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Tiêu đề Biochemical characterization of Bacillus subtilis type II isopentenyl diphosphate isomerase, and phylogenetic distribution of isoprenoid biosynthesis pathways
Tác giả Ralf Laupitz, Stefan Hecht, Sabine Amslinger, Ferdinand Zepeck, Johannes Kaiser, Gerald Richter, Nicholas Schramek, Stefan Steinbacher, Robert Huber, Duilio Arigoni, Adelbert Bacher, Wolfgang Eisenreich, Felix Rohdich
Trường học Technische Universität München
Chuyên ngành Biochemistry
Thể loại Research article
Năm xuất bản 2004
Thành phố Garching, Germany
Định dạng
Số trang 12
Dung lượng 641,68 KB

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The analysis of the occurrence of idi-1 and idi-2 genes in conjunction with the mevalonate and nonmevalonate pathway in 283 completed and unfinished prokaryotic genomes revealed 10 differe

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Biochemical characterization of Bacillus subtilis type II isopentenyl diphosphate isomerase, and phylogenetic distribution of isoprenoid biosynthesis pathways

Ralf Laupitz1, Stefan Hecht1, Sabine Amslinger1, Ferdinand Zepeck1, Johannes Kaiser1, Gerald Richter1, Nicholas Schramek1, Stefan Steinbacher2, Robert Huber3, Duilio Arigoni4, Adelbert Bacher1,

Wolfgang Eisenreich1and Felix Rohdich1

1

Lehrstuhl fu¨r Organische Chemie und Biochemie, Technische Universita¨t Mu¨nchen, Garching, Germany;2Division of Chemistry and Chemical Engineering, California Institute of Technology, Pasadena, CA, USA;3Abteilung fu¨r Strukturforschung,

Max-Planck-Institut fu¨r Biochemie, Martinsried, Germany;4Laboratorium fu¨r Organische Chemie, Eidgeno¨ssische Technische Hochschule Zu¨rich, Switzerland

An open reading frame (Acc no P50740) on the Bacillus

subtilis chromosome extending from bp 184 997–186 043

with similarity to the idi-2 gene of Streptomyces sp CL190

specifying type II isopentenyl diphosphate isomerase was

expressed in a recombinant Escherichia coli strain The

recombinant protein with a subunit mass of 39 kDa was

purified to apparent homogeneity by column

chromatog-raphy The protein was shown to catalyse the conversion of

dimethylallyl diphosphate into isopentenyl diphosphate and

vice versa at rates of 0.23 and 0.63 lmolÆmg)1Æmin)1,

respectively, as diagnosed by1H spectroscopy FMN and

divalent cations are required for catalytic activity; the highest

rates were found with Ca2+ NADPH is required under

aerobic but not under anaerobic assay conditions The

enzyme is related to a widespread family of

(S)-a–hydroxy-acid oxidizing enzymes including flavocytochrome b2 and

L-lactate dehydrogenase and was shown to catalyse the formation of [2,3-13C2]lactate from [2,3-13C2]pyruvate, albeit

at a low rate of 1 nmolÆmg)1Æmin)1 Putative genes specifying type II isopentenyl diphosphate isomerases were found in the genomes of Archaea and of certain eubacteria but not in the genomes of fungi, animals and plants The analysis of the occurrence of idi-1 and idi-2 genes in conjunction with the mevalonate and nonmevalonate pathway in 283 completed and unfinished prokaryotic genomes revealed 10 different classes Type II isomerase is essential in some important human pathogens including Staphylococcus aureus and Enterococcus faecaliswhere it may represent a novel target for anti-infective therapy

Keywords: isoprenoids, mevalonate, deoxyxylulose, Idi-2, FMN

Isoprenoids are one of the largest groups of natural

products comprising more than 35 000 reported

com-pounds [1] Numerous representatives of the terpenoid

family have important physiological functions such as light

perception (retinal), light protection (carotenoids), energy

transduction (retinal, chlorophyll), signal transduction

(ret-inoic acid, steroids), membrane fluidity modulation

(ster-oids, hopanoids), predator repulsion and pollinator or mate

attraction [1]

Despite their enormous structural and functional

com-plexity, all terpenoids are assembled from two simple

precursors, isopentenyl diphosphate (IPP) and dimethylallyl

diphosphate (DMAPP) (Fig 1) The biosynthesis of these universal terpene precursors via the mevalonate pathway has been studied in considerable detail in yeast and animals These classical studies established the formation of IPP from three acetate moieties via mevalonate (reviewed in [2–5]) IPP is then converted into DMAPP by an isopentenyl diphosphate isomerase which is essential in all organisms using the mevalonate pathway (reviewed in [6,7])

The elucidation of the mevalonate pathway culminated in the development of the statin type drugs which inhibit 3-hydroxy-3-methylglutaryl-CoA reductase and reduce car-diovascular morbidity and mortality by reduction of blood cholesterol levels and probably also by down-regulation

of inflammatory processes [8,9] Certain statins such as Lipitor and Zocor are record holders with regard to current drug sales

A second isoprenoid biosynthesis pathway starting with 1-deoxy-D-xylulose 5-phosphate has been discovered in the last decade (reviewed in [10–14]) The linear carbohydrate precursor is transformed into a branched polyol derivative, 2C-methyl-D-erythritol 4-phosphate [15] which is further converted into 1-hydroxy-2-methyl-2-(E)-butenyl 4-diphos-phate by the consecutive action of enzymes specified by the ispCDEFG genes (Fig 1) [16–21] The reduction of

Correspondence to F Rohdich and W Eisenreich, Lehrstuhl fu¨r

Organische Chemie und Biochemie, Technische Universita¨t,

Lichtenbergstr 4, D-85747 Garching, Germany.

Fax: + 49 89 289 13363, Tel.: + 49 89 289 13364 and

+49 89 289 13336, E-mail: felix.rohdich@ch.tum.de and

wolfgang.eisenreich@ch.tum.de

Abbreviations: DMAPP, dimethylallyl diphosphate; IPP, isopentenyl

diphosphate.

(Received 26 March 2004, revised 27 April 2004,

accepted 30 April 2004)

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1-hydroxy-2-methyl-2-(E)-butenyl 4-diphosphate catalysed

by IspH protein affords both IPP and DMAPP [22–26]

Accordingly, the participation of an IPP isomerase is in

principle not required in this pathway Nevertheless,

numerous prokaryotes endowed with these genes display

IPP isomerases which may act as salvage enzymes in order

to adjust the ratio of DMAPP and IPP to the specific

requirements of the downstream terpenoid metabolism [27]

A recently discovered IPP isomerase (designated type II)

from Streptomyces sp CL190 [28] is devoid of sequence

similarity to the previously known IPP isomerases of yeast

and animal origin which are now designated type I

Whereas type I isomerases only require divalent cations

for catalytic activity, the type II isomerase of Streptomyces

sp CL190 has been reported to require FMN and NADPH

as well as divalent metals [28] The structure of a type II IPP

isomerase from Bacillus subtilis has been elucidated by

X-ray crystallography [29] This paper reports on the

biochemical properties of the recombinant enzyme from

B subtilis Phylogenetic patterns of IPP isomerases in the

archaeal and eubacterial kingdoms with respect to the two

IPP/DMAPP biosynthesis pathways were analysed by

bioinformatic methods

Experimental procedures

Materials

IPP, DMAPP and [3,4,5-13C3]DMAPP were prepared

by published procedures [30,31] [U-13C3]acetone and

[2,3-13C]pyruvate were obtained from Isotec (Miamisburg,

OH, USA) Restriction enzymes were purchased from New England Biolabs (Frankfurt, Germany) Oligonucleotides were custom synthesized by MWG Biotech (Ebersberg, Germany) NADPH and NADH were purchased from Biomol (Hamburg, Germany) FMN was obtained from Sigma (Steinheim, Germany)

Cloning and expression of theidi-2 gene from B subtilis

A DNA segment extending from bp position 184 997–

186 043 of the B subtilis chromosome was amplified by PCR using chromosomal B subtilis DNA as template and the oligonucleotides 5¢-TTGGTGGGATCCGTGACTCG AGCAGAACGAAAAAGAC-3¢ and 5¢-GGCTTTGTCG ACTTATCGCACACTATAGCTTGATG-3¢ as primers (restriction sites are underlined and start- and stop-codons are in bold type) The amplificate was purified, treated with the restriction enzymes BamHI and SalI, and ligated into the His-tag-encoding expression vector pQE30 (Qiagen, Hilden, Germany) which had been treated with the same enzymes The resulting plasmid pQEidi2 was electrotrans-formed into Escherichia coli strains XL1-Blue (Stratagene [32]) and M15 (pREP4) [33] affording the recombinant strains XL1-pQEidi2 and M15-pQEidi2

Preparation of the recombinant Idi-2 protein The recombinant E coli strain M15-pQEidi2 was grown in Luria-Bertani broth containing ampicillin (180 mgÆL)1) and kanamycin (50 mgÆL)1) Cultures were incubated at 37C with shaking At an optical density of 0.7 (600 nm), isopropyl thio-b-D-galactoside was added to a final concen-tration of 2 mM, and the culture was incubated for 5 h The cells were harvested by centrifugation, washed with 0.9% (w/v) sodium chloride, and stored at)20 C under anaer-obic conditions

The following steps were carried out under anaerobic conditions Frozen cell mass (4 g) was thawed in 38 mL of

100 mM Tris hydrochloride, pH 8.0, containing 0.5M sodium chloride and 20 mMimidazole hydrochloride The suspension was passed through a French press and was then centrifuged To the supernatant (60 mL), 40 mL of water were added, and the mixture was applied to a column

of Ni-chelating Sepharose FF (column volume, 11 mL; Amersham Pharmacia Biotech) which had been equili-brated with 100 mMTris hydrochloride, pH 8.0, containing 0.5M sodium chloride and 20 mM imidazole (flow rate,

2 mLÆmin)1) The column was washed with 90 mL of

100 mM Tris hydrochloride, pH 8.0, containing 0.5M sodium chloride and 20 mM imidazole and was then developed with a gradient of 20–500 mM imidazole in

150 mL of 100 mMTris hydrochloride, pH 8.0, containing 0.5Msodium chloride Fractions were combined (retention volume of Idi-2 protein, 20 mL), dialyzed overnight against

100 mMTris hydrochloride, pH 8.0 and stored at)80 C Assay of IPP isomerase activity

Unless otherwise specified, assay mixtures contained

100 mMTris hydrochloride, pH 8.0, 10 mMMgCl2, 10 lM FMN, 2 mMsodium acetate, 10.8 mMDMAPP or IPP, and protein The mixtures were incubated at 37C under

Fig 1 Biosynthesis of IPP and DMAPP.

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anaerobic conditions The reaction was terminated by the

addition of EDTA to a final concentration of 26 mM After

the addition of D2O to a final concentration of 10% (v/v),

the samples were analysed by NMR spectroscopy

Assay of lactate dehydrogenase activity

Assay mixtures containing 100 mM Tris hydrochloride,

pH 8.0, 17 mM NADH, 1.6 mM [2,3-13C2]pyruvate, 10%

(v/v) D2O and 2.0 mg of Idi-2 protein (but without added

FMN) in a total volume of 0.7 mL were incubated at 37C,

and13C NMR spectra were recorded at intervals

Sequence determination

DNA was sequenced by the automated dideoxynucleotide

method using a 377 Prism sequencer from Perkin Elmer,

Norwalk, USA [34] N-terminal peptide sequences were

obtained by Pulsed-Liquid Mode using a PE Biosystems

Model 492 (Perkin Elmer, Weiterstadt, Germany)

NMR spectroscopy

1H and13C NMR spectra were recorded with a DRX 500

AVANCE spectrometer from Bruker Instruments,

Karls-ruhe, Germany

Analytical ultracentrifugation

Hydrodynamic studies were performed with an analytical

ultracentrifuge Optima XL-I (Beckman Instruments, Palo

Alto, CA) equipped with ultraviolet and interference

optics Experiments were performed with double sector

cells equipped with aluminum centerpieces and sapphire

windows Partial specific volumes and buffer densities

were estimated according to published procedures [35]

Samples contained 100 mM Tris hydrochloride, pH 8.0

Mass spectrometry

Mass spectra were recorded with a Biflex III MALDI-TOF

mass spectrometer from Bruker Instruments, Karlsruhe,

Germany Samples contained 25 mM Tris hydrochloride,

pH 8.0, 33% (v/v) CH3CN, saturated

a-cyanohydroxycin-namic acid, 0.1% (v/v) trifluoracetic acid and 0.7 mg of IPP

isomerase per mL

Bioinformatics

Similarity searches in the GenBank database of completed

and unfinished prokaryotic genomes (among them not yet

specifically assigned genomes) (http://www.ncbi.nlm.nih

gov) were performed with the programs BLASTP and

TBLASTNusing the gappedBLASTandPSI-BLASTalgorithms

[36] Nucleic acid sequences of unfinished genomes were

downloaded from the GenBank database, and open reading

frames were identified and translated into amino acid

sequences with the programPCGENE(IntelliGenetics,

Uni-versity of Geneva, Switzerland) Alignments were

construc-ted using the programPILEUP(GCG, Madison, Wisconsin)

Phylogenetic analyses of the aligned amino acid sequences

were performed using the Phylogeny Interference Package

PHYLIP3.57c [37] andPHYLO_WIN [38] Phylogenetic trees were constructed by the Neighbor-joining method Dayh-off’s PAM 001 matrix was used to calculate the distances between pairs of protein sequences [39] A bootstrapping analysis using 1000 iterations was performed [40] Only groups with bootstrap probablity values >50% were retained

Results

Cloning and expression of theidi-2 gene from B subtilis

An open reading frame (Acc no P50740) extending from

bp position 184 997–186 043 on the B subtilis chromosome with similarity to the idi-2 gene of Streptomyces sp CL190 [28] (37% identical amino acid residues; Fig 2A) was amplified by PCR and was cloned into the plasmid pQE30 affording the recombinant plasmid pQEidi2 (see Experi-mental procedures) An E coli strain carrying this plasmid produced copious amounts of a 39 kDa protein as judged

by SDS electrophoresis (Fig 3)

The recombinant protein was purified by affinity chro-matography on Nickel-chelating sepharose and appeared homogeneous as judged by SDS/PAGE (Fig 3) Partial N-terminal Edman degradation afforded the amino acid sequence MRGSHHHHHHGSVTRAE in agreement with the sequence of the recombinant gene MALDI-TOF mass spectrometry showed a relative mass of 38 463 Da in good agreement with the calculated mass of 38 455 Da (data not shown)

Hydrodynamic studies on Idi-2 protein ofB subtilis X-ray structure analysis of the B subtilis enzyme in the presence of FMN indicated a D4symmetric homooctamer structure with a relative mass of 309 kDa [29] In order to check for the potential influence of substrates and cofactors

on the quaternary structure of the enzyme, we performed boundary sedimentation experiments under different experi-mental conditions In the absence of substrates and cofactors, the enzyme sedimented as a single, symmetrical boundary with an apparent sedimentation coefficient of 10.0 S which is well in line with the published octamer structure [29] In the aerobic assay mixture, however, the enzyme sediments with an apparent rate of 4.0 S which indicates dissociation under substrate turnover conditions Catalytic properties of Idi-2 protein fromB subtilis The reaction catalysed by the recombinant enzyme could be monitored conveniently by NMR spectroscopy (Table 1) The1H NMR and13C NMR signals of IPP and DMAPP have been assigned previously on the basis of1H13C and

13C13C correlation spectroscopy with13C-labelled samples [22] The 1H NMR assignments of DMAPP shown in Table 1 were confirmed by two-dimensional NOESY experiments indicating strong NOE interactions between the methyl signal at 1.79 p.p.m (E-methyl group) and the signal at 5.47 p.p.m (methine group) It should be noted that some confusion with respect to these assignments reigns

in the literature Whereas the correct1H NMR assignments are given in the text of [41], reversed assignments of the

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methyl signals are given in footnote 26 of that paper and in [28] When the enzyme was incubated with IPP as substrate under aerobic conditions in the presence of NADPH and

Fig 2 Amino acid residues essential for

functionality of Idi-2 protein (A) Amino acid

sequence comparison of Idi-2 proteins.

Sequences included in this analysis were

B subtilis Idi-2 protein and Idi-2 proteins

from major human pathogens Residues

absolutely conserved in all Idi-2 amino acid

sequences available in the GenBank database

are labelled by open triangles Residues

involved in FMN binding (as found in the

crystallographic structure of the B subtilis

protein, see below) are shown by filled

triangles (B) Stereo representation of the

FMN-binding site of B subtilis Idi-2 protein.

The disordered regions between Met256 and

Phe263 and Tyr211 and Arg226, respectively,

are indicated by dotted lines The latter region

is expected to cover FMN Conserved residues

are shown in blue.

Fig 3 SDS/PAGE (A) Molecular mass markers; (B) cell extract of

recombinant E coli M15-pQEidi2 hyperexpressing the idi-2 gene from

B subtilis; C, recombinant Idi-2 protein of B subtilis after nickel

chelating affinity chromatography.

Table 1 NMR data of isopentenyl diphosphate and dimethylallyl diphosphate.

Position

Chemical shifts (p.p.m.)

Coupling constants (Hz)

1 H a 13 C a J HH J PH J CCb

Isopentenyl diphosphate

Dimethylallyl diphosphate

a

Referenced to external trimethylsilylpropane sulfonate;bobserved with [3,4,5-13C 3 ]DMAPP and [2,3-13C 3 ]IPP.

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FMN, we observed the appearance of the signals of both

methyl groups and of the methine group of the

enzymat-ically formed DMAPP (Fig 4A) Concomitantly, the

signals of IPP were progressively diminished Using acetate

as an internal standard, the signal integrals afforded the

concentrations of IPP and DMAPP as a function of time

(Fig 5)

Figure 4B illustrates the reverse reaction, i.e the

conver-sion of DMAPP into IPP The1H NMR spectrum observed

at equilibrium was virtually identical with that obtained in

the experiment mentioned above (cf Figure 4A) Under the

experimental conditions described (10.8 mM IPP or DMAPP, respectively, and 0.2 mg of enzyme per mL), the conversion of IPP into DMAPP and vice versa approached

a state of equilibrium after a reaction period of about 4 h (Fig 5)

The rates based on1H NMR analysis for the conversion

of IPP into DMAPP and vice versa with Mg2+as cofactor were 0.63 ± 0.042 and 0.23 ± 0.007 lmolÆmg)1Æmin)1, respectively (Table 2) These values agree with the activities

of the E coli Idi-1 protein reported earlier [27] Their ratio is similar to the equilibrium constant for the reversible reaction reported earlier [41]

The isomerization reaction could also be monitored by

13C NMR spectroscopy using [3,4,5-13C]DMAPP as

Fig 4.1H-NMR assay of type II IPP isomerase from B subtilis.

A, part of the 1 H NMR spectrum of the reaction mixture (lower lane)

obtained from IPP (1H NMR signals, see upper lane) by the catalytic

action of Idi-2 protein under aerobic conditions B, part of the 1 H

NMR spectrum of the reaction mixture (lower lane) obtained from

DMAPP (1H NMR signals, upper lane) by the catalytic action of Idi-2

protein under aerobic conditions Assay mixtures contained 100 m M

Tris hydrochloride, pH 8.0, 10 m M MgCl 2 , 1 m M dithiothreitol,

2.5 m M NADPH, 10 l M FMN, 2 m M sodium acetate, and 10.8 m M

IPP and 10.8 m M DMAPP, respectively; *, internal standard (acetate).

Fig 5 Catalytic rates of the reversible conversion of IPP into DMAPP catalyzed by Idi-2 protein from B subtilis under aerobic conditions Numerical simulations were performed using the DYNAFIT software [58] Assay mixtures contained 100 m M Tris hydrochloride, pH 8.0,

10 m M MgCl 2 , 1 m M dithiothreitol, 2.5 m M NADPH, 10 l M FMN,

2 m M or 3 m M sodium acetate, and 10.8 m M IPP or DMAPP.

j, formation of DMAPP from IPP; s, formation of IPP from DMAPP.

Table 2 Catalytic rates of Idi-2 protein under different conditions Reaction mixtures contained MgCl 2 and were prepared as described under Experimental procedures.

Procedure/condition

Specific activity (lmolÆmin)1Æmg)1) Conversion of IPP into DMAPP

Conversion of DMAPP into IPP

Conversion of [3,4,5-13C 3 ]DMAPP into [3,4,5-13C 3 ]IPP

Conversion of [2,3- 13 C 2 ]pyruvate into [2,3- 13 C 2 ]lactate

a

Reaction mixtures contained NADPH and dithiothreitol;

b activities were calculated from rate constants (Fig 5); c reaction mixtures contained NADH.

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substrate The decrease of the13C-coupled signals of the

Z- and E-methyl groups resonating at 17.0 and

24.7 p.p.m., respectively, as well as that of the quaternary

carbon atom resonating at 139.7 p.p.m was accompanied

by the appearance of three new 13C-coupled signals at

143.4, 111.3 and 21.4 p.p.m assigned as the carbon atoms

3, 4, and 5 of IPP, respectively (cf Table 1 and Fig 6)

Within the limits of experimental accuracy the catalytic

rates determined with this assay were the same as those

described above

Whereas NADPH or NADH was required for catalytic

activity under aerobic conditions, the reaction could

proceed without NADPH under anaerobic conditions using

enzyme which had been purified under anaerobic

condi-tions FMN, however, was required under aerobic as well as

under anaerobic conditions The reaction rates were similar

under aerobic and anaerobic conditions (Table 2)

Photo-metric analysis gave no evidence for reduction of FMN in

aerobic or anaerobic assay mixtures (data not shown) The

recombinant enzyme has an absolute requirement for a

divalent metal ion for catalytic activity; the highest rates

were found with Ca2+(Table 3) A different order for the

catalytic activation by such ions has been reported

previ-ously for Streptomyces type II isomerase [28]

Orthologs of Idi-2 protein

An exhaustiveBLASTsearch of 283 completed and

unfin-ished prokaryotic genomes in the GenBank database

recovered 91 genes specifying proteins with close similarity

to Idi-2 protein of B subtilis This set characterized by an

expect value < 2e-27 is proposed to comprise all type II

isomerases in the set of 283 prokaryotic genomes analysed

for reasons that will become obvious in the following

paragraphs Additionally to this set, 10 orthologous

sequence entries of microrganism were found in GenBank

whose genomes were not available in their entirety All 102

putative Idi-2 orthologs of microrganisms studied here share a significant degree of sequence similarity Their lengths range from 330 to 360 amino acid residues (Fig 2A) Twenty-one amino acid residues are absolutely conserved (marked by triangles in Fig 2) Notably, all amino acid residues shown by the X-ray structure to be involved in the binding of the FMN cofactor [29] are absolutely conserved (Fig 2A,B) These residues (marked

by filled triangles in Fig 2A) are located in four different segments of the peptide chain Specifically, the residues Gly66, Gly258, Gly259 contact the phosphate moiety of FMN via hydrogen bonds (Fig 2B) The isoalloxazine ring

is coordinated by residues Thr64 (N5), Ser93 (O4), Asn122 (N3) and Lys184 (N1, O2) The amino acid residues His147, Asn149, Gln152 and Glu153 (marked by open triangles in Fig 2) in the direct neighborhood of the FMN binding site are also absolutely conserved (Fig 2B)

Type II isomerases are restricted to the archaeal and eubacterial kingdoms With the exception of Halobacterium

sp NRC-1, Mycobacterium marinum and Photorhabdus luminescensfeaturing both a putative idi-1 and a putative idi-2gene, the distribution of type I and type II isomerases in the prokaryotic kingdom appears to be mutually exclusive Genes specifying type II isomerases were found in 19 of 20 (95%) archaebacterial species Nanoarchaeum equitans is devoid of IPP biosynthesis as well as of idi genes In the group of 263 eubacterial genomes studied, 35 (13%) carry

an idi-1 gene, 72 (27%) carry an idi-2 gene, 2 carry both an idi-1and idi-2 gene, and 154 (59%) appear to be devoid of IPP isomerases

Phylogenetic analyses of 102 type II isomerases were performed as described under Experimental procedures The final consensus phylogenetic tree (majority rule) shows the major phylogenetic grouping of 76 type II isomerases in the archaeal and eubacterial kingdoms as illustrated in Fig 7 Bacillales and Lactobacillales form a cluster which is separated from other lineages with statistical relevance (bootstrap value: 100%) Some actinobacteria (Streptomy-cessp CL190, Kitasatospora griseola and Actinoplanes sp A40644) group also within this cluster The separation of the archaeabacterial from the eubacterial kingdom was not found to be statistically relevant (bootstrap values < 50%)

In the eubacterial kingdom, Cyanobacteria with the exception of Crocosphaera watsonii and Synechocystis sp PCC6803 (which group together with the sulfur bacterium Chloroflexus aurantiacus; bootstrap value of 67), some actinobacteria (Mycobacterium avium, Mycobacterium

Fig 6.13C NMR signals of DMAPP and IPP A [3,4,5-13C 3 ]DMAPP;

B, mixture of [3,4,5- 13 C 3 ]DMAPP and [3,4,5- 13 C 3 ]IPP obtained from

[3,4,5- 13 C 3 ]DMAPP by the catalytic action of Idi-2 protein of B subtilis

under aerobic conditions Assay mixtures contained 100 m M Tris

hydrochloride, pH 8.0, 10 m M MgCl 2 , 1 m M dithiothreitol, 2.5 m M

NADPH, 10 l M FMN, and 5.2 m M [3,4,5- 13 C 3 ]DMAPP.

Table 3 Activation of Idi-2 protein by divalent cations Reaction mix-tures were prepared as described under Experimental Procedures Metal ions were added to a final concentration of 10 m M

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marinumand Mycobacterium smegmatis) together with the

Deinococcus-Thermus group, and some proteobacteria

form separate lineages (bootstrap values of 95, 100 and

79%, respectively) The remaining orders among the

eubacterial kingdom did not reveal any statistically

suppor-ted relationship In the archaeal kingdom, the

Methanos-arcinales together with the Archaeoglobales (bootstrap

value: 69%) and the Thermococcales together with the

Methanobacteriales (bootstrap value: 77%) are grouped

into clusters

The presently available data suggest that

Cyano-bacteria, Bacillales and Lactobacillales with the exception

of B halodurans, Geobacillus stearothermophilus and Pasteuria nishizawae use exclusively type II isomerase (Table S1) Type II isomerases are also found in the Actinobacteria group (M avium, M marinum and

M smegmatis), and in the a-subgroup of proteobacteria (Mesorhizobium loti and Rickettsia spp.) Only very few representatives from other bacteria groups possess idi-2 genes (Dichelobacter nodosus, Legionella pneumophila and

P luminescens, all three c-proteobacteria; and the Spiro-chete Borrelia burgdorferi) Interestingly, the idi-2 gene of

P luminescens, whose genome specifies the enzymes of the deoxyxylulose phosphate pathway together with both the

Fig 7 Consensus cladogram of Idi-2 proteins from various microorganisms The simplified tree (majority rule) was deduced by Neighbor-joining analysis based on the alignment of the amino acid sequences of 76 Idi-2 proteins Gaps were removed from the alignment, and the total number of positions taking into account was 320 The numbers at the nodes are the statistical confidence estimates computed by the bootstrap procedure Only groups with bootstrap probablity values

>50% were retained The bar represents 0.137 PAM distance.

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idi-1 as well as the idi-2 genes, is interrupted by a

counterclockwise located transposase gene effectively

knocking it out

Type I isomerases are found preferably in the

Actino-bacteria group (Corynebacterium sp., Mycobacterium sp

and Streptomyces sp.), but also in the Bacteroidetes group

(Cytophaga hutchinsonii), and some in the a-subgroup

(Rhodobacter sphaeroides and Silicibacter pomeroyi) and in

the c-subgroup (Coxiella burnetii, Erwinia carotovora,

Escherichia sp., Klebsiella pneumoniae, P luminescens,

Salmonellaand Shigella sp.) of the proteobacteria group

Phylogenetic pattern of Idi proteins

It is now in order to analyse the distribution of the two

isomerase types in relation to the two isoprenoid

biosyn-thetic pathways, i.e the classical mevalonate pathway and

the more recently discovered nonmevalonate pathway via

deoxyxylulose phosphate (Fig 1) Of the 16 possible

combinations, 10 were actually found in the group of 283

completed and unfinished sequenced prokaryotic genomes

(Fig 8) Among 204 prokaryotic genomes studied using

exclusively the deoxyxylulose phosphate pathway, 147 carry

no idi gene, 35 carry idi-1 genes, 20 carry idi-2 genes and 2

carry both an idi-1 and idi-2 gene The majority of

prokaryotes using exclusively the mevalonate pathway

(total number, 64) possess type II isomerases (total number,

62 including several important human pathogens) (Fig 8)

As an exception, the genomes of Listeria monocytogenes and

Listeria innocua carry complete sets of genes for both

pathways in conjunction with type II isomerases The

combination of mevalonate pathway genes together with

type I isomerase is found exclusively in the genome of the

eubacterium Coxiella burnetii The genomes of obligate

intracellular parasitic Rickettsia spp (total number, 4) are

devoid of an isoprenoid pathway, but carry idi-2 genes

With the exception of Mycoplasma gallisepticum R,

Myco-plasma penetransand Spiroplasma kunkelii, the genomes of

the members of the mollicutes group (total number, 7 out of

10) are devoid of genes for the biosynthesis of IPP and DMAPP The same is true for the genome of the archaeon

N equitans.No putative orthologs of the type II isomerase were found in any eukaryotic species including plants, fungi and animals On the other hand, all completely sequenced eukaryotic genomes comprise putative orthologs of the type

I isomerase Highest degrees of similarity were found to Idi-1 proteins of the c-proteobacteria A vinelandii and

P luminescens and to Idi-1 protein of the Bacteroid

C hutchinsonii (expect values 3e-22, 5e-19 and 8e-20, respectively)

Paralogs of Idi-2 proteins Database searches conducted with the BLASTP program retrieved a considerable number of proteins with substantially lower similarity (Expect value >1e-10) to the

B subtilis Idi-2 protein which was used as search motif Notably, type II isomerase shows weak, but significant similarity with a family of FMN dependent (S)-a-hydroxy-acid dehydrogenases (pfam database accession no PF01070) including flavocytochrome b2 from yeast [EC 1.1.2.3 (FCb2)] [42], long chain hydroxyacid oxidase from mammals [43], glycolate oxidase from spinach [EC 1.1.3.15 (GOX)] [44], L-lactate dehydrogenases from bacteria (EC 1.1.1.27) [45] (S)-mandelate dehydrogenase from P putida (MD) [46] and inosine 5¢-monophosphate dehydrogenases (EC 1.1.1.205) [47] over the entire length of their respective sequences (Fig 9A) (expect values 0.002, 1.1, 0.083, 2e-06, 0.47 and 0.001, respectively)

The members of this enzyme family display a TIM-barrel fold Superposition of Idi-2 with the known structures of GOX [48], FCb2 [49] and MD [50] demonstrates that Idi-2 protein shares a very similar three-dimensional fold in the C-terminal half of the protein but with significant deviations

in the N-terminal half (Fig 9B) This finding is reflected by rms deviations of 1.3–1.5 A˚ for only about 200 matching residues out of 349 when comparing Idi-2 to GOX and FCb2 The rms deviation is only 1.0 A˚ for about 300 matching residues when comparing GOX with FCb2 or MD

In addition, the fraction of identical residues for the significantly lower number of matching residues is below 30% in the first case compared to above 40% in the second (Table 4)

In addition to a conserved sequence motif SNHG[AG]RQ [PROSITE database (http://www.expasy org/prosite)], GOX, FCb2 and MD share a number of conserved active site residues (Tyr24, Tyr129, Arg124, His254 and Arg257, corresponding to the amino acid sequence of GOX) (Fig 9A) Both the sequence motif and these active site residues are missing in Idi-2 On the other hand, Idi-2 proteins encompass a set of conserved amino acid residues (His147, Asn149, Gln152 and Glu153, corres-ponding to the amino acid sequence of the B subtilis protein) in the direct vicinity of the FMN binding site which

is not present in the other members of the family

Thus, under structural aspects, Idi-2 appears as a fairly distant relative of the FMN-dependent a-hydroxyacid-oxidizing enzymes However, the sequence similarity in conjunction with the TIM barrel fold and the conserved FMN binding site leave no doubt about the evolutionary relatedness of Idi-2 with the dehydrogenase superfamily

Fig 8 Distribution of isoprenoid biosynthesis pathways and IPP

isomerases in 283 completed and unfinished prokaryotic genomes MEV,

mevalonate pathway genes; DXP, deoxyxylulose pathway genes.

Trang 9

L-Lactate dehydrogenase activity of Idi-2 protein

fromB subtilis

Partial sequence similarity of the Idi-2 gene and the

paralogous lldD gene specifying -lactate dehydrogenase

together with similarities in the TIM-barrel fold and FMN binding site of the two respective proteins promp-ted a search for the presence of a residual redox activity

in type II isomerase In order to obtain maximum sensitivity in combination with maximum selectivity, we used [2,3-13C2]pyruvate as substrate Using NADH as cofactor, we observed the formation of [2,3-13C2]lactate

at a rate of 1 nmolÆmg)1Æmin)1 by 13C NMR spectro-scopy (Table 2) The addition of FMN did not increase the catalytic activity A protein sample prepared from an

E coli strain harbouring the expression vector without insert and eluted from the Nickel affinity column with the same volume as compared to the recombinant Idi-2 protein did not show any lactate dehydrogenase activity This result clearly indicated that the lactate dehydroge-nase activity displayed by Idi-2 protein did not result from E coli wildtype background activities caused by protein impurities

Fig 9 Structural relationshipof Idi-2 with a-hydroxyacid dehydrogenases A, structure based sequence alignment of glycolate oxidase (gox), flavocytochrome b 2 (cy), S-mandelate dehydrogenase (md; Acc no P20932) and Idi-2 (idi) The sequence of L -lactate dehydro-genase was added based on a sequence alignment to glycolate oxidase Secondary structures and sequence numbers refer to gox (top lines) and idi (bottom lines), respectively The eight ba modules of the TIM-barrel domain are colored yellow and named S1 to S8 and H1 to H8, respectively Active site residues

of the a-hydroxyacid dehydrogenase family are shown in red, the characteristic

NHG[GA]RQL-motif is boxed (note that it is not conserved in IDI proteins) FMN-binding residues are coloured blue and residues con-served in IDI proteins located close to FMN in green The similarity between the a-hydroxy-acid dehydrogenase and Idi-2 proteins is most pronounced in the C-terminal half which har-bours the standard phosphate binding site (SPB) B, stereo-view of the superposition of Idi-2 (N-terminal extension in yellow, TIM-barrel in grey, C-terminal extension in green) and glycolate dehydrogenase (N-terminal extension in dark blue, TIM-barrel in light blue, C-terminal extension in purple) Secon-dary structure elements of the TIM-barrel superimpose very well, especially for modules b7/a7 and b8/a8 which harbor the standard phosphate binding site (SPB) Deviations are found for the N- and C-terminal extensions FMN is depicted in orange (Idi-2) and green (GOX) In addition, the GOX structure dis-plays a bound active site inhibitor [4-carboxy-5-(1-pentenyl)hexylsulfanyl-1,2,3-triazole (TACA)] in pink His254 and Arg257 of the signature motif NHG[AG]RQL of GOX are depicted as ball and stick.

Table 4 Structure superimposion of Idi-2 protein with

(S)-a hydroxy-acid dehydrogenases Rmsd in A˚, # of matching residues, % identity for

matching residues The structures have been superimposed with

TOP3D using the PDB entries 1al8 (glycolate dehydrogenase), 1ltd

(flavocytochrome b 2 ), 1p5b (S-mandelate dehydrogenase) and 1pno

(Idi-2).

Idi-2 1.5, 201, 27.9% 1.3, 203, 27.1% 1.5, 202, 24.3

Trang 10

The type I IPP isomerases which have been known for a

long time require only divalent metal cations for activity

[6,7] On the other hand, the type II isomerases of

Streptomyces sp CL190 and Staphylococcus aureus have

been reported to require FMN and NADPH in addition to

divalent metal cations for activity under aerobic as well as

under anaerobic conditions [28] Under aerobic conditions,

the type II IPP isomerase of B subtilis requires NADPH

as well as FMN for activity in close similarity with the

Streptomyces enzyme However, when the enzyme is

purified and assayed under anaerobic conditions, NADPH

is not required The catalytic activities observed with IPP as

substrate are similar under aerobic and anaerobic

condi-tions, in the range of 0.6 lmolÆmg)1Æmin)1 No evidence was

obtained for redox cycling of FMN The amino acid

residues involved in the FMN binding site are absolutely

conserved throughout a large number of orthologs

(Figs 2,9) This suggests an essential role for FMN despite

the low affinity of the enzyme for that cofactor and the

apparent absence of a redox process as part of the catalytic

cycle The substrate binding site of the type II enzyme

remains veiled However, a patch of absolutely conserved

amino acid residues comprising the polar amino acid

residues H147, N149, Q152 and E153 in close proximity

to the FMN binding site suggests that the substrates could

be bound in close proximity to the isoalloxazine moiety In

the absence of direct evidence for the involvement of a redox

process it is tempting to postulate that the cofactor might act

as a dipole stabilizing a cationic intermediate or transition

state of the reaction A similar role has been postulated for

tryptophan 121 in the case of E coli Idi-1 [51]

During the preparation of this manuscript three groups

reported the catalytic properties of recombinant type II IPP

isomerases from B subtilis [52] and the two Archaea

Methanothermobacter thermoautothrophicus[53] and

Sulf-olobus shibatae [54] These enzymes were found to be

dependent on NADPH and Mg2+as cofactors Anaerobic

conditions were not tested in these studies In contrast to

these findings [52–54], the catalytic activity of the

recom-binant enzyme studied here was maximal with Ca2+ In

addition, we show for the first time, that, under anaerobic

conditions, the enzyme did not require NADPH However,

unchanging of NADPH-dependency was claimed earlier for

the respective enzymes of Streptomyces sp CL190 and

Staphylococcus aureusunder anaerobic conditions [28]

Idi-2 protein is clearly a member of a superfamily of

(S)-a-hydroxyacid dehydrogenases, and the coenzyme pattern

of Idi-2 protein, where the roles of FMN and of NADPH

required under aerobic conditions are at present not

understood, may ultimately find an explanation by the

evolutionary relationship with oxidoreductases

Numerically, bacteria using the deoxyxylulose phosphate

pathway without any isomerase (147 out of 283) and

bacteria using the mevalonate pathway in conjunction with

type II isomerase (62 out of 283) constitute the largest sets

within the prokaroytic kingdom (Fig 8) The combination

of the deoxyxylulose phosphate pathway with type I

isomerase (35 out of 263) and with type II isomerase (20

out of 263) occur with lower frequency This situation could

be the result of a differential gene loss, in which some

microorganisms have either retained Idi-1 or Idi-2, or of a lateral gene transfer similar to that reported for 3-hydroxy-3-methylglutaryl coenzyme A reductase [55,56] It is inter-esting in this context that both types of isomerases are found

in the Actinobacteria group The anomalous positions for some eubacterial species (e.g Cyanobacteria and Actino-bacteria) observed here (cf Figure 7) may be explained by a loss of evolutionary constraints due to nonessential func-tions of Idi-2 proteins in bacteria using the deoxyxylulose phosphate pathway

With regard to the complex distribution of the two different terpenoid pathways and of the two different isomerase types in the eubacterial kingdom, it is relevant to emphasize that certain highly pathogenic Gram-positive cocci including Enterococcus and Staphylococcus species use type II isomerases in conjunction with the mevalonate pathway which has an absolute requirement for isomeriza-tion of IPP in order to generate DMAPP Hence, the type II isomerase is an essential enzyme in this group of human pathogens

Enterococciand Staphylococci have a dramatic history of resistance development against virtually all currently avail-able antibiotics Most notably, many strains are multidrug resistant, and the rapidly spreading resistance against vancomycin and methicillin constitutes a life-threatening problem in affected patients [57] Clearly, there is an urgent requirement for novel therapeutic strategies directed at these microorganisms As the human type I IPP isomerase and the type II isomerase of the microorganisms mentioned have no detectable similarity, it should be possible to develop inhibitors for the bacterial enzyme which have little

or no significant cross-inhibitory activity for the human enzyme

Acknowledgements

We thank the Deutsche Forschungsgemeinschaft, the Fonds der Chemischen Industrie, and the Hans Fischer Gesellschaft for support Financial support by Novartis International AG, Basel (to D A.) is gratefully acknowledged We thank Fritz Wendling and Katrin Ga¨rtner for skillful assistance, and Angelika Werner for expert help with the preparation of the manuscript.

References

1 Sacchettini, J.C & Poulter, C.D (1997) Creating isoprenoid diversity Science 277, 1788–1789.

2 Bach, T.J (1995) Some new aspects of isoprenoid biosynthesis in plants –a review Lipids 30, 191–202.

3 Bloch, K (1992) Sterol molecule: structure, biosynthesis, and function Steroids 57, 378–382.

4 Bochar, D.A., Friesen, J.A., Stauffacher, C.V & Rodwell, V.W (1999) Biosynthesis of mevalonic acid from acteyl-CoA.

In Comprehensive Natural Product Chemistry (Cane, D., ed.), Vol 2, pp 15–44 Pergamon, Oxford.

5 Qureshi, N & Porter, J.W (1981) Conversion of acetyl-coenzyme

A to isopentyl pyrophosphate In Biosynthesis of Isoprenoid Compounds (Porter, J.W & Spurgeon, S.L., eds), Vol 1, pp 47–

94 John Wiley, New York.

6 Poulter, C.D & Rilling, H (1981) Conversion of farnesyl pyro-phosphate to squalene In Biosynthesis of Isoprenoid Compounds (Porter, J.W & Spurgeon, S.L., eds), Vol 1, pp 162–209 John Wiley, New York.

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