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Tiêu đề Crystal structure of a subtilisin-like serine proteinase from a psychrotrophic vibrio species reveals structural aspects of cold adaptation
Tác giả Jóhanna Arnórsdóttir, Magnús M. Kristjánsson, Ralf Ficner
Trường học Georg-August Universität Göttingen
Chuyên ngành Molecular Structure Biology
Thể loại báo cáo khoa học
Năm xuất bản 2004
Thành phố Göttingen
Định dạng
Số trang 14
Dung lượng 651,83 KB

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Nội dung

The cold-adapted Vibrio proteinase was compared with known three-dimensional structures of homologous enzymes of meso- and thermo-philic origin, proteinase K and thermitase, to which it

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a psychrotrophic Vibrio species reveals structural aspects

of cold adaptation

Jo´hanna Arno´rsdo´ttir1, Magnu´s M Kristja´nsson2and Ralf Ficner1

1 Abteilung fu¨r Molekulare Strukturbiologie, Institut fu¨r Mikrobiologie und Genetik, Georg-August Universita¨t Go¨ttingen, Germany

2 Department of Biochemistry, Science Institute, University of Iceland, Reykjavı´k, Iceland

Microorganisms inhabit the most diverse environments

on earth Extremophiles are microorganisms that have

adapted to environmental conditions regarded by

humans as falling outside the normal range in terms of

temperature, pressure, salinity or pH Extremophiles

have had to develop strategies to deal with

environ-mental stress, mainly by molecular adaptation of their

cell inventory Of major importance in adapting to

extreme environmental conditions is the optimization

of protein function and stability Enzymes from

extremophiles are essentially like their mesophilic

counterparts, sharing the same overall fold and

catalysing identical reactions via the same mechanisms, while having adopted different traits regarding kinetic and structural properties Therefore, they provide excellent tools for examining the molecular basis of different protein properties, as well as the relation between structure and function in enzymes Regarding temperature, organisms have been isolated from places with temperatures as high as 113C [1] and biological activity has been detected in microbial samples at tem-peratures as low as )20 C [2] Thermo- and hyper-thermophiles face the challenge of keeping their macromolecules functional under the environmental

Keywords

cold adaptation; crystal structure;

psychrotrophic; subtilase; thermostability

Correspondence

R Ficner, Abteilung fu¨r Molekulare

Strukturbiologie, Institut fu¨r Mikrobiologie

und Genetik, Universita¨t Go¨ttingen,

Justus-von-Liebig-Weg11, 37077 Go¨ttingen,

Germany

Fax: +49 551 391 4082

Tel: +49 551 391 4072

E-mail: rficner@gwdg.de

Database

The coordinates and structure factors for

the final structure of Vibrio proteinase at

1.84 A ˚ resolution have been deposited in

the Protein Data Bank under the accession

number 1SH7.

(Received 30 September 2004, revised 26

November 2004, accepted 9 December

2004)

doi:10.1111/j.1742-4658.2005.04523.x

The crystal structure of a subtilisin-like serine proteinase from the psychro-trophic marine bacterium, Vibrio sp PA-44, was solved by means of molecular replacement and refined at 1.84 A˚ This is the first structure of a cold-adapted subtilase to be determined and its elucidation facilitates examination of the molecular principles underlying temperature adaptation

in enzymes The cold-adapted Vibrio proteinase was compared with known three-dimensional structures of homologous enzymes of meso- and thermo-philic origin, proteinase K and thermitase, to which it has high structural resemblance The main structural features emerging as plausible determi-nants of temperature adaptation in the enzymes compared involve the char-acter of their exposed and buried surfaces, which may be related to temperature-dependent variation in the physical properties of water Thus, the hydrophobic effect is found to play a significant role in the structural stability of the meso- and thermophile enzymes, whereas the cold-adapted enzyme has more of its apolar surface exposed In addition, the cold-adap-ted Vibrio proteinase is distinguished from the more stable enzymes by its strong anionic character arising from the high occurrence of uncompen-sated negatively charged residues at its surface Interestingly, both the cold-adapted and thermophile proteinases differ from the mesophile enzyme in having more extensive hydrogen- and ion pair interactions in their struc-tures; this supports suggestions of a dual role of electrostatic interactions

in the adaptation of enzymes to both high and low temperatures The Vibrio proteinase has three calcium ions associated with its structure, one

of which is in a calcium-binding site not described in other subtilases

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stress imposed by extreme thermal motion As a

response, they have evolved enzymes that are highly

stable against heat and other denaturants The

increased stability of enzymes from thermo- and

hyper-thermophiles is considered to reflect structural rigidity,

which in turn would account for their poor catalytic

efficiency at low temperatures The properties of

ther-mophilic enzymes have aroused great interest as they

have potential in biotechnology and diverse industrial

processes [3,4] In addition, the production of

thermo-philic recombinant enzymes is facilitated by their

relat-ively straightforward overexpression and purification,

which makes them feasible candidates for various

bio-chemical experiments as well as for crystal structure

determination These factors have enhanced research

on thermostability, which has been studied extensively

in the past, mainly by comparing the structural

proper-ties of thermo- and mesophilic enzymes, as well as

by using mutagenic experiments [5] In contrast to

enzymes from thermophiles, cold-adapted enzymes are

relatively poorly examined, in particular considering

their extensive distribution and occurrence in our

bio-sphere Organisms occupying permanently cold areas

that dominate the earth’s surface, collectively called

psychrophiles, have to rely on enzymes that can

com-pensate for low reaction rates at their physiological

temperatures The properties that characterize and

dis-tinguish cold-adapted enzymes from enzymes

origin-ating at higher temperatures are their increased

turnover rate (kcat) and inherent higher catalytic

effi-ciency (kcat⁄ Km) at low temperatures [6] It is assumed

that optimization of the catalytic parameters in

cold-adapted enzymes is accomplished by developing

increased structural flexibility, allowing the

conforma-tional changes required for catalysis at low

tempera-tures [7] In recent years, a few crystal structempera-tures of

cold-adapted enzymes have been determined [8–16]

These structures have served as a basis in comparative

studies on structural aspects of cold adaptation Also,

information from site-directed mutagenesis

experi-ments, homology modelling and directed evolution has

been used in an effort to shed light on the molecular

principles underlying the adaptation of enzymes to low

temperatures [17–24] In general, regardless of whether

research is directed at thermo- or psychrophilic

adap-tation, the results show that each protein family adopts

its own strategies for coping at extreme temperatures

Although no general rules have been found to apply in

temperature adaptation in enzymes, some structural

tendencies have emerged The most frequently reported

features related to temperature adaptation, going from

higher to lower temperatures, are a reduced number of

noncovalent intra- and intermolecular interactions, less

compact packing of the hydrophobic core, an increased apolar surface area, decreased metal ion affinity, longer surface loops and a reduced number of prolines in loops [5,6,8,25–28] In general, in naturally occurring enzymes, a correlation is seen between cata-lytic efficiency at low temperatures and susceptibility

to heat and other denaturants [29] However, using directed evolution methods, mutants have been obtained with changes in one of the properties, stabil-ity or catalytic efficiency, indicating that these pro-perties are not essentially interlinked [22,23] The observed instability of cold-adapted enzymes is regar-ded not as a selected for property, but rather as a consequence of the reduction in stabilizing features arising from the need for increased flexibility to main-tain catalytic efficiency at low temperatures [30] Structural flexibility in cold-adapted enzymes is, as yet, a poorly defined term for which little direct experi-mental evidence is available Attempts to assess and compare the structural flexibility of a psychrophilic a-amylase and more thermostable homologues using dynamic fluorescence quenching supported the idea of

an inverse correlation between protein stability and structural flexibility [31] Comparisons of hydrogen– deuterium exchange rates as a way of estimating flexi-bility in enzymes originating at different temperatures [32] have supported the idea of ‘corresponding states’ [33], which assumes that, at their physiological temper-atures, enzymes possess comparable flexibility and a structural stability adequate to maintain their active conformation

In order to improve the understanding of the struc-tural principles of temperature adaptation we studied a subtilisin-like serine proteinase from the psychrotrophic marine bacterium, Vibrio sp PA-44 The Vibrio prote-inase belongs to the proteprote-inase K family and has a high sequence identity of 60–87% with several meso- and thermophilic family members [34] Furthermore, it has 41% sequence identity and 57% similarity with protein-ase K, the best characterized representative of this pro-tein family, the three-dimensional structure of which has been determined to atomic resolution [35] The Vibrio proteinase has been identified as showing clear cold-adaptive traits in comparison with its meso- and thermophilic homologues [36] Thorough sequence and computer model comparisons performed on the Vibrio proteinase and its most closely related meso- and thermophilic enzymes have revealed some differences, possibly relevant to temperature adaptation [34] The results have given rise to ongoing mutagenic research in which single and combined amino acid substitutions aimed at increasing the stability of the Vibrio ase are being tested Elucidation of the Vibrio

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protein-ase structure, the first structure of a cold-adapted

subti-lase to be determined, enables a more focused

examina-tion of plausible determinants of different temperature

adaptation among subtilases

We crystallized the cold-adapted Vibrio proteinase in

the presence of bound inhibitor,

phenyl-methyl-sulfo-nate, and the structure was refined at 1.84 A˚

resolu-tion In order to identify parameters that might be

important with respect to cold adaptation we analysed

and compared structural features in Vibrio proteinase

and the two most closely related enzymes of known

three-dimensional structure, proteinase K from the

mesophilic fungi Tritirachium album Limber and

thermi-tase from the thermophilic eubacterium

Thermoactino-mycetes vulgaris

Results

The crystal structure of the Vibrio proteinase

The obtained Vibrio proteinase crystals formed clusters

of needles, which transformed into thin platelets within

a few days The crystals belong to space group P21with

unit cell dimensions of a¼ 43.2 A˚, b ¼ 36.9 A˚, c ¼

140.5 A˚ and b¼ 97.8 The Matthews coefficient [37]

(Vm¼ 1.9 A˚3⁄ Da) suggested two molecules in the

asymmetric unit with a solvent content of 36.3% The

structure was determined by molecular replacement

using a homology model based on the known structure

of proteinase K (PDB accession number, 1IC6) as a

search model The crystallized 30 kDa catalytic domain

of Vibrio proteinase encompasses amino acids 140–420

of the 530 amino acid prepro-enzyme [34] The model

was refined at a resolution of 1.84 A˚ with an R-factor

of 14.1% and an Rfreevalue of 19.6% (Table 1)

Figure 1 shows the three-dimensional structure of

Vibrio proteinase, hereafter referred to as 1SH7

according to its PDB accession number The structure

shows the a⁄ b scaffold characteristic of subtilisin-like

serine proteinases It consists of six a helices, one

3⁄ 10 helix, a b sheet made of seven parallel strands

and two b sheets made of two antiparallel strands

(Fig 1B) Determination of the structure confirms the

presence of three previously predicted disulfide bonds,

Cys67–Cys99, Cys163–Cys194 and Cys277–Cys281

[34] Three calcium-binding sites are found in 1SH7,

two of which were predicted based on sequence

align-ments and one as yet not described in other subtilases

The active site of 1SH7 consists of the catalytic triad

Asp37, His70 and Ser220, and substrate recognition

and binding sites that are well conserved among

subti-lases [38] The substrate-binding site in 1SH7 appears

on the surface as a relatively distinct cleft (see below,

‘Surface properties and packing’) in which the sub-strate is accommodated by forming a triple-stranded antiparallel b sheet with residues of the S4- and S3-binding sites (nomenclature of subsites, S4–S2¢, is according to Schechter and Berger [39]) The bottom

of the S1 substrate-binding pocket is made up of resi-dues A154–A155–G156 and the oxyanion hole residue N157 The substrate-binding cleft appears to be relat-ively open with T105 at the rim of S4; in many subti-lases this site is occupied by a larger residue, typically

a tyrosine (e.g subtilisin and proteinase K), which is assumed to form a flexible lid on the S4 pocket [40]

Overall structure comparison with related enzymes from meso- and thermophiles

A 0.98 A˚ resolution structure of proteinase K (PDB accession number 1IC6) and a 1.37 A˚ resolution struc-ture of thermitase (PDB accession number 1THM), were used for structural comparison with 1SH7 The high resolution of all three structures allows reasonable comparison with respect to the quality of the models Pairwise least square superposition of the three

Table 1 Data collection and refinement statistics for 1SH7 Num-bers in parenthesis refer to the highest resolution shell.

Data collection

Unit cell parameters

a ¼ 43.2 A˚

b ¼ 36.9 A˚

c ¼ 140.5 A˚

? ¼ 97.80

Refinement statistics

Rcryst⁄ R freeb(%) 14.1(22.6)⁄ 19.6(29.8) Rms deviation from ideality

Bonds (A ˚ ) ⁄ angles () 0.014 ⁄ 1.521 Average B-values (A ˚ 2 )

Protein ⁄ water ⁄ PMSF ⁄ Ca 2+ 13.3⁄ 25.4 ⁄ 34.1 ⁄ 11.9 Ramachandran plotc

Most favoured, additional, generously allowed (%)

89.9⁄ 9.9 ⁄ 0.2

a Rsym¼ 100ÆS h S i |Ii(h) – < I(h) > | ⁄ S h I(h), where Ii(h) is the ith meas-urement of the h reflection and < I(h) > is the average value of the reflection intensity.bR cryst ¼ S|F o – F c | ⁄ S |F o |, where F o and F c are the observed and calculated structure factors, respectively Rfreeis

R cryst with 10% of test set structure factors c Calculated with PRO-CHECK [82].

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structures, with a cut-off distance of 3.5 A˚ showed that

85–93% of the Ca-atoms lie at common positions and

gave a root mean square deviation of 0.84–1.21 A˚

(Table 2, Fig 2) The structural resemblance with

regard to root mean square deviation, fraction of com-mon Ca-atoms and the amino acid sequence identity,

is in the order 1SH7–1IC6 > 1SH7–1THM > 1IC6– 1THM The distance deviations of the superposed structures and the locations of insertions and⁄ or dele-tions are restricted to a few parts of the structure The most distinct differences are seen in the N- and C-ter-minal regions, where 1THM aligns poorly with both 1SH7 and 1IC6 The C-termini of 1IC6 and 1SH7 also diverge; the last four residues of 1IC6 are not equival-ent to residues in 1SH7 Furthermore, 1SH7 has an extended C-terminus relative to 1IC6 The four regions that deviate considerably owing to multiple residue insertions and deletions are marked in Fig 2 as des-cribed below First, a surface loop region, Phe57– Asn68 in 1SH7 does not align with 1IC6 This loop is identical in 1SH7 and 1THM and hosts a calcium-binding site that has been described as a medium– strong calcium-binding site in thermitase [41] Second, relative to both 1THM and 1SH7, 1IC6 has an inser-tion in an extended surface loop, residues 119–125 in 1IC6 This surface loop in 1IC6 contains some plaus-ible stabilizing features, a disulfide bridge, Cys34– Cys123, and a salt bridge, Asp117–Arg121 Third, a loop region connecting a helices E, carrying the Ser of the catalytic triad, and the succeeding a helix F is not well conserved among the enzymes and the structures are accordingly variable Fourth, 1SH7 contains a new calcium-binding site This part of the structure is noticeably different from the corresponding regions in proteinase K and thermitase If the allowed distance between equivalent Ca-atoms is defined as being within

2 A˚, the ratio of Ca-atoms common to 1SH7 and the other two structures remains > 80% The high struc-tural homology of these enzymes which originate at different temperatures gives an opportunity to examine structural features that might contribute to their differ-ent temperature adaptation

Charged residues and ion pairs Thermitase contains 30 charged side chains, whereas proteinase K and the Vibrio proteinase each contain

38 The Vibrio proteinase differs from the enzymes with which it is compared in that it has a higher pro-portion of negatively charged side chains (Table 3) Charged residues reside on the protein surface in regions that are the least conserved Superposition of 1SH7, 1IC6 and 1THM revealed that at seven sites there are identically charged side chains in all three proteins Also, each pair of enzymes, 1SH7–1IC6, 1SH7–1THM and 1IC6–1THM, has 4–6 side chains with the same charge in equivalent positions Thus,

Fig 1 (A) Model of the crystal structure of the Vibrio proteinase.

The residues of the catalytic triad, S220, H70 and D37 are shown

in yellow, the calcium ions as green spheres and the disulfide

brid-ges in orange (B) A topology diagram of the Vibrio proteinase

structure.

Table 2 Pairwise superposition of Ca-atoms in 1SH7, 1IC6 and

1THM with a cut-off of 3.5 A ˚

1SH7–1IC6 1IC6–1THM 1SH7–1THM Number of residues 281–279 279–279 281–279

Aligned residues 261 (93%) 238 (85%) 246 (88%)

Root mean square

deviation (A ˚ )

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conservation of charged residues is comparable with

the overall homology of these structures, being in the

range of 30–40%

The tendency for more salt-bridges with increasing temperature of origin, which is frequently observed when comparing related structures, cannot be con-firmed for the enzymes in this study Ionic interactions,

as defined here, are restricted to two oppositely charged residues (Asp, Glu, Arg and Lys) within a dis-tance of 4 A˚ The meso- and psychrophilic structures have the same number of salt-bridges and only two fewer than the thermophilic structure (Table 3) An important aspect of the proposed contribution of salt-bridges to protein stability resides in their location and distribution Bae and Phillips [13] recently defined as

‘critical ion pairs’ for temperature adaptation, those ion pairs that are not conserved between the structures compared and bridging residues of distant regions (> 10 residues) of the polypeptide chain Four non-conserved ion pairs in 1IC6 link residues that are four

or fewer residues apart in the polypeptide chain In contrast, all the salt-bridges in 1SH7 and all but one

in 1THM, involve residues more than 10 residues apart (Table 4) The higher number of critical ion pairs in 1SH7 and 1THM, which contain seven such inter-actions each, compared with three in 1IC6, supports the possible significance of salt-bridges in the adapta-tion of enzymes to hot as well as to cold environments

Fig 2 Stereoview of the superposition of the cold-adapted Vibrio proteinase (1SH7, blue) with (A) proteinase K (1IC6, green) and (B) thermitase (1THM, red) Calcium ions (same colour as the protein they belong to) and a sodium ion (beige) bound to thermitase are shown as spheres The numbering relates to the four regions that deviate due to multiple insertion and deletions as described in the text.

Table 3 Comparison of structural features of 1SH7, 1IC6 and

1THM.

Number of noncompensated

charged residues

(D + E) ⁄ (R + K) (16 ⁄ 7) (10⁄ 13) (7 ⁄ 8)

Number of hydrogen bonds

Exposed surface areab(A˚2) 10 115 10 079 9822

Buried surface area b (A˚2 ) 31 695 32 013 31 714

a

An interaction is assigned to a salt bridge where distance

between atoms of opposite charge is within 4 A ˚ Interactions

invol-ving histidine are not included b Solvent accessible surface area for

residues 1–275 of each enzyme c Carbon and sulphur atoms.

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[42] There is one common ion pair, Asp183–Arg10

(numbers relate to 1SH7), in all three enzymes,

con-necting sites that are otherwise not well conserved in

1THM relative to 1SH7 and 1IC6 1SH7 and 1THM

share an ion pair arrangement, Asp56–Arg95 and

Asp59–Arg95 (numbers relate to 1SH7), connecting

the surface loop that hosts their common

calcium-binding site to a site proximate to the

substrate-bind-ing site (Fig 3) Critical ion pairs are found in both

1IC6 and 1THM bridging the a helices C and D, which

are directly connected to the substrate-binding loops

In 1THM, the ion pair network formed by Asp188–

Arg270, Asp257–Arg270 and Asp257–Lys275 tethers

the C-terminus Such tethering has been suggested to

contribute to increased stability in other proteins

[43] Thus, by observing single ion pair interactions,

differences emerge that cannot be seen merely by counting interactions In the context of estimating the effect of salt-bridges on protein stability, their accessi-bility to solvent is highly important We thus checked solvent accessibility in the ion pairs forming salt-brid-ges in the three protein structures, but such compari-sons did not reveal any trends in terms of the temperature adaptation of the enzymes

Hydrogen bonds Due to their large number, hydrogen bonds play a substantial role in the stability of proteins The num-ber and type of hydrogen bonds are frequently repor-ted as factors correlarepor-ted to temperature adaptation in proteins [44,45] but the evidence is far from conclusive [46,47] The total number of hydrogen bonds in the cold-adapted 1SH7 is higher than in 1IC6 and compar-able with the number in 1THM (Tcompar-able 3) Further-more, the number of side chain-side chain and main chain-side chain hydrogen bonds was found to

be lowest in the mesophilic structure, 1IC6

Calcium-binding sites The presence of bound calcium ions is a feature shared

by members of the subtilisin superfamily, where cal-cium binding has been shown to be essential for correct folding and structural stability [48,49] Consid-ering the stabilizing effect of binding metal ions in many proteins, it might be expected that increased affinity and the number of bound metal ions should correlate with the thermostability of proteins Differ-ences in stability and kinetic properties between meso-and psychrophilic enzymes have, in fact, been related

to fewer or weaker metal ion binding sites in the latter [50–52] In the case of thermitase, differences in

Table 4 Listing of salt-bridges and the shortest distances between

charged atoms Salt-bridges are restricted to a distance of 4 A˚

between charged atoms of the residues: Asp, Glu, Arg and Lys.

Conserved ion pairs are in the upper row Critical ion pairs [13] with

respect to both of the compared enzymes are underlined.

D183–R10 2.74 A˚ D187–R12 2.77 A˚ D188–K17 3.91 A˚

(E27–87 4.65 A ˚ ) a

E28–K95 2.79 A˚ D138–R169 3.02 A ˚ E48–R80 3.93 A˚ D124–K153 3.20 A˚

E236–R252 2.83 A˚ E50–R52 2.95 A˚ D188–R270 2.81 A˚

E255–K267 2.81 A˚ D98–K94 2.75 A˚ D201–R249 3.41 A˚

D260–R185 2.92 A ˚ D112–R147 2.76 A˚ E253–R249 2.98 A˚

D274–R14 3.28 A˚ D117–R121 2.94 A˚ D257–R270 2.80 A˚

D184–R188 3.02 A˚ D257–K275 2.78 A˚ D260–R12 3.02 A ˚

a

The criterion of conserved ion pairs is when the distance between

corresponding charged residues is within 6 A ˚ Therefore, although

not defined here as a salt bridge, this interaction excludes the

cor-responding ion pair in 1THM from being critical in this comparison.

Fig 3 Comparison of the distribution of salt-bridges in the Vibrio proteinase (1SH7, blue), proteinase K (1IC6, green) and thermitase (1THM, red) Yellow spheres represent critical salt-bridges, i.e nonconserved interactions between oppositely charged groups more than 10 residues apart in the polypeptide chain, and grey spheres represent noncritical salt-bridges The catalytic triad, the disulfide bridges (orange) and the calcium ions (spheres) are also displayed as reference points.

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calcium binding were considered as one of the major

reasons for the enhanced stability of the enzyme as

compared with its mesophilic counterparts [53]

Surpri-singly, three calcium ions are found associated with

the structure of 1SH7, whereas 1IC6 and 1THM have

two each (Figs 1 and 2) At one of the binding sites,

Ca1, which is analogous to the known strong

calcium-binding site Ca1 in proteinase K [54], the calcium ion

in 1SH7 is coordinated by Od1 and Od2 of Asp196,

the carbonyl-oxygen of Pro171 and Gly173 and two

water molecules According to sequence alignments,

this site is well conserved among members of the

pro-teinase K family, including enzymes of thermo- and

mesophilic origin most related to the Vibrio proteinase

The second calcium-binding site in 1SH7 corresponds

to the described, second or medium strength

calcium-binding site, Ca2, of 1THM [53] Od1 and Od2 of

Asp61, Od1 of Asp56, the carbonyl oxygen of Asp63

and three water molecules coordinate the calcium ion

According to sequence alignments, this

calcium-bind-ing site should also be present in the highly

homolog-ous proteinases from Vibrio alginolyticus and Vibrio

cholerae, but absent in the thermophilic proteinase

from Thermus Rt41a and aqualysin I from Thermus

aquaticus The third, additional calcium-binding site of

1SH7, Ca3 (Fig 1), has not yet been found in known

proteinase structures The calcium ion links the a helix

A and residues of the succeeding surface loop and it is

coordinated by the side chain and carbonyl oxygen of

Asp9, the side chains of Asp12, Gln13, Asp19, the

car-bonyl oxygen of Asn21 and one water molecule in a

pentagonal bipyramidal manner (Fig 4A) Sequence

alignments indicate that this new calcium-binding site

is most likely present in the closest relatives (Fig 4B)

Calcium binding plays a critical role in the stability of

the Vibrio proteinase, as in the case of related enzyme

(M.M Kristja´nsson, unpublished results) From the

structural comparisons carried out here it is difficult,

however, to deduce how or whether differences in

calcium-binding sites contribute to temperature

adap-tation in the enzymes involved

Surface properties and packing

The chemical properties of the groups comprising

pro-tein surfaces are expected to be important for

adapta-tion of protein funcadapta-tion to both high and low

temperatures, as these determine the important

inter-actions of the protein with water; interinter-actions which

are highly dependent on temperature as a result of

changes in the structure of water [55–57] A larger

frac-tion of polar surface in a number of thermophilic

pro-teins has been suggested to contribute to their increased

stability [46,58,59] In several cases, differences in sur-face charge distributions or an increase in nonpolar surface area have been suggested as relevant in the adaptation to low temperatures [8,12,14,15,52] In cit-rate synthases adapted to different temperatures a clear trend was observed in the reduced exposure of apolar surfaces in proceeding from psychrophile to hyper-thermophile structures [60] Thermo-, and in particular hyperthermophilic, proteins have been reported to have improved packing and fewer and smaller cavities in their protein core relative to mesophiles [46] Other sta-tistical approaches analysing structural parameters in large samples of dissimilar proteins regarding the origin and temperature range, do not show significant trends regarding the polarity of protein surfaces or different degrees of packing [42,44,47]

Cold-adapted 1SH7 and mesophilic 1IC6 have a lar-ger solvent accessible surface area and a larlar-ger non-polar surface area than 1THM (Table 3) Thus, among these enzymes the recurring trend in thermophilic enzymes to reduce their exposed apolar surfaces is observed The total area of buried surfaces is similar for the three enzymes, but their composition is differ-ent in that 1SH7 buries significantly less apolar surface than either 1IC6 or 1THM By the same token, more buried surface in the cold enzyme is polar than in either the meso- or thermophilic enzyme (Table 3) The larger buried apolar surface of 1IC6 and 1THM

Fig 4 (A) Stereoview of the new calcium-binding site, Ca3, found

in the structure of the Vibrio proteinase The calcium ion is coordi-nated in a pentagonal bipyramidal manner by the carboxyl groups

of D9 and N21, the side chain oxygen atoms of D9, D12, Q13, D19 and one water molecule (B) Sequence containing the residues forming Ca3 (shaded with yellow) in the Vibrio proteinase is well conserved among the most related enzymes of meso- (proteinases from Vibrio alginolyticus, Vibrio cholerae, Kytococcus sedentarius and Streptomyces coelicolor) and thermophilic origin (aqualysin I from Thermus aquaticus and proteinase from Thermus sp Rt41a).

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would be expected to contribute to the higher stability

of these enzymes via the hydrophobic effect The effect

of the larger buried apolar surface can be estimated to

be in the range 5.7 to 15.6 kcalÆmol)1 between 1IC6

and 1SH7 and 5.3 to 14.3 kcalÆmol)1 between 1SH7

and 1THM, when calculated as suggested by Criswell

et al [61] Thus, the cold-adapted enzyme would be

less dependent on the hydrophobic effect for stability

than its counterparts adapted to higher temperatures

In fact, Kristjansson and Magnusson [62] reached the

same conclusion from their study of the effects on

lyotropic salts on the stability of Vibrio proteinase,

proteinase K and the thermophilic homologue,

aqua-lysin I It remains debateable, however, whether this

observation, as well as reported cases of larger exposed

apolar surfaces in cold enzymes, is merely a

conse-quence of a diminished hydrophobic effect at low

tem-perature, or if it is part of a molecular strategy of cold

adaptation Because of the ordering of water structure

at low temperature (i.e below approximately the

tem-perature of maximum stability) the entropic penalty

for exposing apolar surfaces is reduced and so too is

the hydrophobic effect [57] At these low temperatures

destabilization of the protein structure is therefore

enthalpically controlled, both as a result of the ordered

water structure [57], and via interactions of water

with both apolar and polar groups of the protein

[55,56,63,64] Hence the entropically driven

hydropho-bic effect would be expected to contribute less to the

overall stability of the proteins at low temperatures, or

to destabilize them locally or globally, which, in effect,

may lead to more open and resilient structures

A notable difference in the surfaces of the proteins

compared here is their different surface electrostatic

potentials (Fig 5) Reflecting the different occurrence

of noncompensated negative charges, as mentioned

above and shown in Table 3, large parts of the surface

of 1SH7 are negatively charged, whereas 1IC6 and

1THM have less charged or positively charged

surfa-ces Furthermore, the substrate-binding cleft of 1SH7

differs from that of 1IC6 and 1THM in shape, being

seemingly deeper and more distinct, and in being more

negatively charged than the binding pockets of 1IC6

and 1THM (Fig 5) The biological implication of this

difference with respect to different temperature

adapta-tion is not clear Interestingly, however, Vibrio

protein-ase shares its anionic character with several other

cold-adapted enzymes [34,43] The more anionic charge

of rat trypsinogen, compared with the bovine

homo-logue, has been suggested as a source of increased

flexibility in the former [65] Also, a group of highly

flexible proteins, the natively unfolded proteins, are

characterized by a large (predominantly negative) net

charge [66] It has been suggested that a higher number

of uncompensated charged residues on protein surfaces may contribute to cold adaptation by providing stron-ger interaction energy with the highly ordered water structure at low temperatures [42] As reflected in a significant increase in surface tension and viscosity at low temperatures, water is optimally hydrogen bonded The energetic cost of the dissolution of a protein under such conditions, arising from the unfavourable disrup-tion of the optimized hydrogen bond network, may be offset by favourable electrostatic interactions of the charged groups with water at the protein surface [42] Among amino acid residues, only Arg is more soluble than Glu or Asp [67] Thus, endowing the protein

Fig 5 Comparison of the electrostatic surface potentials of (A) 1SH7, (B) 1IC6 and (C) 1THM On the right-hand side, the mole-cules have been rotated 180 about the y-axis The approximate locations of substrate binding pockets, S1–S4 (nomenclature according to [39]) and the oxyanion hole residue, N157, are labelled

on the surface of the Vibrio proteinase (A) The positive potential is

in blue and the negative potential is in red The electrostatic surface potential was calculated with Delphi [81] and the graphical presen-tations were made in PYMOL

Trang 9

surface with their hydrophilic nature may enhance

favourable electrostatic interaction with water at low

temperature and, at the same time, result in an anionic

character, which may favour a more disordered or

flex-ible structure

Disulfide bridges

There are three disulfide bridges in the structure of

1SH7 (Fig 1) In 1SH7 Cys67–Cys99 connects the

loop carrying the Ca2-binding site and the loop

con-taining the residues of substrate-binding pocket S4

The second disulfide bridge in 1SH7, Cys163–Cys194,

bridges residues next to the Ca1-binding site and a

region carrying residues of the substrate-binding

pocket S1 According to sequence alignment, these two

disulfide bridges are highly conserved among the

enzymes most closely related to the Vibrio proteinase

including aqualysin I The third disulfide bridge in

1SH7, Cys277–Cys281, is at the C-terminus The

struc-ture of 1IC6 contains two disulfide bridges that,

although not identical to those found in 1SH7, also

link parts of the structure directly connected to the

substrate-binding sites There is no disulfide bridge in

1THM The higher number of disulfides in 1SH7

relat-ive to its related enzymes and the absence of such

bonds in 1THM is not evidence of disulfides playing a

critical role in the different temperature adaptation of

the enzymes compared here This is also consistent

with what is seen in a psychrophilic subtilisin that

con-tains the same or higher numbers of disulfide bridges

as highly homologous mesophiles [51,52] Only in rare

cases has the introduction of disulfide bridges by

muta-genesis resulted in increased stability [68,69] Based on

comparison of the reactivity towards sulfitolysis and

dithiothreitol, the disulfide bridges of the Vibrio

prote-inase were previously suggested to be more accessible

to solvent than proteinase K and the thermophilic

aqu-alysin I [36] This is confirmed by anaqu-alysing the surface

accessibility of the disulfide bridges in the structures

compared here, 1SH7 and 1IC6, and hence is assumed

to also apply to aqualysin I, which contains the two

conserved disulfide bridges of 1SH7 The disulfide

bridges in those enzymes are found in regions where

many supposedly stabilizing features, such as

calcium-binding sites and ion pairs come together, and they

have both sequential and spatial proximity to parts

involved in substrate binding This, although crucial

for the active conformation of the Vibrio proteinase

[36], might have some relevance to temperature

adap-tation First, it might reflect a tendency for the more

stable enzymes to protect critical parts of the structure

by decreasing their solvent accessibility Second, the

absence of disulfide bridges in THM is in line with the observed tendency of thermophilic enzymes to have a reduced occurrence of thermolabile residues [5]

Discussion

From the comparison of the three subtilases in this study, we observe some structural differences that may

be important for their temperature adaptation First, whereas the overall exposed surface areas of the psy-chro- and the mesophilic enzymes are larger than for the thermophile enzyme, mainly as a result of larger area of apolar atoms, the meso- and thermophilic enzymes bury significantly more apolar surface in their folded structures than the cold-adapted enzyme We, therefore, conclude that the higher number of hydro-phobic interactions in the meso- and thermophilic pro-teins contributes to their increased stability relative

to the cold-adapted Vibrio proteinase This is in line with previous experimental results on the effects of lyotropic salts on the conformational stability of the Vibrio proteinase, proteinase K and the thermophilic relative, aqualysin I, in which the cold enzyme was shown to be less dependent on hydrophobic inter-actions for structural stability than its counterparts of higher temperature origin [62] Furthermore, this find-ing was supported by comparative sequence analysis [34] These results also agree with the thermodynamics

of the hydrophobic effect in protein stabilization, being enforced by increasing temperature and thus stabilizing structures at high temperatures, at least to a certain extent, but diminishing in strength at lower tempera-tures The diminished hydrophobic effect at low tem-peratures may account for the larger exposure of apolar surfaces observed in the Vibrio proteinase and several other reported cold-adapted enzymes [8,15, 50,70], relative to enzymes adapted to higher tempera-tures To address questions regarding the proposed role of the increased exposed apolar surface as a mech-anism of cold adaptation, it should be considered that interactions of such surfaces with water at low temper-atures may be quite different to what might be observed at higher temperatures as a result of tempera-ture dependence of the properties of water According

to Robinson and Cho [64] polar surface groups give rise to a lower entropy and lower enthalpy in the sur-rounding water, whereas apolar groups would have the opposite effect Whether this proposed effect of apolar groups in promoting less order in the highly ordered water structure at low temperatures influences protein motions remains to be determined

Another surface property in which the Vibrio protei-nase differs from the other enzymes compared in this

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study is its increased anionic character Cold-adapted

enzymes are frequently found to be more anionic than

their homologues adapted to higher temperatures It is

not clear, however, whether this property makes any

contribution to cold adaptation Anionic character has

been suggested to promote flexibility in trypsinogens,

but a possible mechanism for this observation was

not provided [65] Kumar and Nussinov [42] have

pointed out the possible dual roles of electrostatics in

the adaptation of protein to both high and low

tem-peratures In cold- adapted enzymes it was suggested

that charges could ensure proper solvation against the

higher surface tension and viscosity characterizing

water at low temperatures, and might also impart

greater flexibility, especially in active site regions [42]

Interestingly, analysis of the amount and pattern of

electrostatic forces in the enzymes compared here

sup-ports this view

Interactions at the protein–water interface are

cru-cial for the function and stability of proteins These

interactions are affected by temperature, not least

because of changes in the structure, and consequently

the properties, of water Thus, some of the molecular

strategies in the temperature adaptation of proteins

must be aimed at accommodating the

temperature-dependent changes in the structure and physical

prop-erties of water Clearly, more information is needed in

this area to gain a better insight into the forces that

facilitate cold adaptation in proteins

Experimental procedures

Expression and purification

Production of Vibrio proteinase for crystallization

prepara-tions was based on the previously established expression

sys-tem [34] and the purification protocol described for the

proteinase from Vibrio strain PA-44 [36], with the following

modifications Expression of the Vibrio proteinase gene

cloned in the pBAD TOPO vector was carried out in 12 L

cultures of Escherichia coli strain Top10 (Invitrogen,

Carls-bad, CA) at 18C in a bioreactor (Applikon Biotechnology,

Schiedam, the Netherlands) Cells were harvested 12 h after

induction with 0.025% l-arabinose and addition of CaCl2

to a final concentration of 10 mm For one preparation, the

cell pellet from 6-L culture was suspended in 300 to 400 mL

of basic buffer (buffer A: 25 mm Tris, pH 8.0 containing

10 mm CaCl2) and disrupted by running it five times, with

5 min intermediate incubations on ice, through a

microfluid-iser (MicrofluidicsTM) at 550 kPa pressure The crude cell

extract was centrifuged at 15 000 g for 15 min at 4C The

protein in the supernatant was precipitated by a 75%

sat-uration of ammonium sulfate and centrifuged at 15 000 g

for 30 min at 4C The pellet was redissolved in buffer A containing 1 m (NH4)2SO4and centrifuged at 100 000 g for

1 h at 4C to remove insoluble impurities Subsequent puri-fication steps were carried out at 4C using the A¨kta system (Amersham Biosciences, Freiburg, Germany) The protein solution was loaded onto a phenyl⁄ Sepharose column (16⁄ 10 Amersham Biosciences) equilibrated with buffer A containing 1 m (NH4)2SO4 Elution was achieved by a 20 column volume gradient of 1 to 0 m (NH4)2SO4 and frac-tions were tested for activity with succinyl-AlaAlaProPhe-p-nitroanilide The fractions containing proteolytic activity were pooled and applied to a 2 mL N-carbobenzoxy-d-phenylalanyl-triethylenetetramine⁄ Sepharose column [71] equilibrated with buffer A After washing with 0.5 m NaCl, the Vibrio proteinase was eluted with buffer A containing

2 m GdmCl Fractions of 2.5 mL were collected into tubes containing 2 mL of 3 m (NH4)2SO4in buffer A The pooled fractions containing proteolytic activity were loaded onto a

5 mL phenyl⁄ Sepharose column (Hitrap Phenyl FF, Amer-sham Bioscience) equilibrated with buffer A containing 1 m (NH4)2SO4and eluted with a 20 column volume gradient of

1 to 0 m (NH4)2SO4 The purified 40 kDa Vibrio proteinase was concentrated to 3 to 6 mgÆmL)1by of salting out with 75% saturated ammonium sulfate, adding 3 parts of a satur-ated ammonium sulphate solution to 1 part of protein solu-tion The solution was centrifuged and the precipitate resuspended with buffer A at a concentration of 5 mgÆmL)1

At this point, the protein was divided into aliquots, flash cooled in liquid nitrogen and stored at )80 C Aliquots containing the purified 40 kDa Vibrio proteinase were incu-bated at 40C for 50 min to give the mature 30 kDa enzyme, which was then inhibited with phenylmethylsulfo-nyl fluoride in a final concentration of 1 mm and applied onto a Superdex 75 column (HR 10⁄ 30, Amersham Bio-sciences) equilibrated with 10 mm Tris pH 8.0 and 10 mm CaCl2 Fractions containing the 30 kDa Vibrio proteinase were pooled and concentrated in centrifugal concentrators (Centricon and Minicon from Millipore) for crystallization trials

Crystallization and data collection

Recombinant Vibrio proteinase was crystallized using the sitting drop method The protein solution used in the initial crystallization trials was 2.5 mgÆmL)1 protein in 10 mm Tris⁄ Cl pH 8.0 and 10 mm CaCl2 A promising condition was found using the Hampton Crystal Screen 1 condition

41 (10% 2-propanol, 20% PEG 4000, 0.1 m Hepes pH 7.5) where clusters of needles grew overnight After variations

of temperature, pH and concentrations of the precipitant and protein solutions, well-diffracting crystals were obtained by mixing in equal volumes of a protein solution

of 6 mgÆmL)1 and a precipitant solution containing 15% PEG 4000, 10% isopropanol, 0.1 m Tris⁄ Cl pH 8.0 at

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