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In medium containing BrdU, BAR1 expression was further enhanced, associ-ated with more marked disruption of nucleosome positioning on the pro-moter region.. In this study, we examined wh

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genes with disruption of nucleosome positioning

Kensuke Miki1, Mitsuhiro Shimizu2, Michihiko Fujii1, Shinichi Takayama1,

Mohammad Nazir Hossain1and Dai Ayusawa1

1 Department of Genome System Science, Yokohama City University, Yokohama, Kanagawa, Japan

2 Department of Chemistry, Meisei University, Hino, Tokyo, Japan

Introduction

5-Bromodeoxyuridine (BrdU), which is frequently used

to measure DNA synthesis immunochemically in living

cells, is also well known to modulate various biological

functions when incorporated into DNA as

5-bromoura-cil instead of thymine Previously we have found that

BrdU very clearly induces a senescent-like phenomenon

in every type of mammalian cell and also in yeast cells

[1,2] Historically, BrdU has been used as a modulator

of cellular differentiation with cAMP and butyrate

[3,4] The latter two are found to target protein kinase

A and histone deacetylase, respectively, leading to func-tional understanding of cell signaling and gene expres-sion BrdU is thought to influence the expression of key genes involved in cellular differentiation However, the molecular mechanism underlying the actions of BrdU still remains a mystery in spite of many efforts [5]

We have extensively characterized genes up- or down-regulated by the addition of BrdU in various cell

Keywords

AT-tract; 5-bromodeoxyuridine; BAR1;

nucleosome positioning; transcriptional

derepression

Correspondence

D Ayusawa, Department of Genome

System Science, Yokohama City University,

Seto 22-2, Kanazawa-Ku, Yokohama,

Kanagawa 236-0027, Japan

Fax: +81 45 787 2193

Tel: +81 45 787 2193

E-mail: dayusawa@yokohama-cu.ac.jp

(Received 5 January 2010, revised 2 August

2010, accepted 2 September 2010)

doi:10.1111/j.1742-4658.2010.07868.x

5-Bromodeoxyuridine (BrdU) modulates the expression of particular genes associated with cellular differentiation and senescence when incorporated into DNA instead of thymidine (dThd) To date, a molecular mechanism for this phenomenon remains a mystery in spite of a large number of stud-ies Recently, we have demonstrated that BrdU disrupts nucleosome posi-tioning on model plasmids mediated by specific AT-tracts in yeast cells Here we constructed a cognate plasmid that can form an ordered array of nucleosomes determined by an a2 operator and contains the BAR1 gene as

an expression marker gene to examine BAR1 expression in dThd-auxotro-phic MATa cells under various conditions In medium containing dThd, BAR1 expression was completely repressed, associated with the formation

of the stable array of nucleosomes Insertion of AT-tracts into a site of the promoter region slightly increased BAR1 expression and slightly destabi-lized nucleosome positioning dependent on their sequence specificity In medium containing BrdU, BAR1 expression was further enhanced, associ-ated with more marked disruption of nucleosome positioning on the pro-moter region Disruption of nucleosome positioning seems to be sufficient for full expression of the marker gene if necessary transcription factors are supplied Incorporation of 5-bromouracil into the plasmid did not weaken the binding of the a2⁄ Mcm1 repressor complex to its legitimate binding site, as revealed by an in vivo UV photofootprinting assay These results suggest that BrdU increases transcription of repressed genes by disruption

of nucleosome positioning around their promoters

Abbreviations

BrdU, 5-bromodeoxyuridine; dThd, thymidine; MNase, micrococcal nuclease; TK, thymidine kinase.

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lines using PCR-based cDNA subtractive hybridization

and DNA microarray analysis [6,7] Such

BrdU-responsive genes behave similarly in normal human

fibroblasts undergoing replicative senescence BrdU

decondenses particular regions of chromosomes after

incorporation into DNA [8], suppresses position effect

variegation [9] and restores expression of silenced

genes [10] Consistent with this, BrdU-responsive genes

are located on particular regions of human

chromo-somes, forming clusters on or nearby Giemsa-dark

bands of human chromosomes [11,12] AT-tract minor

groove binders, such as distamycin A, netropsin,

Hoe-chst 33258 and the AT-hook protein HMG-I, have all

been shown to markedly potentiate the effects of BrdU

[11,13] On the basis of the above observations, we

suggest that BrdU targets certain types of AT-rich

sequence and alters the chromatin structure to induce

particular genes

In eukaryotes, DNA fibers exist as regularly arrayed

beads of nucleosomes Nucleosomes restrict the

acces-sibility of transcription factors to promoters and

regu-latory sequences of genes Thus, an alteration of

nucleosome positioning is an essential step for the

transition from a repressed state to an active state by

aid of chromatin remodeling complexes [14–16] In our

previous studies, we addressed the effect of BrdU on

nucleosome positioning in vivo using TALS plasmids

[17], which have been successfully utilized to study

nucleosome positioning in Saccharomyces cerevisiae

We have clearly shown that 5-bromouracil

incorpo-rated into the plasmids disrupts nucleosome

position-ing by inducposition-ing A-form-like DNA conformation in

yeast cells [18] In mammalian cells, histones were

shown to bind more tightly to 5-bromouracil-substi-tuted DNA in vitro than normal DNA containing thy-mine [19,20]

In this study, we examined whether BrdU induces the expression of genes associated with the destabiliza-tion of nucleosome posidestabiliza-tioning with model plasmids containing the BAR1 gene as a marker gene We showed that BrdU increases the transcription of repressed genes by disruption of nucleosome position-ing around their promoters These data will facilitate the understanding of the role of BrdU and AT-tracts

in the induction of particular genes

Results

Construction of model plasmids

In yeast MATa cells, the a2⁄ Mcm1 repressor binds to the a2 operator and acts to repress MATa cell-spe-cific genes such as BAR1, STE2 and STE6 [21–25] Previously, our high-resolution mapping of micrococ-cal nuclease (MNase) cleavage sites has indicated that nucleosomes were well positioned around the pro-moter region of the genomic BAR1 gene [26] and such nucleosome positioning was required for its full repression [27] Here, we constructed a plasmid pRS-BAR1 that contains the pRS-BAR1 gene as a model sys-tem (Fig 1) to easily examine the role of BrdU in gene expression and nucleosome positioning in vivo

To further examine the effects of AT-tracts on nucle-osome positioning, we inserted them into a KpnI site adjacent to the TATA box These plasmids were introduced into a thymidine (dThd)-auxotrophic

Inserts at the Kpn I site pRS001-BAR1 pRS801-BAR1

: T3CCT6CT5GCT5CT7 : A34

BAR1 -UTR -UTR pRS-BAR1

TATA

2 op

mRNA Kpn I

+1 130

– 219 – 66 +95 +255

I II III IV V

60 –500

Xho I

– 235 – –

+420 –158

EcoRV

+1667

BAR1

Fig 1 Schematic representation of pRS-BAR1 and its derivatives Plasmid pRS-BAR1 contains a genomic fragment spanning the BAR1 gene regulated by a2 ⁄ Mcm1 repressor The a2 operator (a2 op), the TATA box (TATA) and the BAR1 coding sequence are denoted by hatched, filled and dotted boxes, respectively The transcriptional initiation site is indicated by a bent arrow The site of KpnI, to which AT-tracts are inserted, is indicated by a vertical line The probes A and B are indicated by gray boxes Positions of stable nucleosomes are shown by shadowed ellipses with numbers.

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mutant of yeast cells to ensure quantitative

incorpora-tion of BrdU into DNA [2]

Nucleosome positioning on pRS-BAR1

We first examined whether pRS-BAR1 forms a stable

and precise array of nucleosomes in dThd medium

using the indirect end-labeling method DNA samples

were completely digested with XhoI after partially

digestion with MNase, and were subjected to Southern

blot analysis with probe A Five MNase cleavage sites

indicated by *a, *b, *c, *d and *e were observed in the

naked DNA sample, and these sites were protected in

a chromatin sample of pRS-BAR1 (Fig 2A, lanes 1

and 2) Also, the cleavage sites showed equal intervals

(140–150 bp) in the chromatin sample (Fig 2A, lane

1) These results showed that at least five stable

nucle-osomes were precisely and stably formed from the a2

operator to the coding region of the BAR1 gene on

pRS-BAR1 in vivo (Fig 1, bottom panel)

We then examined how BrdU affects nucleosome

positioning on the pRS-BAR1 plasmid When the

MATa cells were cultured in BrdU medium, the two

bands (*b and *c) that were protected in the chromatin

sample prepared in dThd medium were evident in the

chromatin sample prepared in BrdU medium (Fig 2A,

lanes 1 versus 3) Densitometrical measurement clearly

showed these differences (Fig 2B) Consistently, the

band corresponding to the linker region between

nucle-osomes II and III became broader in the same sample

(Fig 2A, lane 3) Because there was no significant

dif-ference in MNase cleavage patterns between naked

DNA samples prepared in dThd and BrdU medium,

BrdU did not affect the specificity and sensitivity to

MNase (Fig 2A, lanes 2 versus 4) These results

indi-cate that BrdU destabilized nucleosome positioning

We then examined the sequence in nucleosome II, and

found the following: the AT-content of the sequence

was 69%, whereas in other nucleosomes it was

approx-imately 60%, and An, Tn or (AT)n tracts (n‡ 6) were

found in six sites in nucleosome II, but not in

nucleo-some III Therefore, BrdU seemed to destabilize the

positioning of nucleosome II through AT-rich

sequences in nucleosome II, which then led to the

destabilization of nucleosome III The same results

were obtained when nucleosome positioning was

deter-mined from the EcoRV site with probe B (Fig 1, S1,

lanes 1–4)

Effects of AT-tracts on nucleosome positioning

We examined two derivatives of pRS-BAR1,

pRS001-BAR1 and pRS801-pRS001-BAR1, containing T3CCT6CT5

GCT5CT7 and A34, respectively, in a promoter region

of the BAR1 gene (Fig 1A) Insertion of these AT-tracts did not affect MNase cleavage patterns in naked DNA samples (Fig 2A, even-numbered lanes) The MNase cleavage pattern in a chromatin sample of pRS001-BAR1 was similar to that in the chromatin sample of pRS-BAR1 in dThd medium, although the bands *c and *d were more evident in the former sam-ple (Fig 2A, lanes 1 versus 5) This suggests that the positioning of nucleosomes I and II on pRS001-BAR1 was less stable than on pRS-BAR1 in dThd medium The bands *b, *c and *e in the chromatin sample of pRS001-BAR1 were more marked in BrdU medium than in dThd medium (Fig 2A, B, lanes 5 versus 7), although the MNase cleavage patterns in the naked DNA sample were similar between dThd and BrdU medium Similar results were obtained when nucleo-some positioning was mapped in the opposite direction from the EcoRV site with probe B (Fig S1, lanes 5–8)

We next analyzed the nucleosome positioning on pRS801-BAR1 Interestingly, the MNase mapping of nucleosomes with probe A showed that the three bands *b to *d (especially band *c) were not protected

in a chromatin sample of pRS801-BAR1 (Fig 2A, lane 9), indicating that insertion of A34disrupts nucleosome positioning more remarkably than that of

T3CCT6CT5GCT5CT7 In BrdU medium, all of the bands that corresponded to the linker regions were eliminated and the bands *a, *b and *e were more clearly detected than in dThd medium (Fig 2B, lanes 9 versus 11) These results were also confirmed by indi-rect end-labeling with probe B (Fig S1, lanes 9–12) Taken together, these observations indicate that nucleosome positioning on pRS001- and pRS801-BAR1 were modestly and almost completely disrupted, respectively, by BrdU

Effect of BrdU on BAR1 expression

We measured the expression of the BAR1 gene on the model plasmids by Northern blot analysis In dThd medium, the BAR1 mRNA level for pRS-BAR1 was

30 times less in the MATa cells than in the MATa cells (Fig 3A) These results indicate that the episomal BAR1gene is regulated similarly to the genomic BAR1 gene In the MATa cells, the mRNA level was 2.4 times higher in BrdU medium than in dThd medium (Fig 3B) Insertion of T3CCT6CT5GCT5CT7 (pRS001-BAR1) slightly increased the mRNA level in dThd medium, but additionally increased it in BrdU medium (Fig 3B) On the other hand, insertion of A34 (pRS801-BAR1) significantly increased the mRNA level in dThd medium and markedly increased it in

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a b c d

a

b

c

d

2 op

c d

a b

c d

a b

c

e

a b

c

e

a b

pRS-BAR1

C D

C D

pRS001-BAR1

pRS801-BAR1

M

2.0

1.5

1.0

0.5

(kb)

I

II

III

IV

V

A

Lane

1

3

5

7

9

11

Fig 2 Nucleosome positioning on pRS-BAR1 and its derivatives Chromatin (indi-cated by C) and naked DNA (indi(indi-cated by D) samples were prepared from cells

transfect-ed with the plasmid indicattransfect-ed and culturtransfect-ed

in dThd or BrdU medium as indicated The samples were partially digested with

5 UÆmL)1(odd-numbered lanes) or 0.5 UÆmL)1(even-numbered lanes) MNase, completely digested with XhoI, and sub-jected to the indirect end-labeling analysis with probe A as described in Materials and Methods At least three independent analy-ses for each plasmid gave similar results (A) Autoradiography of MNase cleavage pat-terns DNA size markers (M), positions of nucleosomes I–V and a2 operator (a2 op) are shown to the left Specific cleavage sites on naked DNA samples are marked with *a, *b, *c, *d and *e Ellipses with dotted lines indicate nucleosomes whose positioning is unstable Open stars on some lanes denote bands that changed in BrdU medium The transcriptional start site and the KpnI site are indicated by a bent arrow and arrowheads, respectively (B) Densito-metric profiles of autoradiography The odd-numbered lanes in (A) were

densitometrical-ly scanned The vertical dotted lines denote the bands (*a, *b, *c, *d and *e) specific to naked DNA The positions of stable nucleo-somes are shown at the top.

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BrdU medium (Fig 3B) We also examined the effect

of BrdU on pYBT1 containing the thymidine kinase

(TK) gene driven by the yeast constitutive ADH1

pro-moter as a control plasmid BrdU did not significantly

affect the expression of the gene (Fig 3B) These

results show that the levels of expression of the BAR1

gene are parallel to those of the disruption of

nucleo-some positioning in dThd and BrdU medium

To confirm that our experimental conditions can

induce specific genomic genes, we examined the

expres-sion of some genomic genes having an AT-tract on

their promoter regions The DED1 gene, having

T3CCT6CT5GCT5CT7, and the MAK16 gene, having

T24, were significantly up-regulated by the addition of

BrdU (Fig 4), suggesting that the mechanism found in

the episomal genes also operates in the genomic genes

Effect of BrdU on a2/Mcm1 repressor–operator

complex

We examined whether the a2⁄ Mcm1 repressor changes

its binding to the a2 operator on pRS-BAR1 upon

incorporation of 5-bromouracil by an in vivo UV

photofootprinting assay The a2⁄ Mcm1 operator has

numerous thymine bases necessary for recognition by

the repressor [28] and thus its binding to the repressor

may be disturbed by substitution of 5-bromouracil In

the noncoding strand of a naked DNA sample, several

thymine dimers were found around the a2 operator

(Fig 5A, lane 2), but three sites (marked with + in Fig 5A) were protected in chromatin samples (Fig 5A, lanes 1 and 3) Although slight differences in the thymine dimers formed were observed in the naked DNA samples containing thymine or 5-bromouracil (Fig 5A, lanes 2 versus 5), the three thymine dimers were equally protected in the chromatin samples con-taining thymine or 5-bromouracil (Fig 5A, lanes 4 and 6) Similar results were obtained with the coding strand (Fig 5B) These results show that BrdU does not weaken the formation of the a2⁄ Mcm1 repressor– operator complex, excluding the possibility that the

DED1

MAK16

ACT1

Fig 4 Northern blot analysis of genomic genes Total RNA sam-ples prepared from the MATa cells cultured in dThd or BrdU med-ium were subject to Northern blot analysis with probes derived from the genes indicated.

T B pRS-BAR1 0

2 4 6

T B pRS001-BAR1

##

T B pRS801-BAR1

###

T B

pYBT1

20

0 MAT MATa

pRS-BAR1 10

30

Fig 3 Gene expression profiles of pRS-BAR1, its derivatives and a reference plasmid (A) BAR1 mRNA levels in MATa and MATa cells Total RNA and DNA samples were prepared from the cell type indicated in dThd medium and subjected to Northern and Southern blot analy-ses as described in Materials and Methods BAR1 mRNA levels were expressed relative to actin mRNA levels after normalization by copy numbers of plasmids (B) Effects of BrdU on BAR1 and TK mRNA levels Total RNA and DNA samples were prepared from the MATa cells transfected with the plasmid indicated and cultured in dThd (T) or BrdU medium (B), and processed as in (A) ***P < 0.001 compared with the values in dThd medium.##P < 0.01 and###P < 0.001 compared with the values of pRS-BAR1 in dThd medium Histograms represent means ± standard error At least four independent analyses carried out for each plasmid gave similar results.

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disruption of nucleosome positioning by BrdU is due

to a decrease in the formation of the a2⁄ Mcm1

com-plex [18]

Effect of BrdU on pRS-BAR1 lacking the promoter activity

We addressed whether the above changes in nucleo-some positioning are affected by the promoter activity

of the BAR1 gene, because transcription factors can affect nucleosome positioning We constructed a plas-mid, pRS-DTA⁄ BAR1, in which the TATA box of BAR1 was disrupted (Fig 6A) [23] With pRS-DTA⁄ BAR1 we were able to determine the change in nucleo-some positioning without the effect of the expression

of BAR1 (Fig 6B)

Disruption of the TATA box caused the disappear-ance of band *d in naked samples of pRS-DTA⁄ BAR1 prepared in dThd and BrdU medium Band *d corre-sponds to the nuclease hypersensitive site located at the TATA box on pRS-BAR1 (Fig 2A, lanes 2 and 4 versus Fig 6C, lanes 2 and 4) as described in the pre-vious reports by Shimizu et al [26] and Cooper et al [23] In dThd medium, the MNase cleavage pattern in

a chromatin sample of pRS-DTA⁄ BAR1 showed well-ordered nucleosome positioning (Fig 6C, lane 1), which was identical to that on the chromatin sample

of pRS-BAR1 (Fig 2A, lane 1), except for the pres-ence of band *d

In BrdU medium, the bands corresponding to the linker regions between nucleosomes I–III were broader, and the two bands (*b and *c) that were pro-tected in the chromatin sample prepared in dThd med-ium became more evident (Fig 6C, lane 3) When

dThd

C D C

BrdU

C D C

+

+

+

+

+

+

+

+ +

C D C

BrdU

C D C

+

+

+

+

+

+

B

1 2 3 4 5 6 7 8 9 10 11 12

Fig 5 In vivo UV photofootprinting of a2 operator on pRS-BAR1.

Intact cells (indicated by C) and naked DNA (indicated by D) were

irradiated with UV at dosages of 250 mJ (lanes 1, 4, 7 and 10),

500 mJ (lanes 3, 6, 9 and 12) and 60 mJ (lanes 2, 5, 8 and 11) UV

photoproducts were analyzed using primer extension mapping on

the noncoding (A) and the coding (B) strands as described in

Mate-rials and Methods The a2 operator sequence is shown to the left

of each panel The thymine bases protected from UV damage are

indicated by +.

a

b c

e I

II III IV

V

a

b c

e

BrdU

CAGTATAAAAGTG

TATA pRS-BAR1

CAGTGGATCCGTG

Bam H I pRS-ΔTA/BAR1

pRS-ΔTA/BAR1 dThd

2 op Lane 1 2 3 4

B

ACT1

BAR1

/BA

R1

pRS-C D

C D

Fig 6 Nucleosome positioning on p0RS-DTA ⁄ BAR1 (A) Sequences of the TATA box and disrupted TATA box of the BAR1 pro-moter (B) Northern blot analysis of the BAR1 gene (C) Autoradiography of MNase cleavage patterns Nucleosome positioning was analyzed as in Fig 2.

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cultured in dThd or BrdU medium, the overall band

patterns were almost identical in the chromatin

sam-ples between pRS-BAR1 and pRS-DTA⁄ BAR1, except

for the presence of band *d These results indicate that

BrdU changes nucleosome positioning in the absence

of the transcription factors involved In line with this

observation, we have shown that incorporation of

BrdU into DNA converts the DNA structure into an

unusual conformation [18] It is thus reasonable to

suggest that such a structural change in DNA induced

by BrdU affects nucleosome positioning and results in

altered gene expression at particular regions

Discussion

We were able to show a positive correlation between

gene expression and the disruption of nucleosome

posi-tioning in yeast cells harboring minichromosomes and

cultured with BrdU as the only source of thymine To

validate this observation, BrdU must directly affect

DNA structure, but not interactions between DNA

and DNA-binding proteins, to induce a change in

nucleosome positioning In our model plasmids used,

the binding of a2⁄ Mcm1 repressor to its legitimate

binding site has a critical role in the formation of

sta-bly ordered nucleosomes As expected, BrdU did not

significantly affect the formation of the a2⁄ Mcm1

repressor–operator complex [18] In support of this,

expression of the genomic STE2 gene regulated by the

a2⁄ Mcm1 complex was not affected by the addition of

BrdU (data not shown) In addition, some

DNA-bind-ing proteins and enzymes examined to date cannot

functionally distinguish between 5-bromouracil and

thymine on DNA [29–32] These results suggest that

an unusual DNA conformation induced by BrdU is

the primary cause of altered nucleosome positioning

In fact, we have demonstrated that the incorporation

of 5-bromouracil into DNA reduces the bending of

DNA [11] and converts to A-form-like DNA or a rigid

DNA structure [18] However, a possibility cannot be

ruled out that a change in interactions between

5-bro-mouracil-substituted DNA and specific proteins may

additionally affect nucleosome positioning

We showed here that levels of BAR1 expression are

parallel to those of the destabilization of nucleosome

positioning with the use of model plasmids In our

pre-vious study employing different minichromosomes [18],

disruption of nucleosome positioning by BrdU was

shown to depend on the length and sequence specificity

of AT-tracts located at particular sites of

minichromo-somes In this study, destabilization of nucleosome

positioning by BrdU did not require the presence of

the promoter or expression of the BAR1 marker gene

As shown in the Results, AT-tracts alone can destabi-lize nucleosome positioning in dThd medium How-ever, not all AT-tracts have the ability to induce the destabilization of nucleosome positioning or expression

of the marker genes

The nucleosome disruption did not lead to full expression of the BAR1 gene This can be explained by the absence of an activator function of Mcm1 in MATa cells Mcm1 acts as an activator of a-cell-spe-cific genes in MATa cells, whereas it acts as a repressor

in MATa cells Taken together, nucleosome position-ing seems to be sufficient for full repression of genes [27] Also, disruption of it seems to be sufficient for full derepression of genes if necessary transcription fac-tors are supplied

Can our findings obtained with the episomal genes apply to genomic loci? The episomal BAR1 gene was shown to behave similarly to the genomic BAR1 gene [27] when A34was inserted into their promoters Simi-larly, the genomic DED1 gene was significantly up-reg-ulated by BrdU, as the episomal BAR1 gene has the AT-tract derived from the DED1 promoter Further-more, the genomic MAK16 gene having T24on its pro-moter region was also up-regulated by BrdU These results suggest that episomal and genomic genes behave similarly, and prove to be useful in studying gene regulation controlled by the higher-order struc-ture of chromatin

However, the presence of AT-tracts does not always affect the expression of their adjacent genes if the genes have a strong promoter Their promoter regions seem to be reluctant to form a stable nucleosome structure In these genes, BrdU does not seem to cause

an additional change in the nucleosome structure sur-rounding the genes and increase their expression For example, the expression of the TK gene driven by the yeast constitutive ADH1 promoter on pYBT1 was not affected by BrdU (Fig 3B) In the case of TALS-GFP, EGFP expression is driven by the ADH1 promoter (Fig S2) pOM801-GFP, having A34 inserted in the ADH1 promoter region (Fig S2), showed significantly increased promoter activity even in dThd medium (Fig S4) However, BrdU did not further increase the expression of GFP (Fig S4) or the state of the already opened nucleosome structure in pOM801-GFP (Fig S3) These results support our hypothesis that BrdU induces the expression of genes through the dis-ruption of nucleosome positioning on their promoter regions

In mammalian cells, BrdU is thought to induce the expression of genes when AT-tracts are located adja-cent to their promoters, similar to yeast systems [11]

In contrast to yeast genes, most of the mammalian

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genes are silenced during lifetime, embedded in

con-densed chromatin As described previously, BrdU can

restore the expression of silenced genes [9,10], and

BrdU-responsive genes are frequently located on

inac-tive chromatin regions, such as AT-rich Giemsa-dark

bands of human chromosomes [11,12] In this context,

BrdU seems to disrupt nucleosome positioning around

AT-rich condensed chromatin and results in the

induc-tion of the expression of silenced or repressed genes

Finally, the data of this study may lead to a new

understanding of the molecular mechanism of BrdU

They may answer the new and old question of why

BrdU modulates the expression of particular genes

associated with cellular differentiation and senescence

Materials and methods

Plasmids and yeast strains

To construct BAR1 expression plasmid pRS-BAR1, the

)500 to +2064 sequence containing the promoter with a

KpnI site ()158 to )154) [27], a coding sequence and

300 bp of 3¢-UTR of the BAR1 gene, were cloned into the

XhoI-SacI site of pRS424DKpnI in which one KpnI site was

filled in Plasmid pRS-BAR1 derivatives were constructed

by inserting oligonucleotides into the KpnI site of

pRS-BAR1 to yield pRS001-pRS-BAR1 (T3CCT6CT5GCT5CT7) and

pRS801-BAR1 (A34)

To disrupt a TATA box in pRS-BAR1, the sequence of

the TATA box at )134 (TATAAAA) was changed to a

sequence (TGGATCC) that contained a BamHI site by

amplifying a KpnI-BglII sequence of pRS-BAR1 with the

following two primers: 5¢ -TATTGGTACCGTGTGTTTT

TTGATAACAGTGGATCCGTG-3¢ and 5¢-GTGGAAGA

TCTATGCTCATTATAAGTACTC-3¢ The amplified

sequence was digested with KpnI and BglII, and cloned into

the KpnI–BglII site of pRS-BAR1 to yield pRS-DTA⁄

-BAR1, which lacks a functional TATA box

These plasmids were introduced into the yeast

dThd-autotrophic strains YKH2 (MATa ura2-52 trp1 his3 leu2

cdc21::LEU2 pYBT1) or YKH4 (MATa ura2-52 trp1 his3

leu2 cdc21::LEU2 pYBT1) established as described

previ-ously [2] Plasmid pYBT1 contains the herpes simplex virus

TKgene driven by the yeast ADH1 promoter

Chromatin preparation and nuclease digestion

Yeast cells harboring plasmids were selected in SC medium

(2% glucose, 0.67% yeast nitrogen base without amino

acids) supplemented with appropriate amino acids (except

for tryptophan) and 1 mm dThd Cells were grown in

30 mL of medium containing 1 mm dThd or BrdU at 30C

for 15 h to an optimal density of 0.6–1.0 at 600 nm

Chro-matin and naked DNA samples were prepared according to

the method of Balasubramanian & Morse [33] Each sample was digested with MNase (Takara, Kyoto, Japan) at 37C for 10 min The reactions were initiated by the addition of 0.15% Nonidet P-40, and halted by the addition of SDS and proteinase K Samples were purified with a phe-nol⁄ chloroform extraction and ethanol precipitation

Indirect end-labeling DNA samples were completely digested with XhoI or EcoRV together with RNase A, run on a 1.4% agarose gel and transferred on to a Nylon membrane (Biodyne B, Pall, Port Washington, NY, USA) followed by cross-linking with

UV light (Stratalimker 2400, Stratagene, La Jolla, CA, USA) The membrane was incubated at 65C for 16 h in hybridization solution (0.5 m Na-Pi, 1 mm ETDA and 7% SDS] containing a probe labeled with [a-32P] dCTP using a random-primed DNA labeling kit (Mega-prime, Amersham, Piscataway, NJ, USA) The XhoI–MspI fragment of pRS-BAR1 was used as a probe to detect nucleosomes in sam-ples digested with XhoI Likewise, a sequence amplified with the primers ATCTTATAATTATCGAGATCG-3¢ and 5¢-AAGTGTTCCACTGTCTAGTTTG-3¢ from pRS-BAR1 was used as a probe to detect nucleosomes in samples digested with EcoRV After washing, the membrane was subjected to autora-diography and densitometric analysis using an image analyzer FLA-5000 (FUJIFILM, Tokyo, Japan)

Gene expression analysis Total RNA samples were prepared, subjected to electropho-resis on a 1% formaldehyde agarose gel and blotted on to

a Nylon membrane as described previously [34] To deter-mine copy numbers of plasmids in yeast cells, DNA sam-ples were prepared as described above The DNA samsam-ples were digested with XhoI to linearize and run on a 1% aga-rose gel and blotted on to a Nylon membrane The mem-brane was hybridized with appropriate 32P-labeled BAR1,

TK, DED1, MAK16 or ACT1 coding sequences as probes After washing, the membrane was subjected to autoradiog-raphy followed by imaging analysis as described above

In vivo UV photofootprinting

An in vivo UV photofootprinting assay was performed as described previously [35,36] Yeast cells harboring a plasmid were grown in SC (Trp)) medium containing 1 mm dThd or BrdU at 30C for 15 h, irradiated with UV at 254 nm using

a Stratalinker 2400, and DNA samples purified As controls, DNA samples were purified from unirradiated cells and irra-diated similarly Sites and levels of the formation of UV photoproducts were determined by primer extension map-ping, using the IR-Dye-800-labeled MS-9 primer ()387 to )358 of the BAR1 coding strand) and MS-10 primer ()87 to

Trang 9

)116 of the BAR1 noncoding strand) with a LI-COR 4000L

DNA sequencer (LI-COR, Lincoln, NE, USA) [26,37]

Acknowledgements

This work was supported in part by Grants-in-Aid for

Scientific Research from the Ministry of Education,

Science and Culture of Japan

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Supporting information

The following supplementary material is available: Fig S1 Nucleosome positioning on pRS-BAR1 and its derivatives

Fig S2 Schematic representation of TALS-GFP and its derivative

Fig S3 Nucleosome positioning on TALS-GFP and pOM801-GFP

Fig S4 Effects of BrdU on EGFP mRNA levels This supplementary material can be found in the online version of this article

Please note: As a service to our authors and readers, this journal provides supporting information supplied

by the authors Such materials are peer-reviewed and may be re-organized for online delivery, but are not copy-edited or typeset Technical support issues arising from supporting information (other than missing files) should be addressed to the authors

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