,

it is possible to assume that in the presence of the donor

substrate, ThDP will possess higher affinity to apoTK The

present study is devoted to elucidation of a possible

regulatory role of donor substrates in holoTK formation

Similarly to other ThDP

capable of using, as cofactors, various bivalent cations

Certain kinetic properties of TK are known to depend on

bivalent cations, which are used as cofactors [5] Comparing

the same kinetic characteristics obtained for

ThDP-depend-ent enzymes with diverse bivalThDP-depend-ent cations as cofactors is a

matter of undebatable interest Only Mg2+has been used as

a cofactor in the studies of all ThDP-dependent enzymes

(including, until recently, TK) This is the reason why Mg2+

was also used as a cofactor in this work

Materials and methods

Materials

ThDP and glycyl-glycine were purchased from Serva

Electrophoresis

acid (HPA), xylulose 5-phosphate (X5P), MgCl2, racemic

glyceraldehyde and ribose 5-phosphate were from Sigma

Chemical Co; N3¢-pyridyl-ThDP was synthesized as

des-cribed previously [15] Other chemicals were of the highest

quality commercially available

Purification of TK

Recombinant bakers yeast TK, with a specific activity of

22 UÆmg)1, was isolated by a method described previously

[16] The enzyme was obtained as apoTK and was

determined to be homogeneous by SDS/PAGE The TK

concentration was determined spectrophotometrically,

using an A1%1 cmof 14.5 at 280 nm [17]

Measurement of TK activity

The activity of TK was determined spectrophotometrically

at 25C by measuring the rate of NAD+reduction in a

coupled system with glyceraldehyde-3-phosphate

dehydro-genase [1]

Determination of ThDP concentration

ThDP concentration was determined

spectrophotometri-cally at 272.5 nm using a molar extinction coefficient of

7800 [18]

Absorption spectra

Absorption spectra were recorded at 25C using an

AMINCO DW 2000 spectrophotometer

Instru-ments, Rochester, NY, USA) (optical path length of

1 cm) Medium components were 1 mgÆmL)1TK, 50 mM

glycyl-glycine buffer (pH 7.6), 2.5 mM MgCl2, 0.5 mM

ThDP or 0.04 mMN3¢-pyridyl-ThDP, and 2.5 mM HPA,

if indicated The difference spectra of holoTK in the

presence or absence of HPA were obtained by subtraction

of the individual spectra of apoTK, ThDP or N3¢-pyridyl-ThDP, and HPA, correspondingly

Spectrophotometric titration The binding of ThDP to apoTK, and the formation of a catalytically active holoenzyme, is accompanied by the appearance of a new absorption band (in the 290–340 nm range), the intensity of which is strictly correlated with the amount of coenzyme bound in the active centers of TK [3,4] This approach was used to determine the affinity of the coenzyme to TK [4,9] and to study the interaction of ThDP with the apoenzyme In this way, the kinetics of the distinct stages during this process were measured [9] In the present study, this method was used to investigate holoTK recon-stitution in the presence of substrates The donor substrate does not provide an essential contribution to the absorption spectrum of holoTK at 320 nm (curves 1 and 2 in Fig 1) Therefore, titration of apoTK with ThDP was carried out at

320 nm The acceptor substrate, however, has no effect on the absorption spectrum of the holoenzyme The registra-tion was conducted in a two-wavelength mode (k¼

320 nm, k¼ 400 nm), using an AMINCO DW 2000 spectrophotometer

ApoTK

7 (3 mL; 0.7 mgÆmL)1) in 50 mM glycyl-glycine buffer, pH 7.6, containing 2.5 mMMgCl2, was added to a quartz cuvette After recording the initial absorption at

320 nm, the first 10 lL (2.3 lM) of ThDP was added

8

and any absorption change was registered The next 10–

40 lL (4.5–452.6 lM) of ThDP

the absorption change No further change in absorption after addition of the final sample of ThDP was used as a sign

of full reconstitution of holoTK

characterizes the amount of holoTK with ThDP bound in two active sites A typical experiment is presented in Fig 2 The influence of the substrate on reconstitution of holoTK

Fig 1.

17 Difference absorption spectra of transketolase from Saccharo-myces cerevisiae (TK) (1 mgÆmL)1) in 50 m M glycyl-glycine buffer,

pH 7.6, containing 2.5 m M MgCl 2 , at 25 °C (1) Holoenzyme of transketolase (holoTK) in the absence of substrate after subtraction of the spectra of the transketolase apoenzyme (apoTK) and thiamin diphosphate (ThDP); (2) holoTK in the presence of 2.5 m M

hydroxypyruvic acid (HPA) after subtraction of the spectra of apoTK, thiamin diphosphate (ThDP) and HPA; (3) complex of TK with N3¢-pyridyl-ThDP after subtraction of the spectra of apoTK and N3¢-pyridyl-ThDP.

Trang 3

was studied when apoenzyme was incubated in the presence

of 2.5 mMHPA and 2.5 mMMgCl2prior to the addition of

ThDP

Reconstitution of the TK–N3¢-pyridyl-ThDP (an inactive

analogue of ThDP) complex, was monitored by a change in

the optical density at 345 nm (absorption maximum of the

band induced as a result of formation of the complex

3 in Fig 1) Spectrophotometric titration was carried out

similarly to the experiments with ThDP, using a

two-wavelength mode (k¼ 345 nm, k ¼ 420 nm), on an

AMINCO DW 2000 spectrophotometer in 50 mM

glycyl-glycine buffer, pH 7.6, containing 2.5 mMMgCl2 Binding

of N3¢-pyridyl-ThDP (Ki¼ 1.3 nM[19]) is impaired in the

presence of 20 mMinorganic sodium diphosphate, resulting

in a decrease of the apparent binding constant In

experi-ments where substrate influence on the reconstitution of

complex TK with N3¢-pyridyl-ThDP was studied, the

enzyme was incubated in the presence of 2.5 mM HPA

and 2.5 mMMgCl2, prior to the addition of analogue

Determination ofKDfor ThDP in the presence and

absence of HPA

Based on the spectrophotometric titration data, the

disso-ciation constants of ThDP binding to each of the enzyme’s

active centers were determined At a saturating

concentra-tion of ThDP, the maximum alteraconcentra-tion in the absorbance at

320 nm corresponds to 100% formation of holoTK The

KDfor ThDP was calculated using the programSCIENTIST

Calculation

12 based on the model for two active centers,

according to Dixon’s method [20]

½holoTK ¼0:5 ½TK  ½ThDPfree

½ThDPfree þ K1

D

þ0:5 ½TK  ½ThDPfree

½ThDPfree þ K2

D :

The concentration of free ThDP was determined according

to the following equation:

½ThDPfree ¼ ½ThDPtotal  ½ThDPbound where [ThDPbound] is equivalent to the concentration of the active centers occupied by ThDP

Determination ofKdfor ThDP in the presence and absence of X5P

The apoTK (1–2 lgÆmL)1) was preincubated at 25C in

50 mM glycyl-glycine buffer, pH 7.6, containing 2.5 mM MgCl2 and 0.1% BSA (for TK stabilization) at different concentrations of ThDP (0.5–120 lM) in the presence or absence of 0.5 mM X5P The reconstitution reaction was allowed to proceed for 90–150 min, which was the time usually required for completion of the process After-wards, the activity of the holoenzyme was measured by adding all the components necessary for determining

TK activity (1 mM sodium arsenate, 0.37 mM NAD+,

3 U glyceraldehyde 3-phosphate dehydrogenase, 3.2 mM dithiothreitol, 1 mM ribose 5-phosphate) The changes in optical density at 340 nm were measured as described above

Based on the data, the Kd for ThDP in the presence and absence of X5P was determined using the program SCIENTIST Calculation

m¼0:5 Vmax ½ThDP

½ThDP þ K1 þ0:5 Vmax ½ThDP

½ThDP þ K2 :

Results

Influence of the donor substrate on the reconstitution

of apoTK with ThDP The influence of the donor substrate on the binding of ThDP to apoTK in the presence of Mg2+, as investigated

by the spectrophotometric titration method, is shown in Fig 3 The affinity exhibited by the two active centers of apoTK to ThDP in the absence of substrate (curve 1) is

Fig 2.

18 Reconstitution of holotransketolase

from apotransketolase (0.7 mgÆmL)1) and

thiamin diphosphate (ThDP) (0–0.453 m M ).

Data of spectrophotometric titration in

50 m M glycyl-glycine buffer, pH 7.6, in the

presence of 2.5 m M MgCl 2 , at 25 C.

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the same in both cases, revealing K1D¼ K2D¼ 5.2 lM

(Table 1) The addition of HPA (an artificial donor

substrate for TK, cleaved in an irreversible manner) caused

a significant increase in the affinities of the two active centers

to ThDP (Fig 3, curve 2 and Table 1); moreover, these

affinities were exhibited to different degrees: for one active

center, the dissociation constant (K2

D) decreased to 1.6 lM, while the affinity of the other increased to such an extent

that K1Dcould not be determined under the experimental

conditions used The affinity of the first active center of TK

for ThDP could not be estimated by the method employed

herein because the affinity was too high: all the ThDP added

to the sample was stoichiometrically bound to the first active

center Thus, in the presence of HPA, the affinity of apoTK

to ThDP increased and a negative cooperative effect on coenzyme binding was induced that is not observed in the absence of substrate (Table 1)

In order to study the influence of the native donor substrate, X5P (which is cleaved by the enzyme in reversible manner), on the affinity of the coenzyme to apoTK, the enzymatic activity of apoTK was measured after preincu-bation with different concentrations of ThDP in the presence or absence of X5P

are presented in Fig 4 and Table 1 Both active centers of apoTK showed the same affinity to ThDP in the absence of X5P, displaying a Kd of 4.6 lM (curve 1, Fig 4) In the presence of X5P (curve 2, Fig 4) the values K1d¼ 0.22 lM and K2d¼ 4.4 lM for ThDP were determined (Table 1) Thus, the addition of X5P caused a significant increase in ThDP affinity to one of the two TK active centers Consequently, as in the case of HPA, X5P not only increases the affinity of TK to ThDP, but also causes nonequivalency of the enzyme’s active centers in coenzyme binding In contrast, the acceptor substrates (glyceraldehyde and ribose 5-phosphate) exert no influence on the affinity

of the enzyme’s active centers to ThDP (Table 1)

The interaction of N3¢-pyridyl-ThDP with apoTK N3¢-pyridyl-ThDP is an inactive analogue of ThDP, in which the N1¢ atom is replaced with CH [19,21] The presence of the induced band in the difference absorption spectrum on the TK-N3¢-pyridyl-ThDP complex (curve 3, Fig 1) enabled us to measure the binding of this analogue

to the apoenzyme using the spectrophotometric titration method Figure 5 shows the formation of an inactive complex of TK with N3¢-pyridyl-ThDP in the presence or

Table 1 Dissociation constants of thiamin diphosphate (ThDP) of the

two active sites of transketolase from Saccharomyces cerevisiae in the

presence of Mg 2+ , as determined by using spectrophotometric titration

(K D ) and by assaying the holoenzyme activity (K d ) The data were

cal-culated

17 using the program SCIENTIST K D and K d were determined

based on the data presented in Figs 3 and 4, respectively.

Substrate

K1D

(l M )

K2D

(l M )

K1d

(l M )

K2d

(l M )

No substrate 5.2 5.2 4.6 4.6

2.5 m M HPA a 1.6b – –

0.5 m M X5P – – 0.22 b 4.4 b

10 m M glyceraldehyde 5.4 5.4 – –

0.7 m M ribose 5-phosphate – – 4.8 4.8

a In the experiments using hydroxypyruvic acid (HPA), the affinity

of ThDP to TK is so high that the method for determination of K D

for the first active site is not qualified.bIn this case, the value of the

dissociation constant is apparent, i.e the value was determined in

the presence of donor substrate.

Fig 4.

20 Activity of the transketolase holoenzyme (holoTK), reconstituted

at different concentrations of thiamin diphosphate (ThDP) in 50 m M

glycyl-glycine buffer, pH 7.6, in the presence of 2.5 m M MgCl 2 at 25 °C The activity of holoTK was determined as described in the Materials and methods: (1) reconstitution of holoTK without substrate; and (2) reconstitution of holoTK in the presence of 0.5 m M xylulose 5-phos-phate (X5P) The concentrations of TK used are 2 and 1 lgÆmL)1and the ThDP concentrations used ranged from 0 to 20 l M and from 20 to

120 l M , respectively The data were fitted to the TK concentration of

1 lgÆmL)1 The points are obtained experimentally; the lines are cal-culated for a set of parameters presented in Table 1.

Fig 3.

19 Influence of the donor substrate on the formation of transketolase

holoenzyme (holoTK) from the transketolase apoenzyme (apoTK)

(0.7 mgÆmL)1) and thiamin diphosphate (ThDP) in 50 m M glycyl-glycine

buffer, pH 7.6, in the presence of 2.5 m M MgCl 2 , at 25 °C (1) in the

absence of substrate and (2) in the presence of 2.5 m M hydroxypyruvic

acid (HPA) The formation of holoenzyme was observed by the change

in absorbance at 320 nm, as described in the Materials and methods.

The points are obtained experimentally; the lines are calculated for a

set of parameters presented in Table 1 Insertion shows the initial part

of the curves.

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absence of 2.5 mMHPA As shown, this compound had

no influence on the affinity of N3¢-pyridyl-ThDP to TK,

indicating that the donor substrate affects the formation of

the catalytically active holoenzyme, but not the formation of

the catalytically inactive complex of TK with

N3¢-pyridyl-ThDP

Discussion

In the presence of Mg2+, the two active centers of TK have

the same affinity for ThDP (Table 1) Donor substrates,

converted both reversibly (X5P) and irreversibly (HPA),

enhance the affinity of the coenzyme for apoTK In the

presence of any donor substrate during holoTK

reconsti-tution, the affinity for the cofactor ThDP increased together

with the manifestation of a negative cooperativity between

the active sites in this process Research on the influence of

aldoses (glyceraldehyde and ribose 5-phosphate) on the

reconstitution of holoTK have shown that the acceptor

substrate, in contrast to the donor substrate, exerts no

influence on the affinity of the enzyme’s active centers for

ThDP (Table 1)

It is suggested that an enhancement of ThDP affinity for

apoTK in the presence of donor substrates may be

explained by the formation of the TK reaction intermediate,

DHEThDP, which exhibits a higher affinity than ThDP to

TK [14] This conclusion was supported by the experiment

with the inactive coenzyme analogue N3¢-pyridyl-ThDP,

which is similar to the native coenzyme except for the lack of

activity Indeed, with respect to ThDP, the

N3¢-pyridyl-ThDP is a competitive inhibitor of TK [19] and of other

thiamin diphosphate-dependent enzymes [22,23] The

inhi-bition constant of N3¢-pyridyl-ThDP for TK is 1.3 nM[19]

Binding of N3¢-pyridyl-ThDP to the active sites of TK is

accompanied by the appearance of a new absorption band

in the same region of the CD spectrum, in which it appears

on the interaction of TK with the native coenzyme [19,21] This fact points to the competent (correct) binding of this analogue to TK and indicates the same microenvironment

of the analogue in the active site, as in the case of ThDP The X-ray crystallography structure of the TK-N3¢-pyridyl-ThDP complex shows that after reconstitution, the ana-logue displays the same V-conformation typical of ThDP in the holoenzyme In the active site of TK from Saccharo-myces cerevisiae, N3¢-pyridyl-ThDP interacts with con-served amino acid residues, as does the native coenzyme, except for a hydrogen bond emerging between the first nitrogen atom of the aminopyrimidine ring of ThDP (lacking in the analogue) and Glu418 [24] This distinction

is, in fact, the reason for the inactivity of the analogue The donor substrate has no effect on the binding of this analogue to TK (Fig 5)

Conversion of HPA resulted in a significant increase in the affinities of the two active centers to ThDP; however, the affinities of the two centers were different (Table 1) These data are in agreement with the results of X-ray analysis, which show the appearance of DHEThDP in both active centers of TK [13] On the other hand, the nonequivalency

of the enzyme’s active centers in the intermediate complex suggests that different states of the active centers occur during catalysis

15

Consequently, the influence of the donor substrate on the reconstitution of the holoenzyme is dependent on the ability

of the reconstituted complex to form DHEThDP or the corresponding intermediate of any analogue We were able

to predict the data obtained as the same effect has been shown on the pyruvate dehydrogenase complex from Escherichia coli[22,23] Moreover, the efficient reconstitu-tion of holoTK in the presence of donor substrate has previously been reported

result was the appearance of the negative cooperativity on the binding of ThDP to apoTK in the presence of the donor substrates

In the presence of X5P (a reversible donor substrate), the affinity of ThDP increases in one of the two active centers (Table 1), i.e the reaction intermediate DHEThDP, having

a high affinity to the enzyme, is formed at one active site only Thus, the cooperativity as a result of this half-of-the-site reactivity becomes apparent The data obtained do not contradict previous results on the reversible converted donor substrate protection of only one active site from the chemical modification [26]

When the concentration of ThDP in the cell is low, only

a proportion of TK is represented by the holoenzyme (the catalytically active form of the enzyme) The donor substrate increases the amount of holoTK by increasing, for example, the affinity of ThDP for apoTK As a result, the total TK activity increases

Hence, based on the data obtained, a mechanism may be postulated for the efficient regulation of TK by the donor substrate at a low concentration of coenzyme

The proposed mechanism explains various data reporting the coenzyme’s affinity to apoTK in the presence of magnesium ions in the literature [5–7,25] Some authors report a negative cooperativity, albeit slightly pronounced [5], while others call into question the nonequivalency of the enzyme’s active centers on ThDP binding [6,7] All of these

Fig 5.

21 Influence of the donor substrate on the formation of an inactive

complex of transketolase (TK) (0.7 mgÆmL)1) with N3¢-pyridyl-thiamin

diphosphate (ThDP) in 50 m M glycyl-glycine buffer, pH 7.6, in the

presence of 2.5 m M MgCl 2 at 25 °C Curve 1 was measured in the

absence of substrate; curve 2 was measured in the presence of 2.5 m M

hydroxypyruvic acid (HPA) Owing to the high affinity of

N3¢-pyridyl-ThDP to the transketolase apoenzyme (apoTK) (K i ¼ 1.3 n M ) [20],

20 m M inorganic diphosphate was added as described in the Materials

and methods.

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data were received in the absence of the donor substrate and

were correlated with the results obtained Strongly

pro-nounced negative cooperativity on the binding of ThDP to

apoTK [25] was shown in the presence of the donor

substrate and could be explained by the different influence

of the donor substrate on the affinity of the TK active

centers to ThDP

Acknowledgements

This research was supported by a grant from the Russian Foundation

for Basic Research (03-04-49025).

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