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Báo cáo khoa học: Knock-out of the chloroplast-encoded PSI-J subunit of photosystem I in Nicotiana tabacum PSI-J is required for efficient electron transfer and stable accumulation of photosystem I pot

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Thus, PSI-J does not appear to participate directly in binding of Pc or Cyt c6, but plays a role in maintaining a precise recognition site for the N-terminal domain of PSI-F required for

Trang 1

of photosystem I in Nicotiana tabacum

PSI-J is required for efficient electron transfer and stable

accumulation of photosystem I

Andreas Hansson1, Katrin Amann2, Agnieszka Zygadlo1, Jo¨rg Meurer2, Henrik V Scheller1

and Poul E Jensen1

1 Plant Biochemistry Laboratory, Department of Plant Biology, Faculty of Life Sciences, University of Copenhagen, Frederiksberg, Denmark

2 Department Biologie I, Botanik, Ludwig-Maximilians-Universita¨t-Mu¨nchen, Germany

The photosystem I (PSI) complex of higher plants

con-sists of at least 19 different polypeptides [1–3] PSI

mediates light-driven electron transfer from reduced

plastocyanin (Pc) in the thylakoid lumen to oxidized

ferredoxin in the stroma The PSI core in higher plants

contains at least 15 different subunits named PSI-A to

PSI-L, PSI-N to PSI-P Two subunits present in

cyanobacteria, PSI-M and PSI-X, are missing from

plants In addition to the PSI core, higher plants

con-tain a peripheral antenna associated with PSI, also

known as light-harvesting complex I (LHCI), which is mainly composed of four different Lhca proteins The major subunits of PSI, PSI-A and PSI-B, form a heterodimer, which binds the components of the elec-tron-transfer chain: the primary electron donor P700 and the electron acceptors A0, A1 and Fx [1,4,5] The two remaining electron acceptors, FAand FB, are bound

to the PSI-C subunit PSI-C is located towards the stro-mal side of PSI and, together with PSI-D and PSI-E, provides the docking side for soluble ferredoxin [5,6]

Keywords

antenna size; electron transport;

photosynthesis; plastocyanin kinetics;

thylakoid membrane

Correspondence

P E Jensen, Plant Biochemistry Laboratory,

Department of Plant Biology, Faculty of Life

Sciences, University of Copenhagen, 40

Thorvaldsensvej, DK-1871 Frederiksberg C,

Denmark

Fax: +45 35 28 33 33

Tel: +45 35 28 33 40

E-mail: peje@life.ku.dk

(Received 30 August 2006, revised 21

December 2006, accepted 31 January 2007)

doi:10.1111/j.1742-4658.2007.05722.x

The plastid-encoded psaJ gene encodes a hydrophobic low-molecular-mass subunit of photosystem I (PSI) containing one transmembrane helix Ho-moplastomic transformants with an inactivated psaJ gene were devoid of PSI-J protein The mutant plants were slightly smaller and paler than wild-type because of a 13% reduction in chlorophyll content per leaf area caused by an  20% reduction in PSI The amount of the peripheral antenna proteins, Lhca2 and Lhca3, was decreased to the same level as the core subunits, but Lhca1 and Lhca4 were present in relative excess The functional size of the PSI antenna was not affected, suggesting that PSI-J

is not involved in binding of light-harvesting complex I The specific PSI activity, measured as NADP+ photoreduction in vitro, revealed a 55% reduction in electron transport through PSI in the mutant No significant difference in the second-order rate constant for electron transfer from reduced plastocyanin to oxidized P700 was observed in the absence of

PSI-J Instead, a large fraction of PSI was found to be inactive Immunoblot-ting analysis revealed a secondary loss of the luminal PSI-N subunit in PSI particles devoid of PSI-J Presumably PSI-J affects the conformation of PSI-F, which in turn affects the binding of PSI-N This together renders a fraction of the PSI particles inactive Thus, PSI-J is an important subunit that, together with PSI-F and PSI-N, is required for formation of the plast-ocyanin-binding domain of PSI PSI-J is furthermore important for stabil-ity or assembly of the PSI complex

Abbreviations

Chl, chlorophyll; Cyt, cytochrome; LHC, light-harvesting complex; Pc, plastocyanin; PS, photosystem.

Trang 2

In plants, the three low-molecular-mass subunits,

PSI-F, PSI-G and PSI-N, have been implicated in the

interaction between PSI and Pc [7–9] PSI-F contains

one transmembrane helix and is exposed to both the

lumen and the stroma: its rather large N-terminal

domain is situated in the lumen [10], whereas the

C-terminus is in contact with PSI-E on the stromal

side [6] The N-terminal part of PSI-F and luminal

interhelical loops of PSI-A and PSI-B form a docking

site for Pc or cytochrome (Cyt) c6 [11–15] In plants,

which only use Pc as an electron donor to PSI, a

longer N-terminal domain contributes to a helix–

loop–helix motif [10], which specifically enables more

efficient Pc binding and, as a result, two orders of

magnitude faster electron transfer from Pc to P700

[16] PSI-N is unique to eukaryotic PSI and is entirely

located in the thylakoid lumen However, the recently

published structural model of higher-plant PSI based

on a crystal structure at 4.4 A˚ does not reveal the

pres-ence of PSI-N [10], and cross-linking experiments have

shown little interaction between PSI-N and other small

PSI subunits [17]

PSI-J is a hydrophobic low-molecular-mass subunit

composed of 44 amino acids with one transmembrane

helix that is located close to PSI-F [5,10] The

N-termi-nus of PSI-J is located in the stroma, and the

C-termi-nus is located in the lumen [6] In cyanobacteria, PSI-J

binds three chlorophylls (Chls) and is in hydrophobic

contact with carotenoids [5], whereas in plants only

two Chl molecules are bound (Fig 1), which has been

proposed to be important for energy transfer between

LHCI and the PSI core [10]

In cyanobacteria, PSI-J interacts with PSI-F [18] A

psaJ knock-out in Synechocystis PCC 6803 contained

only 20% PSI-F subunit compared with wild-type [19]

The corresponding psaJ knock-out in Chlamydomonas

contained wild-type levels of PSI-F and PSI, and the cells were able to grow photoautotrophically A large fraction of the mutant PSI complexes displayed slow kinetics of electron donation from Pc or Cyt c6 to P700 The absence of PSI-J did not alter the half-lives

of the different kinetic phases, but led to the formation

of two subpopulations of PSI complexes which differed with respect to electron transfer to P700+ One popu-lation behaved like wild-type with fully functional PSI-F, and the other population had characteristics similar to a PSI-F-deficient strain [20] It was conclu-ded that, in 70% of the PSI complexes lacking PSI-J, the N-terminal domain of PSI-F is unable to provide

an efficient binding site for either Pc or Cyt c6and was explained by a displacement of this domain Thus, PSI-J does not appear to participate directly in binding

of Pc or Cyt c6, but plays a role in maintaining a precise recognition site for the N-terminal domain of PSI-F required for fast electron transfer from Pc and Cyt c6to PSI [20]

To determine the role of PSI-J in plants, we gener-ated homoplastomic psaJ knock-outs in tobacco Transplastomic transformants were obtained and ana-lyzed for differences in electron transport and antenna function In contrast with results obtained with PSI-J-deficient Chlamydomonas, the content of PSI was reduced by 20% and the remaining PSI had a decreased in vitro NADP+-photoreduction activity A secondary loss of the luminal subunit, PSI-N, was seen when PSI complexes were analysed and kinetic analysis revealed a large fraction of inactive PSI Thus, we pro-pose a dual function of PSI-J in higher plants; one for assembly of the PSI core complex and the other for integrity and stabilization of a luminal domain invol-ving at least PSI-N and the N-terminal part of PSI-F which is required for efficient electron transfer

Fig 1 Alignment of PSI-J sequences representing cyanobacteria, algae and higher plants In total, 44 full-length PSI-J sequences were aligned using CLUSTAL W In the alignment shown are the sequences from plants [Arabidopsis thaliana (ARATH) and Nicotiana tabacum (TOBAC)], algae [Chlamydomonas reinhardtii (CHLRE) and Porphyra purpurea (PORPU)] and cyanobacteria [Synechcoccus elongatus (SYNEL) and Prochlorococcus marinus (PROMA)] Amino-acid residues involved in Chl binding [W (Trp), E (Glu) and H (His)] are indicated with green arrows Note that the histidine residue is only conserved in cyanobacteria, in agreement with the notion that PSI-J of cyanobacteria is involved in binding three Chls, whereas plant PSI-J only binds two Amino-acid residues making contact with b-carotene [I (Ile) and R (Arg)] are indicated with orange arrows The underlined residues are completely conserved in plants, algae and cyanobacteria.

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Targeted inactivation of the tobacco chloroplast

psaJ gene

To determine the function of PSI-J in plants, we have

taken a reverse genetics approach and constructed a

knock-out allele for targeted disruption of the tobacco

psaJ (Fig 2A) The knock-out allele was introduced

into the tobacco plastid genome by particle

bombard-ment-mediated chloroplast transformation [21]

From 10 bombarded leaf samples, 19 chloroplast

transformants were selected and verified by PCR

and DNA gel blot analysis (data not shown) Two

independent transplastomic lines were subjected to

additional rounds of regeneration on

spectinomycin-containing medium to obtain homoplastomic tissue In

Fig 2B, an example of PCR verification of one of the

homoplastomic psaJ knock-out lines is shown

Nor-thern blot analysis was also performed to demonstrate

that the psaJ gene was disrupted by the insertion of

the aadA cassette (Fig 2C) Finally, PSI particles (PSI

holocomplexes) were prepared from wild-type and

plants disrupted in the psaJ gene and subjected to

immunoblot analysis An antibody originally raised

against electroeluted PSI-I [22] and subsequently found

to recognize both PSI-I and PSI-J [17] was used to confirm the absence of PSI-J protein from the mutant (Fig 2D) Altogether this clearly shows that the psaJ gene has been disrupted causing elimination of the PSI-J protein

Plants devoid of PSI-J are fully viable and fertile but display a clear phenotype

When plants lacking PSI-J were transferred to soil, they grew photoautotrophically and were fully fertile (Fig 3) The original transformed lines were self-polli-nated, and the seeds produced were germinated directly on soil The resulting offspring displayed the same characteristics as the first generation (results not shown)

Tobacco plants lacking PSI-J were slightly smaller than wild-type plants (Fig 3) This was observed for plants grown in either a growth-chamber or a green-house and suggests that elimination of the PSI-J pro-tein from PSI affects the overall photosynthetic performance

Besides being slightly smaller than wild-type, the psaJ knock-out plants were visibly paler Pigment

4 7

16 17 34 45 55 105 kDa

564

947 831 1375 1584 2027/1904 3530

3.7 kb

1.9 kb

250-bp

ScaI

(ScaI/SmaI)

(PsaJ)

(HindIII/ScaI)

AadA

ΔJ ΔJ

Fig 2 (A) Construction of the plastid trans-formation vector Schematic map of the 2.53-kb chloroplast genomic fragment con-taining the psaJ gene The aadA cassette is inserted in a ScaI site within the coding sequence of psaJ in the sense orientation (B) PCR confirmation that the aadA cassette has inserted in the psaJ gene M, marker;

1, total DNA from transgenic plant as tem-plate; 2, plasmid DNA used to transform the plants as template; 3, total DNA from wild-type tobacco as template (C) Northern blot showing that there is no wild-type-sized psaJ mRNA (as a loading control the left hand side shows the stained and the right hand side the actual Northern blot) (D) Immunoblot analysis of PSI complexes from wild-type and DpsaJ plants The panel on the left is the stained gel, and the panel on the right is an immunoblot using an antibody directed against a mixture of PSI-I and PSI-J The arrow indicates PSI-J.

Trang 4

extraction of leaf discs using boiling ethanol and

spec-trophotometric quantification showed a 13% reduction

in the content of Chl per leaf area compared with

wild-type (Table 1) Estimated from the leaf extracts,

the Chl a⁄ b ratio was 2.95 in the psaJ knock-out leaves

compared with 3.25 in the wild-type leaves This

differ-ence was caused by a bigger decrease in Chl a (15%

less) and a smaller decrease in Chl b (6% less) in the

mutant (Table 1) Similar measurements on several

independent preparations of thylakoids also revealed a

lower Chl a⁄ b ratio in the mutant, although the

abso-lute numbers were different The reduced Chl a⁄ b ratio

suggests that plants without PSI-J either have less of

the core complexes or increased content of the Chl b

containing peripheral antenna

To monitor the photosynthetic electron flow through PSI during steady-state photosynthesis in vivo, we esti-mated the redox state of P700 in the light by measuring oxidation of P700 in the leaf as DA at 810 minus 860 nm

as described in Experimental procedures The light dependence of the P700 oxidation ratio (DA⁄ DAmax) was examined, and, in both the wild-type and DPSI-J plants, P700 oxidation was almost linearly related to increasing light intensity However, in the DPSI-J plants the redox state of P700 was higher than wild-type at all light intensities (Fig 4) This means that P700 stays more oxidized in the absence of PSI-J This usually sug-gests that electron donation from Pc to P700+is affec-ted Comparison of the curves suggested that about 20% of the PSI has very inefficient electron donation from Pc in the absence of PSI-J

Table 1 Chl a and b content per leaf area, Chls per PSI reaction centre, PSI activity, and the plastoquinone redox state under different light conditions.

[lmol NADP + Æs)1Æ(lmol P700))1]

a Mean of three independent thylakoid preparations *P < 0.05; ***P < 0.001.

Fig 3 Phenotype of homoplastomic DpsaJ plants grown under

growth chamber conditions Note that the DpsaJ plant is slightly

smaller and paler than the wild-type plant.

Light intensity ( μE)

0.0 0.1 0.2 0.3 0.4 0.5 0.6 0.7

WT ΔJ

Fig 4 P700 oxidation state in leaves of wild-type and DpsaJ plants Light response of P700 oxidation ratio (DA ⁄ DA max ) in leaves of wild-type (WT) and DPSI-J plants (DJ) All data points are mean ± SD (n ¼ 3), but in some cases the error bars are covered

by the marker.

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The PSII excitation pressure (estimated as 1–qP) was

subsequently measured in vivo in the growth chamber

under the light conditions to which the plants were

adapted Under these conditions 1–qP was increased

1.7-fold in the plants lacking PSI-J (Table 1),

indica-ting that the PSII excitation pressure was significantly

increased as the result of a more reduced

plastoqui-none pool Measuring 1–qP under greenhouse

condi-tions on either a cloudy or a sunny day confirmed the

higher excitation pressure in plants without PSI-J,

especially under conditions where the plants have to

cope with higher light intensities (Table 1) This is in

agreement with a restriction of electron flow at PSI

The amount of PSI is reduced in the absence

of PSI-J

To analyze the content of PSI further, the amount of

P700 was determined in solubilized thylakoids using

flash-induced absorption changes in P700 at 834 nm

The number of Chls per P700 reaction centre was

esti-mated to be 435 ± 17 for wild-type and 531 ± 32 for

thylakoids from the PSI-J-less plants (Table 1) Similar

values were obtained using chemical oxidation and

reduction of P700 (data not shown) This clearly

indi-cates an  20% reduction in P700 in plants lacking

PSI-J

To investigate this by an independent method and

also to analyze whether the absence of PSI-J caused

changes in photosynthetic complexes, we performed

immunoblot analysis of thylakoid proteins using a

variety of antibodies directed against subunits of the

PSI, PSII and ATP synthase complexes (Fig 5) The

gels were loaded with proteins corresponding to equal

amounts of Chl This analysis showed that subunits of

PSII and the ATP synthase were present in amounts

equal or close to the amounts found in wild-type

(Fig 5) In contrast, the amounts of the analysed

sub-units of the PSI core were consistently reduced by 15–

25% compared with the wild-type (Fig 5A) This

shows that there are fewer PSI core complexes in the

absence of PSI-J and confirms the spectroscopic

deter-mination of Chl per P700 above Together this

sug-gests that PSI-J is implicated in stable accumulation of

PSI because of a requirement for this subunit either

during assembly or subsequently for the stability of

the PSI complex

To analyse the effect of the absent PSI-J in more

detail, immunoblot analysis of PSI particles purified

using sucrose density gradient centrifugation was also

performed (Fig 6) This revealed that most of the

sub-units analysed were present in the complex of the

mutant in amounts similar to that found in the

wild-type This included the PSI-F subunit, which is known

to be located next to PSI-J in the complex [5,10] Sur-prisingly, the only subunit that was reduced in content was PSI-N, which was reduced to 30–40% of the wild-type level

Fig 5 Immunoblot analysis of proteins in thylakoids prepared from DpsaJ and wild-type plants (A) Content of a range of PSI core pro-teins and ATP synthase (CF 1 -b) Thylakoids were prepared from leaves from two to four different wild-type or DpsaJ plants A dilu-tion series containing protein corresponding to 1.0, 0.5, and 0.25 lg Chl of the wild-type and 1.0–0.5 lg Chl of the mutant was separated by SDS ⁄ PAGE, blotted and analyzed with the antibodies indicated Wild-type (WT) and DpsaJ dilutions were run side by side, and, for each antibody, the resulting signal was quantified using the LabWorks software as described in Experimental proce-dures Quantification was performed on two independent prepara-tions of both wild-type and DpsaJ thylakoids (B) Content of light-harvesting Chl a ⁄ b proteins of PSI Thylakoid proteins were separ-ated as above and the blots were incubsepar-ated with antibodies as indi-cated The Lhca2 antibody also detects Lhcb4 (CP29) (C) Content

of light-harvesting Chl a ⁄ b proteins of PSII and PSII core proteins Thylakoid proteins were separated as above, and the blots were incubated with antibodies as indicated.

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PSI-J is not involved in binding LHCI

The four Lhca proteins, which constitute the major

part of the peripheral antenna of PSI (LHCI), were

not reduced to the same extent as the core subunits

Lhca1 and Lhca4 were present in near wild-type

amounts, and Lhca2 and Lhca3 were reduced by

15–25% compared with wild-type (Fig 5B) This

indi-cates that some of the Lhca proteins are present in

rel-ative excess of the PSI core complexes

The antenna properties were further analysed by

fluorescence emission measurements at low

tempera-ture Fluorescence emission spectra between 650 and

800 nm during excitation at 435 nm at 77 K using

intact leaves of wild-type plants and plants devoid of

PSI-J are shown in Fig 7 The spectra revealed that,

in the absence of PSI-J, there is a 2–3 nm blue shift in

the far-red emission originating from PSI–LHCI The

blue shift suggests a perturbation of the peripheral

antenna, which is because either PSI-J plays a

func-tional role in the binding⁄ function of the LHCI

antenna or free Lhca complexes are present in the

membrane However, low-temperature fluorescence

emission measurements on PSI–LHCI particles

enriched using sucrose density gradient centrifugation

as shown in Fig 8 did not display the 2–3 nm blueshift

(data not shown), indicating that the blue shift is

caused by excess free Lhca complexes in the thylakoid

membrane

This was further supported by estimation of the

functional antenna size of PSI using light-induced

P700 absorption changes at 810 nm after very gentle solubilization of the thylakoid membrane using digito-nin as described in Experimental procedures We have

WT ΔJ

B

PSI-D

PSI-E

PSI-F

PSI-K PSI-H

PSI-L PSI-J PSI-C

PSI-N

0 20 40 60 80 100

ΔJ

A

Fig 6 Immunoblot analysis of proteins in

PSI particles prepared from DpsaJ and

wild-type plants (A) Quantification of the signals

obtained in the immunoblot analysis (B)

Representative example of the signals with

the various PSI antibodies.

Emission wavelength (nm)

660 680 700 720 740 760 780

0.0 0.5 1.0 1.5 2.0

WT

ΔJ

Fig 7 Low-temperature fluorescence emission Shown are the spectra of intact leaves from a wild-type plant (WT) and a DpsaJ plant (DJ) Leaves from several individual plants of both genotypes were measured, and the mutant consistently showed a 3-nm blue shift in the far-red florescence emission peak Excitation wave-length was 435 nm, and the spectra were normalized to the peak

at 685 nm.

Trang 7

previously used this method to successfully detect

changes in PSI antenna caused by association with

LHCII during state transitions [23] or genetic

elimin-ation of individual Lhca proteins in Arabidopsis [24]

The functional PSI antenna size was expressed by the

t1 ⁄ 2 value which is defined as the time it takes to

oxidize 50% of the P700 in the sample and was

esti-mated at three different intensities of actinic light At

all three light intensities, there was no significant

dif-ference in t1⁄ 2 in the samples lacking PSI-J compared

with the values obtained with wild-type samples

(Table 2), suggesting that the PSI antenna size is unaffected by the elimination of PSI-J and further-more ruling out the possibility that PSI-J is strictly required for binding of any of the Lhca antenna proteins

The presence of free Lhca1 and Lhca4 in the thylakoid membrane was verified by gentle solubiliza-tion of the various thylakoid membrane complexes using dodecyl-b-d-maltoside and subsequent separation

of the complexes using sucrose density gradient centrif-ugation After separation, the gradients were harvested

in 0.5-mL fractions, and the individual fractions were analysed by gel electrophoresis and immunoblotting using antibodies against the four Lhca proteins and the PSI-F subunit (Fig 8) This revealed that signifi-cant amounts of free Lhca1 and Lhca4 proteins indeed were found in the fractions where mainly LHCII trim-ers and⁄ or Lhcb monomers are normally found How-ever, this analysis also suggested that PSI–LHCI complexes devoid of PSI-J are slightly more sensitive

to the detergent treatment, as some free Lhca2 and Lhca3 proteins were also detected

Table 2 Measurements of antenna size using time course of P700

photo-oxidation in solubilized thylakoid preparations from wild-type

and DPSI-J plants lE, l moles photonÆm)2Æs)1.

lE

t1⁄ 2(ms)

1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17 18 19 20 21 22 23 24 25

wt

ΔJ

PSI-LHCI

PSII-core

LHCII trimers and monomers

wt Lhca1

wt Lhca2

wt Lhca3

wt Lhca4

ΔJ Lhca2

ΔJ Lhca3

ΔJ Lhca4

ΔJ Lhca1

wt PsaF

ΔJ PsaF

Fig 8 Analysis of the distribution of Lhca proteins in the thylakoid membrane of DpsaJ (DJ) plants Shown is the centrifuga-tion tubes after separacentrifuga-tion of the solubilized membrane complexes in a sucrose density gradient (top panels) and an immunoblot analysis using the four Lhca antibodies and

a PSI-F antibody on individual fractions har-vested from the sucrose density gradient fraction (bottom part).

Trang 8

PSI-J is important for proper electron transfer

On the basis of work with mutants of Chlamydomonas

lacking PSI-J, it has been proposed that the function

of PSI-J is to maintain PSI-F in the correct

orienta-tion, facilitating fast electron transfer from Pc or

Cyt c6 to P700 [20] A similar role for PSI-J in higher

plants is likely, and, in order to analyse this, NADP+

photoreduction was determined using thylakoids

puri-fied from plants without PSI-J and wild-type plants In

our standard assay with 2 lm Pc, an activity of

24.8 ± 2.0 lmol NADPHÆs)1Æ(lmol P700))1 was

obtained with thylakoids from wild-type and

11.1 ± 1.0 lmol NADPHÆs)1Æ(lmol P700))1 with

thyl-akoids devoid of PSI-J (Table 1) Thus, PSI devoid of

PSI-J only has 45% of the NADP+ photoreduction

activity of the wild-type

This result clearly suggests that PSI-J affects electron

transport As indicated from work with green algae [20]

and the in vivo measurement of the P700 redox level in

Fig 4, the most obvious step to be affected is the

elec-tron transfer from Pc to P700 To investigate the kinetics

of the Pc–P700 interaction, flash-induced P700

tion transients were determined by following the

absorp-tion at 834 nm in the presence of Pc Flash excitaabsorp-tion of

PSI results in a very rapid absorption increase at 834 nm

caused by photo-oxidation of P700 to P700+, followed

by a slower absorption decrease due to reduction of

P700+ by Pc The reaction between Pc and P700 is a

multistep reaction, which can be divided into three major

steps: binding of Pc to P700, electron transfer within a

complex between Pc and P700, and release of oxidized

Pc from the complex between Pc and P700 The

absorp-tion decrease at 834 nm can be modelled as the sum of

three exponential decays discerned as a fast phase

corres-ponding to the electron transfer between preformed Pc–

PSI complexes, an intermediate phase corresponding to

the bimolecular reaction between Pc in solution and PSI,

and a slow phase corresponding to inactive PSI and a

contribution from absorption of oxidized Pc at 834 nm

[25–27] For analysis of wild-type and mutants lacking

PSI-J, Pc concentrations of 5 an 25 lm were used, and

the first 20 ls of the data were ignored With 5 and

25 lm Pc, the fast reduction of P700+by Pc bound to

PSI before photo-oxidation is negligible Therefore, good fits to the experimental data could be obtained using a sum of two exponential decays The results show that there is no difference between wild-type and mutant in the apparent second-order rate constants (Table 3), sug-gesting that PSI-J does not affect the electron transfer from Pc to PSI directly However, the amplitude of the intermediate phase is 80% in wild-type and only 63% in the PSI-J-less samples, indicating that the absence of PSI-J results in 20% more inactive PSI compared with wild-type Thus, the observed decrease in NADP+ pho-toreduction can, at least in part, be explained by a larger fraction of inactive PSI in the absence of PSI-J

Discussion

PSI-J is a subunit of PSI in almost all photosynthetic organisms studied so far However, the unicellular cyanobacterium, Gleobacter violaceus PCC 7421, appears to have a PSI without PSI-J [28,29] The func-tion of PSI-J in higher plants has so far not been investigated We have successfully generated transgenic Nicotiana tabaccum plants devoid of the J subunit of PSI and been able to investigate the role of PSI-J in higher plants The PSI-J-less plants were analysed with various biochemical and physiological methods

PSI-J is required for stable accumulation of PSI

In the absence of PSI-J, the steady-state accumulation

of PSI is reduced by  20%, as evidenced by the esti-mates of Chl⁄ P700, the immunoblotting analysis of thylakoid proteins (Fig 5), and the lower Chl a⁄ b ratio (Table 1) This suggests that PSI-J is implicated in sta-bility or assembly of the PSI complex in tobacco This

is in contrast with results reported for Chlamydomonas lacking PSI-J, where it was concluded that steady-state accumulation of PSI does not require the PSI-J sub-unit [20] Differences between higher plants and green algae with respect to PSI stability and function have also been reported after removal of PSI-F, which in Arabidopsis resulted in severe destabilization of PSI and especially loss of stromal subunits such as PSI-C, PSI-D and PSI-E [8] In contrast, a deletion of PSI-F

Table 3 Apparent second-order rate constant (k) for the reduction of P700 + by plastocyanin The rate constants were obtained from a curve-fitting analysis of flash-induced absorption transients recorded at 834 nm in samples of dodecyl-b- D -maltoside-solubilized thylakoids.

Percentage of amplitudes relative

to the total amplitude

Trang 9

in Chlamydomonas did not affect the stability of the

PSI complex [11,20]

Transgenic Arabidopsis plants without PSI-N, PSI-H,

PSI-K and PSI-L compensate for a poorly functioning

PSI by making 15–20% more PSI [7,30–32] Apparently,

the plants devoid of PSI-J cannot compensate in a

sim-ilar way, which again suggests that PSI-J affects the

sta-bility or assembly in a different way from the absence of

PSI-N, PSI-H, PSI-K and PSI-L In some aspects,

plants devoid of PSI-J display certain similarities to

plants devoid of PSI-G [9,33,34] In the absence of

PSI-G, less PSI core, a relative excess of LHCI, and a

less stable PSI is also observed To distinguish whether

it is the stability or the assembly of the PSI complex that

is affected needs further investigation

The reduced content of PSI was readily revealed by

the appearance of the transgenic tobacco plants, which

were slightly smaller and paler than wild-type Plants

devoid of PSI-G or PSI-K have been reported to be

reduced in mean size [34], and plants devoid of PSI-G

have a 40% reduction in content of PSI [33] and also

a slightly lighter pigmentation [34] Thus, there is good

correlation between the amount of PSI, plant size, and

pigmentation, although one would not expect a 20%

reduction in PSI to affect the growth to the extent seen

for the tobacco plants without PSI-J However,

com-bined with a less efficient PSI, as both the in vitro

NADP+ measurements and the in vivo estimations of

the PSII excitation pressure indicate, the observed

growth phenotype is explainable

PSI-J is not necessary for binding of the

peripheral light-harvesting antenna

The two Chls bound to PSI-J in higher plants are

sug-gested to be important for the energy transfer between

LHCI and the PSI core [10] However, the functional

PSI antenna size is unaffected by the elimination of

PSI-J from the PSI complex (Table 2) Thus, PSI-J

is not required for binding or the function of the

peripheral antenna, or at least the PSI that is formed

is unaffected by the missing PSI-J The measurements

of the functional antenna size using P700 oxidation

rates do not allow enough time resolution to tell

whe-ther the absence of the two Chl molecules bound to

PSI-J causes inefficient transfer of excitation energy

from the peripheral antenna to the core

In vitrothe absence of PSI-J affects the stability of the

PSI–LHCI complex The results of the fractionation of

mildly solubilized thylakoid membrane complexes as

pre-sented in Fig 8 indicate that some Lhca proteins, mainly

Lhca1 and Lhca4 are present in relative excess compared

with the core subunits, as also indicated in the

immuno-blot analysis on nonsolubilized thylakoids (Fig 5) and the 77 K fluorescence emission measurements on detached leaves (Fig 7) Alternatively, the solubilization with detergent affects the PSI-J-deficient complexes more than the wild-type complexes, and thereby more of the Lhca proteins are released from the complex

PSI-J is required for efficient electron transfer PSI-J affects the electron transport through PSI Meas-ured as in vitro NADP+ photoreduction activity, a 55% decrease in the steady-state electron transport in the absence of PSI-J was observed The kinetic analysis

of the reaction between Pc and P700 did not reveal any significant difference in the second-order rate con-stant between wild-type and PSI-J-deficient plants that can explain the observed decrease in PSI activity The kinetic parameters of the reaction between Pc and P700 was also found to be unaffected when PSI from DPSI-J and wild-type Chlamydomonas was analysed [20], and it therefore seems that PSI-J does not partici-pate directly in the binding of Pc in either plants or green algae In Chlamydomonas, the amplitude of the PSI-F-dependent second-order kinetics was 76% and 42% of the total amplitude with wild-type and PSI-J-deficient thylakoid membranes, respectively [20], which correspond to a 45% decrease This decrease is thought to be caused by an increased proportion of PSI complexes incompetent for fast electron transfer in the absence of PSI-J and has been suggested to be due

to a stabilizing effect of PSI-J on PSI-F [20] Similar to this, we observe a 20% decrease in the amplitude of the second-order component of electron transfer with plant thylakoids devoid of PSI-J Thus, in plants, there

is also an increased proportion of PSI complexes that are incompetent for efficient electron transfer Interest-ingly, the immunoblotting analysis of PSI particles purified using sucrose density gradient centrifugation after solubilization with dodecyl-b-d-maltoside clearly suggested that binding of the luminal PSI-N to PSI was affected in the absence of PSI-J (Fig 6) This loss

of PSI-N is probably due to increased sensitivity to detergent during preparation of the PSI particles, but, despite this, it strongly suggests a perturbation of the luminal side of PSI involving PSI-F and PSI-N The absence of PSI-J might affect the conformation of PSI-F, which, in turn, changes the binding of PSI-N PSI-F provides part of the Pc-binding site in plants [16], and it is known that the depletion of PSI-F by antisense suppression of the corresponding gene leads

to a secondary loss of PSI-N [8], indicating an interac-tion between these two subunits PSI-N has further been shown to be necessary for the efficient interaction

Trang 10

with Pc, as the second-order rate constant was reduced

by 40% in the absence of PSI-N [7]

The increase in the pool of inactive PSI observed in

plants devoid of PSI-J is not caused by the absence of

PSI-N because mutants lacking PSI-N clearly have a

changed second-order rate constant for Pc–P700

inter-action but not an increased proportion of inactive PSI

complexes [7] Furthermore, the immunoblotting

ana-lysis of thylakoid proteins (Fig 5) clearly indicates that

PSI-N is present in amounts similar to the other PSI

core subunits Instead it seems plausible that the

chan-ged conformation of PSI-F in the absence of PSI-J

renders a fraction of the PSI complexes inactive

The in vivo measurements of the P700 redox level

indicate that P700 in the DPSI-J plants constantly

stays more oxidized, which is usually caused by a

limi-tation of electron-transfer activities on the donor or

lu-minal side of PSI The 20% permanently oxidized PSI

estimated from the in vivo experiment is in excellent

agreement with the 20% inactive PSI determined with

the flash excitation At the same time, the

plastoqui-none pool is more reduced, as indicated by the

increased PSII excitation pressure These observations

are consistent with a greater pool of inactive PSI

cen-tres in the absence of PSI-J in vivo

However, the 20% increase in the pool of inactive

PSI complexes in the absence of PSI-J does not explain

the dramatic reduction in PSI activity measured by

NADP+ photoreduction activity The kinetic analysis

clearly indicates that the second-order rate constant

for electron transfer from Pc to P700 is unaffected

However, the release of oxidized Pc has been shown to

limit electron transfer between the cytochrome b6f

complex and PSI in vivo [35], and the absence of PSI-J

may affect the koffvalue, so that oxidized Pc stays

lon-ger in the active site and thereby blocks efficient

exchange with reduced Pc Alternatively, the changed

conformation of PSI-F in the absence of PSI-J could

affect proper functioning of stromal subunits in

con-tact with PSI-F, such as PSI-E or PSI-D These

sub-units are involved in docking and efficient electron

transfer to ferredoxin [6], and, from the structures, it is

known that PSI-E is in contact with the C-terminus of

the PSI-F subunit [5] Changes in binding or amounts

of any of the stromal subunits of PSI were not

detec-ted in our immunoblot analysis; however, a subtle

change in arrangement of the subunits is still possible

In conclusion, PSI-J is needed for stable accumulation

of the PSI core complex and proper electron transfer

Despite the location of PSI-J close to the rim of the core

complex facing LHCI, it is not needed for correct

inter-action with the peripheral antenna complexes Clearly

the luminal side of PSI is perturbed, probably because

of destabilization of PSI-F in the absence of PSI-J, resulting in an increased pool of inactive PSI

Experimental procedures

Vector construction, chloroplast transformation, and plant material

The region of the tobacco chloroplast genome containing

700 bp upstream and downstream of the psaJ reading frame was amplified using PCR The 1535-bp fragment was ligated into the SacI and BamHI sites of pUC19 The psaJ knock-out allele was created by digestion of this construct with ScaI, and a chimeric aadA gene conferring resistance

to aminoglycoside antibiotics [21] was inserted into this ScaI site to disrupt psaJ and to facilitate selection of chloroplast transformants ScaI causes disruption of the 132-bp psaJ coding region after nucleotide 38 A plasmid clone carrying the aadA gene in the same orientation as psaJyielded the transformation vector pPsaJ (Fig 2) Chloroplasts of N tabaccum cv Petit Havanna were transformed by particle bombardment of leaves [21] Selec-tion and culture of transformed material as well as assess-ment of plastome segregation and the homoplastomic state were performed essentially as described by De Santis-Maciossek et al [36] and Swiatek et al [37] Essentially, 10 leaves were used for particle bombardment, and 19 antibi-otic resistant transformants were selected The material was maintained on agar-solidified MS medium [38] containing 2% sucrose, and grown in 12 h dark⁄ light cycles at 25 C and 20 lmol photonsÆm)2Æs)1 and, under selective condi-tions, 500 lgÆmL)1 spectinomycin For thylakoid isolation and physiological measurements, wild-type and transformed plants (originating from two independent transplastomic lines) were planted in compost and kept in growth chamber conditions in 8 h light and 120–140 lmol photonsÆm)2Æs)1

Isolation of thylakoid membranes and PSI particles from tobacco

Leaves from 6–8-week-old plants were used for isolation of thylakoids as described previously [7] PSI particles were iso-lated from thylakoids after solubilization with dodecyl-b-d-maltoside and sucrose density gradient ultracentrifugation as described in [31] Chl content and the Chl a⁄ b ratio were determined in 80% acetone as described previously [39] The samples were frozen in liquid nitrogen and stored at)80 C

RNA gel blot analysis Northern blot analysis of total leaf RNA was performed using DNA probes and was carried out as described by Meurer et al [40] A 33P-labelled DNA fragment corres-ponding to the psaJ gene was used as probe

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