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Tiêu đề Production and characterization of a secreted, C-terminally processed tyrosinase from the filamentous fungus Trichoderma reesei ppt
Tác giả Emilia Selinheimo, Markku Saloheimo, Elina Ahola, Ann Westerholm-Parvinen, Nisse Kalkkinen, Johanna Buchert, Kristiina Kruus
Trường học VTT Technical Research Centre of Finland; University of Helsinki
Chuyên ngành Biochemistry
Thể loại Research article
Năm xuất bản 2006
Thành phố Espoo
Định dạng
Số trang 14
Dung lượng 1,52 MB

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Nội dung

reesei tyrosinase gene tyr2, encoding a protein with a putative signal sequence.. The purified TYR2 protein had a sig-nificantly lower molecular mass 43.2 kDa than that calculated from the

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C-terminally processed tyrosinase from the filamentous fungus Trichoderma reesei

Emilia Selinheimo1, Markku Saloheimo1, Elina Ahola2, Ann Westerholm-Parvinen1, Nisse Kalkkinen2, Johanna Buchert1and Kristiina Kruus1

1 VTT Technical Research Centre of Finland, Espoo, Finland

2 Protein Chemistry Research Group and Core Facility, Institute of Biotechnology, University of Helsinki, Finland

Tyrosinase (monophenol, o-diphenol:oxygen

oxidore-ductase, EC 1.14.18.1) is a copper-containing

metallo-protein that is ubiquitously distributed in nature

Tyrosinases are found in prokaryotic as well as in

eukaryotic microorganisms, and in mammals,

inverte-brates and plants Tyrosinase is a mono-oxygenase and

a bifunctional enzyme that catalyzes the

o-hydroxyla-tion of monophenols and subsequent oxidao-hydroxyla-tion of o-diphenols to quinones [1,2] The activities are also referred to as cresolase or monophenolase and catecholase or diphenolase activities, respectively Tyrosinase thus accepts monophenols and diphenols as substrates, and the monophenolase activity is the ini-tial rate-determining reaction [2,3]

Keywords

fungal; secreted; Trichoderma reesei;

tyrosinase

Correspondence

E Selinheimo, VTT Technical Research

Centre of Finland, PO Box 1000,

Espoo FIN-02044 VTT, Finland

Fax: +358 20 722 7071

Tel: +358 20 722 7135

E-mail: Emilia.Selinheimo@vtt.fi

(Received 16 May 2006, revised 7 July

2006, accepted 27 July 2006)

doi:10.1111/j.1742-4658.2006.05429.x

A homology search of the genome database of the filamentous fungus Trichoderma reeseiidentified a new T reesei tyrosinase gene tyr2, encoding

a protein with a putative signal sequence The gene was overexpressed

in the native host under the strong cbh1 promoter, and the tyrosinase enzyme was secreted into the culture supernatant This is the first report on

a secreted fungal tyrosinase Expression of TYR2 in T reesei resulted in good yields, corresponding to approximately 0.3 and 1 gÆL)1 tyrosinase in shake flask cultures and laboratory-scale batch fermentation, respectively

T reesei TYR2 was purified with a three-step purification procedure, consisting of desalting by gel filtration, cation exchange chromatography and size exclusion chromatography The purified TYR2 protein had a sig-nificantly lower molecular mass (43.2 kDa) than that calculated from the putative amino acid sequence (61.151 kDa) According to N-terminal and C-terminal structural analyses by fragmentation, chromatography, MS and peptide sequencing, the mature protein is processed from the C-terminus

by a cleavage of a peptide fragment of about 20 kDa The T reesei TYR2 polypeptide chain was found to be glycosylated at its only potential N-gly-cosylation site, with a glycan consisting of two N-acetylglucosamines and five mannoses Also, low amounts of shorter glycan forms were detected at this site T reesei TYR2 showed the highest activity and stability within a neutral and alkaline pH range, having an optimum at pH 9 T reesei tyros-inase retained its activity well at 30C, whereas at higher temperatures the enzyme started to lose its activity relatively quickly T reesei TYR2 was active on both l-tyrosine and l-dopa, and it showed broad substrate specificity

Abbreviations

TYR2, tyrosinase 2 from Trichoderma reesei; Q-TOF, quadrupole time-of-flight.

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In mammals, tyrosinase catalyzes reactions in the

multistep biosynthesis of melanin pigments, being

responsible, for instance, for skin and hair

pigmenta-tion [4] Tyrosinases play an important role in

regula-tion of the oxidaregula-tion–reducregula-tion potential, and the

wound-healing system in plants [5,6] They are also

related to browning reactions of fruit and vegetables

[7] Tyrosinase activity has an essential role in some

plant-derived food products, e.g tea, coffee, raisins

and cocoa, where it produces distinct organoleptic

properties [8] Most commonly, tyrosinase-mediated

reactions in plants, however, are related to the

brown-ing reactions that are considered harmful [9] To date,

the information on the physiologic role of tyrosinases

in microorganisms is very limited The most extensively

investigated fungal tyrosinases, from both a structural

and a functional point of view, are from Agaricus

bisporus [10] and Neurospora crassa [1] Studies with

N crassa have shown that the enzyme is completely

absent in the vegetative stage However, under stress

conditions high levels of the enzyme can be induced

[1] This suggests that tyrosinases are not essential to

the metabolism of the fungi, but improve the survival

and competence of the fungi by producing melanins

Tyrosinases have been shown to share a similar

act-ive site with catechol oxidase and hemocyanin, a

pro-tein involved in oxygen transport in arthropods and

molluscs [11] These proteins are type 3 copper

pro-teins with a diamagnetic spin-coupled copper pair in

the active center Each of the two copper atoms is

coordinated by three conserved histidine residues [12]

Molecular oxygen is used as an electron acceptor and

it is reduced to water in tyrosinase-catalyzed reactions

On the basis of thorough chemical and spectroscopic

analyses of tyrosinases, the binuclear active site is

known to exist in three states: oxy-tyrosinase,

met-tyrosinase and deoxy-met-tyrosinase [13–15]; a catalytic

cycle, in which these states are alternated, has been

proposed [16] The met state is the resting state of the

enzyme, and in the absence of substrate about 85–90%

of the enzyme is in this state [16] Both the met and

oxy states of tyrosinases can catalyze the

diphenoloxi-dase reaction, whereas the monohydroxylase reaction

requires the oxy state Just recently, the first tyrosinase

structure from Streptomyces castaneoglobisporus [17]

became available, and will enable more detailed

analy-sis of the exact reaction mechanisms The tyrosinase

structure, wherein the active site was located at the

bottom of a large vacant space and one of the six

his-tidine ligands appeared to be highly flexible, was

deter-mined with a help of a caddie protein, ORF378, at

1.2–1.8 A˚ resolution [17] Knowledge of fungal

tyrosin-ases is still limited, and the work has been hampered

by relatively low production yields of the enzymes In this article, the cloning, production and characteriza-tion of a novel tyrosinase from the filamentous fungus

T reesei is reported

Results

Isolation of a tyrosinase gene from Trichoderma reesei

A homology search was performed against the genome sequence of T reesei (http://gsphere.lanl.gov/trire1/ trire1.home.html) This revealed two uncharacterized genes showing clear similarity with known tyrosinase sequences Analysis of the deduced protein sequence encoded by the tyr2 gene with the program signalp [24] indicated that the protein has a signal sequence, and should thus be a secreted enzyme The T reesei tyr2 gene and the corresponding cDNA were cloned

by PCR and sequenced in order to verify the sequence

at the genome website, to exclude PCR mutations and

to localize the introns The gene is interrupted by seven short introns The encoded protein consists of 571 amino acids, including a predicted signal sequence of

18 amino acids, and three potential N-glycosylation sites The closest homologs of the T reesei TYR2 pro-tein are putative tyrosinases from the fungi Gibberella zeae (46% amino acid identity), N crassa (35% iden-tity) and Magnaporthe grisea (34% ideniden-tity) All these three proteins are predicted by the signalp program to have a signal sequence; however, none of them has been characterized at the protein level The amino acid identity of TYR2 to the intracellular tyrosinase from Pycnoporus sanguineus is 34% [25], whereas the amino acid identity to other fungal tyrosinases is around 25–33% Alignment of T reesei TYR2 with the

G zeaetyrosinase places the suggested signal sequence cleavage sites of both enzymes precisely at the same location (Fig 1) When these two sequences are aligned with the intracellular tyrosinase characterized from N crassa [26], the suggested N-termini of the mature secreted enzymes coincide with the N-terminus

of the intracellular enzyme (Fig 1) The segments in the N-terminal portion around the copper ligand amino acids of the active site are well conserved between the proteins, whereas the C-terminal domains are less conserved

Overexpression of the tyrosinase gene in Trichoderma reesei

Tyrosinase production by an untransformed T reesei strain was tested, but no tyrosinase activity in culture

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supernatants or in cell lysates could be detected.

Therefore, the tyrosinase was overexpressed in T

ree-sei An expression construct in which the

protein-coding region of the genomic tyr2 is between the cbh1

promoter and terminator was made by in vivo

recombi-nation with the Gateway recombirecombi-nation system The

cbh1 promoter is a strong inducible promoter and

act-ive throughout cultivation The construct (pMS190)

was transformed into T reesei, and the transformants

were tested with a plate activity assay with tyrosine as

the indicator substrate A number of transformants

developing a stronger brown color around the streaks

than the parental strain were found (data not shown)

These uninucleate clones were isolated and tested for

tyrosinase production in shake flask cultures The best

transformant produced 40.1 nkatÆmL)1 of tyrosinase activity The first test cultures were made with 0.1 mm CuSO4 in the medium The effect of copper concentra-tion on the producconcentra-tion level was studied by using 0–6 mm CuSO4 in the medium in cultures of the best transformant The optimal copper concentration was

2 mm, but relatively good production was obtained at 1–4 mm The highest tyrosinase production obtained

in shake flask cultures was 96 nkatÆmL)1 The best tyr2-overexpression transformant was grown in a laboratory fermenter in a volume of 20 L The enzyme production increased continuously during culture, and the activity level of 300 nkatÆmL)1was reached after 6 days of cultivation Although the activity was still increasing, the fermentation had to be stopped because

Fig 1 Alignment of T reesei TYR2 (TrTYR2) amino acid sequence with the putatively secreted tyrosinase of Gibberella zeae (GzTYR) and the intracellular tyrosinase characterized from Neurospora crassa (NcTYR1) Identical amino acids are indica-ted by asterisks, conserved substitutions by colons, and similar amino acids by dots The hisitidines acting as ligands for the Cu atoms A and B are shaded The Cys–His thioester bond in the active site is indicated Signal sequence cleavage sites of TrTYR2 and GzTYR are indicated by arrows The last amino acids of processed TrTYR2 and NcTYR1 are marked by triangles Putative N-glycosylation sites are in bold.

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of foaming problems According to the specific activity

of the purified tyrosinase (300 nkatÆmg)1; Table 1),

the highest activity obtained in fermentation, 300

nkatÆmL)1, corresponds to about 1 g of the enzyme

per liter of culture supernatant

Enzyme purification

Enzyme purification was started with desalting by gel

filtration (Sephadex G25) The following cation

exchange chromatography was performed in 10 mm

Tris⁄ HCl, pH 7.3 Tyrosinase eluted at an NaCl

con-centration of 120 mm Because of the high pI of

T reesei TYR2, most of the Trichoderma cellulases

and hemicellulases could be separated from the

tyro-sinase-containing fractions The final purification step

was carried out with gel filtration (Sephacryl S-100)

The overall recovery of activity in the three-step

purifi-cation procedure was 15% (Table 1)

Biochemical characterization

IEF of the purified T reesei TYR2, and subsequent

staining with l-dopa, showed a band in the gel

corres-ponding to a pI around 9.5 The purified T reesei TYR2 appeared as a double protein band on SDS⁄ PAGE gel (Fig 2), with an apparent molecular mass of 43 kDa, which is far below the theoretical value of 61 151 Da calculated from the encoded amino acid sequence (including the signal sequence) The result suggested that T reesei TYR2 is processed, as also described for several other fungal tyrosinases [25– 28] The purified tyrosinase had an absorption maxi-mum at around 350 nm, which is an indication of a T3-type copper pair in its oxidized form with a brid-ging hydroxyl moiety, assigned as an O22–fi Cu2+ charge transfer transition [29]

For molecular characterization, the purified enzyme was first subjected to reversed-phase chromatography, where it eluted as one symmetric peak (Fig 3) Further analysis of the reversed-phase purified protein

by SDS⁄ PAGE still gave a double protein band corresponding to a molecular mass of about 43 kDa

Table 1 Purification of T reesei TYR2.

Purification step

Total activity (nkat)

Total protein (mg)

Specific activity (nkatÆmg)1)

Activity yield (%)

Purification factor

Cation exchange

chromatography

MW 4

3 2 1

203.6 116.1 92.3 50.4

37.0 28.9

20.0 6.9

Fig 2 Purification of T reesei TYR2 as analyzed by SDS ⁄ PAGE

(12% Tris ⁄ HCl gel) Gel lanes: MW, molecular mass markers; 1,

culture filtrate; 2, desalted culture filtrate, enzyme preparation after

cation exchange; 3, enzyme preparation after gel filtration.

min 50

2.5 2.0 1.5 1.0 0.5 0.0

MW 1 kDa 97.0 66.0 45.0 30.0 20.1 14.4

AU 214 nm

Fig 3 Reversed-phase chromatographic analysis of T reesei TYR2 from the last gel filtration purification step Chromatography was performed on a 1 · 20 mm TSKgel TMS-250 column using a linear gradient of acetonitrile (3–100% in 60 min) in 0.1% trifluoroacetic acid and a flow rate of 40 lLÆmin)1 The eluted protein peak was collected in two fractions, which were analyzed by 12% SDS ⁄ PAGE (insert) Gel lanes: 1 and 2, equal samples from the first half and second half of the peak, respectively; MW, molecular mass markers.

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By MALDI-TOF MS, the reversed-phase purified

pro-tein gave a single peak corresponding to an average

molecular mass of 43.3 kDa (not shown)

Further-more, in an electrospray MS analysis, using a

quadru-pole time-of-flight (Q-TOF) instrument, which has

a considerably better resolution, the same protein

preparation resulted in a set of masses among which

43 124.0 Da and 43 204.0 Da were dominant (not

shown) The results thus indicate that the protein exists

in different post-translationally modified forms To

further characterize the molecule, the reversed-phase

purified protein was subjected to N-terminal

sequen-cing by Edman degradation No amino acid

deriva-tives comparable to the amount of analyzed protein

(200 pmol) could be obtained, suggesting that the

protein has a blocked N-terminus For further

char-acterization, the protein was alkylated and

fragmen-ted by trypsin The tryptic peptides obtained were

first directly analyzed by MALDI-TOF peptide mass

fingerprinting, where most of the obtained peptide

masses could be correlated with theoretical tryptic

peptide masses calculated from the deduced protein

sequence Notably, no tryptic peptide masses

correla-ting with the C-terminal part after Lys394 (Fig 1) of

the encoded sequence could be found The most

N-terminal tryptic peptide found that correlated with

the theoretical tryptic peptide map of the deduced

protein was QNINDLAK (m¼ 914.482 Da),

indica-ting that the possible signal sequence cleavage site is

located N-terminally to this peptide Homology

comparisons suggested that the signal sequence

clea-vage site could be at the A(18)–Q(19) bond in the

deduced sequence (shown by an arrow in Fig 1)

Often, this kind of cleavage is followed by cyclization

of the N-terminal glutamine to form pyroglutamic

acid The tryptic peptide mass fingerprint of TYR2

contained a peptide mass of 2136.108 Da, which was

suggested to correspond to the N-terminal blocked

tryptic peptide (< QGTTHIPVTGVPVSPGAAVPLR,

m¼ 2136.196 Da) The identity of this peptide was

then confirmed by MALDI-TOF⁄ TOF fragment ion

analysis, where partial sequences of this peptide were

obtained from the ladders of b-fragment and

y-frag-ment ions Subsequently, for specifying the

C-termi-nus of the protein, the most C-terminal tryptic

peptide, as compared with the theoretical tryptic

pep-tide map of the deduced protein, was found to be

SQAQIK (m¼ 673.376 Da) The mass of the

follow-ing theoretical tryptic peptide

(SSVTTIINQLYGP-NSGK, m¼ 1777.927 Da) could not be found,

indicating that the C-terminus of the protein is within

this sequence In the peptide mass fingerprint, a mass

corresponding to the peptide SSVTTIINQLYGPNSG

(m¼ 1649.826 Da) was found The identity of this C-terminal peptide was further confirmed by MALDI-TOF⁄ TOF fragment ion analysis as well as

by Edman degradation of the corresponding peptide after purification by reversed-phase chromatography During the search for and confirmation of the N-ter-minus and C-terN-ter-minus of the protein, many other pep-tides were also analyzed, and the results confirmed most of the remaining deduced amino acid sequence Edman sequencing of purified tryptic peptides covered 39.1% of the sequence The molecular masses of the tryptic peptides, either from the mass fingerprint or purified peptides, covered 60.3% of the sequence, and the masses of cyanogen bromide fragments, including the N-terminal and C-terminal ones, covered 94.9% of the sequence Together with the results from the glyco-peptide analysis (see below), the glyco-peptide analyses com-pletely confirmed the deduced amino acid sequence of the secreted protein The calculated average molecular mass of the polypeptide chain of TYR2 with the deter-mined N-terminus and C-terminus is 41 862.7 Da, whereas the mass determined by MS is about

43 200 Da Thus, there is a mass difference of about

1300 Da between the determined and calculated mass, due to post-translational modifications The purified polypeptide chain contains one potential N-glycosyla-tion site in the tryptic peptide SGPQWDLYVQA MYNMSK (m¼ 2016.907 Da) In order to analyze the possible N-glycosylation, the protein was digested with trypsin and the potential glycopeptides were bound to a ConA column MALDI-TOF MS analysis

of the eluted material revealed a few peptides, of which the largest had a molecular mass of 3255.309 Da This could correspond to the sodium adduct of the above-mentioned tryptic peptide with a high-mannose-type glycan, (GlcNAc)2(Hex)5, attached to it Further MALDI-TOF⁄ TOF MS analysis of this peptide selec-ted as the precursor ion (Fig 4) revealed a ladder of b-ions corresponding to the suggested glycopeptide with a sequential loss of Na+, five hexoses, and two N-acetylglucosamines, respectively The resulting prot-onated mass of 2017.0 Da fits well with the mass of the nonglycosylated peptide Further downstream in the fragment ion spectrum, a b-ion ladder correspond-ing to the amino acid sequence LYVQAM was detec-ted, which confirmed the identity of the peptide Thus, the purified protein is N-glycosylated at the asparagine residue (N62, Fig 1) having a high-mannose-type glycan consisting of two N-acetylglucosamines and five hexoses From the ConA eluate, other masses corres-ponding to the same tryptic peptide but with a shorter glycan (e.g peptide + 1 · GlcNAc, m ¼ 2220.12 Da) were also detected, which indicates that the presence of

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other shorter glycan structures cannot be excluded.

N-Glycans with five mannoses have been found as the

predominant form previously in the Cel7A cellulase of

T reesei [30] In the same study, N-glycosylation sites

with a single GlcNAc were also found

In order to clarify the reason for the existence of

TYR2 as a double band in SDS⁄ PAGE, the two

pro-tein bands were individually cut out and ‘in-gel’

diges-ted with trypsin Mass fingerprint analysis of the

tryptic fragments by MALDI-TOF MS did not show

significant differences in the mass fingerprints, thus

leaving the reason for the double band unclear

T reesei TYR2 was shown to be almost fully active

within a pH range of 6–9.5, with an optimum at pH 9

Considering the stability of T reesei TYR2 within a

pH range of 2–8, the enzyme showed good stability at

neutral and alkaline pH When the pH was under 7,

the enzyme started to lose activity; after 1 h at pH 5,

activity loss was 50%, and after 1 h at pH 4.0, the

enzyme had totally lost its activity Although

l-tyro-sine was chosen as the substrate to diminish the

sub-strate auto-oxidation effect in the pH optimum and

stability determination, the disturbance of

auto-oxida-tion could not be totally eliminated, because l-tyrosine

is first hydroxylated to diphenolic l-dopa and then

further oxidized to quinones in tyrosinase-catalyzed

reactions An alkaline environment also changes the redox potential of the phenolic substrates, making them more easily oxidized Therefore, the pH profile reflects not only the optimal behavior of the enzyme, but also changes in the substrate With regard to tem-perature stability, T reesei TYR2 was found to be sta-ble up to 30C However, at higher temperatures it started to lose its activity relatively quickly; the enzyme showed half-lives of 18 h, 3 h 45 min and

15 min at 30C, 40 C and 50 C, respectively Among the tested substrates (Table 2), the highest affinity of T reesei TYR2 was observed with p-tyrosol (Km¼ 1.3 mm), followed by p-coumaric acid (Km¼ 1.6 mm) and l-dopa (Km¼ 3.0 mm) The highest turn-over number, kcat, was observed with l-dopa, at

22 s)1 Substrate specificity determination for T reesei TYR2 showed that the enzyme was able to oxidize various substituted monophenols, which had the OH group in the para position (Table 3) The activity of the enzyme on diphenols was substantially higher than

on monophenols; for example, catechol was oxidized approximately 10 times faster than phenol Interest-ingly, aniline, containing no hydroxyl groups in the aromatic ring, but an amino group, was also oxidized

by the tyrosinase, although slowly Any side chain ortho to the phenolic hydroxyl group prevented

8.5-

8.0-

7.5-

7.0-

6.5-

6.0

5.5-

5.0-

4.5-

4.0-

3.5-

3.0-

2.5-

2.0-

1.5-

1.0-

0.5-

600 800 1000 1200 1400 1600 1800 2000 2200 2400 2600 2800 3000 3200

m/z

Abs Int × 1000

Δ m GlcNAc = 203.20 Da

Δ m Hexose = 162.14 Da c

3256.309

- L Y V Q A M Y -

670.931

783.800 945.792 1174.001 1045.842 1244.723

1375.497 1539.592

Fig 4 The MALDI-TOF MS ⁄ MS spectrum of a ConA affinity-purified tryptic glycopeptide from T reesei TYR2 The peptide with a deter-mined monoisotopic protonated mass of 3256.309 Da (shown in the insert) was selected as the precursor ion and analyzed in the LID-LIFT mode without collision gas The resulting fragment ions correspond to a sequential loss of one Na + , five hexoses and two N-acetylglucosa-mines, resulting in a molecule with a protonated mass of 2017.0 Da The fragmentation ion ladder at the lower molecular mass range corres-ponds to a sequence LYVQAMY, which confirms the identity of the glycopeptide.

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oxidation of the substrate, presumably because of

ster-ic hindrance The presence and the position of an

amine group in the substrate structure appeared to be

critical, considering the oxidation of the substrate by

T reesei TYR2 The closer to the hydroxyl group of

phenol the amino group was, the slower was the

oxida-tion of the substrate For most of the substrates

stud-ied, increasing the substrate concentration from

2.5 mm to 10 mm (or to 20 mm, data not shown) did

not substantially affect the activity as calculated

relative (%) to l-dopa (Table 3) However, different

stereo-forms of catechin behaved differently As the

concentration of (+)-catechin and (–)-catechin was

increased from 2.5 to 10 mm, (–)-catechin was oxidized

faster, suggesting that T reesei TYR2 has a lower Km

value for (–)-catechin than for (+)-catechin

Further-more, T reesei TYR2 was found to be stereospecific; it

oxidized the l-forms of dopa and tyrosine noticeably better than the d-forms (Table 4)

Various potential inhibitors of T reesei TYR2 were tested (Table 5) Kojic acid and b-mercaptoethanol were the most effective inhibitors, even at low concen-trations Sodium chloride and EDTA did not inhibit the enzyme very efficiently Glutathione caused only moderate inhibition, inhibiting the enzyme with 20% efficiency, as measured with the oxygen consumption assay However, as measured with the spectrophoto-metric assay, inhibition efficiency was 100%, suggest-ing that glutathione does not inhibit the enzymatic reaction, but has more effect on the subsequent non-enzymatic reactions, as also reported in other studies [31]

T reeseiTYR2 was able to oxidize the tested model peptides glycine–tyrosine and glycine–glycine–tyrosine (Table 6) The oxidation rate was dependent on the

Table 2 Determination of K m and k cat values for T reesei TYR2 on

L-dopa, p-coumaric acid and p-tyrosol.

k cat ⁄ K m (s)1ÆmM)1)

Table 3 Substrate specificity of T reesei TYR2 as determined

relative to L-dopa ND, not determined due to the low solubility.

Relative activity (%) on monophenols and polyphenols from oxygen

consumption (nmolÆL)1Æs)1) was calculated according to the

stoichio-metry that one monophenol molecule needs one oxygen molecule,

and one polyphenol molecule needs half an oxygen molecule, in

the reaction to form a quinone c, substrate concentration.

Substrate

Activity (%) relative to L-dopa

Table 4 Stereospecificity of T reesei TYR2.

Substrate (2.5 mM)

Activity (%) relative to L-dopa and L-tyrosine

Table 5 Degree of inhibition of T reesei TYR2 as determined in the presence of 15 mM L-dopa, as analyzed by oxygen consumption and spectrophotometric assay.

Inhibitor

Inhibitor (mM)

Degree (%) of inhibition

as analyzed by oxygen consumption assay

Degree (%) of inhibition

as analyzed by spectro-photometric assay

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length of the peptide, the tripeptide being more readily

oxidized than the dipeptide

Discussion

Tyrosinase enzymes and their genes have previously

been characterized from bacteria, fungi, plants and

mammals The most extensively investigated fungal

tyrosinases, from both a structural and a functional

point of view, are from Agaricus bisporus [10] and

N crassa [1] Also, a few bacterial tyrosinases have

been reported, of which Streptomyces tyrosinases are

the most thoroughly characterized [32,33] In addition,

tyrosinases have been reported, for example, from

Pseudomonadacae [34], Bacillus, Myrothecium [35],

Mucor [36], Miriococcum [37], Aspergillus,

Chaetoto-mastia, Ascovaginospora [38], Trametes [39] and

Pyc-noporus [40] Our aim was to discover novel fungal

tyrosinases, and we used the genome sequence of a

well-known industrial enzyme producer T reesei for

the search A homology search in the genome database

of this fungus revealed a new tyrosinase gene tyr2,

which, according to sequence analysis, has a signal

sequence The gene was overexpressed in the native

host; thus, the gene product was verified to be

secre-ted This is exceptional in this class of enzymes, as all

the plant, animal and fungal tyrosinases studied thus

far have been intracellular The characterized

Strep-tomycestyrosinases are secreted but do not have signal

sequences; their secretion is assisted by a second

pro-tein that has a signal sequence [41,42] It appears that

other ascomycetous fungi also have secreted

tyrosin-ases, because the three closest homologs of T reesei

TYR2 from G zeae, N crassa and Magnaporthe grisea

have putative signal sequences In fact, N crassa has

both secreted and intracellular tyrosinases; the enzyme

cloned and studied previously is intracellular [26]

From an industrial point of view, a naturally secreted

tyrosinase can be considered beneficial, as such an

enzyme is likely to be compatible with the secretory

system of the host organism in attempts to produce

substantial amounts of enzyme for applications

Although microbial tyrosinases have been produced

heterologously, e.g in Eschericia coli [32,43,44] and in

Saccharomyces cerevisiae [45], the expression levels reported thus far have been relatively low The avail-ability of the enzyme has hampered its detailed charac-terization as well as testing it in various applications The T reesei TYR2 tyrosinase gene was expressed in

T reesei under the strong cbh1 promoter In shake flasks, the highest production level was approximately

320 mgÆL)1, whereas production levels were over three times higher than this in fermenter conditions The addition of copper to the T reesei medium had a positive effect on tyrosinase production Because the tyrosinase was expressed under the cbh1 promoter, which is not activated by copper, the improved production levels were presumably not caused by higher transcription rates In addition, no effect of copper addition on fungal growth was observed, which implies that the higher enzyme yields may have been due to improved folding of the active enzyme in the presence

of elevated copper concentrations

The importance of high copper concentrations has been reported in laccase production in S cerevisiae, where the overexpression of two copper-trafficking enzymes from Trametes versicolor led to significantly improved recombinant laccase yields [46] Added cop-per can improve correct folding of recombinant lac-case, as previously detected in Aspergillus nidulans and Aspergillus niger expressing a laccase from Ceriporiop-sis subvermispora [47], and in T reesei producing the laccase of Melanocarpus albomyces [48]

C-terminal processing of fungal tyrosinases has been reported previously, and also the molecular mass of purified TYR2 tyrosinase, 43.2 kDa, suggested exten-sive processing of the protein The intracellular tyro-sinases from N crassa [26–28] and Agaricus [28] and Pycnoporus species [25] have an additional C-terminal domain that is proteolytically released from the cata-lytic domain It has been postulated that the function

of the C-terminal domain is to keep the enzyme in-active until the activity is needed [26] According to our results, the secreted T reesei TYR2 is also C-ter-minally processed (after Gly410) (Fig 1) However, in this case the peptidase performing the cleavage must reside in the secretory pathway or be extracellular The precise processing site has previously been determined only for the N crassa tyrosinase (after Phe408) (Fig 1) According to the alignment of T reesei TYR2 and the N crassa tyrosinase, the positions of the pro-cessing sites in these two enzymes coincide exactly, even though the sequences are not conserved in that region This is compatible with the idea that this site is

at a domain border that would be susceptible to pro-teases The processed N crassa tyrosinase ends with a phenylalanine, and thus it was assumed that it is

Table 6 Activity of T reesei TYR2 on 2.5 mM dipeptides and

trip-eptides in relation to L-tyrosine (%) Y, tyrosine; G, glycine.

Substrate

Activity (%) relative to Y

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cleaved by a chymotrypsin-like enzyme [26] The

Agaricus bisporus tyrosinase can be processed in vitro

by the serine proteases trypsin and subtilisin [28] The

processed T reesei TYR2 ends with a glycine residue

Analysis of the whole sequence with the program

pep-tidecutter [49], which searches for all known

pepti-dase cleavage sites, did not indicate that the protein

could be cleaved at that site The C-terminal glycine of

the mature TYR2 is followed in the sequence by the

amino acids Lys–Lys–Arg This contains a recognition

sequence for the KEX2⁄ furin-type protease, which

resides in the Golgi complex and processes a number

of secreted enzymes and other proteins after dibasic

recognition sites [50] The putatively secreted

tyrosin-ase of G zeae has Lys–Arg at the same position

(Fig 1) For these reasons, it is possible that TYR2 is

first cleaved by a T reesei KEX2-type endopeptidase

during secretion and is further processed by an

exo-peptidase Further analyses are needed to elucidate the

role of the C-terminal processing

As for the T reesei TYR2, the pH optimum in the

alkaline pH range has been reported for

Thermomicro-bium roseum (pH 9.5) [51] and pine needle tyrosinase

(9–9.5) [52] Many fungal tyrosinases have their pH

optima at neutral and slightly acidic pH, e.g N crassa

and Aspergillus flavipes at pH 6.0–7.0 [53,54] and

Pyc-noporus sanguineus at pH 6.5–7 [40] T reesei TYR2

was not able to retain substantial activity at

tempera-tures above 30C Longer half-lives have been

repor-ted, e.g 2 h at 50C for P sanguineus tyrosinase

However, at 60C, P sanguineus tyrosinase was also

inactivated completely within 20 min [40] In general,

mammalian and plant-derived tyrosinases are not very

thermostable; even a short incubation at 70–90C

inac-tivates the enzymes completely [52,55] Also,

inactiva-tion of A flavipes [54] and N crassa [56,57] tyrosinases

at relatively low temperatures has been reported

The enzyme showed relatively high Kmvalues for all

tested substrates, l-dopa, p-coumaric acid, and

p-tyro-sol The values were in accordance with values reported

in the literature Kmvalues for l-dopa were 3.0 mm for

T reesei TYR2, 0.74–1.09 mm for N crassa [1,58],

5.0 mm for A flavipes [54], 5.97 mm for Streptomyces

glaucescens[59] and 8.7–10 mm for pine needle [52]

Trichoderma reeseiTYR2 showed surprisingly broad

substrate specificity and higher oxidation activity for

diphenols than for monophenolic substrates Ferulic

acid, as well as other compounds with a side group

orthoto the phenolic hydroxyl group, was not oxidized

by the enzyme, presumably because of steric

hin-drance The substituted phenols, such as

2-aminophe-nol and 4-nitrophe2-aminophe-nol, or benzene derivatives, such as

benzoic and naphthoic acids, have been reported to be

efficient tyrosinase inhibitors, and the inhibitory mech-anism is suggested to be competitive docking, due to the similarity between the structures of these inhibitory compounds and those of phenol or tyrosine [60,61] For instance, Piquemal et al [61] showed in a theoret-ical study that 2-aminophenol forms a more stable and energetically favored complex with tyrosinase than phenol does The amine group also seemed to act as a substrate analog for T reesei TYR2 Also, a thiol group in the phenolic ring inhibited the enzyme Tous-saint and Lerch [62] and Ga˛sowska et al [63] showed that N crassa tyrosinase oxidizes aromatic amines and o-aminophenols, structural analogs of monophenols and ortho-diphenols Similar catalytic reactions, ortho hydroxylation and oxidation, took place, although the reaction rates observed for aromatic amines were relat-ively slow as compared to those for monophenols

T reesei TYR2 was also found to oxidize phenylalan-ine, although extremely slowly

The tyrosyl residue was oxidized by T reesei TYR2

in the dipeptide glycine–tyrosine and the tripeptide glycine–glycine–tyrosine The relative oxidation rate increased as the length of the peptide increased Simi-larly, protein-bound tyrosyl was oxidized by the enzyme, and subsequent protein crosslinking was observed, as analyzed by SDS⁄ PAGE (data not shown) Because of difficulties in the production and purifi-cation of microbial tyrosinases in sufficient amounts, knowledge of their structure–function relationships and exact reaction mechanisms is still limited The availability of the enzyme has also hampered its testing and use in applications We have reported here for the first time the production, purification and characteriza-tion of a novel tyrosinase from the well-known protein producer T reesei The high production levels of the tyrosinase also allow the testing of the enzyme for applications

Experimental procedures

Isolation of the tyrosinase gene from Trichoderma reesei

The tyr2 gene was amplified from genomic T reesei DNA with the following primers: forward, GGG GAC AAG TTT GTA CAA AAA AGC AGG CTA TCA TGC TGT TGT CAG GTC CCT CTC G; and reverse, GGG GAC CAC TTT GTA CAA GAA AGC TGG GTC AGT GGT GGT GGT GGT GGT GCA GAG GAG GGA TAT GGG GAA CGG CAA A The PCR reaction was done with the Dyna-zyme EXT thermostable polymerase (FinnDyna-zymes, Helsinki, Finland) in a reaction mixture recommended by the manu-facturer The PCR program comprised an initial

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denatura-tion step of 3 min at 94C, followed by 25 cycles of 30 s at

94C, 45 s at 52 C and 2.5 min at 72 C This was

followed by a final elongation step of 5 min at 72C The

tyr1 gene fragment was cloned into the pCR2.1TOPO

vector with the TOPO-TA Cloning Kit (Invitrogen,

Carls-bad, CA, USA) and subsequently transferred into the

pDONR221 vector (Invitrogen) with a BP recombination

reaction carried out with the Gateway Recombination kit

(Invitrogen)

The tyr2 cDNA was isolated by RT-PCR from a cDNA

expression library of T reesei RutC-30 [18] with primers

that were designed to create an N-terminal His6 tag and

add EcoRI and KpnI restriction endonuclease sites to the 5¢

and 3¢ ends, respectively The primers used were as follows:

forward primer, GTT GGA ATT CCA TCA TCA TCA

TCA TCA TCA GGG CAC GAC ACA CAT CCC C; and

reverse primer, GAT CGG TAC CTC ATT ACA GAG

GAG GGA TAT GGG GAA C The PCR reaction was

done as described above The amplified PCR product was

inserted into the EcoRI and KpnI sites of the vector

pPIC-Za´A (Invitrogen) and the sequence of the product was

verified

Overexpression of the tyrosinase gene

in Trichoderma reesei

The genomic tyr2 gene fragment was transferred by an LR

recombination reaction from the pDONR221 vector to the

T reeseiexpression vector pMS186, giving rise to the

plas-mid pMS190 The pMS186 contains the Gateway reading

frame cassette C (RfC) inserted between the cbh1

(cello-biohydrolase 1) promoter and terminator, and a

hygromy-cin resistance cassette The LR recombination reaction was

done with the Gateway Recombination kit (Invitrogen)

according to the manufacturer’s instructions

The plasmid pMS190 was transformed into the T reesei

strain VTT-D-00775, essentially as described [19], and

transformants were selected for hygromycin resistance on

plates containing 125 lgÆmL)1of hygromycin B The

trans-formants were streaked on the selective medium for three

successive rounds and tested for tyrosinase activity with a

plate assay In the assay plates, Trichoderma minimal

med-ium [19] with 2% lactose as a carbon source, 1% potassmed-ium

phthalate as a buffering agent (pH 5.5), 0.1 mm CuSO4and

1% tyrosine as an indicator substrate was used The

trans-formants were streaked on the plates and grown for 7 days,

and tyrosinase activity was observed on the plates as a

brown color appearing around the streaks Positive

trans-formants were isolated by single-spore cultures In order to

quantify tyrosinase production in liquid cultures, the

trans-formants positive in the plate assay were grown in shake

flasks for 8 days in 50 mL of Trichoderma minimal medium

[19] supplemented with 4% lactose, 2% spent grain,

100 mm piperazine-N-N¢-bis(3-propanesulfonic acid) and

0.1–2 mm CuSO4, and tyrosinase activity was measured

with 3,4-dihydroxy-l-phenylalanine (l-dopa) substrate as described below

Trichoderma reesei was cultivated in a Braun Biostat C-DCU 3 fermenter (B Braun Biotech International, GmbH, Melsungen, Germany) in 20 L of a medium con-taining (gÆL)1): lactose (20), distiller’s spent grain (10), and

KH2PO4 (15), and 2 mm CuSO4.5H2O The medium pH was adjusted to 5.5–6 with NH4OH and H3PO4, and the cultivation temperature was 28C The dissolved oxygen level was kept above 30% with agitation at 450 r.p.m., aer-ation at 8 LÆmin)1 and 0–30% O2 enrichment of incoming air Foaming was controlled by automatic addition of Struktol J633 polyoleate antifoam agent (Schill & Seilacher, Hamburg, Germany) After fermentation, cells were har-vested by centrifugation and the culture supernatant was concentrated 2.5 times by ultrafiltration

Protein and enzyme activity assays

Tyrosinase activity was measured according to Robb [2] with a few modifications, using 15 mm l-dopa and 2 mm

l-tyrosine as substrates Activity assays were carried out in 0.1 m sodium phosphate buffer (pH 7.0) at 25C, monitor-ing dopachrome formation at 475 nm (edopachrome¼

3400 m)1Æcm)1) Tyrosinase activity was also determined by following the consumption of the cosubstrate oxygen with a single-channel oxygen meter (Precision Sensing GmbH, Regensburg, Germany) The activity was determined by measuring the oxygen consumption during the reaction in a sealed and a fully filled sample vial (1.8 mL) at 25C The reaction was initiated by addition of the enzyme to the sub-strate solution, and the oxidation rate (nmolÆL)1Æs)1) was calculated from the linear part of the oxygen consumption curve The protein concentration was determined with the Bio-Rad DC protein assay kit (Bio-Rad, Richmond, CA, USA), with BSA as standard During enzyme purification,

to estimate protein contents for pooling fractions, protein content determinations were done by monitoring absorb-ance at 280 nm

Enzyme purification

The concentrated culture supernatant was first desalted on

a Sephadex G-25 Coarse column (2.6· 27 cm; Pharmacia Biotech, Uppsala, Sweden) in 10 mm Tris⁄ HCl buffer,

pH 7.3 The subsequent purification steps were carried out with an A¨KTApurifier (Amersham Biosciences, Uppsala, Sweden) The sample was applied to a HiPreptm

16⁄ 10 CM Sepharose Fast Flow column, in 10 mm Tris⁄ HCl buffer,

pH 7.3 Bound proteins were eluted with a linear NaCl gra-dient (0–180 mm in six column volumes) in the equilibra-tion buffer Tyrosinase-positive fracequilibra-tions were pooled, concentrated with a Vivaspin concentrator (20 mL, 10 000 molecular weight cut-off; Vivascience, Hannover, Ger-many), and subjected to gel filtration in a Sephacryl S-100

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